Food and Chemical Toxicology最新文献_第2页

Study on the role of mitophagy and pyroptosis induced by nano-silver in testicular injury.

IF 3.9 3区医学

Food and Chemical Toxicology Pub Date : 2025-01-07 DOI: 10.1016/j.fct.2025.115245

Deyu Zhu, Yingyi Li, Min Liu, Yufen Yang, Jiayu Fu, Liyu Su, Feng Wang, Yuyan Cen, Yanna Zhou, Yan Li

引用次数: 0

POLYSORBATE 80 AND CARBOXYMETHYLCELLULOSE: A DIFFERENT IMPACT ON EPITHELIAL INTEGRITY WHEN INTERACTING WITH THE MICROBIOME.

IF 3.9 3区医学

Food and Chemical Toxicology Pub Date : 2025-01-06 DOI: 10.1016/j.fct.2025.115236

Alicia Bellanco, Teresa Requena, M Carmen Martínez-Cuesta

{"title":"POLYSORBATE 80 AND CARBOXYMETHYLCELLULOSE: A DIFFERENT IMPACT ON EPITHELIAL INTEGRITY WHEN INTERACTING WITH THE MICROBIOME.","authors":"Alicia Bellanco, Teresa Requena, M Carmen Martínez-Cuesta","doi":"10.1016/j.fct.2025.115236","DOIUrl":"https://doi.org/10.1016/j.fct.2025.115236","url":null,"abstract":"The consumption of dietary emulsifiers, including polysorbate 80 (P80) and sodium carboxymethylcellulose (CMC), has raised safety concerns due to its interaction with the intestinal microbiome. This study demonstrated that increasing concentrations of P80 and CMC added to a dynamic four-stage gut microbiota model (BFBL gut simulator) altered the microbiome composition and impacted epithelial integrity in a dose-dependent manner. 16S rDNA amplicon-based metagenomics analysis revealed that these emulsifiers increased microbial groups with proinflammatory capacities while decreasing microbial taxa known to enhance barrier function. Increasing doses of P80 significantly decreased Bacteroides dorei and Akkermansia, taxa associated with anti-inflammatory potential, while increasing doses of CMC were linked to a higher abundance of Ruminococcus torques and Hungatella, which negatively impact barrier function. Both emulsifiers displayed a different impact on epithelial integrity when interacting with the microbiome. On one hand, supernatants from the BFBL simulator fed with P80 disrupted epithelial integrity to a lesser extent than the additive alone. On the other hand, both the microbiota and the supernatants from the BFBL simulator fed with CMC diminished the epithelial integrity, though the additive itself did not. These findings highlight the need to incorporate the gut microbiome in the risk assessment of these additives.","PeriodicalId":317,"journal":{"name":"Food and Chemical Toxicology","volume":" ","pages":"115236"},"PeriodicalIF":3.9,"publicationDate":"2025-01-06","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"142942073","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":3,"RegionCategory":"医学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}

引用次数: 0

Evaluation of 9,10-anthraquinone contamination in tea products from Indonesian manufacturers and its carcinogenic risk to consumer health.

IF 3.9 3区医学

Food and Chemical Toxicology Pub Date : 2025-01-06 DOI: 10.1016/j.fct.2025.115239

Harmoko Harmoko, Rahmana Emran Kartasasmita, Hasim Munawar, Dadan Rohdiana, Fransiska Kurniawan, Daryono Hadi Tjahjono, Amadeo R Fernández-Alba

{"title":"Evaluation of 9,10-anthraquinone contamination in tea products from Indonesian manufacturers and its carcinogenic risk to consumer health.","authors":"Harmoko Harmoko, Rahmana Emran Kartasasmita, Hasim Munawar, Dadan Rohdiana, Fransiska Kurniawan, Daryono Hadi Tjahjono, Amadeo R Fernández-Alba","doi":"10.1016/j.fct.2025.115239","DOIUrl":"10.1016/j.fct.2025.115239","url":null,"abstract":"This study aimed to determine 9,10-anthraquinone (AQ) levels in Indonesian tea products from different manufacturers and assess the AQ's associated health risks. AQ levels increased significantly during withering and drying stages, using pinewood as a heat source. Generally, black tea was highly contaminated by AQ followed by green tea, oolong tea, and white tea. Out of a total of 116 samples from manufacturers using wood pellets as a heat source, 13% (15/116) of samples were contaminated with AQ exceeding the EU maximum residue level (MRL), and after accounting for measurement uncertainty, this value decreased to only 2% (2/116) that were deemed non-compliant. In contrast, 88% (57/65) and 50% (7/14) of tea samples were contaminated with AQ exceeding the EU MRL when manufacturers used pinewood and palm kernel shells as heat sources, respectively. However, based on our estimation, the risk level due to AQ exposure from Indonesian tea is still manageable, as indicated by calculating incremental lifetime cancer risk, <10⁻⁶ across all conditions studied (age group, type of tea, and heat source).","PeriodicalId":317,"journal":{"name":"Food and Chemical Toxicology","volume":" ","pages":"115239"},"PeriodicalIF":3.9,"publicationDate":"2025-01-06","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"142942070","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":3,"RegionCategory":"医学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}

