Advances in Nutrition最新文献

{"title":"Invitation for Nominations for 2027","authors":"","doi":"10.1016/j.advnut.2026.100748","DOIUrl":"10.1016/j.advnut.2026.100748","url":null,"abstract":"","PeriodicalId":7349,"journal":{"name":"Advances in Nutrition","volume":"17 10","pages":"Article 100748"},"PeriodicalIF":12.7,"publicationDate":"2026-10-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"149212331","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":1,"RegionCategory":"医学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"OA","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}

{"title":"Perspectives on the Nutritional Paradox in Uremia: A “Sensitize-Then-Feed” Framework for Protein-Energy Wasting","authors":"Maoting Li, Zewei Chen, Nanmei Liu, Naiying Lan, Fanzhou Zeng, Cheng Xue, Bo Yang","doi":"10.1016/j.advnut.2026.100728","DOIUrl":"10.1016/j.advnut.2026.100728","url":null,"abstract":"<div><div>Protein-energy wasting (PEW) drives significant morbidity and mortality in advanced chronic kidney disease (CKD). Despite the implementation of comprehensive clinical guidelines and medical nutrition therapy, a subset of uremic patients may exhibit a defining “nutritional paradox.” Unlike simple starvation, which responds to caloric and protein supplementation, CKD-associated cachexia is characterized by a potential systemic anabolic resistance. This renders standard nutritional interventions—including oral supplements and intradialytic parenteral nutrition—often insufficient. To elucidate the molecular hierarchy driving this barrier to nutritional therapy, we propose a model in which, during central dysregulation, gut-microbiome–derived uremic toxins, notably tryptophan metabolites, such as indoxyl sulfate, have been shown in experimental models to impair the blood–brain barrier integrity. This breach triggers significant hypothalamic neuroinflammation. Concurrently, proinflammatory cytokines (e.g., interleukin-1<em>β</em>, tumor necrosis factor–<em>α</em>) and stress-responsive circulating factors like growth differentiation factor 15 (GDF15) aberrantly reset the central metabolic set point via the melanocortin system, potentially contributing to anorexia and altered energy expenditure. Peripherally, the failure of critical nutrient-sensing axes—specifically, the ghrelin–ghrelin O-acyltransferase and insulin/insulin-like growth factor–1 signaling pathways—may impair the anabolic phosphoinositide 3-kinase/Akt/mechanistic target of rapamycin complex 1 cascade. This peripheral desensitization not only blunts the synthetic response to dietary amino acids but also precipitates a disruption of muscle proteostasis through ubiquitin–proteasome system hyperactivation and forkhead box O–mediated transcription. Concurrently, skeletal muscle undergoes significant mitochondrial bioenergetic dysfunction, further impairing nutrient utilization. By reframing PEW not simply as a state of nutrient deficiency, but as a broader dysregulation of hormonal signaling, we advocate a translational “sensitize-then-feed” therapeutic paradigm. We critically evaluate emerging targeted agents—including GDF15 antagonists, myostatin inhibitors, and ghrelin agonists. We propose that these agents could be investigated as metabolic modulators to potentially enhance the efficacy of tailored nutritional support in the uremic microenvironment.</div></div><div>Protein-energy wasting (PEW) drives significant morbidity and mortality in advanced chronic kidney disease (CKD). Despite the implementation of comprehensive clinical guidelines and medical nutrition therapy, a subset of uremic patients may exhibit a defining “nutritional paradox.” Unlike simple starvation, which responds to caloric and protein supplementation, CKD-associated cachexia is characterized by a potential systemic anabolic resistance. This renders standard nutritional interventions—including oral supplement","PeriodicalId":7349,"journal":{"name":"Advances in Nutrition","volume":"17 10","pages":"Article 100728"},"PeriodicalIF":12.7,"publicationDate":"2026-10-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"148921934","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":1,"RegionCategory":"医学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"OA","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}

