European Journal of Neuroscience最新文献

{"title":"Alpha-Band Brain Activity Shapes Online Perceptual Learning of Concurrent Speech Differentially in Musicians vs. Nonmusicians","authors":"Jessica MacLean,&nbsp;Jack Stirn,&nbsp;Gavin M. Bidelman","doi":"10.1111/ejn.70100","DOIUrl":"https://doi.org/10.1111/ejn.70100","url":null,"abstract":"<div>\u0000 \u0000 <p>Plasticity from auditory experience shapes the brain's encoding and perception of sound. Though stronger neural entrainment (i.e., brain-to-acoustic synchronization) aids speech perception, underlying oscillatory activity may uniquely interact with long-term auditory experiences (i.e., music training) and short-term plasticity during concurrent speech perception. Here, we explored oscillatory activity during rapid auditory perceptual learning of concurrent speech sounds in normal-hearing young adults who differed in their amount of self-reported music training (defined as “musicians” and “nonmusicians”). Participants learned to identify double-vowel mixtures during ~45 min training sessions with concurrent high-density EEG recordings. We analyzed alpha-band power (7–12 Hz) following a rhythmic speech-stimulus train (~9 Hz) preceding behavioral identification to determine whether increased (brain-to-speech entrainment) or decreased alpha activity (alpha-band suppression) corresponded with task success. Source and directed functional connectivity analyses of EEG data probed whether behavior was driven by group differences in auditory-motor coupling. Both groups improved in behavioral identification with training. Listeners' alpha-band power prior to target speech predicted behavioral identification performance; surprisingly, stronger alpha oscillations were observed preceding incorrect compared to correct trial responses. We also found stark hemispheric biases in auditory-motor coupling, with greater auditory-motor connectivity in right compared to left hemisphere for musicians (R &gt; L) but not in nonmusicians (R = L). Stronger alpha activity preceding incorrect behavioral responses supports the notion that alpha-band (~10 Hz) suppression is an important modulator of trial-by-trial success in perceptual processing. Our findings suggest that neural oscillations and auditory-motor connectivity interact with music training to impact speech perception.</p>\u0000 </div>","PeriodicalId":11993,"journal":{"name":"European Journal of Neuroscience","volume":"61 9","pages":""},"PeriodicalIF":2.7,"publicationDate":"2025-04-28","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"143884012","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":4,"RegionCategory":"医学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}

{"title":"Lower Cerebral Blood Flow Following Surgical Treatment of Acute Type A Dissection: An Arterial Spin Labelling Study","authors":"Guozhong Chen,&nbsp;Xinying Wu,&nbsp;Xiaobin Huang,&nbsp;Fuhua Huang,&nbsp;Jian Li,&nbsp;Yujia Gao,&nbsp;Hai Lin,&nbsp;Jianmin Yuan,&nbsp;Xiaoxue Liu,&nbsp;Xindao Yin,&nbsp;Xin Chen","doi":"10.1111/ejn.70075","DOIUrl":"https://doi.org/10.1111/ejn.70075","url":null,"abstract":"<div>\u0000 \u0000 <p>Postoperative permanent neurological dysfunction remains a challenging complication in type A acute aortic dissection (TAAAD). Researches evaluating cerebral perfusion and altered blood flow in postoperative patients with TAAAD using non-invasive imaging techniques, such as arterial spin labelling are scarce. This study aims to assess cerebral blood flow (CBF) in postoperative patients with TAAAD, using arterial spin labeling (ASL). This study enrolled 22 postoperative patients and 25 healthy control subjects (HC), they all underwent a three-dimensional pseudo-continuous ASL MRI scanning. Voxel-based comparison of normalized CBF was conducted. The relationship between CBF variation and clinical scale assessment was further analysed. Compared with HC subjects, postoperative patients with TAAAD exhibited lower CBF levels in the right middle frontal gyrus, the right orbit of frontal gyrus, the bilateral fusiform gyrus, the right middle temporal gyrus, the bilateral inferior temporal gyrus, the bilateral lateral occipital cortex and the bilateral cerebellum (<i>t</i> &gt; 3.0 and <i>p</i> &lt; 0.05, FDR corrected at cluster level). These variations were also significantly correlated with multiple clinical rating scales about cognition and emotion. Postoperative patients with TAAAD exhibit abnormalities in several brain regions. The affected areas involve important component of neuro-networks, including psychological cognitive activity, attention and emotion regulation and high-level visual function.</p>\u0000 </div>","PeriodicalId":11993,"journal":{"name":"European Journal of Neuroscience","volume":"61 8","pages":""},"PeriodicalIF":2.7,"publicationDate":"2025-04-28","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"143879935","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":4,"RegionCategory":"医学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}

