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Engineered IL1R1-CD3 Tregs modulate neuroimmune responses and attenuate neuropathic pain in a rat CCI model. 在大鼠CCI模型中，工程IL1R1-CD3 Tregs调节神经免疫反应并减轻神经性疼痛。
IF 3.1 4区 医学
Immunologic Research Pub Date : 2026-04-27 DOI: 10.1007/s12026-026-09781-3
Jun Chen, Junfeng Li, Qin Qin, Le Zheng
{"title":"Engineered IL1R1-CD3 Tregs modulate neuroimmune responses and attenuate neuropathic pain in a rat CCI model.","authors":"Jun Chen, Junfeng Li, Qin Qin, Le Zheng","doi":"10.1007/s12026-026-09781-3","DOIUrl":"https://doi.org/10.1007/s12026-026-09781-3","url":null,"abstract":"","PeriodicalId":13389,"journal":{"name":"Immunologic Research","volume":"74 1","pages":""},"PeriodicalIF":3.1,"publicationDate":"2026-04-27","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"147770316","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":4,"RegionCategory":"医学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
引用次数: 0
Innovative biomaterial solutions for enhancing the efficacy of tumor immunotherapy. 提高肿瘤免疫治疗效果的创新生物材料解决方案。
IF 3.1 4区 医学
Immunologic Research Pub Date : 2026-04-27 DOI: 10.1007/s12026-026-09762-6
Pengwei Zhu
{"title":"Innovative biomaterial solutions for enhancing the efficacy of tumor immunotherapy.","authors":"Pengwei Zhu","doi":"10.1007/s12026-026-09762-6","DOIUrl":"https://doi.org/10.1007/s12026-026-09762-6","url":null,"abstract":"","PeriodicalId":13389,"journal":{"name":"Immunologic Research","volume":"74 1","pages":""},"PeriodicalIF":3.1,"publicationDate":"2026-04-27","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://www.ncbi.nlm.nih.gov/pmc/articles/PMC13121201/pdf/","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"147770282","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":4,"RegionCategory":"医学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"OA","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
引用次数: 0
FTO-mediated m6A demethylation of TRIM21 regulates macrophage polarization and attenuates myocardial ischemia-reperfusion injury. fto介导的TRIM21的m6A去甲基化调节巨噬细胞极化，减轻心肌缺血再灌注损伤。
IF 3.1 4区 医学
Immunologic Research Pub Date : 2026-04-25 DOI: 10.1007/s12026-026-09765-3
Zheng Wan, Yanchun Ding
{"title":"FTO-mediated m6A demethylation of TRIM21 regulates macrophage polarization and attenuates myocardial ischemia-reperfusion injury.","authors":"Zheng Wan, Yanchun Ding","doi":"10.1007/s12026-026-09765-3","DOIUrl":"https://doi.org/10.1007/s12026-026-09765-3","url":null,"abstract":"","PeriodicalId":13389,"journal":{"name":"Immunologic Research","volume":"74 1","pages":""},"PeriodicalIF":3.1,"publicationDate":"2026-04-25","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"147770300","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":4,"RegionCategory":"医学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
引用次数: 0
Epitope prediction and immunogenicity assessment of Leishmania donovani TryS protein as a potential vaccine candidate. 多诺瓦利什曼原虫TryS蛋白作为潜在候选疫苗的表位预测和免疫原性评价。
IF 3.1 4区 医学
Immunologic Research Pub Date : 2026-04-18 DOI: 10.1007/s12026-026-09775-1
Ehasanullah Khan, Shiv Kumar, Manash Sarma, Prasoon Madhukar, Shyam Sundar, Rajiv Kumar, Vikash Kumar Dubey
{"title":"Epitope prediction and immunogenicity assessment of Leishmania donovani TryS protein as a potential vaccine candidate.","authors":"Ehasanullah Khan, Shiv Kumar, Manash Sarma, Prasoon Madhukar, Shyam Sundar, Rajiv Kumar, Vikash Kumar Dubey","doi":"10.1007/s12026-026-09775-1","DOIUrl":"https://doi.org/10.1007/s12026-026-09775-1","url":null,"abstract":"","PeriodicalId":13389,"journal":{"name":"Immunologic Research","volume":"74 1","pages":""},"PeriodicalIF":3.1,"publicationDate":"2026-04-18","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"147716686","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":4,"RegionCategory":"医学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
引用次数: 0
Inverse relationship between Epstein-Barr virus serostatus and anti-glutamic acid decarboxylase autoantibody levels. eb病毒血清状态与抗谷氨酸脱羧酶自身抗体水平呈反比关系。
IF 3.1 4区 医学
Immunologic Research Pub Date : 2026-04-18 DOI: 10.1007/s12026-026-09778-y
