Clinical Neuropsychologist最新文献

{"title":"Prevalence of post-traumatic hypopituitarism in active-duty service members receiving comprehensive care for traumatic brain injury (TBI) and psychological health: A military mild TBI cohort study.","authors":"Bryson J Hewins, Sara M Lippa, Rachael D C Jones, J Kent Werner, Carol B Moore, Kimbra Kenney","doi":"10.1080/13854046.2026.2614576","DOIUrl":"10.1080/13854046.2026.2614576","url":null,"abstract":"<p><p><b>Objective:</b> The prevalence of neuroendocrine dysfunction (NED) following mild traumatic brain injury (mTBI) remains obscure, with widely varying prevalence estimates. This study aimed to determine prevalence of NED among central hypogonadism, central hypothyroidism, and growth hormone deficiency (GHD) in active-duty military service members (SMs) receiving comprehensive TBI and psychological health care and characterize TBI burden, neurobehavioral symptom severity, and NED associations. <b>Methods:</b> Retrospective analysis of baseline, fasting morning serum screening labs of thyroid, gonadal, and growth hormone axes obtained from SMs attending a 4-week treatment program for TBI. NED prevalence was characterized in those who completed full tri-axis screening as well as GHD and central hypogonadism screening independently. <b>Results:</b> Of 1,832 TBI-screened SMs, 493 completed full neuroendocrine testing. Dual-clinician review confirmed NED in 45 (9.1%; 95% CI [6.9%, 12.0%]). Central hypogonadism (<i>n</i> = 33, 6.7%) was most common, followed by central hypothyroidism (<i>n</i> = 10, 2.0%); Screen-positive suspected GHD was 0.2% (1/493); no cases were confirmed by dynamic stimulation testing, so the true prevalence of GHD cannot be determined in this cohort. NED was associated with increased time since injury (<i>OR</i> = 4.15, <i>p</i><.001) and fewer lifetime TBIs (<i>OR</i> = 0.66, <i>p</i>=.008), but not neurobehavioral symptoms or cognitive function. <b>Conclusions:</b> In the largest military TBI cohort with full NED screening to date, confirmed post-traumatic hypopituitarism prevalence was 9.1%. GHD was least common by IGF-1 (0.2%) screening, but its true prevalence remains indeterminate without systematic stimulation testing. Findings refine prevalence estimates for NED after military mTBI and emphasize the need for standardized diagnostic approaches prioritizing gonadal and thyroid axes.</p>","PeriodicalId":55250,"journal":{"name":"Clinical Neuropsychologist","volume":" ","pages":"2383-2402"},"PeriodicalIF":2.6,"publicationDate":"2026-11-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"145999480","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":3,"RegionCategory":"心理学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}

{"title":"Neuroendocrine dysfunction after TBI: A guide for the clinical neuropsychologist.","authors":"Stefanie N Howell, Brent E Masel","doi":"10.1080/13854046.2025.2537335","DOIUrl":"https://doi.org/10.1080/13854046.2025.2537335","url":null,"abstract":"<p><strong>Objective: </strong>This review aims to provide neuropsychologists with a comprehensive understanding of neuroendocrine dysfunction following traumatic brain injury (TBI), emphasizing post-traumatic hypopituitarism (PTHP) and its implications for cognitive function and rehabilitation.</p><p><strong>Method: </strong>A critical review of current literature on neuroendocrine dysfunction following TBI, including studies on pituitary function, hormone deficiencies, and their effects on cognition, mood, and fatigue. The review integrates findings from endocrinology, neuroscience, and clinical neuropsychology to highlight the importance of interdisciplinary management.</p><p><strong>Results: </strong>Research indicates that a high proportion of moderate to severe TBI cases may develop PTHP, with growth hormone deficiency (GHD) as the most prevalent dysfunction. While less extensively studied, evidence suggests that individuals with persistent post-mild TBI symptoms may exhibit neuroendocrine dysfunction at similar rates to moderate to severe TBI patients. The impact of hormonal dysregulation on cognition and neuropsychological performance underscores the need for targeted assessment and intervention. Emerging data also suggest that metabolic disturbances, such as altered gut microbiota and amino acid imbalances, may contribute to post-TBI fatigue and cognitive impairment.</p><p><strong>Conclusions: </strong>Neuropsychologists play a vital role in identifying and referring patients with suspected neuroendocrine dysfunction for appropriate testing and treatment. Recognizing PTHP as a potential contributor to chronic TBI symptoms may lead to improved patient outcomes through targeted hormonal therapies and interdisciplinary rehabilitation strategies. Increased awareness of the relationship between TBI, pituitary dysfunction, and cognition is essential for optimizing clinical care.</p>","PeriodicalId":55250,"journal":{"name":"Clinical Neuropsychologist","volume":"40 8","pages":"2347-2360"},"PeriodicalIF":2.6,"publicationDate":"2026-11-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"149247665","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":3,"RegionCategory":"心理学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}

