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Cell Adhesion & Migration最新文献

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Nhe1 is required for directional sensing in vegetative Dictyostelium cell migration. Nhe1是营养盘形骨细胞迁移过程中定向传感所必需的。
IF 3.3 3区 生物学
Cell Adhesion & Migration Pub Date : 2025-12-01 Epub Date: 2025-06-03 DOI: 10.1080/19336918.2025.2514374
Uri Han, Nara Han, Taeck Joong Jeon
{"title":"Nhe1 is required for directional sensing in vegetative <i>Dictyostelium</i> cell migration.","authors":"Uri Han, Nara Han, Taeck Joong Jeon","doi":"10.1080/19336918.2025.2514374","DOIUrl":"10.1080/19336918.2025.2514374","url":null,"abstract":"<p><p>Na+/H+ exchanger 1 (Nhe1) is a crucial regulator of pH homeostasis and the present study demonstrates that Nhe1 is required for directional sensing in vegetative <i>Dictyostelium</i> cell migration. <i>nhe1</i> null cells exhibited severe defects in directional sensing and migration in response to folate gradients and applied electric fields.Specifically, <i>nhe1</i> null cells failed to exhibit proper translocation of phosphatidylinositol (3,4,5)-trisphosphate (PIP3) to the cell cortex and showed abnormal intracellular calcium signaling upon folate stimulation. Additionally, cells lacking Nhe1 exhibited a reduced capacity to engulf bacteria and latex beads. In contrast, <i>nhe1</i> null cells in the developed stage displayed a defect in cAMP-directed chemotaxis, but normal translocation kinetics of PIP3 to the cell cortex upon cAMP stimulation.</p>","PeriodicalId":9680,"journal":{"name":"Cell Adhesion & Migration","volume":"19 1","pages":"2514374"},"PeriodicalIF":3.3,"publicationDate":"2025-12-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://www.ncbi.nlm.nih.gov/pmc/articles/PMC12143694/pdf/","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"144215029","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":3,"RegionCategory":"生物学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"OA","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
引用次数: 0
N-Acetylcysteine relieving hydrogen peroxide-induced damage in granulosa cells of sheep. n -乙酰半胱氨酸减轻过氧化氢诱导的绵羊颗粒细胞损伤。
IF 3.3 3区 生物学
Cell Adhesion & Migration Pub Date : 2025-12-01 Epub Date: 2025-03-30 DOI: 10.1080/19336918.2025.2484182
Hao Chen, Jine Wang, Bingzhu Zhao, Yahua Yang, Chongfa Yang, Zhijie Zhao, Xiaona Ding, Yang Li, Taojie Zhang, Zhaxi Yingpai, Shengdong Huo
{"title":"N-Acetylcysteine relieving hydrogen peroxide-induced damage in granulosa cells of sheep.","authors":"Hao Chen, Jine Wang, Bingzhu Zhao, Yahua Yang, Chongfa Yang, Zhijie Zhao, Xiaona Ding, Yang Li, Taojie Zhang, Zhaxi Yingpai, Shengdong Huo","doi":"10.1080/19336918.2025.2484182","DOIUrl":"10.1080/19336918.2025.2484182","url":null,"abstract":"<p><p>Sheep ovarian granulosa cells (GCs) play a unique role in the ovary. Damage to GCs can affect the normal development of oocytes. The oxidative stress model was constructed by H<sub>2</sub>O<sub>2</sub>to study the biological changes. Specifically, pathological characteristic was assessed by immunohistochemistry (IHC), while signaling pathway was studied using western blot, quantitative RT-PCR, and immunofluorescence. Theresults showed that the oxidative damage model was successfully constructed by 200 μmol/LH<sub>2</sub>O<sub>2</sub> for 12 h. NAC can protect the proliferation of GCs under H<sub>2</sub>O<sub>2</sub>-induced oxidative stress and reduce apoptosis. It can also promote the secretion of E<sub>2</sub> and P<sub>4</sub> by GCs and reduce the inflammatory response of GCs. NAC can enhance the expression of NRF2, PI3K and Akt. These findings suggest that NAC alleviates H<sub>2</sub>O<sub>2</sub>-induced oxidative stress injury through NRF2/PI3K/AKT signaling pathways. Provide ideas for studying the poor quality of mammalian oocytes.</p>","PeriodicalId":9680,"journal":{"name":"Cell Adhesion & Migration","volume":"19 1","pages":"2484182"},"PeriodicalIF":3.3,"publicationDate":"2025-12-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://www.ncbi.nlm.nih.gov/pmc/articles/PMC11959897/pdf/","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"143751373","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":3,"RegionCategory":"生物学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"OA","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
