Journal of Neuropsychology最新文献

{"title":"Facial emotion recognition, affective empathy and psychosocial functioning in euthymic BD-I","authors":"Susan Zyto,&nbsp;Nienke Jabben,&nbsp;Annet Nugter,&nbsp;Peter F. J. Schulte,&nbsp;Ralph W. Kupka,&nbsp;Sigfried Schouws","doi":"10.1111/jnp.12417","DOIUrl":"10.1111/jnp.12417","url":null,"abstract":"<p>There is emerging evidence of social cognitive impairments in bipolar disorders (BD). Less evident is the question if social cognitive impairments are predictive of psychosocial functioning, independently of neurocognitive impairment. The aims of the study were to investigate if patients with BD-I showed impairments in facial emotion recognition and alterations in affective empathy, in relation to healthy controls, and if these impairments would predict psychosocial functioning, after accounting for neurocognitive impairments. Thirty-seven patients diagnosed with BD-I, in an euthymic state, and 37 matched healthy controls underwent an assessment including a facial recognition test (ERT) and a self-report scale of affective empathy (BEES). Patients additionally underwent an extensive neuropsychological assessment consisting of traditional tests. Patients with BD-I were significantly less able to recognize the emotion fear compared to healthy controls. However, the lower ability to recognize fear did not predict psychosocial functioning. In addition, it was not related to any of the other neuropsychological variables. The degree of self-reported empathy did not differ between patients and healthy controls. The results add to the evidence of a specific deficit in recognizing fear in BD-I; however, a link with psychosocial functioning was lacking. It is possible that the ability to recognize fear is related to a more narrow concept of interpersonal functioning than to the broad concept of psychosocial functioning. Future research should be directed towards aspects of social functioning in relation to social cognitive impairments, while taking account of subgroups of social cognitive functioning.</p>","PeriodicalId":197,"journal":{"name":"Journal of Neuropsychology","volume":"19 3","pages":"486-498"},"PeriodicalIF":1.8,"publicationDate":"2025-02-17","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://bpspsychub.onlinelibrary.wiley.com/doi/epdf/10.1111/jnp.12417","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"143439810","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":4,"RegionCategory":"心理学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"OA","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}

{"title":"The role of spontaneous strategy use in verbal episodic memory impairment in adult ADHD","authors":"Matti Laine,&nbsp;Anton Kunnari,&nbsp;Tilda Eräste,&nbsp;Liisa Ritakallio,&nbsp;Benjamin Hedberg,&nbsp;Juha Salmi","doi":"10.1111/jnp.12416","DOIUrl":"10.1111/jnp.12416","url":null,"abstract":"<p>Attention deficit hyperactivity disorder (ADHD) is associated with diverse cognitive deficits of which problems related to memory and learning are well-established but poorly understood. In an online experiment, we studied whether verbal memory impairment in adult ADHD is related to differences in spontaneous use of memory strategies that hinge upon metacognitive and executive skills. Eighty-one ADHD adults and 209 neurotypical controls performed a Word List Learning task where the same words were presented three times, each time coupled with an open strategy report. Bayesian analyses indicated that the ADHD group recalled less words, did not differ from controls in efficacy of strategy use, but exhibited more limited strategy use than the controls. This suggests that less versatile strategy employment, possibly related to core executive deficits, may play a role in verbal memory impairment in adults with ADHD. Concerning treatment implications, future research could probe to what extent external strategy instruction might increase the versatility of mnemonic strategy use in adult ADHD, and thereby partly compensate for their verbal memory deficits.</p>","PeriodicalId":197,"journal":{"name":"Journal of Neuropsychology","volume":"19 3","pages":"454-472"},"PeriodicalIF":1.8,"publicationDate":"2025-02-13","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"143412615","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":4,"RegionCategory":"心理学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}