{"title":"Global Clock Coordination by the Brain Clock in the Suprachiasmatic Nucleus Through Relay and Amplification of Diffusible and Neural Signaling","authors":"Rae Silver,&nbsp;Yifan Yao,&nbsp;Jihwan Myung","doi":"10.1111/ejn.70097","DOIUrl":"https://doi.org/10.1111/ejn.70097","url":null,"abstract":"<div>\u0000 \u0000 <p>The brain clock located in the suprachiasmatic nucleus (SCN) of the hypothalamus receives direct retinal input, thereby providing the entire body with an internal representation of external solar time. The pathways by which this small nucleus signals so broadly involve co-occurring nervous and diffusible output signals, although the latter are less understood. Portal pathways, such as the well-known pituitary portal pathway, provide a mechanism, whereby signals of neural origin can reach local, specialized targets without suffering dilution in the systemic blood supply. Newly discovered vascular pathways involve direct connections between each of the sensory circumventricular nuclei at its point of attachment to the brain. These nuclei line the brain's ventricles, and their leaky blood vessels and large perivascular spaces represent a route, whereby secretions from the SCN can be relayed and then amplified, providing a pathway to achieve global coordination of circadian clock signaling. This review provides a narrative that incorporates our understanding of SCN neural and diffusible output signals, with particular emphasis on the contribution of brain fluidic compartments and the fluids therein.</p>\u0000 </div>","PeriodicalId":11993,"journal":{"name":"European Journal of Neuroscience","volume":"61 8","pages":""},"PeriodicalIF":2.7,"publicationDate":"2025-04-25","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"143871889","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":4,"RegionCategory":"医学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}

{"title":"Melatonin and Methylprednisolone Combination Ameliorates Inflammation and Enhances Recovery After Sciatic Nerve Crush Injury","authors":"Gökçenur Yalçın,&nbsp;Başak Bilir Kaya,&nbsp;Emre Ata,&nbsp;Cumaali Demirtaş,&nbsp;Hakan Beyaztas,&nbsp;Gülnihal Ay,&nbsp;Pınar Engin Zerk,&nbsp;Eray Metin Guler","doi":"10.1111/ejn.70116","DOIUrl":"https://doi.org/10.1111/ejn.70116","url":null,"abstract":"<div>\u0000 \u0000 <p>This sham-controlled animal study aimed to investigate the effects of melatonin and methylprednisolone combination on motor function, nerve conduction and histopathological and biochemical findings in rats with sciatic nerve crush injury. Forty-eight male Sprague–Dawley rats were divided into 6 groups (<i>n</i> = 8): CT: control, VEH: sciatic nerve injury, LMP: 15-mg/kg methylprednisolone, high-dose methylprednisolone (HMP): 30-mg/kg methylprednisolone, MEL: 15-mg/kg melatonin, MMP: 15-mg/kg methylprednisolone+15-mg/kg melatonin. The rats were evaluated with Sciatic Functional Index (SFI), nerve conduction study, interleukin-1β <b>(</b>IL-1ß), nerve growth factor (NGF), total antioxidant status (TAS), total oxidant status (TOS) and histopathological scores. There were no significant intergroup differences in baseline tests. SFI significantly improved in all treated groups with no significant intergroup differences. Motor amplitude improved most in MMP, LMP and MEL, respectively. Nerve conduction velocity significantly improved in MMP compared to VEH. There were no significant intergroup differences regarding serum NGF, TAS and TOS. Tissue NGF levels were higher in LMP, HMP and MEL. IL-1ß levels were significantly lower in CT and MMP. Tissue oxidative stress levels were significantly lower in treated groups compared to VEH, with no significant difference among them. MMP showed greater histopathological improvement. Melatonin combination therapy in sciatic nerve crush injury provided adequate functional improvement, superior electrophysiological and histopathological recovery compared to high-dose methylprednisolone and exhibited better anti-inflammatory activity through IL-1ß.</p>\u0000 </div>","PeriodicalId":11993,"journal":{"name":"European Journal of Neuroscience","volume":"61 8","pages":""},"PeriodicalIF":2.7,"publicationDate":"2025-04-25","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"143871892","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":4,"RegionCategory":"医学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}