{"title":"Virtual reality-based eye tracking throughout typical concussion recovery.","authors":"Gregory C Edwards, Nick Fogt, James A Onate, Rebecca A Bliss, Catherine C Quatman-Yates, Jaclyn B Caccese","doi":"10.1080/13854046.2025.2503328","DOIUrl":"10.1080/13854046.2025.2503328","url":null,"abstract":"<p><p><b>Objective:</b> The purpose of this study was to determine the effects of concussion on visual and oculomotor function using a virtual reality-based eye tracking system throughout recovery. <b>Design:</b> A prospective, longitudinal cohort design assessed at the subacute, return-to-activity, and 6-month timepoints was used. <b>Methods:</b> Recently concussed (<i>n</i> = 21; age = 30 ± 14) and healthy control participants (<i>n</i> = 20; age = 29 ± 11) completed visual and oculomotor testing at three timepoints (subacute, return-to-activity, and 6-months) using a virtual reality-based head mounted display eye tracking system sampled at 100 Hz. Symptoms and clinical assessments were collected using the Sport Concussion Assessment Tool-5^th Edition (SCAT-5). <b>Results:</b> No statistically significant visual and oculomotor group differences were observed (<i>p</i> > 0.01), although individuals within the concussion group demonstrated highly variable outcomes at the subacute timepoint. <b>Conclusions:</b> While our findings did not reveal significant group-wide differences in visual and oculomotor function, the high inter-individual variability within the concussion group underscores the need for individualized assessment approaches. The ability of eye-tracking technology to detect subtle, yet potentially clinically relevant impairments at the individual level suggests its potential as a valuable adjunct to symptom-based concussion assessments. Future studies should explore the integration of symptom-based tools, clinical oculomotor assessments, and objective eye-tracking metrics to improve our understanding of concussion-related impairments.</p>","PeriodicalId":55250,"journal":{"name":"Clinical Neuropsychologist","volume":" ","pages":"2330-2346"},"PeriodicalIF":2.6,"publicationDate":"2026-11-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"144259426","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":3,"RegionCategory":"心理学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}

{"title":"Vestibular dysfunction after mild traumatic brain injury: A systematic review.","authors":"Monica-Rae Owens, Eve Larkin, Samuel A Tenhoeve, Jason R Olcott, Nathan Barber, Matthew C Findlay, Sarah T Menacho, Mohammed Sbai, Ramesh Grandhi","doi":"10.1080/13854046.2025.2536694","DOIUrl":"10.1080/13854046.2025.2536694","url":null,"abstract":"<p><p><b>Objective:</b> Vestibular system dysfunction (VD), a condition characterized by impairments in balance, spatial orientation, and eye movement control, is frequently encountered after mild traumatic brain injury (mTBI). We reviewed the existing literature on the various forms of post-traumatic VD and discuss the epidemiology and natural history, diagnostic methods, assessment tools, and therapeutic interventions crucial to identifying and managing these impairments. <b>Method:</b> We examined diagnostic techniques, therapeutic strategies, and outcomes reported in the literature on patients with mTBI with VD. Studies were excluded if they did not address VD resulting from mTBI or if they focused primarily on post-concussion syndrome. <b>Results:</b> Of the 927 studies screened, 59 met the inclusion criteria. Although most research prioritized symptom alleviation, some evaluated outcomes after treatment. Common vestibular symptoms identified included dizziness, imbalance, headache, postural and gait disturbances, vertigo, vestibulo-ocular dysfunction, hearing loss or tinnitus, and nystagmus. Diagnostic methods comprised various techniques, including diverse clinical assessments and specialized vestibular tests, and therapeutic strategies included vestibular rehabilitation therapy and pharmacological treatments, leading to varied clinical outcomes and quality-of-life improvements. <b>Conclusions:</b> The current approaches to diagnosing, assessing, and treating VD in patients with mTBI underscore the importance of a multidisciplinary approach with targeted therapeutic strategies to effectively manage this complex and heterogeneous condition. Further research is needed to investigate the complex interplay between VD and neuropsychiatric symptomology.</p>","PeriodicalId":55250,"journal":{"name":"Clinical Neuropsychologist","volume":" ","pages":"2284-2310"},"PeriodicalIF":2.6,"publicationDate":"2026-11-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"144709986","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":3,"RegionCategory":"心理学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}

