International journal of oncology最新文献

[Corrigendum] Dual targeting of glioblastoma multiforme with a proteasome inhibitor (Velcade) and a phosphatidylinositol 3‑kinase inhibitor (ZSTK474). [勘误]用蛋白酶体抑制剂（Velcade）和磷脂酰肌醇3激酶抑制剂（ZSTK474）双重靶向多形性胶质母细胞瘤。

IF 4.5 3区医学

International journal of oncology Pub Date : 2025-09-01 Epub Date: 2025-07-19 DOI: 10.3892/ijo.2025.5777

Lehang Lin, Daria Gaut, Kaishun Hu, Haiyan Yan, Dong Yin, Phillip H Koeffler

{"title":"[Corrigendum] Dual targeting of glioblastoma multiforme with a proteasome inhibitor (Velcade) and a phosphatidylinositol 3‑kinase inhibitor (ZSTK474).","authors":"Lehang Lin, Daria Gaut, Kaishun Hu, Haiyan Yan, Dong Yin, Phillip H Koeffler","doi":"10.3892/ijo.2025.5777","DOIUrl":"https://doi.org/10.3892/ijo.2025.5777","url":null,"abstract":"Following the publication of the above article, an interested reader drew to the attention of the Editorial Office that GAPDH bands featured for the U87 cell line (left‑hand panels) in Fig. 5 on p. 561 were strikingly similar to the GAPDH bands for the U118 cell line (right‑hand panels) shown in Fig. 1 on p. 559, even though the experiments shown in these figures were performed under different experimental conditions. Upon examining their data, the authors have realized that Fig. 5 was presented incorrectly; specifically, the cell lines ('U87' and 'U118') in Fig. 5 were mistakenly labeled in reverse, and the GAPDH bands from the right‑hand panels of Fig. 1 were inadvertently re‑used in the left‑hand panels of Fig. 5. The authors have now corrected the cell line labels and replaced the GAPDH bands in the left‑hand panels of Fig. 5 with alternative data from a repeated experiment. The revised version of Fig. 5 is shown below. It is important to note that this error did not affect the overall conclusions reported in the study. The authors are grateful to the Editor of International Journal of Oncology for allowing them this opportunity to publish a Corrigendum, and all the authors agree with its publication. Furthermore, the authors deeply apologize to the readership for any inconvenience caused. [International Journal of Oncology 44: 557‑562, 2014; DOI: 10.3892/ijo.2013.2205].","PeriodicalId":14175,"journal":{"name":"International journal of oncology","volume":"67 3","pages":""},"PeriodicalIF":4.5,"publicationDate":"2025-09-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"144667623","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":3,"RegionCategory":"医学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}

引用次数: 0

Inhibiting SSBP1 enhances ferroptosis and improves the effectiveness of sorafenib treatment for liver cancer. 抑制SSBP1增强铁下垂，提高索拉非尼治疗肝癌的有效性。

IF 4.9 3区医学

International journal of oncology Pub Date : 2025-09-01 Epub Date: 2025-08-01 DOI: 10.3892/ijo.2025.5778

Sai Li, Xinyu Yang, Haoxuan Gao, Xiuya Hu, Danni Wang, Qiqi Zhang, Juan Xu, Jiaqi Zhang, Lu Zhu, Zihan Wang

