发布求助
文献互助 智能选刊 最新文献

Fetal and Pediatric Pathology最新文献

筛选
英文 中文
Challenges Faced by Newborns with Inherited Metabolic Disorders and Their Mothers During Antepartum, Intrapartum, and Postpartum Periods. 产前、产时和产后遗传性代谢紊乱新生儿及其母亲面临的挑战。
IF 0.7 4区 医学
Fetal and Pediatric Pathology Pub Date : 2025-01-01 Epub Date: 2025-01-06 DOI: 10.1080/15513815.2024.2447082
Merve Koç Yekedüz, Gözde Nur Yağci, İlknur Sürücü Kara, Merve Evgin, Engin Kose, Fatma Tuba Eminoğlu
{"title":"Challenges Faced by Newborns with Inherited Metabolic Disorders and Their Mothers During Antepartum, Intrapartum, and Postpartum Periods.","authors":"Merve Koç Yekedüz, Gözde Nur Yağci, İlknur Sürücü Kara, Merve Evgin, Engin Kose, Fatma Tuba Eminoğlu","doi":"10.1080/15513815.2024.2447082","DOIUrl":"10.1080/15513815.2024.2447082","url":null,"abstract":"<p><p>Inherited metabolic disorders (IMDs) pose various obstetric challenges. In this study investigates the prenatal and perinatal profiles of pregnancies affected by IMDs and examines their obstetric outcomes. The most frequently observed antepartum issues identified among 996 patients with IMDs were intrauterine growth restriction (IUGR), intrauterine microcephaly and oligohydramnios. It was notable that mitochondrial disorders are associated with increased incidence of oligohydramnios (<i>p</i> = 0.010), IUGR (<i>p</i> < 0.001), microcephaly (<i>p</i> < 0.001) and intrauterine cardiac issues (<i>p</i> = 0.002). Furthermore, the incidence of intrauterine and natal facial malformations was significantly elevated in the patient groups with mitochondrial (<i>p</i> < 0.001) and lysosomal/peroxisomal diseases (<i>p</i> = 0.037) when compared to the other IMD groups. The mothers of newborns with mitochondrial diseases developed significantly more complications during previous pregnancies than those with other diagnoses (<i>p</i> = 0.040). Identifying risk factors and complications early on can greatly improve outcomes for both mother and infant by facilitating timely intervention and treatment.</p>","PeriodicalId":50452,"journal":{"name":"Fetal and Pediatric Pathology","volume":" ","pages":"53-62"},"PeriodicalIF":0.7,"publicationDate":"2025-01-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"142933450","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":4,"RegionCategory":"医学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
引用次数: 0
Exploring Causal Correlations Between Inflammatory Cytokines and Kawasaki Disease: A Mendelian Randomization. 探索炎症细胞因子与川崎病之间的因果关系：孟德尔随机试验
IF 0.7 4区 医学
Fetal and Pediatric Pathology Pub Date : 2025-01-01 Epub Date: 2024-11-01 DOI: 10.1080/15513815.2024.2414175
Yan Pan, Fuyong Jiao
{"title":"Exploring Causal Correlations Between Inflammatory Cytokines and Kawasaki Disease: A Mendelian Randomization.","authors":"Yan Pan, Fuyong Jiao","doi":"10.1080/15513815.2024.2414175","DOIUrl":"10.1080/15513815.2024.2414175","url":null,"abstract":"<p><strong>Background: </strong>The role of inflammatory cytokines in Kawasaki disease (KD) pathogenesis is known, but causal relationships are unclear. This study investigates these connections using Mendelian randomization (MR).</p><p><strong>Methods: </strong>Genetic variations associated with KD were obtained from a GWAS including 119 cases and 6071 controls of European ancestry. Genetic data on inflammatory cytokines were sourced from a GWAS of 8,293 healthy participants.</p><p><strong>Results: </strong>The study identified significant associations between higher levels of macrophage colony stimulating factor (M-CSF), monocyte chemotactic protein-1 (MCP-1), and tumor necrosis factor-related apoptosis-inducing ligand (TRAIL) and increased risk of KD. The odds ratios (OR) were 1.04 (95% CI: 1.01-1.08, <i>p</i> = 0.010) for M-CSF, 1.03 (95% CI: 1.01-1.05, <i>p</i> = 0.026) for MCP-1, and 1.02 (95% CI: 1.01-1.04, <i>p</i> = 0.027) for TRAIL.</p><p><strong>Conclusion: </strong>This study suggests that M-CSF, MCP-1, and TRAIL are potentially involved in the etiology of KD.</p>","PeriodicalId":50452,"journal":{"name":"Fetal and Pediatric Pathology","volume":" ","pages":"1-13"},"PeriodicalIF":0.7,"publicationDate":"2025-01-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"142559192","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":4,"RegionCategory":"医学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