{"title":"Among common neuropsychological tests, the Paced auditory serial addition test is the strongest predictor of trait fatigue in patients with traumatic brain injury","authors":"Nils Berginström,&nbsp;Johan Thelander,&nbsp;Peter Nordström,&nbsp;Anna Nordström","doi":"10.1111/jnp.12419","DOIUrl":"10.1111/jnp.12419","url":null,"abstract":"<p>Fatigue is one of the most common symptoms following traumatic brain injury (TBI). Despite its prevalence, fatigue remains a challenging concept to define and measure. The aim of the present study was to explore potential relationships between self-rated fatigue in patients with TBI and performance on several widely used neuropsychological tests. In a cross-sectional design, patients with TBI (<i>n</i> = 68) and healthy controls (<i>n</i> = 27) underwent a comprehensive battery of commonly used neuropsychological tests and completed two self-assessment fatigue scales, the Fatigue Severity Scale and the Mental Fatigue Scale. Patients with TBI performed worse on neuropsychological tests of short-term memory, processing speed and executive functioning (inhibition) compared to healthy controls. Within the TBI group, only the Paced Auditory Serial Addition Test (PASAT) and the Stroop—Inhibition task showed significant correlations with measures of fatigue. However, after adjusting for relevant demographic variables, including age, gender, education and TBI severity, only PASAT remained significantly associated with the Mental Fatigue Scale (<i>r</i> = .45, <i>p</i> = .005). Within the healthy control group, no such associations were found. These results highlight an interesting relationship between PASAT performance and self-assessed fatigue. With further research, PASAT, modifications of it or similar measures could potentially help clinicians in evaluating fatigue in patients with TBI.</p>","PeriodicalId":197,"journal":{"name":"Journal of Neuropsychology","volume":"19 3","pages":"473-485"},"PeriodicalIF":1.8,"publicationDate":"2025-02-13","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://bpspsychub.onlinelibrary.wiley.com/doi/epdf/10.1111/jnp.12419","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"143412612","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":4,"RegionCategory":"心理学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"OA","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}

{"title":"Dopamine genetic risk scores and psychiatric symptoms: Interacting risk factors for impulse control behaviours in de novo Parkinson's disease","authors":"Emma Whooley,&nbsp;Vincent Koppelmans,&nbsp;Hayley J. MacDonald,&nbsp;Alison Hall,&nbsp;Marit F. L. Ruitenberg","doi":"10.1111/jnp.12415","DOIUrl":"10.1111/jnp.12415","url":null,"abstract":"<p>Up to 45% of patients with Parkinson's disease (PD) experience impulse control disorders (ICDs), characterized by a loss of voluntary control over impulses, drives or temptations. This study aimed to investigate whether previously identified genetic and psychiatric risk factors interact towards the development of ICDs in PD. A total of 278 <i>de novo</i> PD patients (ICD-free at enrollment) were selected from the Parkinson's Progression Markers Initiative database. ICD presence at baseline and yearly follow-up assessments were evaluated via the Questionnaire for Impulsive-Compulsive Disorders. Symptoms of anxiety and depression at baseline were measured via the State–Trait-Anxiety Inventory and Geriatric Depression Scale, respectively. Furthermore, an individual dopamine genetic risk score was calculated according to polymorphisms in genes coding for dopamine (D1, D2 and D3 receptors and catechol-O-methyltransferase), with higher scores reflecting higher central dopamine neurotransmission. In total, 146 participants (47.5%) developed an ICD with an average onset time of 36 months (range 3–96) from baseline. Results from a Cox proportional hazards model showed a trait anxiety × genetics interaction, suggesting that individuals with both higher baseline trait anxiety scores and higher dopamine genetic risk scores were at increased risk of ICD development. This interaction remained significant after controlling for age, sex and motor symptom severity. Our findings suggest that genetic and psychiatric predictors of impulsivity in PD interact and jointly yield increased ICD risk during the course of the disorder. This implies that early screening of anxiety symptoms in combination with genotyping can be useful to identify those at risk for ICD.</p>","PeriodicalId":197,"journal":{"name":"Journal of Neuropsychology","volume":"19 3","pages":"443-453"},"PeriodicalIF":1.8,"publicationDate":"2025-01-28","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://www.ncbi.nlm.nih.gov/pmc/articles/PMC12301494/pdf/","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"143057396","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":4,"RegionCategory":"心理学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"OA","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}