Cyril Debuysschere, Mahdi Ouafi, Magloire Pandoua Nekoua, Delphine Lobert, Pascal Pigny, Enagnon Kazali Alidjinou, Didier Hober
{"title":"Inverse relationship between Epstein-Barr virus serostatus and anti-glutamic acid decarboxylase autoantibody levels.","authors":"Cyril Debuysschere, Mahdi Ouafi, Magloire Pandoua Nekoua, Delphine Lobert, Pascal Pigny, Enagnon Kazali Alidjinou, Didier Hober","doi":"10.1007/s12026-026-09778-y","DOIUrl":"10.1007/s12026-026-09778-y","url":null,"abstract":"","PeriodicalId":13389,"journal":{"name":"Immunologic Research","volume":"74 1","pages":""},"PeriodicalIF":3.1,"publicationDate":"2026-04-18","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://www.ncbi.nlm.nih.gov/pmc/articles/PMC13091841/pdf/","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"147716626","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":4,"RegionCategory":"医学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"OA","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
引用次数: 0
Lewis Y (Ley) orchestrates leukocyte trafficking and inflammatory remodeling in leprosy patients from the Brazilian Amazon. Lewis Y （Ley）在巴西亚马逊麻风病患者中协调白细胞运输和炎症重塑。
IF 3.1 4区 医学
Immunologic Research Pub Date : 2026-04-09 DOI: 10.1007/s12026-026-09776-0
Sintia Almeida, Ermelinda do Rosário Moutinho da Cruz, Rosane do Socorro Pompeu de Loiola, Tereza C O Corvelo
{"title":"Lewis Y (Le<sup>y</sup>) orchestrates leukocyte trafficking and inflammatory remodeling in leprosy patients from the Brazilian Amazon.","authors":"Sintia Almeida, Ermelinda do Rosário Moutinho da Cruz, Rosane do Socorro Pompeu de Loiola, Tereza C O Corvelo","doi":"10.1007/s12026-026-09776-0","DOIUrl":"10.1007/s12026-026-09776-0","url":null,"abstract":"","PeriodicalId":13389,"journal":{"name":"Immunologic Research","volume":"74 1","pages":""},"PeriodicalIF":3.1,"publicationDate":"2026-04-09","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://www.ncbi.nlm.nih.gov/pmc/articles/PMC13065620/pdf/","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"147645174","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":4,"RegionCategory":"医学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"OA","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
引用次数: 0
Subcutaneous Sweet Syndrome as an uncommon mucocutaneous manifestation in systemic lupus erythematosus: a case report and literature review. 系统性红斑狼疮的皮下甜综合征是一种罕见的粘膜皮肤表现：1例报告并文献复习。
IF 3.1 4区 医学
Immunologic Research Pub Date : 2026-04-07 DOI: 10.1007/s12026-026-09773-3
Erik Cimé-Aké, David Arriaga Escamilla, Javier Ríos-Valencia, Marcela Saeb-Lima, Hilda Fragoso-Loyo
{"title":"Subcutaneous Sweet Syndrome as an uncommon mucocutaneous manifestation in systemic lupus erythematosus: a case report and literature review.","authors":"Erik Cimé-Aké, David Arriaga Escamilla, Javier Ríos-Valencia, Marcela Saeb-Lima, Hilda Fragoso-Loyo","doi":"10.1007/s12026-026-09773-3","DOIUrl":"https://doi.org/10.1007/s12026-026-09773-3","url":null,"abstract":"","PeriodicalId":13389,"journal":{"name":"Immunologic Research","volume":"74 1","pages":""},"PeriodicalIF":3.1,"publicationDate":"2026-04-07","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"147627717","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":4,"RegionCategory":"医学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
引用次数: 0
Epigenetic regulators and inflammation antagonists in familial Mediterranean fever: the role of hsa-miR-335-5p, hsa-miR-26b-5p, hsa-miR-16-5p miRNAs and IL-36Ra levels in pathogenesis. 家族性地中海热的表观遗传调控因子和炎症拮抗剂：hsa-miR-335-5p、hsa-miR-26b-5p、hsa-miR-16-5p mirna和IL-36Ra水平在发病机制中的作用
IF 3.1 4区 医学
Immunologic Research Pub Date : 2026-04-06 DOI: 10.1007/s12026-026-09771-5
Sedef Erdik, Ayla Solmaz Avcikurt, Hilmi Bolat