引用次数: 0
High Genetic Diagnostic Yield of Whole Exome Sequencing in Children with Epilepsy and Neurodevelopmental Disorders. 全外显子组测序在癫痫和神经发育障碍儿童中的高遗传诊断率。
IF 0.7 4区 医学
Fetal and Pediatric Pathology Pub Date : 2025-01-01 Epub Date: 2024-12-08 DOI: 10.1080/15513815.2024.2434919
Turgay Cokyaman, Eda Gül Özcan, Nihan Ecmel Akbaş
{"title":"High Genetic Diagnostic Yield of Whole Exome Sequencing in Children with Epilepsy and Neurodevelopmental Disorders.","authors":"Turgay Cokyaman, Eda Gül Özcan, Nihan Ecmel Akbaş","doi":"10.1080/15513815.2024.2434919","DOIUrl":"10.1080/15513815.2024.2434919","url":null,"abstract":"<p><p><b>Introduction:</b> Nowadays, the diagnostic rate of childhood epilepsies is increasing rapidly in parallel with the advances in genetic technology. In this study, it was aimed to reveal the diagnostic yield of whole exome sequencing (WES) in children with epilepsy and neurodevelopmental disorders (NDDs). <b>Methods:</b> Children aged 1 to 17 years with epilepsy and NDD who underwent WES were included in this retrospective study. Demographic, epilepsy and NDD characteristics, and WES results were recorded. <b>Results:</b> WES was performed in 36.6% of cases. Various single nucleotide variants were detected in 86.3% of cases tested by WES, and the diagnostic yield on a case-by-case basis was found to be 50%. <b>Discussion:</b> The diagnostic yield of WES is quite high in children with epilepsy and NDDs without a definitive diagnosis. Revealing the genetic causes of childhood epilepsy brings up effective and individualized treatment options.</p>","PeriodicalId":50452,"journal":{"name":"Fetal and Pediatric Pathology","volume":" ","pages":"25-39"},"PeriodicalIF":0.7,"publicationDate":"2025-01-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"142796480","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":4,"RegionCategory":"医学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
引用次数: 0
Cytomegalovirus (CMV) Appendicitis in an Immunocompetent Child Masked by Prominent Lymphoid Hyperplasia. 一名免疫功能正常儿童的巨细胞病毒（CMV）阑尾炎被明显的淋巴细胞增生所掩盖。
IF 0.7 4区 医学
Fetal and Pediatric Pathology Pub Date : 2025-01-01 Epub Date: 2024-11-23 DOI: 10.1080/15513815.2024.2430249
Mariel Bedell, Tiarra Price, Qian Wang, Bryan Rea
{"title":"Cytomegalovirus (CMV) Appendicitis in an Immunocompetent Child Masked by Prominent Lymphoid Hyperplasia.","authors":"Mariel Bedell, Tiarra Price, Qian Wang, Bryan Rea","doi":"10.1080/15513815.2024.2430249","DOIUrl":"10.1080/15513815.2024.2430249","url":null,"abstract":"","PeriodicalId":50452,"journal":{"name":"Fetal and Pediatric Pathology","volume":" ","pages":"82-84"},"PeriodicalIF":0.7,"publicationDate":"2025-01-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"142695962","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":4,"RegionCategory":"医学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
引用次数: 0
Placental Mesenchymal Dysplasia with Unique Chromosomal Abnormality and Unusual Histopathology: A Case Report and Literature Review. 胎盘间充质发育不良伴独特的染色体异常和异常的组织病理学：1例报告和文献复习。
IF 0.7 4区 医学
Fetal and Pediatric Pathology Pub Date : 2025-01-01 Epub Date: 2024-11-29 DOI: 10.1080/15513815.2024.2431988
Bushra K Al-Tarawneh, Stefan Kostadinov, Nina Tatevian