{"title":"A Kinetic Ecological Approach to Beauty Perception: A Perspective Review on the Case of Symmetry and the Golden Ratio","authors":"Marco Iosa,&nbsp;Maria Pia Lucia,&nbsp;Claudia Salera","doi":"10.1111/ejn.70119","DOIUrl":"https://doi.org/10.1111/ejn.70119","url":null,"abstract":"<p>In the 1970s, the psychologist J.J. Gibson developed an “ecological approach to visual perception”, suggesting that humans perceive the environment exploiting environmental affordances—surrounding invariant features that define possible individuals–object interactions—without top–down mediation of cognitive processes. Shepard extended this approach, suggesting that common environmental features are internalized defining perceptual constraints, such as the circadian rhythm, three-dimensional space, and gravity. In this perspective review, we apply these concepts to neuroaesthetics and beauty perception, discussing how the internalization of invariants may influence our perception of beauty. Within this framework, special emphasis was placed on symmetry and golden ratio, typically considered as two benchmarks for beauty, and two geometrical features that can be considered as environmental affordances. Moreover, we suggest that kinetic aspects play a role in beauty perception of these proportions, particularly by the internal model of gravity.</p>","PeriodicalId":11993,"journal":{"name":"European Journal of Neuroscience","volume":"61 8","pages":""},"PeriodicalIF":2.7,"publicationDate":"2025-04-25","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://onlinelibrary.wiley.com/doi/epdf/10.1111/ejn.70119","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"143871549","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":4,"RegionCategory":"医学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"OA","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}

{"title":"VEGF-E Attenuates Injury After Ischemic Stroke by Promoting Reparative Revascularization","authors":"Romain Menet,&nbsp;Leila Nasrallah,&nbsp;Maxime Bernard,&nbsp;Anne-Sophie Allain,&nbsp;Ayman ElAli","doi":"10.1111/ejn.70114","DOIUrl":"https://doi.org/10.1111/ejn.70114","url":null,"abstract":"<p>The angiogenic response after stroke correlates with mild injury and an improved recovery. Stimulation of post-stroke angiogenesis using vascular endothelial growth factor (VEGF)-A is associated with an increased risk of vascular destabilization, leading to life-threatening complications. The non-mammalian VEGF-A homolog, VEGF-E, stimulates stable cutaneous vascularization and promotes wound healing. Herein, we posit that VEGF-E represents a potential mediator of reparative revascularization after ischemic stroke. C57BL6/J wildtype mice were subjected to experimental stroke, and VEGF-E or VEGF-A were intranasally delivered during the subacute phase. Our results indicate that VEGF-E improves neurological recovery and increases vascular density without compromising permeability, more efficiently than VEGF-A. We show that VEGF-E-mediated revascularization correlates with normal restoration of brain perfusion, whereas VEGF-A induces cerebral hyperperfusion, indicative of vascular dysfunction. Furthermore, VEGF-E reduces microvascular stalls, increases the density of angiogenic vasculature, and improves the interaction of brain endothelial cell with pericytes, which is critical for vascular stabilization. Using cell-based assays, we demonstrate that stimulation of brain endothelial cells with VEGF-E, but not with VEGF-A, increases the expression of platelet-derived growth factor (PDGF)-D, a potent ligand of PDGFRβ that plays critical roles in regulating the survival and functions of perivascular cells, including pericytes. These effects are associated with activation of extracellular signal-regulated kinase (ERK)1/2 and P38 mitogen-activated protein kinase (MAPK). Finally, we confirm that the secretome of VEGF-E-stimulated brain endothelial cells ameliorates pericyte migration required for vascular recruitment. Our study indicates that VEGF-E promotes a stable and functional revascularization after ischemic stroke, outlining its promises for therapeutic purposes.</p>","PeriodicalId":11993,"journal":{"name":"European Journal of Neuroscience","volume":"61 8","pages":""},"PeriodicalIF":2.7,"publicationDate":"2025-04-25","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://onlinelibrary.wiley.com/doi/epdf/10.1111/ejn.70114","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"143871653","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":4,"RegionCategory":"医学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"OA","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}