{"title":"Inhibiting SSBP1 enhances ferroptosis and improves the effectiveness of sorafenib treatment for liver cancer.","authors":"Sai Li, Xinyu Yang, Haoxuan Gao, Xiuya Hu, Danni Wang, Qiqi Zhang, Juan Xu, Jiaqi Zhang, Lu Zhu, Zihan Wang","doi":"10.3892/ijo.2025.5778","DOIUrl":"https://doi.org/10.3892/ijo.2025.5778","url":null,"abstract":"Liver cancer is the third leading cause of cancer‑related mortality globally, with increasing morbidity and mortality rates. Sorafenib, a multi‑kinase inhibitor, is an effective first‑line therapy for late‑stage liver cancer. However, its effectiveness is hindered by low responsiveness, high drug resistance and significant side effects. The progression and metastasis of liver cancer are associated with alterations in mitochondrial metabolism, including mitochondrial stress responses and defects in oxidative phosphorylation, which are involved in the increased production of reactive oxygen species. Targeting mitochondrial biogenesis and bioenergetics presents a promising therapeutic strategy. Bioinformatics analysis (integrated analysis of The Cancer Genome Atlas, mitochondrial genomes, liver cancer mouse models, and bioinformatics tools) revealed that the expression of single‑stranded DNA‑binding protein 1 (SSBP1) was significantly elevated in liver cancer. In addition, MTT and colony formation assays showed that increased SSBP1 expression notably enhanced cell proliferation, while wound healing and Transwell assays demonstrated enhanced metastasis. Furthermore, flow cytometry, qPCR, and western blotting indicated that SSBP1 knockout impaired mitochondrial function and increased sensitivity to sorafenib, effectively attenuating cancer progression. Clinical correlation analysis demonstrated that higher SSBP1 expression was associated with poorer prognosis in patients with liver cancer. In summary, the present study identified SSBP1 as a potential driver of tumor growth and a promising prognostic biomarker and therapeutic target in liver cancer, thus providing novel insight for improving patient outcomes.","PeriodicalId":14175,"journal":{"name":"International journal of oncology","volume":"67 3","pages":""},"PeriodicalIF":4.9,"publicationDate":"2025-09-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"144760069","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":3,"RegionCategory":"医学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}

引用次数: 0

Knockdown of ACC1 promotes migration and invasion of U251 glioma cells by epigenetically suppressing SDH. 敲低ACC1通过表观遗传抑制SDH促进U251胶质瘤细胞的迁移和侵袭。

IF 4.9 3区医学

International journal of oncology Pub Date : 2025-09-01 Epub Date: 2025-08-01 DOI: 10.3892/ijo.2025.5779

Xixi Wei, Yang Wang, Wanlong Zhao, Wenqian Yang, Jiaping Tang, Baosheng Zhao, Yuzhen Liu

{"title":"Knockdown of ACC1 promotes migration and invasion of U251 glioma cells by epigenetically suppressing SDH.","authors":"Xixi Wei, Yang Wang, Wanlong Zhao, Wenqian Yang, Jiaping Tang, Baosheng Zhao, Yuzhen Liu","doi":"10.3892/ijo.2025.5779","DOIUrl":"https://doi.org/10.3892/ijo.2025.5779","url":null,"abstract":"Glioma is a common and aggressive malignant brain tumor. Despite advances in research, the mechanisms driving glioma initiation and progression remain incompletely understood. The present study aimed to assess the role of acetyl‑CoA carboxylase 1 (ACC1) in glioma, focusing on its mechanistic function in U251 cells and its clinical significance. ACC1 expression was first assessed in four glioma cell lines and then the effects on cellular functions were evaluated. Based on the finding that ACC1 knockdown altered the phenotype of U251 cells, potentially through modulation of succinate dehydrogenase (SDH) activity, further mechanistic assessments were performed. Finally, the association between ACC1 expression and patient prognosis was analyzed. The results demonstrated that ACC1 overexpression inhibited proliferation, migration and invasion in U87 cells. Conversely, ACC1 knockdown promoted these processes in U251, T98G and LN229 cells. Mechanistically, in U251 cells, ACC1 knockdown increased acetyl‑CoA levels, enhancing substrate availability for P300. This led to upregulation of DNA methyltransferase 1 (DNMT1), hypermethylation of the SDH promoter and subsequent SDH downregulation. The resulting increase in reactive oxygen species (ROS) levels promoted U251 cell migration and invasion. Analysis of clinical data revealed a significant correlation between low ACC1 expression and poor survival outcomes in patients with glioma. These findings suggest that ACC1 functions as a tumor suppressor in glioma. Its downregulation promotes a pro‑tumorigenic phenotype via the acetyl‑CoA/P300/DNMT1/SDH/ROS pathway, highlighting its potential as a prognostic marker and therapeutic target. This underscores the importance of developing personalized treatment strategies targeting ACC1 in glioma.","PeriodicalId":14175,"journal":{"name":"International journal of oncology","volume":"67 3","pages":""},"PeriodicalIF":4.9,"publicationDate":"2025-09-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"144760070","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":3,"RegionCategory":"医学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}

引用次数: 0

[Corrigendum] IL‑21R functions as an oncogenic factor and is regulated by the lncRNA MALAT1/miR‑125a‑3p axis in gastric cancer. [更正]IL - 21R作为一种致癌因子，在胃癌中受lncRNA MALAT1/miR - 125a - 3p轴的调控。