{"title":"Placental Mesenchymal Dysplasia with Unique Chromosomal Abnormality and Unusual Histopathology: A Case Report and Literature Review.","authors":"Bushra K Al-Tarawneh, Stefan Kostadinov, Nina Tatevian","doi":"10.1080/15513815.2024.2431988","DOIUrl":"10.1080/15513815.2024.2431988","url":null,"abstract":"<p><p><b>Introduction:</b> Placental mesenchymal dysplasia (PMD), rare vascular and connective tissue placental anomaly can be associated with fetal intrauterine growth restriction (IUGR), stillbirth, Beckwith-Wiedemann syndrome (BWS), some chromosomal abnormalities, or phenotypically and genetically normal fetuses [1]. We reviewed a PMD case from our institution characterized by a previously undescribed chromosomal abnormality along with an unreported histopathologic finding.</p>","PeriodicalId":50452,"journal":{"name":"Fetal and Pediatric Pathology","volume":" ","pages":"69-74"},"PeriodicalIF":0.7,"publicationDate":"2025-01-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"142751659","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":4,"RegionCategory":"医学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
引用次数: 0
Sebaceous Gland Hyperplasia of the Caruncle in a 6-Year-Old Child: A Case Report with Summary of Prior Published Cases. 一名 6 岁儿童的角疣皮脂腺增生症：病例报告及之前发表的病例摘要。
IF 0.7 4区 医学
Fetal and Pediatric Pathology Pub Date : 2024-11-01 Epub Date: 2024-08-26 DOI: 10.1080/15513815.2024.2393367
Xiaojiao Tang, Jin Zhu, Lin Chen
{"title":"Sebaceous Gland Hyperplasia of the Caruncle in a 6-Year-Old Child: A Case Report with Summary of Prior Published Cases.","authors":"Xiaojiao Tang, Jin Zhu, Lin Chen","doi":"10.1080/15513815.2024.2393367","DOIUrl":"10.1080/15513815.2024.2393367","url":null,"abstract":"<p><p><b>Background:</b> Caruncular sebaceous gland hyperplasia (SGH) is an uncommon, benign lesion. Its cause is still unclear. It has not been reported in the pediatric population, with few cases diagnosed in the fourth to eighth decades of life. <b>Case Report:</b> A 6-year-old boy presented with a slowly growing caruncular mass in the right eye. A diagnosis of caruncular SGH was made by histopathology. The clinical, histopathology, treatment, and prognosis are reviewed. <b>Conclusion:</b> This is the first described pediatric case of caruncular SGH that occurs since birth. There are many similarities between adult and pediatric caruncular SGH. Surgical excision is the recommended treatment.</p>","PeriodicalId":50452,"journal":{"name":"Fetal and Pediatric Pathology","volume":" ","pages":"487-491"},"PeriodicalIF":0.7,"publicationDate":"2024-11-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"142057133","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":4,"RegionCategory":"医学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
引用次数: 0
A Comprehensive Consolidation of Data on the Relationship Between Surfactant Protein-B (SFTPB) Polymorphisms and Susceptibility to Bronchopulmonary Dysplasia. 关于表面活性蛋白-B（SFTPB）多态性与支气管肺发育不良易感性之间关系的综合数据。
IF 0.7 4区 医学
Fetal and Pediatric Pathology Pub Date : 2024-11-01 Epub Date: 2024-09-08 DOI: 10.1080/15513815.2024.2400145
Reza Bahrami, Mohammad Golshan-Tafti, Seyed Alireza Dastgheib, Kamran Alijanpour, Maryam Yeganegi, Mohamad Hosein Lookzadeh, Seyed Reza Mirjalili, Sepideh Azizi, Maryam Aghasipour, Amirmasoud Shiri, Mahmood Noorishadkam, Hossein Neamatzadeh
{"title":"A Comprehensive Consolidation of Data on the Relationship Between Surfactant Protein-B (SFTPB) Polymorphisms and Susceptibility to Bronchopulmonary Dysplasia.","authors":"Reza Bahrami, Mohammad Golshan-Tafti, Seyed Alireza Dastgheib, Kamran Alijanpour, Maryam Yeganegi, Mohamad Hosein Lookzadeh, Seyed Reza Mirjalili, Sepideh Azizi, Maryam Aghasipour, Amirmasoud Shiri, Mahmood Noorishadkam, Hossein Neamatzadeh","doi":"10.1080/15513815.2024.2400145","DOIUrl":"10.1080/15513815.2024.2400145","url":null,"abstract":"<p><strong>Background: </strong>This meta-analysis aims to evaluate the potential link between common variations in the Surfactant Protein-B (SFTPB) gene and the risk of bronchopulmonary dysplasia (BPD) in preterm neonates.