{"title":"S-9-PAHSA Attenuates Aβ Accumulation and Improves Cognitive Deficits by Promoting Mitochondrial Autophagy in 5xFAD Mice","authors":"Chenyu Lu,&nbsp;Jiaoqi Ren,&nbsp;Shanshan Huang,&nbsp;Meng Wang,&nbsp;Houguang Zhou,&nbsp;Jingchun Guo","doi":"10.1111/ejn.70118","DOIUrl":"https://doi.org/10.1111/ejn.70118","url":null,"abstract":"<div>\u0000 \u0000 <p>Alzheimer's disease (<span>AD</span>) is a prevalent neurodegenerative disorder characterized by significant cognitive impairment and predominantly affects the elderly. With no effective cure available, research continues to explore novel therapeutic and preventive strategies. Recently, palmitic acid-hydroxystearic acids (PAHSAs), especially their stereochemistry S-configuration, have shown potential as a dietary supplement with anti-inflammatory and anti-diabetic properties. We previously found that one of the PAHSAs, 9-PAHSA, could improve cognitive impairment in the high-fat-diet mice, however, whether it has an equal effect on <span>AD</span>-like mice remains unclear. Since mitochondrial dysfunction is recognized as a significant pathological feature of <span>AD</span>, with impaired mitophagy leading to the accumulation of dysfunctional mitochondria, thus exacerbating disease progression, in this study, we evaluated the effects of the chiral isomer of 9-PAHSA, S-9-PAHSA, on cognitive dysfunction and mitochondrial dysfunction in 5xFAD mice. Three-month-old mice were treated with S-9-PAHSA 30 mg/kg in their drinking water for 3 months. Behavioral studies were conducted using the Morris Water Maze (MWM) and Y-maze tests, followed by assessments of amyloid-beta (Aβ) plaque deposition, neuronal apoptosis, and mitochondrial function. We found that S-9-PAHSA significantly enhanced spatial learning and memory abilities, reduced amyloid plaque deposition, decreased neuronal apoptosis, and improved mitochondrial homeostasis and autophagy in 5xFAD mice. These findings suggest that S-9-PAHSA holds promise as a supplementary preventive and therapeutic strategy for <span>AD</span> treatment.</p>\u0000 </div>","PeriodicalId":11993,"journal":{"name":"European Journal of Neuroscience","volume":"61 8","pages":""},"PeriodicalIF":2.7,"publicationDate":"2025-04-25","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"143871891","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":4,"RegionCategory":"医学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}

{"title":"When Testing Becomes Learning—Underscoring the Relevance of Habituation to Improve Internal Validity of Common Neurocognitive Tests","authors":"Konstantin Warneke,&nbsp;Manuel Oraže,&nbsp;Gerit Plöschberger,&nbsp;Marco Herbsleb,&nbsp;Jose Afonso,&nbsp;Sebastian Wallot","doi":"10.1111/ejn.70117","DOIUrl":"https://doi.org/10.1111/ejn.70117","url":null,"abstract":"<p>Testing neurocognitive function is receiving growing attention in psychological and physical health research. To counteract the costs, reduced accessibility, and complexity of brain imaging (e.g., CT scans and fMRI) or function tests, neurocognitive performance tests (e.g., the Stroop test, the Trail Making Test, or the Choice Reaction Task) are commonly implemented. Although reliability is considered paramount when interpreting intervention effects, a detailed quantification of systematic and random errors is scarce. By recruiting 68 healthy participants from different age groups (7–64 years), we quantified population-specific measurement errors in the aforementioned neurocognitive tasks. The goal was to raise awareness about the impact of learning effects on reliability assessments and their interpretation. By performing five testing sessions with two trials per day, we observed significant learning effects from repeated testing. Trial-to-trial improvements of up to 50% were measured, accompanied by a random measurement error reduction from day to day. These learning effects were task and population specific, highlighting the need for caution when transferring reliability coefficients from other studies. The quantification of systematic and random measurement errors underscores the importance of conducting sufficient habituation sessions in neurocognitive tasks, as test protocols lack validity if they do not ensure reliability. Therefore, sufficient habituation sessions (i.e., until no meaningful learning effects can be observed) may be warranted when testing is repeated within short timeframes.</p>","PeriodicalId":11993,"journal":{"name":"European Journal of Neuroscience","volume":"61 8","pages":""},"PeriodicalIF":2.7,"publicationDate":"2025-04-24","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://onlinelibrary.wiley.com/doi/epdf/10.1111/ejn.70117","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"143871496","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":4,"RegionCategory":"医学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"OA","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}