IF 4.9 3区医学

International journal of oncology Pub Date : 2025-09-01 Epub Date: 2025-08-01 DOI: 10.3892/ijo.2025.5780

Lei Yan, Jing Zhang, Dong Guo, Ji Ma, Shao-Feng Shui, Xin-Wei Han

{"title":"[Corrigendum] IL‑21R functions as an oncogenic factor and is regulated by the lncRNA MALAT1/miR‑125a‑3p axis in gastric cancer.","authors":"Lei Yan, Jing Zhang, Dong Guo, Ji Ma, Shao-Feng Shui, Xin-Wei Han","doi":"10.3892/ijo.2025.5780","DOIUrl":"https://doi.org/10.3892/ijo.2025.5780","url":null,"abstract":"Following the publication of the above article, an interested reader drew to the authors' attention that their paper was found to contain data with a previous article that had been published in the journal Oncotarget (Figs. 2C and 4B), and in a paper that appeared subsequently in the journal Molecular Cancer. All cases involved the sharing of Transwell assay data, and the other papers in question were published by the same authors/the same research group. After having examined their original data, the authors realized that Figs. 2 and 4 had been inadvertently assembled incorrectly in the above paper. Specifically, Figs. 2C and D, and 4B and D, showing the results of Transwell assay experiments indicating the effects of IL‑21R knockdown or co‑transfection with IL‑21R and miR‑125a mimic on cell invasion in HGC‑27 and MKN‑45 cell lines, contained erroneous images. The revised versions of Figs. 2 and 4, featuring replacement data for Figs. 2C and D and 4B and D, showing the correct data obtained for the effects of IL‑21R knockdown or co‑transfection with IL‑21R and miR‑125a mimic on cell invasion, are shown on the next page. All authors confirm that the errors made in Figs. 2C and D and 4B and D did not influence the final conclusions reported in the above article, and they thank the Editor of International Journal of Oncology for granting them the opportunity to publish a Corrigendum. All the authors agree to the publication of this Corrigendum, and apologize for the inconvenience to the readers. [International Journal of Oncology 54: 7‑16, 2019; DOI: 10.3892/ijo.2018.4612].","PeriodicalId":14175,"journal":{"name":"International journal of oncology","volume":"67 3","pages":""},"PeriodicalIF":4.9,"publicationDate":"2025-09-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"144760068","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":3,"RegionCategory":"医学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}

引用次数: 0

[Corrigendum] STAT3 activation in HER2‑overexpressing breast cancer promotes epithelial‑mesenchymal transition and cancer stem cell traits. [勘误]STAT3在HER2过表达乳腺癌中的激活促进上皮-间充质转化和癌症干细胞特性。

IF 4.5 3区医学

International journal of oncology Pub Date : 2025-08-01 Epub Date: 2025-07-19 DOI: 10.3892/ijo.2025.5775

Seyung S Chung, Nolan Giehl, Yanyuan Wu, Jaydutt V Vadgama

{"title":"[Corrigendum] STAT3 activation in HER2‑overexpressing breast cancer promotes epithelial‑mesenchymal transition and cancer stem cell traits.","authors":"Seyung S Chung, Nolan Giehl, Yanyuan Wu, Jaydutt V Vadgama","doi":"10.3892/ijo.2025.5775","DOIUrl":"10.3892/ijo.2025.5775","url":null,"abstract":"Following the publication of the above article, a pair of interested readers drew to the Editor's attention that certain of the western blotting data featured in Figs. 1A and 3A were strikingly similar to data that had appeared in a pair of articles published previously by the same research group. Subsequently, an independent investigation of the data in this paper on the part of the Editorial Office revealed that a pair of the panels showing the results of cell invasion assays in Fig. 4A on p. 405 for the MCF7‑WT cells appeared to contain overlapping sections, such that data which were intended to show results from entirely different microscopic fields had apparently been derived from partly the same original field of view. Upon investigating these matters with the authors, they were able to repeat the experiments concerned (in the case of Figs. 1 and 3). The revised versions of Figs. 1, 3 and 4, now featuring the replacement data for Figs. 1A and 3A and the two completely differentiated microscopic fields of view for Fig. 4, are shown on the next two pages. The authors regret that certain of the data featured in Figs. 1 and 3 of this article were erronoeusly re‑used from a pair of their previous publications, and thank the Editor of International Journal of Oncology for granting them the opportunity to publish this corrigendum. All the authors agree with the publication of this corrigendum; furthermore, they apologize to the readership of the journal for any inconvenience caused. [International Journal of Oncology 44: 403‑411, 2014; DOI: 10.3892/ijo.2013.2195].","PeriodicalId":14175,"journal":{"name":"International journal of oncology","volume":"67 2","pages":""},"PeriodicalIF":4.5,"publicationDate":"2025-08-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://www.ncbi.nlm.nih.gov/pmc/articles/PMC12274169/pdf/","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"144667620","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":3,"RegionCategory":"医学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"OA","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}