引用次数: 0

Impact of Endocrine Disruptors on Key Events of Hepatic Steatosis in HepG2 Cells.

IF 3.9 3区医学

Food and Chemical Toxicology Pub Date : 2025-01-06 DOI: 10.1016/j.fct.2025.115241

Marina F Grosso, Eliška Řehůřková, Ishita Virmani, Eliška Sychrová, Iva Sovadinová, Pavel Babica

{"title":"Impact of Endocrine Disruptors on Key Events of Hepatic Steatosis in HepG2 Cells.","authors":"Marina F Grosso, Eliška Řehůřková, Ishita Virmani, Eliška Sychrová, Iva Sovadinová, Pavel Babica","doi":"10.1016/j.fct.2025.115241","DOIUrl":"https://doi.org/10.1016/j.fct.2025.115241","url":null,"abstract":"Endocrine-disrupting compounds (EDCs) may contribute to the rising incidence of metabolic dysfunction-associated steatotic liver disease (MASLD). We investigated the potential of 10 environmentally relevant EDCs to affect key events of hepatic steatosis in HepG2 human hepatoma cells. Increased lipid droplet formation, a key marker of steatosis, was induced by PFOA, bisphenol F, DDE, butylparaben, and DEHP, within the non-cytotoxic concentration range of 1 nM-25 μM. Cadmium also induced this effect, but at concentrations impairing cell viability (>1 μM). At non-cytotoxic concentrations, these compounds, along with bisphenol A, dysregulated major genes controlling lipid homeostasis. Cadmium, PFOA, DDE, and DEHP significantly upregulated the DGAT1 gene involved in triglyceride synthesis, while butylparaben increased the expression of the FAT/CD36 gene responsible for fatty acid uptake. Bisphenol A downregulated the CPT1A gene involved in fatty acid oxidation. No significant effects on lipid droplet accumulation or lipid metabolism-related genes were observed for PFOS, bisphenol S, and dibutyl phthalate. Among the tested EDCs, lipid accumulation positively correlated with the expression of SREBF1, DGAT1, and CPT1A. These findings provide additional evidence that EDCs can affect MASLD and highlight the utility of in vitro methods in the screening of EDCs with hazardous steatogenic and metabolism-disrupting properties.","PeriodicalId":317,"journal":{"name":"Food and Chemical Toxicology","volume":" ","pages":"115241"},"PeriodicalIF":3.9,"publicationDate":"2025-01-06","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"142942071","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":3,"RegionCategory":"医学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}

引用次数: 0

An update on beverage consumption patterns and caffeine intakes in a representative sample of the US population.

IF 3.9 3区医学

Food and Chemical Toxicology Pub Date : 2025-01-06 DOI: 10.1016/j.fct.2025.115237

Diane C Mitchell, Megan Trout, Ross Smith, Robyn Teplansky, Harris R Lieberman

{"title":"An update on beverage consumption patterns and caffeine intakes in a representative sample of the US population.","authors":"Diane C Mitchell, Megan Trout, Ross Smith, Robyn Teplansky, Harris R Lieberman","doi":"10.1016/j.fct.2025.115237","DOIUrl":"https://doi.org/10.1016/j.fct.2025.115237","url":null,"abstract":"Caffeine is a popular stimulant, predominantly consumed from beverages. The caffeinated beverage marketplace is continually evolving resulting in considerable interest in understanding the impact caffeinated beverages have on levels of intakes. Therefore, estimates of caffeine intakes in the U.S. population were calculated using a recent 2022 beverage survey, the Kantar Worldpanel Enhanced Beverage Service. A nationally representative sample of 49,700 consumers (aged ≥2 years) completed a 1-day beverage intake survey which collected data on beverage type/category, amount and brand. Approximately 69% of the U.S. population consumed at least one caffeinated beverage per day. The mean (±SE) daily caffeine intake of caffeine consumers (age >2 years) from all beverages was 210 ±1.5 mg. Caffeine intake was highest in consumers aged 50-64 years (246±4.5 mg/day) and lowest in children aged 2-5 (42±2.4 mg/day). At the 90th percentile intake was 520 mg/day for all ages combined. Coffee was the largest contributor (69%) to caffeine intake across all age groups followed by carbonated soft drinks (15.4%), tea (8.8%), and energy drinks (6.3%). This study indicates that an increase in caffeine intake has occurred with a corresponding shift in beverage consumption patterns compared to previous surveys.","PeriodicalId":317,"journal":{"name":"Food and Chemical Toxicology","volume":" ","pages":"115237"},"PeriodicalIF":3.9,"publicationDate":"2025-01-06","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"142942069","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":3,"RegionCategory":"医学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}

