Case Reports in Nephrology最新文献_第2页

Disseminated Bartonellosis Masquerading as Autoimmune Glomerulonephritis: A Case Report. 伪装成自身免疫性肾小球肾炎的播散性巴尔通体病1例报告。

Case Reports in Nephrology Pub Date : 2026-02-11 eCollection Date: 2026-01-01 DOI: 10.1155/crin/7727894

Tim Debyser, Mattias Falter, Thomas Vanhoutte, Priyanka Koshy, Melissa Depypere, Katrien De Vusser, Liesbet Henckaerts

{"title":"Disseminated Bartonellosis Masquerading as Autoimmune Glomerulonephritis: A Case Report.","authors":"Tim Debyser, Mattias Falter, Thomas Vanhoutte, Priyanka Koshy, Melissa Depypere, Katrien De Vusser, Liesbet Henckaerts","doi":"10.1155/crin/7727894","DOIUrl":"10.1155/crin/7727894","url":null,"abstract":"Background: Although Bartonella henselae is primarily known for causing self-limiting cat scratch disease in immunocompetent individuals, it can also lead to severe disseminated infections, particularly in immunocompromised patients.Case presentation: We present a rare case of disseminated B. henselae infection in a 70-year-old man with multiple comorbidities, including a recent aortic valve replacement and pacemaker implantation. The patient initially presented with purpuric skin lesions, progressive renal impairment, and pancytopenia, leading to a preliminary diagnosis of autoimmune glomerulonephritis and treatment with immunosuppressants. Subsequent investigations, including kidney and skin biopsies, revealed C3-dominant glomerulonephritis and leukocytoclastic vasculitis, respectively. Further imaging uncovered a concurrent lung malignancy, treated with radiotherapy. Five months later, the patient presented with blood culture-negative endocarditis complicated by septic embolic strokes, persisting pancytopenia, and hepatosplenomegaly. Serology and B. henselae PCR of bone marrow confirmed disseminated Bartonella henselae infection.Conclusions: This case highlights the diagnostic challenges of disseminated Bartonella infections, which can mimic autoimmune diseases and delay appropriate treatment. Clinicians should maintain a high index of suspicion to ensure timely diagnosis and management.","PeriodicalId":9604,"journal":{"name":"Case Reports in Nephrology","volume":"2026 ","pages":"7727894"},"PeriodicalIF":0.0,"publicationDate":"2026-02-11","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://www.ncbi.nlm.nih.gov/pmc/articles/PMC12893391/pdf/","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"146177778","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"OA","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}

引用次数: 0

Light Chain Deposition Disease and Light Chain Cast Nephropathy in a Patient With Multiple Myeloma and HIV Infection: A Case Report. 多发性骨髓瘤合并HIV感染患者轻链沉积病和轻链铸型肾病1例报告。

Case Reports in Nephrology Pub Date : 2026-02-08 eCollection Date: 2026-01-01 DOI: 10.1155/crin/4508810

Tamzyn Huisamen, Liezel Coetzee, Mogamat-Yazied Chothia

{"title":"Light Chain Deposition Disease and Light Chain Cast Nephropathy in a Patient With Multiple Myeloma and HIV Infection: A Case Report.","authors":"Tamzyn Huisamen, Liezel Coetzee, Mogamat-Yazied Chothia","doi":"10.1155/crin/4508810","DOIUrl":"10.1155/crin/4508810","url":null,"abstract":"Kidney dysfunction is a common complication in multiple myeloma (MM), typically presenting with cast nephropathy as a single pattern of injury on kidney biopsy and infrequently occurs in people living with human immunodeficiency virus (PLHIV). A man in his 50s, newly diagnosed with HIV, who was admitted with community-acquired pneumonia was found to have severe acute kidney injury requiring hemodialysis. Due to the lack of renal recovery, a kidney biopsy was performed, revealing fractured, periodic acid-Schiff stain-negative tubular casts with surrounding multinucleated giant cell reaction. Congo red staining was negative, but electron microscopy revealed granular subendothelial electron-dense deposits. Immunofluorescence demonstrated lambda light chain restriction. A diagnosis of light chain deposition disease with concurrent light chain cast nephropathy was made. To the best of our knowledge, this is the first description of concurrent light chain cast nephropathy and light chain deposition disease in a PLHIV and MM and highlights the importance of a kidney biopsy in the evaluation of acute kidney injury in a PLHIV and concomitant myeloma.","PeriodicalId":9604,"journal":{"name":"Case Reports in Nephrology","volume":"2026 ","pages":"4508810"},"PeriodicalIF":0.0,"publicationDate":"2026-02-08","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://www.ncbi.nlm.nih.gov/pmc/articles/PMC12883664/pdf/","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"146156074","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"OA","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}