{"title":"Correction to “From Solo to Collaborative: The Global Increase in Neuroscience Authors Over the Two Decades”","authors":"","doi":"10.1111/ejn.70122","DOIUrl":"https://doi.org/10.1111/ejn.70122","url":null,"abstract":"<p>\u0000 <span>Segreti, M.</span>, <span>Paul, A.</span>, <span>Pani, P.</span>, <span>Genovesio, A.</span> and <span>Brunamonti, E.</span> (<span>2025</span>), <span>From Solo to Collaborative: The Global Increase in Neuroscience Authors Over Two Decades</span>. <i>European Journal of Neuroscience</i>, <span>61</span>: e70092. https://doi.org/10.1111/ejn.70092.\u0000 </p><p>The legend in Figure 2, and the corresponding figure description, currently labels one of the categories as “USA.” This label should be corrected to “America” to ensure consistency with the broader regional classification used throughout the manuscript and the description of this result in the text. Please find attached below the figure correctly labelled.</p><p>Two smaller errors are also present at page 3:</p><p>In the sentence “the two-way mixed ANOVA revealed that the number of publications was significantly influenced by both the country and sub-period factor”, “number of publications”, should be replaced by “number of authors”, as the statistical analysis specifically pertains to authorship trends rather than publication counts.</p><p>In addition, the sentence “South Korea is the only country showing a slight decreasing trend rather than growth” mistakenly refers to Figure 2 rather than Figure 3.</p><p>We apologise for these errors.</p>","PeriodicalId":11993,"journal":{"name":"European Journal of Neuroscience","volume":"61 8","pages":""},"PeriodicalIF":2.7,"publicationDate":"2025-04-23","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://onlinelibrary.wiley.com/doi/epdf/10.1111/ejn.70122","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"143865966","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":4,"RegionCategory":"医学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"OA","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}

{"title":"Inhibition of C3/C3aR Pathway Alleviates Visual and Optic Nerve Impairments in a Rat Model of Neuromyelitis Optica Spectrum Disorder","authors":"Siqi Chen,&nbsp;Yurong Zhang,&nbsp;Kaiqun Liu,&nbsp;Zhiqiang Xiao,&nbsp;Xiaoning Liu,&nbsp;Ziyan Xu,&nbsp;Wei Qiu,&nbsp;Ying Xu,&nbsp;Changyong Tang,&nbsp;Hui Yang","doi":"10.1111/ejn.70103","DOIUrl":"https://doi.org/10.1111/ejn.70103","url":null,"abstract":"<div>\u0000 \u0000 <p>Accumulating evidence suggests that the complement C3/C3a receptor (C3aR) pathway plays a pivotal role in the pathogenesis of neurodegenerative and autoimmune disorders, particularly neuromyelitis optica spectrum disorders (NMOSDs). Despite this, the specific pathogenic effect and mechanism of C3/C3aR pathway in NMOSD remains elusive. In this study, we demonstrated for the first time that the expression of C3aR and its upstream ligand C3 is significantly upregulated in the optic nerve of a rat model of NMOSD-related optic neuritis (NMOSD-ON). Our findings reveal that this upregulation leads to blood–brain barrier (BBB) disruption, demyelination and neuronal damage. Through the use of a novel C3aR inhibitor, JR14a, we demonstrate its effectiveness in reducing C3aR expression and mitigating pathological damage in the optic nerve. Furthermore, our transcriptome sequencing analysis of NMOSD optic nerve tissues reveals extensive enrichment of inflammation- and immune response–related pathways, with particular emphasis on the complement and coagulation cascades pathway. This study not only elucidated the crucial role of the C3-C3aR pathway in NMOSD-ON pathogenesis but also provided a new promising therapeutic target for NMOSD through C3aR pathway inhibition.</p>\u0000 </div>","PeriodicalId":11993,"journal":{"name":"European Journal of Neuroscience","volume":"61 8","pages":""},"PeriodicalIF":2.7,"publicationDate":"2025-04-22","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"143856940","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":4,"RegionCategory":"医学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}