引用次数: 0

RAD51 and PALB2 in precision oncology: Clinical implications for HRD associated breast and ovarian cancers (Review). RAD51和PALB2在精确肿瘤学：HRD相关乳腺癌和卵巢癌的临床意义（综述）。

IF 4.5 3区医学

International journal of oncology Pub Date : 2025-08-01 Epub Date: 2025-07-04 DOI: 10.3892/ijo.2025.5771

Mohd Adnan Kausar, Khalid Farhan Alshammari, Fahaad Alenazi, Sadaf Anwar, Amany Mohammed Khalifa, Tarig Ginawi, Abdulaziz Asiri, Mohammad Zeeshan Najm, Syed Arman Rabbani, Mohamed El-Tanani, Saumyatika Gantayat

{"title":"RAD51 and PALB2 in precision oncology: Clinical implications for HRD associated breast and ovarian cancers (Review).","authors":"Mohd Adnan Kausar, Khalid Farhan Alshammari, Fahaad Alenazi, Sadaf Anwar, Amany Mohammed Khalifa, Tarig Ginawi, Abdulaziz Asiri, Mohammad Zeeshan Najm, Syed Arman Rabbani, Mohamed El-Tanani, Saumyatika Gantayat","doi":"10.3892/ijo.2025.5771","DOIUrl":"10.3892/ijo.2025.5771","url":null,"abstract":"Maintaining genomic stability is essential for reducing the risk of carcinogenesis. Homologous recombination (HR) is a high‑fidelity DNA repair mechanism that addresses double‑strand breaks and interstrand crosslinks. The present review examined two key components of HR: RAD51, the eukaryotic recombinase and PALB2, a scaffolding protein. Their structural and functional roles are explored in the context of breast and ovarian cancer. RAD51 facilitates homology search and strand invasion, while PALB2 links BRCA1 and BRCA2, stabilizing RAD51 filaments. Mutations in these genes compromise HR, increasing susceptibility to various cancers and impacting treatment efficacy by impairing DNA repair. The present review discussed the clinical implications of RAD51 and PALB2 mutations, focusing on risk stratification, PARP inhibitor efficacy and emerging therapies. Additionally, it highlighted the potential of RAD51 and PALB2 as biomarkers and therapeutic targets, contributing to advances in personalized cancer management.","PeriodicalId":14175,"journal":{"name":"International journal of oncology","volume":"67 2","pages":""},"PeriodicalIF":4.5,"publicationDate":"2025-08-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://www.ncbi.nlm.nih.gov/pmc/articles/PMC12270392/pdf/","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"144560140","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":3,"RegionCategory":"医学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"OA","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}

引用次数: 0

[Retracted] N6‑methyladenosine upregulates miR‑181d‑5p in exosomes derived from cancer‑associated fibroblasts to inhibit 5‑FU sensitivity by targeting NCALD in colorectal cancer. [引自]N6 -甲基腺苷上调来自癌症相关成纤维细胞的外泌体中的miR - 181d - 5p，通过靶向结直肠癌中的NCALD抑制5 - FU敏感性。

IF 4.5 3区医学

International journal of oncology Pub Date : 2025-08-01 Epub Date: 2025-06-27 DOI: 10.3892/ijo.2025.5768

Shengli Pan, Yingying Deng, Jun Fu, Yuhao Zhang, Zhijin Zhang, Xianju Qin

引用次数: 0

[Corrigendum] Metastasis suppressor 1 expression in human ovarian cancer: The impact on cellular migration and metastasis. 转移抑制因子1在人卵巢癌中的表达：对细胞迁移和转移的影响。

IF 4.5 3区医学

International journal of oncology Pub Date : 2025-08-01 Epub Date: 2025-07-04 DOI: 10.3892/ijo.2025.5770