{"title":"Interface of Climate and Environmental Change on Food Systems—A Report from the “Agriculture and Diet: Value Added for Nutrition, Translation, and Adaptation in a Global Ecology” (ADVANTAGE) Project Working Group 3","authors":"Robin R White, Matthew R Smith, Lewis Ziska, Kathryn J Fiorella, Daniel J Raiten","doi":"10.1016/j.advnut.2026.100724","DOIUrl":"10.1016/j.advnut.2026.100724","url":null,"abstract":"<div><div>As the climate changes, investigation at the intersection of climate and food systems is critical to understand broader impacts on human nutrition and health. This work seeks to <em>1</em>) identify reciprocal interactions of climate and environmental change (CEC) and food systems, <em>2</em>) illuminate research gaps in CEC interactions with food systems, and <em>3</em>) identify paths to respond to and mitigate the impact of CEC on food systems. We discuss CEC and food system interactions via 4 pathways. Food systems are influenced by CEC through shifting climate normals and altered instances of extreme events. The physical systems necessary to support food systems are also affected by CEC through ocean conditions, freshwater availability, soil systems, ground-level ozone, and surface water quality. The biological systems underpinning food systems are also altered by CEC, through shifts in pest, weed, and pathogen pressure, wild pollinator populations, and biodiversity changes. The agricultural production system driving food systems is also influenced by CEC via labor shifts and productivity changes, disruptions to supply chains, and spoilage and foodborne diseases. Each of these pathways can alter growing conditions for crop- and animal-based foods, leading to shifts in productivity and nutrient composition and altering food safety, distribution, access, and availability. Adapting food systems to CEC requires qualitative and quantitative interventions, emphasizing system adaptability and mitigation of climate change. Although relationships between CEC and food systems are well studied, research gaps highlight the need to assess the impacts of simultaneous climate factors on crop yields, nutrient density, and food quality; to refine cultivars, technologies, and management practices for resilient food production; to identify social, economic, and political strategies to facilitate global food security goals; to utilize bridging data for robust modeling; and to advance nonagricultural nutrient production systems.</div></div><div>As the climate changes, investigation at the intersection of climate and food systems is critical to understand broader impacts on human nutrition and health. This work seeks to <em>1</em>) identify reciprocal interactions of climate and environmental change (CEC) and food systems, <em>2</em>) illuminate research gaps in CEC interactions with food systems, and <em>3</em>) identify paths to respond to and mitigate the impact of CEC on food systems. We discuss CEC and food system interactions via 4 pathways. Food systems are influenced by CEC through shifting climate normals and altered instances of extreme events. The physical systems necessary to support food systems are also affected by CEC through ocean conditions, freshwater availability, soil systems, ground-level ozone, and surface water quality. The biological systems underpinning food systems are also altered by CEC, through shifts in pest, weed, and path","PeriodicalId":7349,"journal":{"name":"Advances in Nutrition","volume":"17 10","pages":"Article 100724"},"PeriodicalIF":12.7,"publicationDate":"2026-10-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"148802244","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":1,"RegionCategory":"医学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"OA","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}