{"title":"Reimagining André Rey's method for recording the copy process of the Rey Complex Figure Test: A commentary","authors":"Maneesh V. Kuruvilla,&nbsp;Angela Blazely","doi":"10.1111/jnp.12414","DOIUrl":"10.1111/jnp.12414","url":null,"abstract":"<p>In 1941, André Rey published the Rey Complex Figure, a widely used test for assessing visual-constructional ability and visual memory. It consists of two parts: copy and recall. Evaluating the copy portion presents challenges, as it requires the administrator to focus on both the process and outcome. The assessor must systematically track how the patient copies the figure in real-time to evaluate their planning, organisation and executive abilities. This ‘clinician's copy’ serves as a record of the patient's approach, aiding later judgements about their cognitive skills. To ensure accuracy, clinicians need a method to record this process for later review or colleague consultation. This paper revisits Rey's suggestion of using different coloured pencils to observe the copy sequence, proposing an alternative. Instead of providing coloured pencils to the patient, we recommend that the administrator use them to record the copy sequence. This method aligns with test norms, reducing potential distractions for the patient while enabling both experienced and novice administrators to easily track and document the sequence of copying.</p>","PeriodicalId":197,"journal":{"name":"Journal of Neuropsychology","volume":"19 3","pages":"649-654"},"PeriodicalIF":1.8,"publicationDate":"2025-01-24","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://bpspsychub.onlinelibrary.wiley.com/doi/epdf/10.1111/jnp.12414","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"143031804","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":4,"RegionCategory":"心理学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"OA","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}

{"title":"Time to align sensitive cognitive assessment with protein biomarkers in Alzheimer's disease","authors":"Jet M. J. Vonk","doi":"10.1111/jnp.12413","DOIUrl":"10.1111/jnp.12413","url":null,"abstract":"&lt;p&gt;In the last decade, Alzheimer's disease research has seen large shifts, particularly regarding diagnostic criteria and the use of protein biomarkers. The Alzheimer's Association (AA) workgroup has recently revised their previous 2018 research criteria (Jack Jr et al., &lt;span&gt;2018&lt;/span&gt;) for diagnosis and staging of Alzheimer's disease to now inform both research and clinical care (Jack Jr et al., &lt;span&gt;2024a&lt;/span&gt;), maintaining a biological definition that primarily relies upon protein biomarkers, such as amyloid and tau levels, to identify the disease even in the absence of cognitive symptoms (e.g. memory and language impairment). In counter-response, the International Working Group (IWG) advocates for an integrative clinical-biological approach, emphasizing that the diagnosis of Alzheimer's disease should not rely solely on protein biomarkers but must also incorporate phenotypic expression such as objective cognitive impairment (Dubois et al., &lt;span&gt;2021&lt;/span&gt;, &lt;span&gt;2024&lt;/span&gt;).&lt;/p&gt;&lt;p&gt;The debate over a biological versus clinical-biological definition of Alzheimer's disease has been ongoing for years but was reignited earlier this year after the release of the revised AA criteria (Jack Jr et al., &lt;span&gt;2024a&lt;/span&gt;). Petersen et al. (Petersen et al., &lt;span&gt;2024&lt;/span&gt;). presented a well-balanced comparison of the overlapping standpoints and differences between the AA and IWG frameworks. Proponents of the biological-only approach have argued that focusing on protein biomarkers, such as amyloid and tau, allows for earlier detection of the disease, providing a window for intervention during the preclinical stage before a significant cognitive decline occurs (Jack Jr et al., &lt;span&gt;2024b&lt;/span&gt;). Jack Jr et al (Jack Jr et al., &lt;span&gt;2024a&lt;/span&gt;) highlight that protein biomarkers offer a more standardized and objectively replicable framework, reducing the variability seen in clinical assessments alone. Critics of the biological framework point out significant limitations and risks. Many researchers caution that a protein biomarker-only approach may lead to overdiagnosis, unnecessary anxiety and stigma for individuals who may never clinically express the underlying disease pathology (Petersen et al., &lt;span&gt;2024&lt;/span&gt;). This perspective reflects concerns about the potential psychological, social and ethical ramifications of labelling asymptomatic individuals as having Alzheimer's disease based purely on protein-biomarker positivity, given the variability in symptom progression and the influence of factors such as cognitive reserve (Glymour et al., &lt;span&gt;2018&lt;/span&gt;; Kiselica et al., &lt;span&gt;2024&lt;/span&gt;).&lt;/p&gt;&lt;p&gt;While a positive protein biomarker test may justify a diagnosis of a disease, the debate raises the question of whether this diagnosis should be ‘Alzheimer's disease’, the same label traditionally associated with progressive cognitive decline and underlying neuropathology. Using the same term for asymptomatic individuals with abnormal prote","PeriodicalId":197,"journal":{"name":"Journal of Neuropsychology","volume":"19 2","pages":"172-175"},"PeriodicalIF":1.8,"publicationDate":"2025-01-19","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://onlinelibrary.wiley.com/doi/epdf/10.1111/jnp.12413","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"142997082","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":4,"RegionCategory":"心理学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"OA","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}