引用次数: 0

Chemopreventive effect of Pistacia vera leaf extract against Mammary Carcinoma Induced by Dimethyl-Benz(a)anthracene in vivo and in vitro: potential role of antioxidant, antiinflammatory and immune mechanisms.

IF 3.9 3区医学

Food and Chemical Toxicology Pub Date : 2025-01-02 DOI: 10.1016/j.fct.2024.115229

Ali G Alkhathami, Esmail M El-Fakharany, Mohamed H El-Sayed, Ahmed Atwa, Fatma Khairallah Ali, Nashwa Hamad, Hussam Askar, Mahmoud Ashry

{"title":"Chemopreventive effect of Pistacia vera leaf extract against Mammary Carcinoma Induced by Dimethyl-Benz(a)anthracene in vivo and in vitro: potential role of antioxidant, antiinflammatory and immune mechanisms.","authors":"Ali G Alkhathami, Esmail M El-Fakharany, Mohamed H El-Sayed, Ahmed Atwa, Fatma Khairallah Ali, Nashwa Hamad, Hussam Askar, Mahmoud Ashry","doi":"10.1016/j.fct.2024.115229","DOIUrl":"https://doi.org/10.1016/j.fct.2024.115229","url":null,"abstract":"This study aimed to define the antitumor effect of ethanolic extract of Pistacia vera leaves (PEE) toward breast cancer both in vitro and in vivo using dimethyl-benz(a)anthracene (DMBA)-induced breast tumor in adult female rats. PEE showed a potent antioxidant effect toward both DPPH (1,1-diphenyl-2-picrylhydrazyl) and ABTS (2,2'-azino-bis(3-ethylbenzthiazoline-6-sulphonic acid) radicals with IC50 values of 72.6 and 107.4 μg/mL, respectively. PEE exerted its cytotoxicity in dose-dependent manners with favorable selectivity toward MCF-7 and MDA cancer cells, sparing normal WI-38 cells. Through considerable decreases in blood CA15.3, CEA, CA19.9, TNF-α, IL1β, IL-4, IL-6, and IL-10 levels, as well as mammary MDA and NO levels, PEE administration effectively improved the damage caused by breast cancer. Additionally, PEE exhibited remarkable increasing in mammary GSH content, GPx, SOD and CAT activities. The histopathological findings demonstrated the therapeutic potential of PEE that successfully improved the mammary gland alterations induced by DMBA and aborted cancer development. PEE has shown intriguing potential as an anti-inflammatory and antioxidant drug by targeting the expression of pro-inflammatory cytokines and oxidative stress indicators, which has helped to successfully treat malignancies in clinical settings. Collectively, our findings support chemo-preventive potential of PEE against DMBA-induced breast tumor in rats via enhancing apoptosis and immune response.","PeriodicalId":317,"journal":{"name":"Food and Chemical Toxicology","volume":" ","pages":"115229"},"PeriodicalIF":3.9,"publicationDate":"2025-01-02","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"142926030","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":3,"RegionCategory":"医学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}

引用次数: 0

Evaluation of the effects of favipiravir (T-705) on the lung tissue of healty rats: An experimental study.