{"title":"Special issue editorial: Multidimensional characterization of somatic, autonomic, and hormonal dysfunction following traumatic brain injury.","authors":"Christine M Mullen","doi":"10.1080/13854046.2026.2738662","DOIUrl":"https://doi.org/10.1080/13854046.2026.2738662","url":null,"abstract":"","PeriodicalId":55250,"journal":{"name":"Clinical Neuropsychologist","volume":"40 8","pages":"2269-2274"},"PeriodicalIF":2.6,"publicationDate":"2026-11-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"149247674","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":3,"RegionCategory":"心理学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}

{"title":"Relationships between vestibular dysfunction and cognitive performance in military veterans with mild to moderate TBI.","authors":"Emanuel M Boutzoukas, Karen M Skop, Marc A Silva","doi":"10.1080/13854046.2025.2598370","DOIUrl":"10.1080/13854046.2025.2598370","url":null,"abstract":"<p><p><b>Objective:</b> Vestibular dysfunction is common following mild-to-moderate traumatic brain injury (TBI) and impacts quality of life. However, little is known regarding the persistence and stability of vestibular symptoms over time and their effects on cognition. This study examined self-reported vestibular symptoms 1 to 2 years following mild-to-moderate TBI in a Military/Veteran cohort. We further evaluated the association of vestibular symptoms with 5-year post-TBI cognitive outcomes. <b>Method:</b> Military service members and Veterans enrolled in the VA TBI Model Systems (TBIMS) completed follow-up interviews at 1-, 2-, and 5-years post-TBI, with valid symptom and performance validity. Study 1 examined vestibular symptom change from 1 to 2 years with the Neurobehavioral Symptom Inventory (<i>N</i> = 76). Study 2 examined the association between year 2 vestibular symptoms and year 5 cognitive performance on Brief Test of Adult Cognition by Telephone (BTACT), controlling for demographics, posttraumatic amnesia duration, and mood symptoms (<i>N</i> = 67). <b>Results:</b> Vestibular symptoms were stable between 1- and 2-year follow-up (88% with no reliable change, 63% with disruptive vestibular symptoms at both time points). Year 2 vestibular symptoms did not predict year 5 BTACT Verbal Memory or Executive Function composites after controlling for covariates. Older age and greater depression symptoms predicted worse executive function. <b>Conclusions</b>: Although vestibular symptoms did not predict cognitive performance, we describe chronic disruptive vestibular symptoms and mood effects on executive functioning for years following mild-to-moderate TBI. Despite study limitations, large effect size differences between TBI-severity groups warrants further exploration to potentially mitigate influence of persistent vestibular symptoms on health outcomes.</p>","PeriodicalId":55250,"journal":{"name":"Clinical Neuropsychologist","volume":" ","pages":"2311-2329"},"PeriodicalIF":2.6,"publicationDate":"2026-11-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"145710346","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":3,"RegionCategory":"心理学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}