{"title":"Epigenetic regulators and inflammation antagonists in familial Mediterranean fever: the role of hsa-miR-335-5p, hsa-miR-26b-5p, hsa-miR-16-5p miRNAs and IL-36Ra levels in pathogenesis.","authors":"Sedef Erdik, Ayla Solmaz Avcikurt, Hilmi Bolat","doi":"10.1007/s12026-026-09771-5","DOIUrl":"https://doi.org/10.1007/s12026-026-09771-5","url":null,"abstract":"<p><p>Familial Mediterranean Fever (FMF) is a hereditary autoinflammatory disease characterized by mutations in the MEFV and an over-activation of the pyrin inflammatory cascade. In this study, the roles of three specific microRNAs (miRNAs) and the inflammatory cytokine antagonist IL-36Ra in the pathogenesis of FMF were investigated due to their potential roles in chronic inflammation. 40 FMF patients and 45 healthy control individuals who applied to the Balikesir University Genetic Disorders Evaluation Center between February 2021 and June 2024 were included in the study. miRNA expression levels were determined by Quantitative Real-Time Polymerase Chain Reaction (qRT-PCR), and IL-36Ra protein levels were determined using the ELISA method. Gene expression levels were analyzed by the 2-ΔΔCt method. A significant decrease (p < 0.005) in hsa-miR-335-5p and hsa-miR-26b-5p expression levels and a significant increase (p < 0.005) in hsa-miR-16-5p and IL-36Ra protein levels were observed in FMF patients. These findings point to the existence of a two-way molecular mechanism in FMF pathogenesis: on one hand, there is a deficit in epigenetic regulators with anti-inflammatory properties, such as hsa-miR-335-5p and hsa-miR-26b-5p, while on the other hand, molecules like hsa-miR-16-5p and IL-36Ra increase as a compensatory response to balance the inflammatory load. These molecules show promise as potential biomarkers for the diagnosis and follow-up of FMF.</p>","PeriodicalId":13389,"journal":{"name":"Immunologic Research","volume":"74 1","pages":""},"PeriodicalIF":3.1,"publicationDate":"2026-04-06","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"147627765","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":4,"RegionCategory":"医学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
引用次数: 0
Effectiveness and safety of ravulizumab in anti-acetylcholine receptor antibody-positive (AChR-Ab+) generalized myasthenia gravis: a systematic review and meta-analysis. ravulizumab治疗抗乙酰胆碱受体抗体阳性（AChR-Ab+）全身性重症肌无力的有效性和安全性：一项系统综述和荟萃分析
IF 3.1 4区 医学
Immunologic Research Pub Date : 2026-04-05 DOI: 10.1007/s12026-026-09770-6
Haneen Sabet, Abdallah Abbas, Abrar AbuHamdia, Esraa Y Salama, Dina Essam Abo-Elnour, Sultan Mohammed Alanazi, Mahmoud G A Saleh, Yasmin Negida
{"title":"Effectiveness and safety of ravulizumab in anti-acetylcholine receptor antibody-positive (AChR-Ab+) generalized myasthenia gravis: a systematic review and meta-analysis.","authors":"Haneen Sabet, Abdallah Abbas, Abrar AbuHamdia, Esraa Y Salama, Dina Essam Abo-Elnour, Sultan Mohammed Alanazi, Mahmoud G A Saleh, Yasmin Negida","doi":"10.1007/s12026-026-09770-6","DOIUrl":"https://doi.org/10.1007/s12026-026-09770-6","url":null,"abstract":"","PeriodicalId":13389,"journal":{"name":"Immunologic Research","volume":"74 1","pages":""},"PeriodicalIF":3.1,"publicationDate":"2026-04-05","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"147622858","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":4,"RegionCategory":"医学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
引用次数: 0
From PD-L1 downregulation to biomarker-driven immunotherapy design: advancing translational application of SALL4-targeted PEN-FFW in breast cancer. 从PD-L1下调到生物标志物驱动的免疫治疗设计：推进sall4靶向PEN-FFW在乳腺癌中的转化应用
IF 3.1 4区 医学
Immunologic Research Pub Date : 2026-04-02 DOI: 10.1007/s12026-026-09774-2
M Vijayasimha, M Srikanth
{"title":"From PD-L1 downregulation to biomarker-driven immunotherapy design: advancing translational application of SALL4-targeted PEN-FFW in breast cancer.","authors":"M Vijayasimha, M Srikanth","doi":"10.1007/s12026-026-09774-2","DOIUrl":"https://doi.org/10.1007/s12026-026-09774-2","url":null,"abstract":"","PeriodicalId":13389,"journal":{"name":"Immunologic Research","volume":"74 1","pages":""},"PeriodicalIF":3.1,"publicationDate":"2026-04-02","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"147591770","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":4,"RegionCategory":"医学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
引用次数: 0
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