{"title":"Comparison of children and adolescents diagnosed with attention deficit hyperactivity disorder with and without autism traits in terms of emotion regulation, clinical characteristics and functionality","authors":"Buket Kılıç,&nbsp;Sadriye E. Ç. Kültür","doi":"10.1111/jnp.12411","DOIUrl":"10.1111/jnp.12411","url":null,"abstract":"<p>This study aims to demonstrate that children and adolescents diagnosed with Attention Deficit Hyperactivity Disorder (ADHD) who exhibit autism traits have a more severe clinical profile in terms of emotion regulation, clinical features related to ADHD, and functionality, compared to those diagnosed with ADHD without these traits. 50 patients with and 64 patients without autism traits between the ages of 8–16 were recruited for the study among the children and adolescents diagnosed with ADHD. The Schedule for Affective Disorders and Schizophrenia for School-Age Children-Present and Lifetime Version, DSM-5-2016-Turkish Adaptation (K-SADS-PL-DSM-5-T) was used to exclude the diagnosis of Autism Spectrum Disorder (ASD) and detect comorbid psychiatric diagnosis. The Social Reciprocity Scale (SRS) was completed by parents to determine groups based on autism traits. Children completed the Childhood Anxiety Sensitivity Index (CASI) and the Affective Reactivity Index (ARI). The Strengths and Difficulties Questionnaire (SDQ), Affective Reactivity Index-Parent Report (ARI-P) and Weiss Functional Impairment Rating Scale-Parent Report (WFIRS-P) were completed by the parents. We found that the group with autism traits had significantly more hyperactivity/inattention, conduct problems, emotional problems, and peer problems and significantly more irritability and frequent separation anxiety disorder. Although there was no significant impairment in functionality in either group, the level of impairment was significantly higher in the group with autism traits. Children diagnosed with Attention Deficit Hyperactivity Disorder who exhibit autism traits experience higher levels of irritability and separation anxiety disorder, as well as greater impairment in functionality, compared to those without these traits.</p>","PeriodicalId":197,"journal":{"name":"Journal of Neuropsychology","volume":"19 3","pages":"425-442"},"PeriodicalIF":1.8,"publicationDate":"2025-01-08","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"142941976","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":4,"RegionCategory":"心理学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}

{"title":"Are patients with Parkinson's disease impaired in the recognition of emotion's authenticity?","authors":"Agnese Anzani,&nbsp;Stefano Zago,&nbsp;Teresa Difonzo,&nbsp;Cristina Scarpazza,&nbsp;Nadia Bolognini,&nbsp;Giulia Franco,&nbsp;Alessio Difonzo,&nbsp;Maria Cristina Saetti","doi":"10.1111/jnp.12410","DOIUrl":"10.1111/jnp.12410","url":null,"abstract":"<p>In recognising emotions expressed by others, one can make use of both embodied cognition and mechanisms that do not necessarily require activation of the limbic system, such as evoking from memory the meaning of morphological features of the observed face. Instead, we believe that the recognition of the authenticity of an emotional expression is primarily based on embodied cognition, for which the mirror system would play a significant role. To verify this hypothesis, we submitted 20 parkinsonian patients and 20 healthy control subjects to the Emotional Authenticity Recognition test, a novel test using dynamic stimuli to evaluate the ability to recognise emotions and their authenticity. Analysis of variance of the test scores shows that Parkinsonian patients perform worse than controls when they had to recognise the authenticity of emotions, although they are able to identify them. Our results confirm a deficit in the recognition of the authenticity of emotions in patients with Parkinson's disease attributable to the disruption of extrapiramidal limbic circuit between ventral striatum and orbitomesial-prefrontal cortex.</p>","PeriodicalId":197,"journal":{"name":"Journal of Neuropsychology","volume":"19 3","pages":"411-424"},"PeriodicalIF":1.8,"publicationDate":"2024-12-26","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://bpspsychub.onlinelibrary.wiley.com/doi/epdf/10.1111/jnp.12410","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"142891079","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":4,"RegionCategory":"心理学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"OA","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}