</p><p><strong>Methods: </strong>All pertinent articles published prior to February 1, 2024, in PubMed, Web of Science, EMBASE, CNKI, and Scopus databases were reviewed.</p><p><strong>Results: </strong>Nineteen case-control studies involving 1149 BPD cases and 1845 non-BPD controls, were analyzed. Combined data indicated a significant link between SFTPB -18 A > C and Intron 4 VNTR polymorphisms with increased BPD susceptibility, while the 1580 C > T polymorphism provides a protective impact on BPD initiation.</p><p><strong>Conclusions: </strong>Pooled data indicated a significant association between SFTPB -18 A > C and Intron 4 VNTR polymorphisms with increased BPD risk, whereas the 1580 C > T polymorphism confers protection. These findings suggest a genetic susceptibility to BPD, underscoring the complex interplay of different genetic elements in its development.</p>","PeriodicalId":50452,"journal":{"name":"Fetal and Pediatric Pathology","volume":" ","pages":"436-454"},"PeriodicalIF":0.7,"publicationDate":"2024-11-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"142156549","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":4,"RegionCategory":"医学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
引用次数: 0
Revisiting Utility of Fetal Autopsy in Genomic Era. 在基因组时代重新审视胎儿尸检的效用。
IF 0.7 4区 医学
Fetal and Pediatric Pathology Pub Date : 2024-11-01 Epub Date: 2024-08-23 DOI: 10.1080/15513815.2024.2393356
Seema Thakur, Chanchal Singh, Preeti Paliwal, Vrunda Appannagri, N Mohit, Gurnihal Singh Chawla, Rounak Bagga
{"title":"Revisiting Utility of Fetal Autopsy in Genomic Era.","authors":"Seema Thakur, Chanchal Singh, Preeti Paliwal, Vrunda Appannagri, N Mohit, Gurnihal Singh Chawla, Rounak Bagga","doi":"10.1080/15513815.2024.2393356","DOIUrl":"10.1080/15513815.2024.2393356","url":null,"abstract":"<p><p><b>Background:</b> Autopsy has been a gold standard in cases of antenatal detected anomalies or fetal demise. This helped clinicians in getting insights into the future management. In current times, ultrasound and genomic testing has become extremely powerful in further refining the etiological basis; however, fetal autopsy still has its role even now. <b>Material and Methods:</b> We have discussed the utility of fetal autopsy in current times by diving the cases in seven groups. <b>Results:</b> Case based discussions to discuss the utility of fetal autopsy. <b>Conclusions:</b> We suggest that fetal autopsy should be the standard of care in case of any abnormal fetal outcomes alongwith fetal genomic testing. Fetal autopsy is complementary to the ultrasound assessment and genomic investigations in reaching the final diagnosis and provides invaluable information regarding recurrence risk which may not be available when couple plans next pregnancy.</p>","PeriodicalId":50452,"journal":{"name":"Fetal and Pediatric Pathology","volume":" ","pages":"510-520"},"PeriodicalIF":0.7,"publicationDate":"2024-11-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"142037600","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":4,"RegionCategory":"医学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
引用次数: 0
Cornelia de Lange Syndrome: Expanding the Neuropathological Spectrum and Clinical Correlations. 科尼莉亚-德-朗格综合征：扩展神经病理学范围和临床相关性。
IF 0.7 4区 医学
Fetal and Pediatric Pathology Pub Date : 2024-11-01 Epub Date: 2024-10-09 DOI: 10.1080/15513815.2024.2412847
Elvio Della Giustina, Tiziana Salviato, Stefania Caramaschi, Luca Fabbiani, Luca Reggiani Bonetti