引用次数: 0
MiR-646 inhibited cell proliferation and migration by targeting P62 in glioma. MiR-646通过靶向胶质瘤中的P62抑制细胞增殖和迁移。
IF 3.3 3区 生物学
Cell Adhesion & Migration Pub Date : 2025-12-01 Epub Date: 2025-07-01 DOI: 10.1080/19336918.2025.2515763
Fangyu Ye, Heng Zhang, Qianqian Chen, Yanping Gui, Geng Tian, Yuting Ye, Xin Chen, Haijuan Dong, Xiangshan Fan, Jun Chen, RuiYing Yuan, Li Zhao, Juping Yu
{"title":"MiR-646 inhibited cell proliferation and migration by targeting P62 in glioma.","authors":"Fangyu Ye, Heng Zhang, Qianqian Chen, Yanping Gui, Geng Tian, Yuting Ye, Xin Chen, Haijuan Dong, Xiangshan Fan, Jun Chen, RuiYing Yuan, Li Zhao, Juping Yu","doi":"10.1080/19336918.2025.2515763","DOIUrl":"10.1080/19336918.2025.2515763","url":null,"abstract":"<p><p>MiR-646, a small non-coding RNA, poorly expressed in a variety of tumors. This study aimed to clarify the role of miR-646 and its underlying mechanisms in glioblastoma (GBM). In our study, we found that miR-646 mRNA levels were lower in tumor tissues than in non-cancer tissues. The ability of glioma cells to proliferate, invade, and migrate is diminished by miR-646 overexpression <i>in vitro and in vivo</i>. Mechanistically, miR-646 targeted sequestosome 1 (p62) in the 3'UTR and affected the Keap1/Nrf2 pathway, thus attenuating the expression of the HO-1 gene. In conclusion, this study provided a novel finding that miR-646 tampered with gliomagenesis by regulating the p62/Keap1/Nrf2 axis, which provides a potential target for GBM therapy.</p>","PeriodicalId":9680,"journal":{"name":"Cell Adhesion & Migration","volume":"19 1","pages":"2515763"},"PeriodicalIF":3.3,"publicationDate":"2025-12-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://www.ncbi.nlm.nih.gov/pmc/articles/PMC12218442/pdf/","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"144539104","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":3,"RegionCategory":"生物学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"OA","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
引用次数: 0
Influences of physical stimulations on the migration and differentiation of Schwann cells involved in peripheral nerve repair. 物理刺激对参与周围神经修复的雪旺细胞迁移和分化的影响。
IF 3.3 3区 生物学
Cell Adhesion & Migration Pub Date : 2025-12-01 Epub Date: 2025-01-16 DOI: 10.1080/19336918.2025.2450311
Qingyan Sun, Xiaodan Mu, Qi Gao, Juncheng Wang, Min Hu, Huawei Liu
{"title":"Influences of physical stimulations on the migration and differentiation of Schwann cells involved in peripheral nerve repair.","authors":"Qingyan Sun, Xiaodan Mu, Qi Gao, Juncheng Wang, Min Hu, Huawei Liu","doi":"10.1080/19336918.2025.2450311","DOIUrl":"10.1080/19336918.2025.2450311","url":null,"abstract":"<p><p>Peripheral nerve injury repair has always been a research concern of scientists. At the tissue level, axonal regeneration has become a research spotlight in peripheral nerve repair. Through transplantation of autologous nerve grafts or other emerging biomaterials functional recovery after facial nerve injury is not ideal in clinical scenarios. Great strides have been made to improve facial nerve repair at the micro-cellular level. Physical stimulation techniques can trigger Schwann cells (SCs) to migrate and differentiate into cells required for peripheral nerve repair. Classified by the sources of physical stimulations, SCs repair peripheral nerves through galvanotaxis, magnetotaxis and durotaxis. This article summarized the activation, directional migration and differentiation of SCs induced by physical stimulations, thus providing new ideas for the research of peripheral nerve repair.</p>","PeriodicalId":9680,"journal":{"name":"Cell Adhesion & Migration","volume":"19 1","pages":"2450311"},"PeriodicalIF":3.3,"publicationDate":"2025-12-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://www.ncbi.nlm.nih.gov/pmc/articles/PMC11740713/pdf/","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"143000787","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":3,"RegionCategory":"生物学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"OA","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