{"title":"Multifactorial interplay on language recovery following left-hemispheric stroke: A retrospective study","authors":"Célise Haldin,&nbsp;Shenhao Dai,&nbsp;Céline Piscicelli,&nbsp;Valérie Marcon,&nbsp;Hélène Lœvenbruck,&nbsp;Dominic Pérennou,&nbsp;Monica Baciu","doi":"10.1111/jnp.12406","DOIUrl":"10.1111/jnp.12406","url":null,"abstract":"<p>Understanding the relative contribution of various factors influencing initial severity of aphasia and recovery after a stroke is essential for optimising neurorehabilitation programmes. We investigated how various significant sociodemographic, cognitive, clinical, stroke-related and rehabilitation-related factors modulate aphasia severity and language recovery following left-hemispheric stroke. Employing an innovative method, we conducted a retrospective analysis of 96 stroke participants to explore the combined impact of these factors. The initial severity of aphasia was categorised into severe, mild/moderate and no aphasia based on the severity of their language deficits in the subacute phase (Aphasia Severity Rating Scale, ASRS). To assess speech-and-language recovery, we classified 53/96 patients with aphasia into high and poor recovery categories using a gain score formula (ASRS_discharge—ASRS_admission)/ASRS_admission. Subsequently, we performed statistical analyses (univariate analyses and forward stepwise logistic regression combined with bootstrap) to identify the determinants of the initial severity of aphasia and the degree of recovery. Our analyses unveiled that more severe aphasia initially was correlated with a more severe stroke (Odds Ratio, OR = .90, <i>p</i> = .041), moderate/severe executive dysfunction (OR = .068, <i>p</i> &lt; .001) and larger lesion size (OR = .068, <i>p</i> &lt; .001). Furthermore, the degree of recovery was associated with the intensity of speech-and-language therapy (OR = 1.47, <i>p</i> = .043). These findings enrich our understanding of the determinants of aphasia severity and language recovery, employing an original methodology to scrutinise the collective effect of multiple variables in a retrospective analysis of stroke participants. A better knowledge of these factors may help implement personalised language rehabilitation programmes to maximise speech-and-language recovery.</p>","PeriodicalId":197,"journal":{"name":"Journal of Neuropsychology","volume":"19 2","pages":"256-273"},"PeriodicalIF":1.8,"publicationDate":"2024-12-23","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://onlinelibrary.wiley.com/doi/epdf/10.1111/jnp.12406","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"142881000","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":4,"RegionCategory":"心理学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"OA","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}

{"title":"Early neuropsychological screening and long-term functional outcome in a sample of patients affected by mild stroke: The ReSCog Project","authors":"Elisa Pini,&nbsp;Fulvio Pepe,&nbsp;Veronica Laini,&nbsp;Nicoletta Ciccarelli,&nbsp;Eugenio Magni","doi":"10.1111/jnp.12408","DOIUrl":"10.1111/jnp.12408","url":null,"abstract":"<p>Stroke causes severe long-term disabilities with a significant reduction in quality of life. This study aims to explore the predictive value of cognitive screening in the acute phase of mild stroke on patients' functional outcome after discharge. A total of 110 patients with mild stroke were recruited. Patients were included in the study if they were discharged directly home from the acute units. The cognitive profile of patients was assessed with the Oxford Cognitive Screen (OCS). The OCS was administered 3–10 days after stroke, providing a five domain-specific cognitive profile. Long-term functional outcomes were evaluated by the Stroke Impact Scale 3.0 (SIS 3.0), a self-reported questionnaire that includes physical, cognitive, emotional and social participation dimensions. All patients completed the survey online on average 10 months after stroke. Our results show that OCS is positively associated with physical and cognitive dimensions, after adjusting for age and stroke severity measured by NIHSS at admission. In conclusion, OCS in acute mild stroke seems to be an independent predictor of long-term functional outcomes and could help clinicians in the long-term management of patients.</p>","PeriodicalId":197,"journal":{"name":"Journal of Neuropsychology","volume":"19 2","pages":"286-298"},"PeriodicalIF":1.8,"publicationDate":"2024-12-22","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"142875846","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":4,"RegionCategory":"心理学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}