Frontiers in Behavioral Neuroscience最新文献

{"title":"An asymptomatic <i>WASF1</i> truncation reveals pathogenic mechanism and therapeutic strategy for neurodevelopmental disorders.","authors":"Shaojuan Song, Zongli Yang, Juanchun Gao, Xiang Wang, Yajing Zhang, Jiaxuan Wang, Shuying Yang, Jiayu Chen, Pu Wang","doi":"10.3389/fnbeh.2026.1822055","DOIUrl":"10.3389/fnbeh.2026.1822055","url":null,"abstract":"<p><strong>Introduction: </strong>Wiskott-Aldrich syndrome protein family member 1 (<i>WASF1</i>) truncating variants, such as c.1516C>T (p.Arg506Ter), are established causes of neurodevelopmental disorders (NDDs), but their underlying pathogenic mechanism remains debated. This study aimed to clarify the disease mechanism and identify potential therapeutic leads.</p><p><strong>Methods: </strong>We characterized a novel, asymptomatic <i>WASF1</i> truncating variant (c.873delA) and compared its clinical and molecular consequences with those of the known pathogenic c.1516C>T variant. To target the likely pathogenic mutant protein, we performed high‑throughput virtual screening of the ZINC20 database.</p><p><strong>Results: </strong>Despite a similar reduction in wild‑type protein levels, the c.873delA variant did not cause neurological symptoms, in contrast to c.1516C>T. This observation supports a dominant‑negative, gain‑of‑function, or altered protein function mechanism rather than simple haploinsufficiency; however, the precise mechanism could not be definitively resolved from the available genetic and protein expression data. Virtual screening identified ZINC000101023849 as a high‑affinity lead compound with favorable drug‑like properties.</p><p><strong>Discussion: </strong>This study provides key evidence that <i>WASF1</i>‑related NDDs likely arise from a non‑haploinsufficiency mechanism and delivers a promising chemical lead for targeted therapy development. Further studies are needed to confirm the exact pathogenic mechanism and to validate the therapeutic potential of the identified compound.</p>","PeriodicalId":12368,"journal":{"name":"Frontiers in Behavioral Neuroscience","volume":"20 ","pages":"1822055"},"PeriodicalIF":3.4,"publicationDate":"2026-06-04","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://www.ncbi.nlm.nih.gov/pmc/articles/PMC13275473/pdf/","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"148294052","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":3,"RegionCategory":"医学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"OA","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}

{"title":"Editorial: Mild cognitive impairment and cognitive aging.","authors":"Eleni Konsolaki, Chrysanthi Nega, Ion Beratis, Miriam A Hickey, Panagiotis Kourtesis","doi":"10.3389/fnbeh.2026.1862932","DOIUrl":"10.3389/fnbeh.2026.1862932","url":null,"abstract":"","PeriodicalId":12368,"journal":{"name":"Frontiers in Behavioral Neuroscience","volume":"20 ","pages":"1862932"},"PeriodicalIF":3.4,"publicationDate":"2026-06-04","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://www.ncbi.nlm.nih.gov/pmc/articles/PMC13275428/pdf/","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"148293999","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":3,"RegionCategory":"医学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"OA","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}

