Revue Veterinaire Clinique最新文献

{"title":"","authors":"G. Pageot","doi":"10.1016/j.anicom.2025.03.003","DOIUrl":"10.1016/j.anicom.2025.03.003","url":null,"abstract":"","PeriodicalId":38077,"journal":{"name":"Revue Veterinaire Clinique","volume":"60 2","pages":"Pages 104-105"},"PeriodicalIF":0.0,"publicationDate":"2025-03-26","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"143881339","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}

{"title":"Pathologie comparée : une nouvelle rubrique","authors":"","doi":"10.1016/j.anicom.2025.03.001","DOIUrl":"10.1016/j.anicom.2025.03.001","url":null,"abstract":"","PeriodicalId":38077,"journal":{"name":"Revue Veterinaire Clinique","volume":"60 2","pages":"Page 63"},"PeriodicalIF":0.0,"publicationDate":"2025-03-25","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"143881325","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}

{"title":"Un cas d’encéphalite limbique féline","authors":"A. Forestier ,&nbsp;M. Guehl ,&nbsp;E. Lyon ,&nbsp;E. Reyes-Gomez ,&nbsp;S. Besnard ,&nbsp;C. Benzimra ,&nbsp;C. Escriou ,&nbsp;S. Blot","doi":"10.1016/j.anicom.2025.03.002","DOIUrl":"10.1016/j.anicom.2025.03.002","url":null,"abstract":"<div><div>Un chat mâle castré de 4,5 ans a été présenté pour des manifestations récurrentes évoquant des crises épileptiques focales associées à une modification du comportement. L’examen IRM de l’encéphale, l’analyse du liquide cérébrospinal, et l’examen électroencéphalographique étaient évocateurs d’une encéphalite limbique, sans pour autant permettre de l’affirmer avec certitude. L’encéphalite limbique a pu être confirmer avec certitude grâce à l’examen anatomopathologique post-mortem couplé au dosage sérique spécifique des anticorps anti-LGI1. L’encéphalite limbique féline à anticorps anti-LGI1 (EL-LGI1) est une forme d’encéphalite auto-immune, récemment reconnue chez les chats, possiblement causée par des anticorps ciblant la protéine LGI1. Similaire à sa forme humaine, l’EL-LGI1 chez les chats se manifeste par des crises épileptiques focales, souvent accompagnées d’automatismes orofaciaux et de changement de comportement comme l’agressivité. Le diagnostic repose sur la détection des anticorps anti-LGI1, bien que ce dosage ne soit pas disponible en routine en médecine vétérinaire et particulièrement en France. Ce cas illustre un cas typique d’encéphalite limbique pour lequel un diagnostic a été établi avec certitude en post-mortem et souligne la difficulté du diagnostic pré-mortem.</div></div><div><div>A 4.5-year-old neutered male cat was presented with recurrent manifestations suggesting focal seizures associated with behavioral changes. MRI of the brain, analysis of cerebrospinal fluid, and electroencephalographic examination were suggestive of limbic encephalitis, although they could not confirm it with certainty. Limbic encephalitis was definitively confirmed through post-mortem histopathological examination combined with specific serum testing for anti-LGI1 antibodies. Feline limbic encephalitis with anti-LGI1 (EL-LGI1) is a form of autoimmune encephalitis recently recognized in cats, possibly caused by antibodies targeting the LGI1 protein. Similar to its human form, EL-LGI1 in cats manifests as focal seizures, often accompanied by oro-facial automatisms and behavioral changes such as aggression. The diagnosis relies on the detection of anti-LGI1 antibodies, although this test is not routinely available in veterinary medicine, especially in France. This case illustrates a typical example of limbic encephalitis for which a definitive diagnosis was established post-mortem and highlights the challenges of pre-mortem diagnosis.</div></div>","PeriodicalId":38077,"journal":{"name":"Revue Veterinaire Clinique","volume":"60 3","pages":"Pages 121-129"},"PeriodicalIF":0.0,"publicationDate":"2025-03-25","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"144679983","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}