引用次数: 0
Acyl-CoA dehydrogenase long chain acts as a tumor-suppressive factor in lung adenocarcinoma progression. 酰基辅酶a脱氢酶长链在肺腺癌进展中作为肿瘤抑制因子。
IF 3.3 3区 生物学
Cell Adhesion & Migration Pub Date : 2025-12-01 Epub Date: 2025-04-22 DOI: 10.1080/19336918.2025.2495676
Tingju Wei, Guowei Fu, Junjie Zhao, Fengan Cao, Danfeng Guo
{"title":"Acyl-CoA dehydrogenase long chain acts as a tumor-suppressive factor in lung adenocarcinoma progression.","authors":"Tingju Wei, Guowei Fu, Junjie Zhao, Fengan Cao, Danfeng Guo","doi":"10.1080/19336918.2025.2495676","DOIUrl":"https://doi.org/10.1080/19336918.2025.2495676","url":null,"abstract":"<p><p>This study investigated the role of long-chain acyl-CoA dehydrogenase (ACADL) in lung adenocarcinoma (LUAD). ACADL was significantly downregulated in human LUAD tissues compared to normal lung tissues. In vitro, ectopic expression of ACADL in murine LLC cells decreased cell viability, migration, and invasion, while ACADL knockdown exhibited the opposite effect. <i>In vivo</i>, ACADL overexpression impeded tumor growth and metastasis. Mechanistically, ACADL hindered tumor progression by inducing cell cycle arrest, promoting apoptosis, and suppressing the epithelial-mesenchymal transition (EMT) process. These findings suggest ACADL acts as a tumor suppressor in LUAD progression.</p>","PeriodicalId":9680,"journal":{"name":"Cell Adhesion & Migration","volume":"19 1","pages":"2495676"},"PeriodicalIF":3.3,"publicationDate":"2025-12-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://www.ncbi.nlm.nih.gov/pmc/articles/PMC12026206/pdf/","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"143977632","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":3,"RegionCategory":"生物学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"OA","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
引用次数: 0
The research trends and hotspots of claudins in the field of cancer. claudin在癌症领域的研究趋势和热点。
IF 3.3 3区 生物学
Cell Adhesion & Migration Pub Date : 2025-12-01 Epub Date: 2025-06-20 DOI: 10.1080/19336918.2025.2520641
Weixin Jing, Linna Long, Xiaorui Zhang, Xia Li, He Huang
{"title":"The research trends and hotspots of claudins in the field of cancer.","authors":"Weixin Jing, Linna Long, Xiaorui Zhang, Xia Li, He Huang","doi":"10.1080/19336918.2025.2520641","DOIUrl":"10.1080/19336918.2025.2520641","url":null,"abstract":"<p><p>Claudins (CLDNs), as the key components of tight junctions, have been implicated as key factors in carcinogenesis and metastasis. A total of 1720 publications on CLDNs in the field of cancer were published from January 2005 to December 2022. The United States dominates the research on CLDNs in cancer, followed by China. China Medical University is the most productive, and Johns Hopkins University has the most citations. Morin PJ is credited with initiating research on CLDNs in cancer. CLDN18, the intestinal barrier, and the intestinal microbiota are the focus and hotspots in this field. The mechanism of CLDN-mediated metastasis still needs further investigation, and the development of CLDN-targeted therapies also needs to be explored in the future.</p>","PeriodicalId":9680,"journal":{"name":"Cell Adhesion & Migration","volume":"19 1","pages":"2520641"},"PeriodicalIF":3.3,"publicationDate":"2025-12-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"144332527","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":3,"RegionCategory":"生物学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
引用次数: 0
Knockdown of HGH1 in breast cancer cell lines can inhibit the viability, invasion and migration of tumor cells. 在乳腺癌细胞系中敲低HGH1可抑制肿瘤细胞的活力、侵袭和迁移。