引用次数: 0

Always Consider a Repeat Kidney Biopsy: Acute Interstitial Nephritis Soon After Membranous Nephropathy. 总是考虑重复肾活检：急性间质性肾炎后不久膜性肾病。

Case Reports in Nephrology Pub Date : 2026-01-31 eCollection Date: 2026-01-01 DOI: 10.1155/crin/2277958

Jarrad Hopkins, James Nolan, Maleeka Ladhani, Chiang Lee

{"title":"Always Consider a Repeat Kidney Biopsy: Acute Interstitial Nephritis Soon After Membranous Nephropathy.","authors":"Jarrad Hopkins, James Nolan, Maleeka Ladhani, Chiang Lee","doi":"10.1155/crin/2277958","DOIUrl":"10.1155/crin/2277958","url":null,"abstract":"Introduction: The heterogeneity of membranous nephropathy is well described in the literature, and its clinical course and response to treatment vary. Similarly, acute interstitial nephritis (AIN) can present in unexpected and unusual ways and should always be considered within the differential diagnosis of worsening renal function. This case study describes the cross-section of these two disease entities.Clinical case: A 68-year-old male with a history of hypertension, chronic obstructive pulmonary disease (COPD), hyperlipidaemia, gout and treated prostate cancer presented with bilateral lower limb swelling and progressive renal dysfunction. Initial laboratory findings demonstrated nephrotic syndrome with impaired renal function. A positive phospholipase A2 receptor (PLA2R) antibody confirmed primary membranous nephropathy. Renal biopsy showed typical findings of membranous nephropathy. Treatment with prednisolone and cyclophosphamide improved renal function initially. However, as prednisolone was tapered, creatinine levels rapidly worsened, leading to a second biopsy. The second biopsy demonstrated AIN superimposed on chronic membranous nephropathy. A drug-induced aetiology was suspected, with pantoprazole, trimethoprim-sulfamethoxazole and frusemide identified as the most likely contributors. Following withdrawal of these agents and reinitiation of high-dose corticosteroids, the patient's renal function improved markedly, obviating the need for dialysis.Conclusion: This case highlights the importance of considering AIN and the need for timely repeat renal biopsy in cases of deteriorating renal function.","PeriodicalId":9604,"journal":{"name":"Case Reports in Nephrology","volume":"2026 ","pages":"2277958"},"PeriodicalIF":0.0,"publicationDate":"2026-01-31","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://www.ncbi.nlm.nih.gov/pmc/articles/PMC12860571/pdf/","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"146104030","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"OA","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}

引用次数: 0

A Rare NPHS2 Mutation (E130K) in Hereditary Steroid-Resistant Nephrotic Syndrome: A Case Report. 遗传性类固醇抵抗性肾病综合征中一罕见的NPHS2突变（E130K）： 1例报告。

Case Reports in Nephrology Pub Date : 2026-01-29 eCollection Date: 2026-01-01 DOI: 10.1155/crin/8289305

Ramzi Hmedan Mujahed, Omar Hammam Salloum, Nimatallah Fares Ishreiteh, Hiba Mofdi Hosheyh, Aisha Dahood Abu Hashem, Yara Fadi Shamestti, Malak Ramzy Hroub