{"title":"Endocrine modulation of stimulant use: bidirectional interactions within the hypothalamic-pituitary-gonadal axis.","authors":"Sally L Huskinson, Amy S Kohtz","doi":"10.3389/fnbeh.2026.1778346","DOIUrl":"10.3389/fnbeh.2026.1778346","url":null,"abstract":"<p><p>Stimulant use disorders are chronic relapsing conditions that differentially affect men and women. Growing evidence has demonstrated that endocrine systems play critical, bidirectional roles in regulating vulnerability to drug use, escalation, withdrawal, and relapse, particularly for women. In the current review we discuss the bidirectional relation between gonadal hormones; focusing on estradiol (E₂), progesterone (P₄), and testosterone (T); and stimulant rewards and reinforcement; focusing on cocaine, amphetamine, and methamphetamine. Most research to date has focused on cocaine and less so on amphetamine or methamphetamine. Similarly, most research has been conducted with rodents and less so with nonhuman primates and humans. Collectively, the literature supports stimulant misuse as an endocrine disorder, particularly for females, emerging from reciprocal interactions between neural and hormone systems. The reciprocal relation between gonadal hormones and stimulant rewards and reinforcement is, for the most part, reliable and robust in female rodents; however, the relation is somewhat mixed with female nonhuman primates and women. These mixed results likely are a result of the wide within- and between-subject variability in menstrual cycles (i.e., duration) and levels of ovarian hormones (e.g., E₂ and P₄ levels) in female nonhuman primates and women. Additionally, it is impossible to determine complete menstrual cycles prior to stimulant exposure in women as it is in female rats or nonhuman primates, though determination of menstrual cycles prior to stimulant exposure with female nonhuman primates is sparse. Female rodents, on the other hand, have much less variability in estrous cycles and corresponding E₂ and P₄ levels, and complete estrous cycles and hormone levels are easily obtainable prior to stimulant exposure. Finally, while the reciprocal relation between gonadal hormones and stimulants in males and men has been studied, the relation appears to be less reliably implicated in stimulant rewards and reinforcement compared with females, women, and ovarian hormones. Recognizing the hypothalamic-pituitary-gonadal (HPG) axis, including effects of stimulants on gonadal hormones and vice versa, is critical for developing individualized pharmacotherapies for stimulant misuse, particularly for women, and for understanding sex-specific stimulant-misuse phenotypes.</p>","PeriodicalId":12368,"journal":{"name":"Frontiers in Behavioral Neuroscience","volume":"20 ","pages":"1778346"},"PeriodicalIF":3.4,"publicationDate":"2026-06-04","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://www.ncbi.nlm.nih.gov/pmc/articles/PMC13275724/pdf/","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"148294029","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":3,"RegionCategory":"医学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"OA","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}

{"title":"Anxiety-like behaviour in mice after mild repetitive head impacts during the subacute phase.","authors":"Andrej Durgala, Marian Horvath, Michaela Skrabanova, Martin Cente, Peter Filipcik","doi":"10.3389/fnbeh.2026.1839876","DOIUrl":"10.3389/fnbeh.2026.1839876","url":null,"abstract":"<p><p>Traumatic brain injury (TBI) is a major health concern in the current population. Animal models are increasingly important for identifying early neurobehavioural changes associated with mild to severe as well as repetitive brain injuries. In this study, we analysed behavioural changes in a mouse model of repetitive mild TBI using unbiased quantification metrics. Our approach enabled automated markerless pose estimation of thigmotaxis together with spatial working memory and locomotor activity assessments specifically during the scarcely-studied subacute phase after brain injury. We found that animals exposed to repetitive mild head impacts developed anxiety-like behaviour, manifested by avoidance of the central zone and increased thigmotaxis in the open field test, while maintaining total distance travelled compared to the control group. However, analysis of exploratory behaviour and spatial working memory in the Y-maze, along with assessment of sensorimotor coordination and balance using rotarod tests, revealed no significant difference between the TBI and control groups. These findings suggest that repetitive mTBI is associated with selective anxiety-like behaviour during the subacute phase after brain injury, without detectable motor or cognitive deficits, reflecting the early affective symptomatology frequently reported in clinical populations with mTBI.</p>","PeriodicalId":12368,"journal":{"name":"Frontiers in Behavioral Neuroscience","volume":"20 ","pages":"1839876"},"PeriodicalIF":3.4,"publicationDate":"2026-06-02","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://www.ncbi.nlm.nih.gov/pmc/articles/PMC13269357/pdf/","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"148270971","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":3,"RegionCategory":"医学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"OA","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}

{"title":"Human sex recognition: a developmental voice-based model of brain circuits.","authors":"Yehuda Salu","doi":"10.3389/fnbeh.2026.1714154","DOIUrl":"10.3389/fnbeh.2026.1714154","url":null,"abstract":"<p><p>Pheromonal, neuroimaging, and comparative evidence converge on a limbic-hypothalamic axis-including the amygdala, bed nucleus of the stria terminalis (BNST), and hypothalamus-that organizes sex-related behaviors. In many mammals, this system is driven by chemosensory input via the vomeronasal organ (VNO) and accessory olfactory bulb (AOB). In humans, however, the functional role of the VNO is uncertain, and no consistent sex pheromone has been identified. The Human Sex Recognition (HSR) model proposes a sensory substitution at the level of input: auditory signals, conveyed through the medial geniculate nucleus (MGN), provide the primary entry point into this conserved system. Within this framework, human voice signals are proposed to serve as a functional analog to pheromonal cues in other mammals. Developmentally, auditory cues, available prenatally and throughout childhood, provide an early scaffold for the acquisition of visual and other sex-related cues through cortical learning. These multimodal representations converge on limbic-hypothalamic circuits, which are reorganized at puberty to support the participation of sex recognition in sex-related behavior. By specifying the primary sensory channel interfacing with a conserved neural architecture, the HSR model offers a biologically grounded alternative to pheromone-based accounts and generates testable predictions regarding the neural and developmental basis of human sex recognition.</p>","PeriodicalId":12368,"journal":{"name":"Frontiers in Behavioral Neuroscience","volume":"20 ","pages":"1714154"},"PeriodicalIF":3.4,"publicationDate":"2026-05-29","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://www.ncbi.nlm.nih.gov/pmc/articles/PMC13260191/pdf/","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"148249754","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":3,"RegionCategory":"医学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"OA","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}