IF 3.3 3区 生物学
Cell Adhesion & Migration Pub Date : 2025-12-01 Epub Date: 2024-12-18 DOI: 10.1080/19336918.2024.2442349
Zeyu Wang, Taiyuan Liu, Kang He, Longyun Wang, Xiaoxuan Ma, Zhaoyun Yang, Yingchao Zhang, Lijing Zhao
{"title":"Knockdown of HGH1 in breast cancer cell lines can inhibit the viability, invasion and migration of tumor cells.","authors":"Zeyu Wang, Taiyuan Liu, Kang He, Longyun Wang, Xiaoxuan Ma, Zhaoyun Yang, Yingchao Zhang, Lijing Zhao","doi":"10.1080/19336918.2024.2442349","DOIUrl":"10.1080/19336918.2024.2442349","url":null,"abstract":"<p><strong>Background: </strong>Research on the function of HGH1 in breast cancer remains lacking.</p><p><strong>Methods: </strong>TCGAand GEO (GSE45827) datasets investigated discrepancies in HGH1 expression in BC. An aggregate of 106 clinical samples were gathered through immunohistochemistry, KM curves were drawn for prognostic analysis, and the function of HGH1 of BC was predicted. Finally, the effects of HGH1 knockdown on MDA-MB-231 and MCF-7 BC cells were verified via CCK8, invasion, wound healing and colony formation assays.</p><p><strong>Results: </strong>HGH1 is highly expressed in BC and is linked to unfavorable prognosis. HGH1 overexpression is connected to keratinization and the cell cycle and is closely related to ER and PR expression and tumor stage in BC patients. Knocking down HGH1 in BC cells inhibited the viability, invasion and migration.</p><p><strong>Conclusion: </strong>Knockdown of HGH1 in breast cancer cell lines can inhibit the viability, invasion and migration of tumor cells.</p>","PeriodicalId":9680,"journal":{"name":"Cell Adhesion & Migration","volume":"19 1","pages":"1-14"},"PeriodicalIF":3.3,"publicationDate":"2025-12-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://www.ncbi.nlm.nih.gov/pmc/articles/PMC11657051/pdf/","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"142846174","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":3,"RegionCategory":"生物学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"OA","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
引用次数: 0
Advancing in vitro cell migration studies: a review of open-source analytical platforms for cancer and wound healing research. 推进体外细胞迁移研究：癌症和伤口愈合研究的开源分析平台综述。
IF 3.3 3区 生物学
Cell Adhesion & Migration Pub Date : 2025-12-01 Epub Date: 2025-04-16 DOI: 10.1080/19336918.2025.2488116
Abhayraj S Joshi
{"title":"Advancing in vitro cell migration studies: a review of open-source analytical platforms for cancer and wound healing research.","authors":"Abhayraj S Joshi","doi":"10.1080/19336918.2025.2488116","DOIUrl":"https://doi.org/10.1080/19336918.2025.2488116","url":null,"abstract":"<p><p>A single cell or cell population exhibits the fundamental phenomenon of cell migration during developmental processes or disease progression. Vast literature suggests that, <i>in vitro</i> 2-dimensional or 3-dimensional cell migration assay is one of the most commonly used assays in cancer, wound healing research, and developmental biology research. The data obtained from this assay are often analyzed using various proprietary or open-source programs. Proprietary software are costly and not always accessible to everyone. Whereas the open-source programs are free, easy to access, and user friendly. However, not all researchers are aware of these open-source programs. Despite the increasing availability of these programs, many researchers still rely on proprietary software, due to a lack of comparative analyses and practical guidance on their implementation. Hence, this review aims to provide insights into these open-source tools and serves as a practical guide to both biologists and computational researchers for their specific analytical needs.</p>","PeriodicalId":9680,"journal":{"name":"Cell Adhesion & Migration","volume":"19 1","pages":"2488116"},"PeriodicalIF":3.3,"publicationDate":"2025-12-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://www.ncbi.nlm.nih.gov/pmc/articles/PMC12006941/pdf/","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"143977934","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":3,"RegionCategory":"生物学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"OA","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