{"title":"A Rare NPHS2 Mutation (E130K) in Hereditary Steroid-Resistant Nephrotic Syndrome: A Case Report.","authors":"Ramzi Hmedan Mujahed, Omar Hammam Salloum, Nimatallah Fares Ishreiteh, Hiba Mofdi Hosheyh, Aisha Dahood Abu Hashem, Yara Fadi Shamestti, Malak Ramzy Hroub","doi":"10.1155/crin/8289305","DOIUrl":"10.1155/crin/8289305","url":null,"abstract":"Hereditary steroid-resistant nephrotic syndrome (HSRNS) due to mutations in the NPHS2 gene (encoding podocin) is a rare genetic condition that typically presents in childhood. We report a case of a 2-year-and-8-month-old male, the seventh child of consanguineous parents, who presented with recurrent fever, febrile tonic-clonic seizures, and periorbital edema. His medical history included multiple hospitalizations in infancy due to suspected sepsis and chest infections. Upon admission, laboratory findings revealed proteinuria, hypoalbuminemia, and hypercholesterolemia, along with mild hepatomegaly. Genetic testing identified compound heterozygous mutations (R138X and E130K) in the NPHS2 gene, confirming a diagnosis of HSRNS. Renal biopsy revealed features consistent with minimal change disease, and immunofluorescence was negative for IgG, IgM, IgA, C3, and C4. The patient was treated with enalapril as part of supportive management. This case highlights the importance of early genetic testing and renal biopsy in diagnosing steroid-resistant nephrotic syndrome, particularly in children with atypical presentations. Understanding the genetic underpinnings of such rare cases is essential for guiding appropriate treatment and providing prognostic insights.","PeriodicalId":9604,"journal":{"name":"Case Reports in Nephrology","volume":"2026 ","pages":"8289305"},"PeriodicalIF":0.0,"publicationDate":"2026-01-29","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://www.ncbi.nlm.nih.gov/pmc/articles/PMC12856216/pdf/","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"146103998","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"OA","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}

引用次数: 0

A Case of Post-Transplantation Lymphoproliferative Disorder Following Kidney Transplantation. 肾移植后淋巴细胞增生性疾病1例。

Case Reports in Nephrology Pub Date : 2026-01-08 eCollection Date: 2026-01-01 DOI: 10.1155/crin/5917161

Punam Ajay Raval, John Otieno Odhiambo, Hanika Patel, Ahmed Sokwala

{"title":"A Case of Post-Transplantation Lymphoproliferative Disorder Following Kidney Transplantation.","authors":"Punam Ajay Raval, John Otieno Odhiambo, Hanika Patel, Ahmed Sokwala","doi":"10.1155/crin/5917161","DOIUrl":"10.1155/crin/5917161","url":null,"abstract":"This was a case of a 65-year-old gentleman known to have diabetes mellitus and hypertension since 2012 and post renal transplantation in 2016. He had also been treated for hepatitis C infection in the past. His regular medications included nebivolol 5 mg once a day, tacrolimus 4 mg twice a day, mycophenolate mofetil 500 mg twice a day, prednisolone 5 mg once a day, and insulin-novomix 14 units in the morning and 6 units at night. He presented to a tertiary teaching hospital in Kenya in October 2023 with abdominal pain, vomiting, and constipation on and off for 6 months, worse in the month prior to presentation. His examination was positive for dehydration, abdominal distension, and generalized abdominal tenderness. He had a normal hematological profile and renal function. A CT scan of the abdomen showed features of small bowel obstruction from the distal ileal to distal jejunal bowel loops. The patient underwent an exploratory laparotomy with intraoperative findings of a midjejunal tumor completely obstructing the lumen with proximal dilatation. The tumor was subsequently excised. Histological specimen confirmed diffuse large B-cell lymphoma (DLBCL) as his final diagnosis.","PeriodicalId":9604,"journal":{"name":"Case Reports in Nephrology","volume":"2026 ","pages":"5917161"},"PeriodicalIF":0.0,"publicationDate":"2026-01-08","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://www.ncbi.nlm.nih.gov/pmc/articles/PMC12780540/pdf/","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"145951325","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"OA","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}

引用次数: 0

Osteitis Fibrosa Cystica: An Unusual Cause of Left-Sided Chest Pain. 囊性纤维性骨炎：左侧胸痛的不寻常原因。

Case Reports in Nephrology Pub Date : 2026-01-08 eCollection Date: 2026-01-01 DOI: 10.1155/crin/3576066