{"title":"The good, the bad, and the in-between: an exploratory study of how different groups may be represented in the brain.","authors":"Jeroen Delplanque, Frank Van Overwalle","doi":"10.3389/fnbeh.2026.1773300","DOIUrl":"10.3389/fnbeh.2026.1773300","url":null,"abstract":"<p><strong>Introduction: </strong>Social categorization is a fundamental aspect of social perception, enabling individuals to process vast amounts of information efficiently. This exploratory fMRI study investigated whether different types of social groups are represented in the brain as stable memory representations.</p><p><strong>Methods: </strong>We used repetition suppression as a proxy-measurement for such stable representation, examining four groups categorized by their warmth and competence into the quadrants of the Stereotype Content Model (SCM). Participants judged the typicality of behavioral sentences associated with these groups, presented in pairs that either repeated the same group, or not.</p><p><strong>Results: </strong>A repetition suppression pattern was observed in the posterior temporal sulcus (pSTS) for groups with mixed warmth and competence; specifically the rich (low warmth, high competence) and the poor (high warmth, low competence). By contrast, no suppression was detected for non-mixed groups (military people and welfare recipients).</p><p><strong>Discussion: </strong>We theorize that mixed groups elicit greater ambiguity, which may prompt the brain to form more robust representations to quickly assess their functional relevance. These findings suggest that stable group representations are not uniformly applied across all group types and may reflect social complexity and motivational salience. The results highlight the pSTS as a key node in group representation and invite further research into its functional role.</p>","PeriodicalId":12368,"journal":{"name":"Frontiers in Behavioral Neuroscience","volume":"20 ","pages":"1773300"},"PeriodicalIF":3.4,"publicationDate":"2026-05-29","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://www.ncbi.nlm.nih.gov/pmc/articles/PMC13260192/pdf/","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"148249814","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":3,"RegionCategory":"医学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"OA","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}