IF 3.9 3区医学

Food and Chemical Toxicology Pub Date : 2025-01-02 DOI: 10.1016/j.fct.2025.115235

Menekşe Ülger, Birkan Ülger, Işıl Tuğçe Turan, Şahin Temel, Arzu Yay, Betül Yalçın, Birkan Yakan

{"title":"Evaluation of the effects of favipiravir (T-705) on the lung tissue of healty rats: An experimental study.","authors":"Menekşe Ülger, Birkan Ülger, Işıl Tuğçe Turan, Şahin Temel, Arzu Yay, Betül Yalçın, Birkan Yakan","doi":"10.1016/j.fct.2025.115235","DOIUrl":"10.1016/j.fct.2025.115235","url":null,"abstract":"Favipiravir, a broad-spectrum RNA-dependent RNA polymerase inhibitor widely used during the COVID-19 pandemic, effectively reduces viral load but has been linked to inflammatory changes in tissues such as the liver and kidneys. High-dose and prolonged use of favipiravir for COVID-19 raises concerns about its potential toxic effects on the lungs, particularly in patients with pre-existing pulmonary conditions. This study investigated favipiravir's effects on lung tissue in healthy rats. Experimental groups included a Control (saline) and three favipiravir doses: Low (200 mg/kg/day loading, 100 mg/kg/day maintenance), Medium (400 mg/kg/day loading, 200 mg/kg/day maintenance), and High (600 mg/kg/day loading, 300 mg/kg/day maintenance), all administered via gavage for 10 days. Histopathological analysis showed normal lung structure in the Control group, while favipiravir-treated groups exhibited Bronchus-Associated Lymphoid Tissue (BALT) enlargement, inflammation, fibrosis, and hemorrhage. Immunohistochemical analysis revealed dose-dependent increases in TNF-α, TGF-β, IL-6, IFN-γ, IL1-β, α-SMA, and collagen-1, especially in the High-dose group (p < 0.05). These findings suggest favipiravir may induce lung inflammation and fibrosis, emphasizing the need for careful evaluation of its safety in clinical settings, particularly for COVID-19 treatment. Future research should investigate the underlying mechanisms of these effects with clinical studies to assess their relevance to humans, high-risk pulmonary patients.","PeriodicalId":317,"journal":{"name":"Food and Chemical Toxicology","volume":" ","pages":"115235"},"PeriodicalIF":3.9,"publicationDate":"2025-01-02","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"142926031","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":3,"RegionCategory":"医学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}

引用次数: 0

Engineered S. cerevisiae-pYD1-ScFv-AFB1 mitigates Aflatoxin B1 toxicity via bio-binding and intestinal microenvironment repair.

IF 3.9 3区医学

Food and Chemical Toxicology Pub Date : 2024-12-31 DOI: 10.1016/j.fct.2024.115232

Hong Huang, Ziyan Li, Zhanghua Qi, Linxi Ma, Gang Hu, Changwei Zou, Tingtao Chen

{"title":"Engineered S. cerevisiae-pYD1-ScFv-AFB1 mitigates Aflatoxin B1 toxicity via bio-binding and intestinal microenvironment repair.","authors":"Hong Huang, Ziyan Li, Zhanghua Qi, Linxi Ma, Gang Hu, Changwei Zou, Tingtao Chen","doi":"10.1016/j.fct.2024.115232","DOIUrl":"https://doi.org/10.1016/j.fct.2024.115232","url":null,"abstract":"The highly toxic aflatoxin B1 (AFB1) is considered one of the primary risk factors for hepatocellular carcinoma, while effective measures after AFB1 exposure remain to be optimized. This study utilized cell-surface-display technique to construct an engineered S. cerevisiae-pYD1-ScFv-AFB1 (S.C-AF) that specifically binds AFB1, and verified the potential mechanism of S.C-AF in vivo through AFB1-induced (gastric perfused with 0.3 mg/kg/d AFB1 per day) liver injury mouse model. In this experiment, the C57BL/6 mouse model of AFB1-induced liver injury was treated with S.C (gastric perfused with 1 × 109 CFU/mL S.C per day) and S.C-AF (gastric perfused with 1 × 109 CFU/mL S.C-AF per day) for 4 weeks, respectively. With probiotic properties optimized, S.C.-AF achieved an in vitro AFB1 binding capacity 1.7 times higher than S. cerevisiae. Furthermore, S.C-AF could alleviate AFB1-induced liver injury by reducing proinflammatory cytokine secretion and apoptotic protein expression, enhancing antioxidative capacity via Nrf2 activation, and simultaneously reversing intestinal tight junction protein deficiency, increasing intestinal barrier permeability, and improving intestinal dysbiosis caused by AFB1 exposure. S.C-AF alleviates AFB1-induced liver lesions, which might be a novel intervention to mitigate aflatoxin toxicity.","PeriodicalId":317,"journal":{"name":"Food and Chemical Toxicology","volume":" ","pages":"115232"},"PeriodicalIF":3.9,"publicationDate":"2024-12-31","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"142918713","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":3,"RegionCategory":"医学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}

引用次数: 0

High-intensity interval training improves hepatic redox status via Nrf2 downstream pathways and reduced CYP2E1 expression in female rats with cisplatin-induced hepatotoxicity.