Rong Liu, Tracey A Martin, Nicola J Jordan, Fiona Ruge, Lin Ye, Wen G Jiang

{"title":"[Corrigendum] Metastasis suppressor 1 expression in human ovarian cancer: The impact on cellular migration and metastasis.","authors":"Rong Liu, Tracey A Martin, Nicola J Jordan, Fiona Ruge, Lin Ye, Wen G Jiang","doi":"10.3892/ijo.2025.5770","DOIUrl":"10.3892/ijo.2025.5770","url":null,"abstract":"Subsequently to the publication of the above article, an interested reader drew to the authors' attention that, concerning the cell invasion assays shown in Fig. 5A on p. 1436, the 'WT' and 'pEF6' data panels contained apparently overlapping sections of data, such that these experiments were apparently derived from the same original source where the results of differently performed experiments were intended to have been portrayed. After re‑examining their original data, the authors have realized that the data panel in Fig. 5A for the 'pEF6' experiment was inadvertently selected incorrectly. The revised version of Fig. 5, showing all the correct data for Fig. 5A, is shown on the next page. The authors are grateful to the Editor of International Journal of Oncology for allowing them this opportunity to publish a Corrigendum, and all the authors agree with its publication. Furthermore, the authors apologize to the readership for any inconvenience caused. [International Journal of Oncology 47: 1429‑1439, 2015; DOI: 10.3892/ijo.2015.3121].","PeriodicalId":14175,"journal":{"name":"International journal of oncology","volume":"67 2","pages":""},"PeriodicalIF":4.5,"publicationDate":"2025-08-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://www.ncbi.nlm.nih.gov/pmc/articles/PMC12270387/pdf/","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"144560139","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":3,"RegionCategory":"医学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"OA","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}

引用次数: 0

[Corrigendum] Dual roles of protein tyrosine phosphatase kappa in coordinating angiogenesis induced by pro-angiogenic factors. [勘误]蛋白酪氨酸磷酸酶kappa在促血管生成因子诱导的血管生成协调中的双重作用。

IF 4.5 3区医学

International journal of oncology Pub Date : 2025-08-01 Epub Date: 2025-07-11 DOI: 10.3892/ijo.2025.5772

Ping-Hui Sun, Gang Chen, Malcolm Mason, Wen G Jiang, Lin Ye

引用次数: 0

Novel non‑metal‑based contrast agents for MR imaging: Emerging approaches and clinical perspectives (Review). 新型非金属造影剂用于磁共振成像：新方法和临床观点（综述）。

IF 4.5 3区医学

International journal of oncology Pub Date : 2025-08-01 Epub Date: 2025-07-19 DOI: 10.3892/ijo.2025.5776

Taoming Du, Haiyang Luo, Huizhen Song, Tao Lin, Qin Yu

{"title":"Novel non‑metal‑based contrast agents for MR imaging: Emerging approaches and clinical perspectives (Review).","authors":"Taoming Du, Haiyang Luo, Huizhen Song, Tao Lin, Qin Yu","doi":"10.3892/ijo.2025.5776","DOIUrl":"10.3892/ijo.2025.5776","url":null,"abstract":"Magnetic Resonance Imaging (MRI) relies on contrast agents to enhance image quality and diagnostic accuracy. Traditional metal‑based agents, such as gadolinium compounds, raise safety concerns due to potential toxicity and long‑term retention in the body. The present review examines recent advancements in non‑metal‑based MRI contrast agents, focusing on fluorine‑19 (19F) compounds, chemical exchange saturation transfer (CEST) agents, nitroxide radicals, and hyperpolarized carbon agents. It discussed the mechanisms by which these agents improve contrast, including their biocompatibility and ability to provide molecular and metabolic information. Key findings highlight the high specificity of19F agents due to negligible background signals, the capacity of CEST agents for molecular imaging without metals, nitroxide radicals' utility in redox‑sensitive imaging, and hyperpolarized 13C compounds' role in real‑time metabolic assessment. Despite challenges such as low sensitivity and technical complexities, these non‑metal‑based agents offer promising, safer alternatives with enhanced diagnostic capabilities, paving the way for more precise and personalized medical imaging.","PeriodicalId":14175,"journal":{"name":"International journal of oncology","volume":"67 2","pages":""},"PeriodicalIF":4.5,"publicationDate":"2025-08-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://www.ncbi.nlm.nih.gov/pmc/articles/PMC12274189/pdf/","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"144667621","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":3,"RegionCategory":"医学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"OA","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}

引用次数: 0