Raahima Habib, Maha Anjum, Ayesha Mahmood, Nabiha Rizvi

{"title":"Osteitis Fibrosa Cystica: An Unusual Cause of Left-Sided Chest Pain.","authors":"Raahima Habib, Maha Anjum, Ayesha Mahmood, Nabiha Rizvi","doi":"10.1155/crin/3576066","DOIUrl":"10.1155/crin/3576066","url":null,"abstract":"We report a case of osteitis fibrosa cystica resulting from secondary hyperparathyroidism in a 21-year-old male patient with end-stage renal disease. The patient presented with persistent, moderate chest pain localized to the left fifth and sixth ribs for eight months. A chest X-ray revealed well-defined, expansile lytic lesions in these ribs. Prior testing showed elevated parathyroid hormone (PTH) levels for five years, along with decreased serum calcium and elevated phosphorus levels. Findings from ultrasound and SPECT scans were consistent with hyperparathyroidism. Repeat laboratory tests showed a PTH level of 939.8 pg/mL (normal: 10-69 pg/mL), calcium level of 8.3 mg/dL (normal: 8.4-10.2 mg/dL), and phosphorus level of 5.1 mg/dL (normal: 2.5-5.0 mg/dL). The patient declined surgical intervention and was managed conservatively with calcium and vitamin D supplementation. Within 4 weeks, symptoms resolved and calcium and phosphorus levels normalized, although PTH levels remained elevated. Osteitis fibrosa cystica can be challenging to diagnose due to its rarity, especially in developed countries, and its nonspecific clinical presentation. This case highlights the importance of considering this diagnosis and outlines an approach to management in resource-limited settings.","PeriodicalId":9604,"journal":{"name":"Case Reports in Nephrology","volume":"2026 ","pages":"3576066"},"PeriodicalIF":0.0,"publicationDate":"2026-01-08","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://www.ncbi.nlm.nih.gov/pmc/articles/PMC12783687/pdf/","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"145951341","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"OA","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}

引用次数: 0

Adaptive Glomerular Hypertrophy in Kidney Transplants Associated With Focal Segmental Glomerulosclerosis: Report of Three Cases. 肾移植伴局灶节段性肾小球硬化的适应性肾小球肥大：3例报告。

Case Reports in Nephrology Pub Date : 2025-12-29 eCollection Date: 2025-01-01 DOI: 10.1155/crin/7613213

Autumn LaRocque, Ujwal Gautam, Nora Ewis, Richard Ugarte, Emily Daniel, John C Papadimitriou, Abdolreza Haririan, Cinthia B Drachenberg

{"title":"Adaptive Glomerular Hypertrophy in Kidney Transplants Associated With Focal Segmental Glomerulosclerosis: Report of Three Cases.","authors":"Autumn LaRocque, Ujwal Gautam, Nora Ewis, Richard Ugarte, Emily Daniel, John C Papadimitriou, Abdolreza Haririan, Cinthia B Drachenberg","doi":"10.1155/crin/7613213","DOIUrl":"10.1155/crin/7613213","url":null,"abstract":"Transplant kidneys face increased functional demands in comparison to physiological conditions, and glomerular hypertrophy (GH) appears to be a common compensatory mechanism. Adaptive GH could contribute to improved graft function, but a so-called \"maladaptive\" response may lead to shortened graft survival. We assessed in three patients the mean glomerular diameter in donor preimplantation wedge biopsies and in subsequent posttransplant biopsies and correlated the glomerular size with Banff scores and clinical course. Mean glomerular size increased from 192.09 ± 28.65, 188.98 ± 19.86, and 168.96 ± 18.5 in preimplantation biopsies to 279.43 ± 50.6 (p < 0.0001), 275.23 ± 68.17 (p < 0.0001), and 266.23 ± 40.5 (p < 0.00001) in the last biopsies, respectively. The proportion of enlarged glomeruli (> 200 µ) increased from 42.86%, 31%, and 5% in the donor biopsies to 93.3% (p0.008), 95.7% (p < 0.0001), and 95.1% (p < 0.00001) in the last biopsy, respectively. Although GH was initially associated with the achievement of good graft function, secondary FSGS and proteinuria developed at 3, 7, and > 17 years posttransplant, respectively. GH (glomerulomegaly) can be easily appreciated by light microscopy, but is not routinely recorded in transplant biopsy reports. Recognition and documentation of GH could help identify factors associated with the \"maladaptive\" phase of this adaptive response and find interventions that can potentially prolong graft survival.","PeriodicalId":9604,"journal":{"name":"Case Reports in Nephrology","volume":"2025 ","pages":"7613213"},"PeriodicalIF":0.0,"publicationDate":"2025-12-29","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://www.ncbi.nlm.nih.gov/pmc/articles/PMC12747116/pdf/","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"145862296","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"OA","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}

引用次数: 0

Calciphylaxis in the Upper GI Tract in an ESRD Patient: An Atypical Presentation and Review of Literature. ESRD患者上消化道的钙化反应：一个不典型的表现和文献回顾。

Case Reports in Nephrology Pub Date : 2025-12-11 eCollection Date: 2025-01-01 DOI: 10.1155/crin/3967671