{"title":"Atrophies progressives de la rétine du chien, rétinopathies pigmentaires de l’homme : similitudes et différences","authors":"G. Chaudieu","doi":"10.1016/j.anicom.2025.02.002","DOIUrl":"10.1016/j.anicom.2025.02.002","url":null,"abstract":"<div><div>Les rétinopathies pigmentaires de l’homme et les atrophies progressives de la rétine du chien, dont les classifications sont initialement rappelées, sont homologues à beaucoup d’égards. Leur phénotype est dominé par une perte progressive de vision avec une cécité terminale très fréquente. Elles présentent une triple hétérogénéité : clinique, génétique et moléculaire. Les données cliniques sont rappelées, comparativement dans les deux espèces, pour les formes isolées et les formes syndromiques. Leur transmission mendélienne est majoritairement autosomique récessive, surtout chez le chien où ce mode est quasi-exclusif. Des gènes communs sont impliqués, leurs mutations sont plus nombreuses chez l’homme, chez qui le pourcentage de cas sporadiques est élevé, alors que le suivi d’une lignée affectée offre une facilité d’étude propre à la structuration génétique de l’espèce canine en races sélectionnées. Ces facteurs contribuent à faire du chien un modèle privilégié transposable à l’homme dans la recherche en thérapie génique rétinienne.</div></div><div><div>Human pigmentary retinopathies and canine progressive retinal atrophies, whose classifications are initially recalled, are homologous in many aspects. Their phenotype is dominated by a progressive vision loss with very frequent terminal blindness. They present a triple heterogeneity: clinical, genetic and molecular. Clinical data are reviewed comparatively in these two species for isolated and syndromic forms. Their mendelian transmission is mostly autosomal recessive, especially in dogs, where this mode is almost exclusive. Common genes are involved, their mutations are most numerous in humans, in whom the percentage of sporadic cases is high, while an affected breeding line following offers an ease of study specific to the genetic structuring of the canine species into selected breeds. These factors contribute to making the dog a privileged model that can be transposed to humans in retina gene therapy research.</div></div>","PeriodicalId":38077,"journal":{"name":"Revue Veterinaire Clinique","volume":"60 2","pages":"Pages 64-82"},"PeriodicalIF":0.0,"publicationDate":"2025-03-19","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"143882050","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}

{"title":"","authors":"B. Cloet-Chabre","doi":"10.1016/j.anicom.2025.02.001","DOIUrl":"10.1016/j.anicom.2025.02.001","url":null,"abstract":"","PeriodicalId":38077,"journal":{"name":"Revue Veterinaire Clinique","volume":"60 3","pages":"Pages 163-164"},"PeriodicalIF":0.0,"publicationDate":"2025-03-05","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"144680033","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}

{"title":"Achalasie cricopharyngée chez un chiot Springer anglais de 6 mois","authors":"C. Humeau,&nbsp;S. Amoyal,&nbsp;T. Meric,&nbsp;J. Gevrey,&nbsp;J. Hernandez","doi":"10.1016/j.anicom.2025.01.009","DOIUrl":"10.1016/j.anicom.2025.01.009","url":null,"abstract":"<div><div>Les dysphagies cricopharyngées sont des maladies rares, dont la suspicion clinique repose essentiellement sur l’observation et la description de la prise alimentaire, et dont l’origine n’est pas toujours complètement élucidée. La vidéofluoroscopie permet d’orienter le diagnostic mais la distinction entre achalasie et asynchronisme cricopharyngés, principales entités à l’origine de dysphagie cricopharyngée, n’est pas évidente à établir. Dans les deux cas, le traitement est chirurgical. Le pronostic de récupération fonctionnelle reste réservé malgré cette prise en charge et semblerait moins favorable en cas d’asynchronisme ; il peut être encore plus réservé en présence de maladie neuromusculaire sous-jacente. Les dysfonctionnements cricopharyngés favorisant les fausses déglutitions, des bronchopneumonies chroniques peuvent également aggraver le pronostic. Ce cas clinique présente la démarche diagnostique et la prise en charge chirurgicale d’un cas d’achalasie cricopharyngée compliquée d’une bronchopneumopathie chronique par fausses déglutitions répétées chez un chiot Springer anglais de 6 mois.</div></div><div><div>Cricopharyngeal dysphagia is a rare disease, the clinical suspicion of which is based essentially on observation and description of food intake, and the cause of which is not always fully understood. Videofluoroscopy helps to orientate the diagnosis, but the distinction between achalasia and cricopharyngeal asynchronism, the main entities causing cricopharyngeal dysphagia, is not easy to establish. In both cases, the treatment is surgical. However, the prognosis for functional recovery remains guarded despite treatment and seems to be worse for asynchronism; it may be even more unfavourable in the presence of underlying neuromuscular disease. Cricopharyngeal dysfunctions, which encourage false swallowing, are often associated with bronchopneumonia, what can also worsen the prognosis. This clinical case presents the diagnostic approach and surgical management of a cricopharyngeal achalasia, complicated by chronic bronchopneumonia due to repeated false swallowing, in a 6-month-old English Springer Spaniel puppy.</div></div>","PeriodicalId":38077,"journal":{"name":"Revue Veterinaire Clinique","volume":"60 2","pages":"Pages 89-95"},"PeriodicalIF":0.0,"publicationDate":"2025-02-12","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"143881341","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}

{"title":"Pronostic à long terme de la réalisation d’hépatectomies multiples sélectives lors de carcinomes hépatocellulaires chez le chien à l’aide d’un dissecteur à ultrasons (CUSA)","authors":"E. Thouvenot Oudart,&nbsp;C. Bismuth","doi":"10.1016/j.anicom.2025.01.003","DOIUrl":"10.1016/j.anicom.2025.01.003","url":null,"abstract":"","PeriodicalId":38077,"journal":{"name":"Revue Veterinaire Clinique","volume":"60 2","pages":"Pages 106-107"},"PeriodicalIF":0.0,"publicationDate":"2025-02-06","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"143881330","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}