{"title":"Physical activity, sedentary behavior, and cognitive function in children and adolescents: a scoping review of dose-response relationships, screen time interactions, and public health implications.","authors":"Ismail Dergaa, Wissem Dhahbi, Mohamed Amine Dergaa, Mortadha Razzak, Halil İbrahim Ceylan, Valentina Stefanica, Raul Ioan Muntean, Noomen Guelmami","doi":"10.3389/fnbeh.2026.1848119","DOIUrl":"10.3389/fnbeh.2026.1848119","url":null,"abstract":"<p><strong>Background: </strong>Physical inactivity among children and adolescents has reached critical levels worldwide, with approximately 81% of school-aged youth failing to meet the World Health Organization recommendation of at least 60 min of moderate-to-vigorous physical activity per day. At the same time, screen-based sedentary behaviors have increased substantially, raising concerns about their combined impact on brain development, cognitive processes, and behavioral regulation. Although extensive research exists, evidence regarding dose-response relationships, neurobiological mechanisms, and the moderating role of screen-related behaviors remains fragmented.</p><p><strong>Objectives: </strong>This scoping review aimed to systematically map the evidence linking physical activity and sedentary behavior with cognitive outcomes in children and adolescents, examine dose-response patterns, synthesize underlying neurobiological mechanisms (including brain-derived neurotrophic factor and neuroplasticity), and evaluate the moderating influence of screen time and smartphone-related behaviors.</p><p><strong>Methods: </strong>The review followed PRISMA-ScR guidelines and the Joanna Briggs Institute methodology. Six electronic databases (PubMed/MEDLINE, Scopus, Web of Science, SPORTDiscus, PsycINFO, and CINAHL) were searched for studies published between January 2000 and March 2026. Eligibility criteria were defined using the Population-Concept-Context framework. Two independent reviewers conducted screening, achieving strong inter-rater reliability (κ = 0.84).</p><p><strong>Results: </strong>Of 2,843 records identified, 60 studies met inclusion criteria across four thematic domains: physical activity and cognitive outcomes, dose-response parameters, neurobiological mechanisms, and screen-based sedentary behavior including nomophobia. Chronic physical activity, particularly in school-based and clinical ADHD settings, was consistently associated with executive function improvements across included systematic reviews and trials. Screen time exceeding 4 h per day was associated with anxiety, depressive symptoms, and attentional deficits in observational data. Evidence on exercise-induced BDNF upregulation, contingent on structured exposure (≥3 sessions/week, ≥12 weeks), is restricted to five pediatric RCTs and warrants cautious interpretation.</p><p><strong>Conclusion: </strong>This scoping review maps consistent associations between physical activity and improved cognitive function in youth, mediated through neurobiological pathways that vary in strength across study designs and populations. Excessive screen exposure is associated with cognitive risk in observational data. Integrated public health frameworks addressing physical activity, screen time, and sleep represent a priority direction for future longitudinal research and policy development.</p>","PeriodicalId":12368,"journal":{"name":"Frontiers in Behavioral Neuroscience","volume":"20 ","pages":"1848119"},"PeriodicalIF":3.4,"publicationDate":"2026-05-29","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://www.ncbi.nlm.nih.gov/pmc/articles/PMC13260285/pdf/","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"148249818","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":3,"RegionCategory":"医学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"OA","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}

{"title":"A holistic review of fatherhood in humans and non-human animals: mechanisms of fatherhood, effects on fathers themselves, and interactions of fathers with others.","authors":"Karen L Bales, Ana Finnerty-Haggerty, Joanna Y Guan, Lauren Meckler, Jasmine Saenz, Meghan J Sosnowski, Shirin Afrakhteh, Christina L Hung, Rani A Purewal, Stephanie L Oliinyk, Felisa J Carbajal, Olufemi Shakuur Nyabingi","doi":"10.3389/fnbeh.2026.1779750","DOIUrl":"10.3389/fnbeh.2026.1779750","url":null,"abstract":"<p><p>Fathers are often the forgotten parent in mammals; due to the necessity of lactation for the survival of infants, mothers and motherhood are by far the better studied sex (a rather unusual phenomenon among most topics). However, the study of fathers has experienced a recent surge in interest. Here we review literature from a neuroscience, endocrine, and psychological perspective, on the mechanisms of fatherhood and roles of fathers across species, including the ways that fathering affects mental health, interactions with others, and partner relationships in humans and animals. Limitations and fundamental gaps remaining in the literature on fatherhood are identified.</p>","PeriodicalId":12368,"journal":{"name":"Frontiers in Behavioral Neuroscience","volume":"20 ","pages":"1779750"},"PeriodicalIF":3.4,"publicationDate":"2026-05-29","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://www.ncbi.nlm.nih.gov/pmc/articles/PMC13261393/pdf/","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"148249822","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":3,"RegionCategory":"医学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"OA","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}