IF 3.9 3区医学

Food and Chemical Toxicology Pub Date : 2024-12-31 DOI: 10.1016/j.fct.2024.115234

Fernanda Santos Portela, Lara Fabiana Luz Malheiro, Caroline Assunção Oliveira, Érika Azenathe Barros Mercês, Lais Mafra De Benedictis, Júlia Mafra De Benedictis, Ana Jullie Veiga Fernandes, Bruna Santos Silva, Júlia Spínola Ávila, Thiago Macêdo Lopes Correia, Márcio Vasconcelos Oliveira, Patrícia da Silva Oliveira, Amélia Cristina Mendes de Magalhães, Telma de Jesus Soares, Fabrício Freire de Melo, Liliany Souza de Brito Amaral

{"title":"High-intensity interval training improves hepatic redox status via Nrf2 downstream pathways and reduced CYP2E1 expression in female rats with cisplatin-induced hepatotoxicity.","authors":"Fernanda Santos Portela, Lara Fabiana Luz Malheiro, Caroline Assunção Oliveira, Érika Azenathe Barros Mercês, Lais Mafra De Benedictis, Júlia Mafra De Benedictis, Ana Jullie Veiga Fernandes, Bruna Santos Silva, Júlia Spínola Ávila, Thiago Macêdo Lopes Correia, Márcio Vasconcelos Oliveira, Patrícia da Silva Oliveira, Amélia Cristina Mendes de Magalhães, Telma de Jesus Soares, Fabrício Freire de Melo, Liliany Souza de Brito Amaral","doi":"10.1016/j.fct.2024.115234","DOIUrl":"10.1016/j.fct.2024.115234","url":null,"abstract":"Cisplatin (CP) is an antineoplastic drug associated with various cytotoxic adverse effects, including hepatotoxicity. Exercise training may offer hepatoprotection by improving redox status. This study compared the effects of light-intensity continuous training (LICT), moderate-intensity continuous training (MICT), and high-intensity interval training (HIIT) on CP-induced hepatotoxicity in female Wistar rats. The rats were divided into five groups (n = 7): sedentary control (C + S), CP and sedentary (CP + S), CP with LICT (CP + LICT), CP with MICT (CP + MICT), and CP with HIIT (CP + HIIT). The training protocols involved eight weeks of treadmill exercise before CP administration (5 mg/kg). Seven days after CP injection, the rats were euthanized to collect blood and liver tissue samples. Our findings demonstrate that HIIT was the most effective protocol in preventing histopathological alterations and reducing oxidative and nitrosative damage markers in macromolecules, including 4-HNE (lipids), nitrotyrosine (proteins), and 8-OHdG (DNA). The reduction in these markers appears to be linked to decreased CYP2E1 levels. Moreover, HIIT activated the Nrf2 pathway and upregulated its downstream antioxidant enzymes, including SOD1, catalase, GPx, and HO-1. In conclusion, HIIT emerged as the most effective protocol for mitigating hepatic damage, likely through CYP2E1 suppression and enhancement of antioxidant defenses via Nrf2 signaling pathway activation.","PeriodicalId":317,"journal":{"name":"Food and Chemical Toxicology","volume":" ","pages":"115234"},"PeriodicalIF":3.9,"publicationDate":"2024-12-31","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"142918748","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":3,"RegionCategory":"医学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}

引用次数: 0

RIFM fragrance ingredient safety assessment, 3-hexenoic acid, CAS Registry Number 4219-24-3.

IF 3.9 3区医学

Food and Chemical Toxicology Pub Date : 2024-12-28 DOI: 10.1016/j.fct.2024.115228

A M Api, A Bartlett, D Belsito, D Botelho, M Bruze, A Bryant-Friedrich, G A Burton, M A Cancellieri, H Chon, M L Dagli, W Dekant, C Deodhar, K Farrell, A D Fryer, L Jones, K Joshi, A Lapczynski, M Lavelle, I Lee, H Moustakas, J Muldoon, T M Penning, G Ritacco, N Sadekar, I Schember, T W Schultz, F Siddiqi, I G Sipes, G Sullivan, Y Thakkar, Y Tokura

引用次数: 0