{"title":"Cornelia de Lange Syndrome: Expanding the Neuropathological Spectrum and Clinical Correlations.","authors":"Elvio Della Giustina, Tiziana Salviato, Stefania Caramaschi, Luca Fabbiani, Luca Reggiani Bonetti","doi":"10.1080/15513815.2024.2412847","DOIUrl":"10.1080/15513815.2024.2412847","url":null,"abstract":"<p><strong>Objectives: </strong>Reporting new neuropathological findings and clinicopathological correlations in Cornelia de Lange syndrome.</p><p><strong>Methods and results: </strong>Cornelia de Lange syndrome has received much attention for its genetics, biochemistry, clinical approach and management, but neuropathological studies are extremely rare. Diffuse hypoplasia of the entire brain, mainly affecting the frontal cortex and, less frequently, the cerebellum, has long been the paradigm for neuropathological findings in rare affected patients. This comprehensive neuropathological study of an affected newborn demonstrates nerve cell heterotopies, poor periventricular matrix and significant hypoplasia of both hippocampi, while Golgi staining of cerebellar tissue samples shows features of nerve cell immaturity.</p><p><strong>Conclusions: </strong>The importance of Cornelia de Lange syndrome as a cohesinopathy and some new neuropathological findings provide an opportunity to discuss and establish interesting clinicopathological correlations, especially with regard to the global intellectual disability of these patients.</p>","PeriodicalId":50452,"journal":{"name":"Fetal and Pediatric Pathology","volume":" ","pages":"477-486"},"PeriodicalIF":0.7,"publicationDate":"2024-11-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"142394901","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":4,"RegionCategory":"医学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
引用次数: 0
Orbital Sarcoma with BCOR Genetic Alterations in the Pediatric Age Group. 儿童年龄组中伴有BCOR基因改变的眼眶肉瘤
IF 0.7 4区 医学
Fetal and Pediatric Pathology Pub Date : 2024-11-01 Epub Date: 2024-09-08 DOI: 10.1080/15513815.2024.2397399
Syed Saad Salman, Aanchal Kakkar, Seema Kashyap, Sameer Rastogi, Rachna Meel
{"title":"Orbital Sarcoma with <i>BCOR</i> Genetic Alterations in the Pediatric Age Group.","authors":"Syed Saad Salman, Aanchal Kakkar, Seema Kashyap, Sameer Rastogi, Rachna Meel","doi":"10.1080/15513815.2024.2397399","DOIUrl":"10.1080/15513815.2024.2397399","url":null,"abstract":"<p><p><b>Introduction:</b> Pediatric orbital tumors encompass a wide spectrum of neoplasms, many of which are malignant small round cell tumors with overlapping histology. Sarcomas with <i>BCOR</i> genetic alterations are undifferentiated round cell sarcomas (URCS) characterized by <i>BCOR</i> rearrangements or internal tandem duplications, having distinct clinical features. Being previously unrecognized in the orbit, they have potential for misdiagnosis. <b>Patients:</b> We describe two cases of orbital sarcomas with <i>BCOR</i> genetic alterations. <b>Results:</b> Both girls, 8 and 16 months of age, respectively, presented with progressive proptosis. Both tumors showed sheets of round to ovoid cells with monomorphic nuclei and frequent mitoses. Delicate branching capillaries and myxoid stroma were absent. Diffuse BCOR, cyclin D1, and SATB2 immunopositivity was present. <b>Conclusion:</b> Orbital sarcomas with BCOR genetic alterations are extremely rare. Pathologists should have high index of suspicion for novel genetically defined entities in the differential diagnosis of pediatric orbital URCS and perform appropriate ancillary tests for accurate diagnosis.</p>","PeriodicalId":50452,"journal":{"name":"Fetal and Pediatric Pathology","volume":" ","pages":"492-499"},"PeriodicalIF":0.7,"publicationDate":"2024-11-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"142156550","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":4,"RegionCategory":"医学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
引用次数: 0
首页 上一页 下一页 尾页
0
×
引用
GB/T 7714-2015
复制
MLA
复制
APA
复制
导出至
BibTeX EndNote RefMan NoteFirst NoteExpress
×
提示
您的信息不完整，为了账户安全，请先补充。
现在去补充
×
提示
您因"违规操作"
具体请查看互助需知
我知道了
×
提示
现在去查看 取消
×
提示
确定
请完成安全验证×
相关产品
×
本文献相关产品
联系我们：info@booksci.cn Book学术提供免费学术资源搜索服务，方便国内外学者检索中英文文献。致力于提供最便捷和优质的服务体验。 Copyright © 2023 布克学术 All rights reserved.
京ICP备2023020795号-1
ghs 京公网安备 11010802042870号
Book学术文献互助
Book学术文献互助群
群 号：481959085
Book学术官方微信