{"title":"Expert Perspectives on Diagnosis and Management of Vitamin B12 Deficiency in Adults","authors":"Jean-Louis Guéant, David H Alpers, Lindsay H Allen, Emmanuel Andres, Angela M Devlin, Ari Green, Ralph Green, Rosa-Maria Guéant Rodriguez, Luciana Hannibal, Dominic Harrington, Joel B Mason, Andrew McCaddon, Joshua W Miller, Michelle Murphy, Ebba Nexo, Helene McNulty, Peter Julian Owen, Edward Quadros, David Rosenblatt, Helga Refsum, Julia L Finkelstein","doi":"10.1016/j.advnut.2026.100726","DOIUrl":"10.1016/j.advnut.2026.100726","url":null,"abstract":"<div><div>There is no universally accepted definition of vitamin B<sub>12</sub> deficiency despite the severity of its medical consequences if not properly diagnosed and treated. Vitamin B<sub>12</sub> deficiency can arise either from inadequate dietary intake, or various causes of impaired absorption or availability. The latter include <em>Helicobacter pylori</em> and autoimmune gastritis, other causes of malabsorption, and drug side effects. Vitamin B<sub>12</sub> deficiency can display diverse clinical signs and symptoms, including hematological, neurological, psychiatric, and thromboembolic manifestations. The minutes and conclusions of 3 workshops at the Federation of American Societies of Experimental Biology (FASEB) “B-vitamins and One-Carbon Metabolism” Scientific Research Conferences (2022 and 2024) and the 2025 International Conference on One-Carbon Metabolism, B-Vitamins and Homocysteine, the examination of the literature and the National Institute for Health and Care Excellence/DELPHI guidelines allowed us to identify gaps and issues on diagnostic criteria and definition, evaluation of causation, and treatment strategies of vitamin B<sub>12</sub> deficiency across different health care settings in adults (>18 y). Our aim is to address these gaps and issues in global guidelines to be reviewed and disseminated by the International Union of Nutritional Sciences. We identified 4 main goals: <em>1</em>) definition of vitamin B<sub>12</sub> deficiency; <em>2</em>) assessment of causality; <em>3</em>) differential diagnosis and evaluation of nutritional and metabolic comorbidities; and <em>4</em>) treatment and follow-up. The objective of such guidelines should be to reduce both underdiagnosis and overdiagnosis of vitamin B<sub>12</sub> deficiency by integrating clinical biomarker assessment and considering cause-specific treatment pathways. This should ultimately improve patient outcomes worldwide and, by arriving at uniform diagnostic criteria, facilitate the conduct, interpretation, and integration of research observations to inform clinical guidelines.</div></div><div>There is no universally accepted definition of vitamin B<sub>12</sub> deficiency despite the severity of its medical consequences if not properly diagnosed and treated. Vitamin B<sub>12</sub> deficiency can arise either from inadequate dietary intake, or various causes of impaired absorption or availability. The latter include <em>Helicobacter pylori</em> and autoimmune gastritis, other causes of malabsorption, and drug side effects. Vitamin B<sub>12</sub> deficiency can display diverse clinical signs and symptoms, including hematological, neurological, psychiatric, and thromboembolic manifestations. The minutes and conclusions of 3 workshops at the Federation of American Societies of Experimental Biology (FASEB) “B-vitamins and One-Carbon Metabolism” Scientific Research Conferences (2022 and 2024) and the 2025 International Conference on One-Carbon Metabolism, B-Vitamin","PeriodicalId":7349,"journal":{"name":"Advances in Nutrition","volume":"17 10","pages":"Article 100726"},"PeriodicalIF":12.7,"publicationDate":"2026-10-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"148820383","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":1,"RegionCategory":"医学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"OA","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}

{"title":"Nutrition Care and Dietetic Practice in Managing Metabolic and Endocrine Dysfunction in Polyendocrine Metabolic Ovarian Syndrome (PMOS): Implications and Opportunities in the Context of the Name Change of Polycystic Ovarian Syndrome to PMOS","authors":"Donna Vine, Lisa Moran, Helena Teede, Mahnaz Bahri-Khomami","doi":"10.1016/j.advnut.2026.100734","DOIUrl":"10.1016/j.advnut.2026.100734","url":null,"abstract":"<div><div>Polyendocrine metabolic ovarian syndrome (PMOS) affects 8‒13% of women across the lifespan. Those with PMOS have reduced quality of life and increased risk of comorbidities, including elevated weight, eating disorders, gestational diabetes, type 2 diabetes, and cardiovascular disease. PMOS is a major population in clinical dietetic practice, and dietary intervention is the first-line approach recommended to target metabolic risk in the international evidence-based guidelines for assessment and management of polycystic ovary syndrome (PCOS). The past name, PCOS, was ovarian-centric and did not accurately reflect the pathophysiology, endocrine, or metabolic dysfunction of the syndrome. The new name, PMOS, has been determined by a rigorous global consensus process including 56 organizations, >14,000 survey responses, and consultation with experts and patients in workshops that identified the need for accurate medical representation of the syndrome and a stigma-reducing name. Dietitians encounter patients with PMOS with increased cardiometabolic risk who also experience high rates of elevated weight, weight stigma, and eating disorders. Nutrition care of patients with PMOS often involves managing these conditions, use of anti-obesity and diabetes medications, conferring with prescribing clinicians, and navigating patient goals and preferences. Improved recognition of the metabolic risks can strengthen alignment with dietary management approaches and referral to nutrition care services as part of the multidisciplinary care needed in those with PMOS. Dietitians can play a key role in managing nutritional misinformation, supporting patient health literacy and nutritional self-management. The new name can contribute to expanded opportunities for nutritional research that targets endocrine and metabolic outcomes to improve the clinical care, health, and well-being of those living with PMOS.</div></div><div>Polyendocrine metabolic ovarian syndrome (PMOS) affects 8‒13% of women across the lifespan. Those with PMOS have reduced quality of life and increased risk of comorbidities, including elevated weight, eating disorders, gestational diabetes, type 2 diabetes, and cardiovascular disease. PMOS is a major population in clinical dietetic practice, and dietary intervention is the first-line approach recommended to target metabolic risk in the international evidence-based guidelines for assessment and management of polycystic ovary syndrome (PCOS). The past name, PCOS, was ovarian-centric and did not accurately reflect the pathophysiology, endocrine, or metabolic dysfunction of the syndrome. The new name, PMOS, has been determined by a rigorous global consensus process including 56 organizations, >14,000 survey responses, and consultation with experts and patients in workshops that identified the need for accurate medical representation of the syndrome and a stigma-reducing name. Dietitians encounter patients with PMOS with increased cardiometab","PeriodicalId":7349,"journal":{"name":"Advances in Nutrition","volume":"17 10","pages":"Article 100734"},"PeriodicalIF":12.7,"publicationDate":"2026-10-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"148984295","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":1,"RegionCategory":"医学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"OA","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}