Pallavi D Shirsat, Kunal Sonavane, Leonardo Ramirez Botana, Bharat Sachdeva

{"title":"Calciphylaxis in the Upper GI Tract in an ESRD Patient: An Atypical Presentation and Review of Literature.","authors":"Pallavi D Shirsat, Kunal Sonavane, Leonardo Ramirez Botana, Bharat Sachdeva","doi":"10.1155/crin/3967671","DOIUrl":"10.1155/crin/3967671","url":null,"abstract":"Calciphylaxis, also referred to as calcific uremic arteriolopathy (CUA), is a rare and life-threatening condition characterized by vascular calcification, ischemic tissue injury, and high morbidity and mortality. It is predominantly observed in patients with end-stage renal disease (ESRD) who are undergoing dialysis and typically presents with painful cutaneous ulcers. We report an exceptional case of gastric mucosal CUA in a dialysis-dependent ESRD patient, presenting with severe upper gastrointestinal bleeding in the absence of preceding skin lesions. Histopathologic examination of gastric biopsies confirmed vascular calcification consistent with CUA. Despite medical intervention, the patient's course was complicated by poor treatment adherence and subsequent fatal outcome. This case underscores the importance of maintaining high index of suspicion for atypical, visceral presentations of CUA in high-risk ESRD patients. This case was previously presented as a poster at the National Kidney Foundation 2025 Spring Clinical Meeting. Trial Registration: ClinicalTrials.gov identifier: NCT02278692.","PeriodicalId":9604,"journal":{"name":"Case Reports in Nephrology","volume":"2025 ","pages":"3967671"},"PeriodicalIF":0.0,"publicationDate":"2025-12-11","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://www.ncbi.nlm.nih.gov/pmc/articles/PMC12747075/pdf/","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"145862368","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"OA","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}

引用次数: 0

Paradoxical Hyperkalemia in Bartter Syndrome: A Case Report of Severe Resistant Hyperkalemia Requiring Hemodialysis. Bartter综合征的矛盾高钾血症：需要血液透析的严重耐药高钾血症1例报告。

Case Reports in Nephrology Pub Date : 2025-12-11 eCollection Date: 2025-01-01 DOI: 10.1155/crin/2644098

Abhishek Vadher, Mehwish Zeb, Shayne Joseph, Sujata Kambhatla, Chadi Saad

{"title":"Paradoxical Hyperkalemia in Bartter Syndrome: A Case Report of Severe Resistant Hyperkalemia Requiring Hemodialysis.","authors":"Abhishek Vadher, Mehwish Zeb, Shayne Joseph, Sujata Kambhatla, Chadi Saad","doi":"10.1155/crin/2644098","DOIUrl":"10.1155/crin/2644098","url":null,"abstract":"(https://ce.mayo.edu/content/abstract-submission-mayo-clinic-nephrology-hypertension-and-kidney-transplantation-update-2). Bartter syndrome is characterized by severe hypokalemia due to renal tubular defects. A female in late 30s with Bartter syndrome Type IV presented with dizziness. She had been managing severe hypokalemia with intravenous potassium supplementation via an implanted port. She missed her scheduled potassium infusion and presented to the emergency department with a critically high potassium level of 8.2 mmol/L. Additionally, she was diagnosed with acute kidney injury, with a GFR of 39 mL/min down from 72 at her baseline. Her EKG showed QRS widening and tall T waves. Despite administration of multiple shifters, her potassium levels remained elevated. Urgent hemodialysis was initiated, temporarily reducing her potassium levels, but with a rebound to 9.1 mmol/L within hours. After three cycles of hemodialysis over 36 h, her potassium levels finally normalized to 4.7 mmol/L, and no further dialysis was required.","PeriodicalId":9604,"journal":{"name":"Case Reports in Nephrology","volume":"2025 ","pages":"2644098"},"PeriodicalIF":0.0,"publicationDate":"2025-12-11","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://www.ncbi.nlm.nih.gov/pmc/articles/PMC12747050/pdf/","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"145862309","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"OA","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}

引用次数: 0

Hydrothorax as a Complication of Peritoneal Dialysis: A Case Report. 腹膜透析并发胸水1例。

Case Reports in Nephrology Pub Date : 2025-11-30 eCollection Date: 2025-01-01 DOI: 10.1155/crin/4365717

Luca Piscitani, Paolo Sipari, Vittorio Di Michele, Lorenzo Ottavio Di Pietro, Vittorio Sirolli, Marilena Tunno

引用次数: 0