{"title":"Sexually dimorphic effects of single prolonged stress on conditioned suppression behavior in Long Evans rats with no detection of altered mRNA expression of VGLUT1, GAD1, and Arc in the ventral hippocampus.","authors":"Stacy R Pitcairn, Andrew E Tapp, Ryan M Drenan, Jeffrey L Weiner","doi":"10.3389/fnbeh.2026.1771722","DOIUrl":"10.3389/fnbeh.2026.1771722","url":null,"abstract":"<p><strong>Introduction: </strong>Women are especially vulnerable to stress-related disorders such as posttraumatic stress disorder (PTSD), yet most preclinical studies have focused on males. Rodents often exhibit sexually-dimorphic coping strategies in fear behaviors, thus complicating neurobiological interpretations. To facilitate comparisons, an operant conditioned suppression paradigm was implemented where the measure of fear is not dependent on the coping strategy. The effect of single prolonged stress (SPS) on fear behavior was assessed. Since stress has been linked to excitatory phenotypes, mRNA expression of vesicular glutamate transporter 1 (VGLUT1) and glutamate decarboxylase (GAD1) was measured to assess excitation and inhibition-related markers in the ventral hippocampus (vHip), a stress-sensitive brain region. Activity-regulated cytoskeleton associated protein (Arc/Arg3.1) was used to identify neurons activated during fear extinction. The goal of this study was to determine whether SPS produces sex differences in conditioned-suppression fear behavior and to identify accompanying molecular changes in the vHip.</p><p><strong>Methods: </strong>Thirty-two adult male and female Long Evans rats (<i>n</i> = 16/sex) were trained to lever press for sucrose on variable-interval (VI) schedules. After stable responding, a subset (8/sex) underwent single prolonged stress (SPS). All rats received fear conditioning with five presentations of a conditioned stimulus (CS)-shock pairing. Extinction sessions included baseline VI-60 responding followed by CS presentations. Fear was defined as the suppression of lever pressing during CS. Tissue containing the vHip was collected following the last session for RNA <i>in situ</i> hybridization (RNA-ISH) to examine mRNA expression of molecular markers of excitation/inhibition (VGLUT1, GAD1) and neuronal activation (Arc).</p><p><strong>Results: </strong>SPS impaired extinction in males on day 1 but had no effect in females. On day 2, males showed greater fear than females regardless of SPS. No significant effects were observed on day 3 or the recall session. No significant effects of sex or stress were observed on vHip mRNA expression of VGLUT1, GAD1, or Arc under the experimental conditions.</p><p><strong>Discussion: </strong>These findings demonstrate sex-specific effects of SPS on fear extinction measured by conditioned suppression. Future studies should explore earlier time points, additional regions (e.g., amygdala and medial prefrontal cortex) hormonal modulation, and interventions that may mitigate SPS effects in males and females.</p>","PeriodicalId":12368,"journal":{"name":"Frontiers in Behavioral Neuroscience","volume":"20 ","pages":"1771722"},"PeriodicalIF":3.4,"publicationDate":"2026-05-28","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://www.ncbi.nlm.nih.gov/pmc/articles/PMC13253521/pdf/","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"148249727","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":3,"RegionCategory":"医学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"OA","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}

{"title":"Sex differences in compulsive sexual behavior disorder.","authors":"Aviv M Weinstein","doi":"10.3389/fnbeh.2026.1821534","DOIUrl":"10.3389/fnbeh.2026.1821534","url":null,"abstract":"<p><strong>Background: </strong>Compulsive sexual behavior is a failure to control intense, repetitive sexual impulses or urges, resulting in sexual behavior that causes marked distress or impairment. Studies on compulsive sexual behavior report a higher prevalence of CSBD among men. There are sex differences in the symptoms and behaviors associated with CSB, like sexual arousal and motivation.</p><p><strong>Methods: </strong>A systematic search in PubMed/Medline and Scopus databases between January 2000 and January 2026, using search terms related to compulsive sexual behavior.</p><p><strong>Results: </strong>While CSBD is more common in men, neuroticism and stress vulnerability contribute importantly to CSBD symptoms in women. Human sexuality is associated with neurotransmitters, hormones, and genes, in particular genes responsible for dopamine, which is associated with novelty seeking, impulsivity, and may influence sexual activity. Neuroimaging studies outlined sex differences in neural responses to sexual stimuli in men in fMRI. Men with CSBD showed an activation of reward areas in the brain in response to sexually explicit stimuli. Their higher sensitivity to the rewarding value of sexual cues may contribute to their increased risk of addictive or compulsive sexual behaviors. Studies on CSBD indicate that impulsivity and psychiatric comorbidity are common across both genders. Finally, childhood trauma and abuse are often reported in individuals seeking treatment for sex addiction, particularly among women.</p><p><strong>Implications: </strong>Little is known about sex differences in CSBD. Although childhood adversities may contribute to the development of CSB in both women and men, further research is needed to determine whether the groups respond differently.</p>","PeriodicalId":12368,"journal":{"name":"Frontiers in Behavioral Neuroscience","volume":"20 ","pages":"1821534"},"PeriodicalIF":3.4,"publicationDate":"2026-05-26","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://www.ncbi.nlm.nih.gov/pmc/articles/PMC13246655/pdf/","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"148216936","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":3,"RegionCategory":"医学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"OA","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}