{"title":"Anthocyanins","authors":"Taylor C Wallace, M Monica Guisti","doi":"10.1016/j.advnut.2026.100692","DOIUrl":"10.1016/j.advnut.2026.100692","url":null,"abstract":"","PeriodicalId":7349,"journal":{"name":"Advances in Nutrition","volume":"17 9","pages":"Article 100692"},"PeriodicalIF":12.7,"publicationDate":"2026-09-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"148364034","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":1,"RegionCategory":"医学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"OA","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}

{"title":"Fibroblast Growth Factor 21: Possible Role in Growth and Metabolic Development during Early Life, Nutritional Interactions and Biological Actions","authors":"Jibran A Wali,&nbsp;José Manuel Ramos-Nieves,&nbsp;Aristea Binia,&nbsp;Eline M van der Beek","doi":"10.1016/j.advnut.2026.100713","DOIUrl":"10.1016/j.advnut.2026.100713","url":null,"abstract":"<div><div>Fibroblast growth factor 21 (FGF21) is a hormone produced mainly in the liver that plays a crucial role in systemic metabolic regulation. Its diverse metabolic functions include promoting glucose uptake in adipocytes, enhancing fat oxidation, and inducing thermogenesis. Studies in rodents have shown that treatment with FGF21 protects against diet-induced obesity and when overexpressed, can even extend lifespan. The relationship between FGF21 and dietary macronutrients is complex and bidirectional. Diets low in protein and high in carbohydrates, particularly simple sugars, strongly stimulate the production of FGF21. Evidence from rodent experiments shows that once secreted, FGF21 acts on the brain, stimulating protein intake and suppressing the preference for sugars. Compared with adults, the role of FGF21 in metabolic physiology and programming during early life is, however, less well understood. In human infants, linear growth is negatively correlated with circulating FGF21 concentrations. In mice, lipids present in breast milk stimulate the production of FGF21 after birth, possibly activating thermogenesis. In this narrative review, we delve into the topic of FGF21 and its significance for metabolic development and growth during early life, the nutritional regulation of FGF21, and its impact on macronutrient preferences. Given the strong link between protein intake and FGF21 signaling, we hypothesize that FGF21-mediated metabolic regulation could provide a mechanistic basis for the early protein hypothesis. This hypothesis posits that higher protein intake in infancy exacerbates weight gain and increases risk of cardiometabolic disease in later life. Here, we aim to contribute to a deeper understanding of FGF21 biology, highlighting its potential to enhance healthspan and longevity through nutritional modulation during early life.</div></div>","PeriodicalId":7349,"journal":{"name":"Advances in Nutrition","volume":"17 9","pages":"Article 100713"},"PeriodicalIF":12.7,"publicationDate":"2026-09-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"148698517","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":1,"RegionCategory":"医学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"OA","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}

{"title":"Human Milk Biology: Illuminating the Path toward the Next Generation of Infant Formula","authors":"Andrew A Bremer","doi":"10.1016/j.advnut.2026.100723","DOIUrl":"10.1016/j.advnut.2026.100723","url":null,"abstract":"","PeriodicalId":7349,"journal":{"name":"Advances in Nutrition","volume":"17 9","pages":"Article 100723"},"PeriodicalIF":12.7,"publicationDate":"2026-09-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"148802311","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":1,"RegionCategory":"医学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"OA","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}