{"title":"The association between concussion and autonomic nervous system responses to a cognitive stressor.","authors":"Mary U Simons, Sophia Musacchio, Blair D Johnson, Daniel L Huber, John Leddy, Hershel Raff, Michael A McCrea, Christopher J Roberts, Lindsay D Nelson","doi":"10.1080/13854046.2026.2653683","DOIUrl":"10.1080/13854046.2026.2653683","url":null,"abstract":"<p><p><b>Objective:</b> Investigate the association between concussion and reactivity of cardiovascular, salivary, and electrodermal indicators of autonomic nervous system (ANS) function in the context of cognitive stress. <b>Method:</b> Individuals (<i>N</i> = 37) with concussion were prospectively recruited from a level 1 trauma center emergency department, along with 23 uninjured community controls (UC). Participants were assessed twice (1-week and 1-month post-injury for the concussion group). Electrocardiogram, impedance cardiography, electrodermal skin conductance, and saliva samples were collected before, during, and after a modified Paced Auditory Serial Addition Test. Analyses evaluated the effect of Group, Visit, and task Event on behavioral and physiological variables. Physiological reactivity to the task was correlated with Rivermead Post Concussive Symptom Questionnaire (RPQ) total scores. <b>Results:</b> The UC group showed augmented heart rate (HR) task responses when compared to the concussion group (effect ranging from <i>d</i> = 0.53 to 0.82). Similarly, the UC group had reduced high-frequency HR variability (HF-HRV) task response, indicating parasympathetic (PNS) withdrawal during cognitive engagement (effects ranging from <i>d</i> = 0.25 to 0.80). The concussion group displayed no significant HF-HRV changes during the task. There were no reliable group differences in sympathetic nervous system measures (i.e. PEP, skin conductance, and salivary alpha amylase). Among concussed individuals, greater increase in HR task response at 1 week was associated with lower concussion symptom burden at 1 month. <b>Conclusions:</b> Findings indicate that blunted ANS, and especially PNS, reactivity to cognitive stress is associated with concussion in community adults and that ANS dysfunction may be prognostic of symptom recovery.</p>","PeriodicalId":55250,"journal":{"name":"Clinical Neuropsychologist","volume":" ","pages":"2437-2455"},"PeriodicalIF":2.6,"publicationDate":"2026-11-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://www.ncbi.nlm.nih.gov/pmc/articles/PMC13177294/pdf/","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"147693679","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":3,"RegionCategory":"心理学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"OA","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}

{"title":"Growth hormone deficiency after moderate traumatic brain injury with normal (or high) IGF-1; a case report demonstrating benefit of replacement therapy and clinical pearls for diagnosis.","authors":"Bryson J Hewins, Rachael D C Jones, Megan E Glazer, Kimbra Kenney, Sara M Lippa","doi":"10.1080/13854046.2025.2562095","DOIUrl":"10.1080/13854046.2025.2562095","url":null,"abstract":"<p><p><b>Objective:</b> Traumatic brain injury (TBI) is a common cause of acquired pituitary dysfunction in adults. The prevalence of anterior pituitary dysfunction after TBI varies widely, but growth hormone deficiency (GHD) is reported as the most common, ranging from 5-20% after mild to severe TBI. GHD can be difficult to diagnose: 1) its neuropsychological symptoms are nonspecific and overlap with many chronic TBI symptoms; 2) GHD frequently remits if present in the first year after TBI; 3) screening laboratories are not reliable; and 4) validated, easy to administer, confirmatory stimulation tests are not widely available. A diagnosis of GHD is often delayed until 5 years or more after injury. Nonetheless, replacement therapy is associated with improvement in GHD related symptoms, including cognition. This study aims to present a case of GHD after moderate TBI. <b>Methods:</b> We present a case of GHD after a moderate TBI and discuss the chronic effects of GH replacement therapy on his neuropsychological testing and symptoms, as well as clinical pearls for the diagnosis of GHD in persistently symptomatic patients with remote TBI. <b>Results:</b> This case demonstrates that clinical suspicion should supersede inconclusive screening results and prompt referral for definitive provocative testing. Even when diagnosed late, targeted GH replacement therapy can yield significant improvements in debilitating fatigue, metabolic health, and specific domains of neurocognition. <b>Conclusions:</b> Enhanced clinical awareness and a more proactive approach to endocrine surveillance by providers can prevent years of morbidity, reduce diagnostic delays, and offer patients a greater potential for functional recovery.</p>","PeriodicalId":55250,"journal":{"name":"Clinical Neuropsychologist","volume":" ","pages":"2403-2417"},"PeriodicalIF":2.6,"publicationDate":"2026-11-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"145139464","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":3,"RegionCategory":"心理学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}