引用次数: 0
Brief biology and pathophysiology of Tekt bundles. Tekt束的生物学和病理生理学简介。
IF 3.3 3区 生物学
Cell Adhesion & Migration Pub Date : 2025-12-01 Epub Date: 2025-02-13 DOI: 10.1080/19336918.2025.2465421
Jun Yin, Min Liu, Xiao Wang, Hongming Miao, Wenjuan He, Wei Liu, Zhongying Yu, Qinghua Zhang, Jialian Bai, Yimei Cheng, Bing Ni
{"title":"Brief biology and pathophysiology of Tekt bundles.","authors":"Jun Yin, Min Liu, Xiao Wang, Hongming Miao, Wenjuan He, Wei Liu, Zhongying Yu, Qinghua Zhang, Jialian Bai, Yimei Cheng, Bing Ni","doi":"10.1080/19336918.2025.2465421","DOIUrl":"10.1080/19336918.2025.2465421","url":null,"abstract":"<p><p>Tektins, a family of microtubule-stabilizing proteins, are critical for cilia and flagella assembly in mammals. They maintain doublet microtubule stability and ciliary/flagellar motility. Loss of Tekt1-5 causes microtubule instability, impaired motility, and diseases like infertility, retinal degeneration, Mainzer-Saldino syndrome, and diabetic nephropathy. Pathophysiological stimuli regulate Tektin expression through transcriptional, posttranscriptional, translational, and posttranslational modifications. This review summarizes the latest findings on Tektin functions and their role in diseases.</p>","PeriodicalId":9680,"journal":{"name":"Cell Adhesion & Migration","volume":"19 1","pages":"2465421"},"PeriodicalIF":3.3,"publicationDate":"2025-12-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://www.ncbi.nlm.nih.gov/pmc/articles/PMC11834534/pdf/","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"143413496","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":3,"RegionCategory":"生物学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"OA","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
引用次数: 0
Dual implication of endothelial adhesion molecules in tumor progression and cancer immunity. 内皮粘附分子在肿瘤进展和癌症免疫中的双重作用。
IF 3.3 3区 生物学
Cell Adhesion & Migration Pub Date : 2025-12-01 Epub Date: 2025-03-12 DOI: 10.1080/19336918.2025.2472308
Louis-Emmanuel Chriqui, Sabrina Cavin, Jean Yannis Perentes
{"title":"Dual implication of endothelial adhesion molecules in tumor progression and cancer immunity.","authors":"Louis-Emmanuel Chriqui, Sabrina Cavin, Jean Yannis Perentes","doi":"10.1080/19336918.2025.2472308","DOIUrl":"10.1080/19336918.2025.2472308","url":null,"abstract":"<p><p>Adhesion molecules are proteins expressed at the surface of various cell types. Their main contribution to immunity is to allow the infiltration of immune cells in an inflamed site. In cancer, adhesion molecules have been shown to promote tumor dissemination favoring the development of metastasis. While adhesion molecule inhibition approaches were unsuccessful for cancer control, their importance for the generation of an immune response alone or in combination with immunotherapies has gained interest over the past years. Currently, the balance of adhesion molecules for tumor promotion/inhibition is unclear. Here we review the role of selectins, intercellular adhesion molecules (ICAM) and vascular cell adhesion molecules (VCAM) from the perspective of the dual contribution of adhesion molecules in tumor progression and immunity.</p>","PeriodicalId":9680,"journal":{"name":"Cell Adhesion & Migration","volume":"19 1","pages":"2472308"},"PeriodicalIF":3.3,"publicationDate":"2025-12-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://www.ncbi.nlm.nih.gov/pmc/articles/PMC11913389/pdf/","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"143603512","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":3,"RegionCategory":"生物学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"OA","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
引用次数: 0
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