{"title":"Urinary tract infection and urolithiasis: potential complications of proximal tubulopathy in a context of canine copper associated hepatopathy","authors":"C. Benoit ,&nbsp;M. Pertuis ,&nbsp;M. Le Dudal ,&nbsp;C. Maurey","doi":"10.1016/j.anicom.2025.01.002","DOIUrl":"10.1016/j.anicom.2025.01.002","url":null,"abstract":"<div><div>A 7-year-old intact female Australian Shepherd was presented with a 5-day history of polyuria-polydipsia, pollakiuria, vomiting, and anorexia. Comprehensive clinical, clinicopathological, ultrasound, cytological, bacteriological, and histological examinations led to a diagnosis of copper associated hepatitis, proximal tubulopathy, and struvite urolithiasis secondary to <em>Staphylococcus pseudintermedius</em> cystitis and potentially pyelonephritis. Treatment, which included infusion, antibiotics, cystotomy, a copper chelator, corticosteroids, and hepatoprotectors, resulted in the resolution of clinical and biological abnormalities. A follow-up 19 months post-diagnosis showed a satisfactory outcome. This is the first documented case of concurrent copper associated hepatopathy and Fanconi's syndrome associated with a urinary tract infection in a dog. This unusual clinical presentation highlights the need for clinicians to stay open-minded about the sometimes variable symptomatology and the intricate interactions among biological systems. This case also raises the question of possible link between the identified Fanconi's syndrome and urinary tract infection, suggesting that such infections might be potential complications of proximal tubulopathy in the context of copper associated hepatopathy. Further studies are needed to confirm this association and assess its potential clinical implications.</div></div><div><div>Une chienne entière berger australien de 7 ans a été présentée pour polyuro-polydipsie, pollakiurie, vomissements et dysorexie évoluant depuis cinq jours. Les examens clinique, biologique, échographique, cytologique, bactériologique et histologique ont permis de diagnostiquer une hépatite cuprique, une tubulopathie proximale et une urolithiase à struvites secondaire à une cystite et potentiellement une pyélonéphrite à <em>Staphylococcus pseudintermedius</em>. La prise en charge, comprenant de la fluidothérapie, une antibiothérapie, une cystotomie, un chélateur de cuivre, un anti-inflammatoire stéroïdien et des hépatoprotecteurs, a conduit à la résolution des anomalies cliniques et biologiques. Un suivi effectué 19 mois après le diagnostic montrait une évolution satisfaisante. Il s’agit du premier cas rapporté d’hépatopathie cuprique et de syndrome de Fanconi concomitants associés à une infection du tractus urinaire chez un chien. Cette présentation clinique atypique invite le clinicien à garder l’esprit ouvert face à des symptomatologies parfois protéiformes et à la complexité des interactions entre les systèmes. Ce cas soulève la question d’un lien possible entre le syndrome de Fanconi et l’infection du tractus urinaire identifiés, suggérant que ces infections pourraient constituer des complications potentielles d’une tubulopathie proximale dans le contexte d’hépatopathie cuprique. Davantage d’études restent nécessaires pour confirmer cette association et évaluer son potentiel impact clinique.</div></div>","PeriodicalId":38077,"journal":{"name":"Revue Veterinaire Clinique","volume":"60 2","pages":"Pages 96-103"},"PeriodicalIF":0.0,"publicationDate":"2025-02-05","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"143881342","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}

{"title":"L’impact de la rétraction du nerf vague sur la paralysie laryngée unilatérale gauche postopératoire après fermeture chirurgicale du canal artériel persistant chez le chien","authors":"S. Manzoni,&nbsp;M. Santos,&nbsp;N. Van Israël,&nbsp;A. Bongartz","doi":"10.1016/j.anicom.2025.01.008","DOIUrl":"10.1016/j.anicom.2025.01.008","url":null,"abstract":"","PeriodicalId":38077,"journal":{"name":"Revue Veterinaire Clinique","volume":"60 2","pages":"Pages 118-120"},"PeriodicalIF":0.0,"publicationDate":"2025-02-04","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"143881338","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}

{"title":"À propos de 10 cas de shunts intrahépatiques traités par chirurgie endovasculaire","authors":"H. Brissot,&nbsp;P. Degail,&nbsp;T. Larcher","doi":"10.1016/j.anicom.2025.01.005","DOIUrl":"10.1016/j.anicom.2025.01.005","url":null,"abstract":"","PeriodicalId":38077,"journal":{"name":"Revue Veterinaire Clinique","volume":"60 2","pages":"Pages 110-111"},"PeriodicalIF":0.0,"publicationDate":"2025-02-04","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"143881340","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}