{"title":"Local Food Interventions for the Management of Moderate Acute Malnutrition (MAM) among Children Aged 6 to 59 Months: A Systematic Review and Non-inferiority Meta-Analysis","authors":"Eskeziaw Abebe Kassahun,&nbsp;Hamid Yimam Hassen,&nbsp;Alinoor Mohamed Farah,&nbsp;Yakob Desalegn Nigatu,&nbsp;Debritu Nane,&nbsp;Emily C Keats,&nbsp;Genet Kiflemariam Paulos,&nbsp;Tesfamichael Awoke Sissay,&nbsp;Kemeria Barsenga,&nbsp;Aweke Kebede,&nbsp;Robert Ackatia-Armah,&nbsp;Paluku Bahwere,&nbsp;Samson Gebremedhin,&nbsp;Seifu Hagos Gebreyesus","doi":"10.1016/j.advnut.2026.100707","DOIUrl":"10.1016/j.advnut.2026.100707","url":null,"abstract":"&lt;div&gt;&lt;div&gt;Moderate acute malnutrition (MAM) remains a major public health challenge among children aged 6 to 59 mo. Specialized nutritious foods (SNFs) are the standard intervention for the management of MAM; however, their use is constrained by supply chain disruptions, high costs, limited local production, and sustainability challenges, highlighting the need for context-specific alternatives. We conducted a systematic review and non-inferiority meta-analysis to compare local food interventions with SNFs for recovery rate and weight gain among children with MAM. We searched Medical Literature Analysis and Retrieval System On Line, Excerpta Medica dataBASE, Cochrane Central Register of Controlled Trials, Web of Science, ProQuest, and grey literature sources. Randomized controlled trials and quasi-experimental studies comparing local food interventions with SNFs were included. Local food interventions were categorized into the following 2 groups: locally available foods (LAFs), prepared from local ingredients using household or community methods, and locally processed foods (LPFs), produced from locally sourced ingredients through processing and fortification. To demonstrate non-inferiority, the predefined non-inferiority margins were set at 10% for recovery rate and 1.2 g/kg/d for weight gain. Non-inferiority was established when the lower bound of the 95% confidence interval (CI) remained above the corresponding margin. A network meta-analysis was performed to compare the relative effectiveness of LAFs, LPFs, fortified blended food, and ready-to-use supplementary food. Twenty-one studies met the eligibility criteria, and 13 were included in the meta-analyses. For recovery rate, LAFs were inferior to SNFs [risk difference (RD): −0.21; 95% CI: −0.33, −0.08], whereas LPFs were non-inferior (RD: −0.03; 95% CI: −0.09, 0.03). For weight gain, both LAFs [mean difference (MD): −0.33; 95% CI: −0.56, −0.11] and LPFs (MD: −0.32; 95% CI: −0.43, −0.21) resulted in lower weight gain than SNFs but met the predefined non-inferiority criterion. In conclusion, LPFs can serve as effective alternatives to SNFs for managing MAM. Further research is needed to optimize the formulation and effectiveness of LAFs for MAM management.&lt;/div&gt;&lt;div&gt;This review was registered at PROSPERO as CRD42024566607.&lt;/div&gt;&lt;/div&gt;&lt;div&gt;Moderate acute malnutrition (MAM) remains a major public health challenge among children aged 6 to 59 mo. Specialized nutritious foods (SNFs) are the standard intervention for the management of MAM; however, their use is constrained by supply chain disruptions, high costs, limited local production, and sustainability challenges, highlighting the need for context-specific alternatives. We conducted a systematic review and non-inferiority meta-analysis to compare local food interventions with SNFs for recovery rate and weight gain among children with MAM. We searched Medical Literature Analysis and Retrieval System On Line, Excerpta Medica dataBASE, Cochrane Cen","PeriodicalId":7349,"journal":{"name":"Advances in Nutrition","volume":"17 9","pages":"Article 100707"},"PeriodicalIF":12.7,"publicationDate":"2026-09-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"149003005","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":1,"RegionCategory":"医学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"OA","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}

{"title":"Reply to C Pang and L Zhao","authors":"Larissa L da Cruz,&nbsp;D Keith Williams,&nbsp;Sarah Sobik,&nbsp;Aline Andres","doi":"10.1016/j.advnut.2026.100712","DOIUrl":"10.1016/j.advnut.2026.100712","url":null,"abstract":"","PeriodicalId":7349,"journal":{"name":"Advances in Nutrition","volume":"17 9","pages":"Article 100712"},"PeriodicalIF":12.7,"publicationDate":"2026-09-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"148995862","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":1,"RegionCategory":"医学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"OA","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}