{"title":"Commentary: Are postconcussive symptoms and exercise testing appropriate tools to identify autonomic impairment after mild traumatic brain injury?","authors":"Ryan Pelo, Melissa Cortez, Peter C Fino, Leland E Dibble","doi":"10.1080/13854046.2025.2492689","DOIUrl":"10.1080/13854046.2025.2492689","url":null,"abstract":"<p><strong>Objective: </strong>After mild traumatic brain injury (mTBI), autonomic nervous system dysfunction is thought to contribute to exercise intolerance and self-reported postconcussive symptoms (e.g. dizziness, lightheadedness, brain fog) but little has been done to establish this relationship in the literature.</p><p><strong>Methods: </strong>Through recent literature review, it appears that few studies have assessed both autonomic function and exercise intolerance, and for those that have utilized varying methodologies making comparison across studies difficult. Some emerging research has identified potential impairment within the sympathetic nervous system after mTBI but no relationship between exercise tolerance testing and postconcussive symptoms has been established.</p><p><strong>Conclusion: </strong>For neuropsychologists, a physiologic understanding of the scope of autonomic dysfunction and appropriate assessment is vitally important, as autonomic nervous system impairment has the potential to impact sleep and mood, and subsequently cognitive function and mental health. When working in collaboration with other disciplines, referrals to exercise intolerance testing and laboratory based autonomic assessments may occur. However, given the lack of an established evidence, the use of exercise intolerance and/or related symptoms to clinically insinuate dysfunction of the autonomic nervous system function is likely premature, and a more thorough assessment of autonomic function <i>via</i> established batteries is more appropriate.</p>","PeriodicalId":55250,"journal":{"name":"Clinical Neuropsychologist","volume":" ","pages":"2275-2283"},"PeriodicalIF":2.6,"publicationDate":"2026-11-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://www.ncbi.nlm.nih.gov/pmc/articles/PMC12353167/pdf/","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"144042114","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":3,"RegionCategory":"心理学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"OA","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}

{"title":"Plasma biomarkers in chronic mild traumatic brain injury: A review.","authors":"Heather E Dark, Sara M Lippa, Jessica M Gill","doi":"10.1080/13854046.2025.2604091","DOIUrl":"10.1080/13854046.2025.2604091","url":null,"abstract":"<p><p><b>Background/Objective</b>: Understanding the biological cascade which results from a mild traumatic brain injury (mTBI) is essential to determine outcomes over time. Acute plasma levels of brain-related injury markers (BRIMS; ubiquitin carboxy-terminal hydrolase L1 [UCH-L1], total tau[t-tau], neurofilament light chain [NfL], S100 calcium-binding protein β [S100β], glial fibrillary acidic protein [GFAP]) are typically elevated in patients post-mTBI, and relate to injury severity, distinguish neuroimaging findings, and to some degree, relate to functional outcomes. However, acute assessment of biomarkers post-injury is not always feasible, and studies assessing biomarkers in the chronic phase of mTBI have yielded less consistent findings. <b>Method</b>: This review aims to (1) summarize the current literature on the most frequently examined BRIMS in mTBI, (2) review prior research in chronic assessment of BRIMS post-mTBI, (3) discuss the relationship between chronically assessed BRIMS and outcomes post-injury, (4) discuss the relationship between chronically assessed BRIMS and cognition including use by neuropsychologists, (5) discuss limitations to chronic assessment, and (6) discuss recent advances in measurement. <b>Results</b>: There is some evidence that NfL and various inflammatory markers may continue to be elevated and differentiate mTBI from controls during the chronic phase of mTBI; however, findings are inconsistent. During the chronic phase, biomarkers such as UCH-L1, S100β, GFAP, and t-tau appear to be mostly comparable between mTBI and controls. <b>Conclusion</b>: Given the limitations in the chronic assessment of plasma biomarkers after mTBI, researchers should continue efforts in discovery-based methods to identify biomarkers which are more reflective of the pathological processes occurring within the chronic phase of mTBI.</p>","PeriodicalId":55250,"journal":{"name":"Clinical Neuropsychologist","volume":" ","pages":"2361-2382"},"PeriodicalIF":2.6,"publicationDate":"2026-11-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"145879452","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":3,"RegionCategory":"心理学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}