Clinical Neurology最新文献

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[A case of heparin-induced thrombocytopenia (HIT) developing into seizure and respiratory arrest caused by continuous intravenous heparin infusion for multiple cerebral infarcts]. 多发脑梗死患者持续静脉滴注肝素致肝素性血小板减少症发展为癫痫和呼吸骤停1例。
Clinical Neurology Pub Date : 2025-04-25 Epub Date: 2025-03-22 DOI: 10.5692/clinicalneurol.cn-002062
Naohiko Seike, Yasuyuki Matsuno, Taiji Ishii, Takashi Mizobe, Hideo Aihara, Kunimitsu Kawahara, Toshiyuki Uehara
{"title":"[A case of heparin-induced thrombocytopenia (HIT) developing into seizure and respiratory arrest caused by continuous intravenous heparin infusion for multiple cerebral infarcts].","authors":"Naohiko Seike, Yasuyuki Matsuno, Taiji Ishii, Takashi Mizobe, Hideo Aihara, Kunimitsu Kawahara, Toshiyuki Uehara","doi":"10.5692/clinicalneurol.cn-002062","DOIUrl":"10.5692/clinicalneurol.cn-002062","url":null,"abstract":"<p><p>A 67-year-old man who was on medication for hypertension and unstable angina pectoris developed mild left hemiparesis on day X-1. He made an emergency call at 4 am on day X due to persistent symptoms. Brain magnetic resonance imaging performing following admission showed multiple acute cerebral infarcts, resulting in the initiation of treatment by continuous intravenous heparin was started. One hour later, after initial heparinization, he developed generalized seizure and stopped breathing on echocardiography. Emergency tracheal intubation was performed, and contrast-enhanced brain computed tomography showed hypoperfusion of the right middle cerebral artery; consequently, we performed mechanical thrombectomy. During emergency thrombectomy, multiple new thrombi were observed on cerebral angiography. We suspected heparin-induced thrombosis (HIT), and therefore changed heparin treatment to argatroban, following which the thrombi disappeared immediately. The diagnosis of HIT was comfirmed by complete blood count and serological examination, which revealed thrombocytopenia and anti-HIT antibodies, respectively. As was observed in our patient, spontaneous HIT without recent exposure to heparin can occur in rare circumstances; as such, heparin treatment should be administered with caution. As mechanical thrombectomy is usually performed under heparinization, it is important to consider the risk of HIT in cases of new thrombi during thrombectomy.</p>","PeriodicalId":39292,"journal":{"name":"Clinical Neurology","volume":" ","pages":"284-289"},"PeriodicalIF":0.0,"publicationDate":"2025-04-25","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"143701828","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
引用次数: 0
[The Japanese term "kishitsu" as the antonym of the functional neurological disorder]. [日语术语“kishitsu”是功能性神经障碍的反义词]。
Clinical Neurology Pub Date : 2025-04-25 Epub Date: 2025-03-27 DOI: 10.5692/clinicalneurol.cn-002065
Tomoko Komagamine, Masahiro Sonoo
{"title":"[The Japanese term \"kishitsu\" as the antonym of the functional neurological disorder].","authors":"Tomoko Komagamine, Masahiro Sonoo","doi":"10.5692/clinicalneurol.cn-002065","DOIUrl":"10.5692/clinicalneurol.cn-002065","url":null,"abstract":"<p><p>The term organic disease has been used as the antonym of functional neurological disorder (FND), which was once called hysteria. Recently, the term \"structural\" is proposed instead of \"organic\". This is because the English word \"organic\" has two meanings: \"related to the living organism\" and \"related to the organ, a part of the body\". When the first meaning is thought of, the traditional usage of the \"non-organic\" seems strange since even FND takes place within the human body. There has been two Japanese translations for the word \"organic\", \"yuki\" and \"kishitsu\". Ogata Koan used the term \"kishitsu-henseibyou\" as the translation for a German word \"desorganisationen\" in 1857. Kawamoto Komin is known to have established the term \"yuki kagaku\" as the translation of organic chemistry in 1861, at the end of the Edo era. Kuwata Kohei used the term \"kishitsu\" as the translation for \"structural\" in 1872. In 1875, Tsuboi Ishun used \"kishitsu\" as the translation of \"organic\". He also used the term \"yuki\" in another book, and it is noteworthy that he used the two words as the translations of \"organic\" in two different meanings depending on the contexts. Thereafter, the lectures of Erwin von Bälz, Jean-Martin Charcot's \"Tuesday Lessons\", and other foreign texts were translated using the term \"kishitsu\" as the antonym of hysteria. Kawahara Hiroshi and Miura Kinnosuke also used \"kishitsu\", and the term was established as the standard. It is surprising and worth praise that the pioneers in late Edo and Meiji periods invented two Japanese terms translating \"organic\" corresponding to the two meanings of this foreign word. Considering these backgrounds, we would like to argue that the term \"kishitsu (sei)\" continues to be an appropriate term in Japanese used for the antonym of FND, and we need not to change the Japanese term to \"kozo (sei)\" in response to the change of the term in English.</p>","PeriodicalId":39292,"journal":{"name":"Clinical Neurology","volume":" ","pages":"260-266"},"PeriodicalIF":0.0,"publicationDate":"2025-04-25","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"143732212","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
引用次数: 0
[A case of unilateral chorea associated with cortical infarction with transient cortical and striatal hyperperfusion]. [单侧舞蹈病合并皮层梗死伴暂时性皮层和纹状体高灌注1例]。
Clinical Neurology Pub Date : 2025-04-25 Epub Date: 2025-03-22 DOI: 10.5692/clinicalneurol.cn-002037
Yuta Madokoro, Hiroyasu Inoue, Teppei Fujioka, Masayuki Mizuno, Masahiro Oomura, Noriyuki Matsukawa
{"title":"[A case of unilateral chorea associated with cortical infarction with transient cortical and striatal hyperperfusion].","authors":"Yuta Madokoro, Hiroyasu Inoue, Teppei Fujioka, Masayuki Mizuno, Masahiro Oomura, Noriyuki Matsukawa","doi":"10.5692/clinicalneurol.cn-002037","DOIUrl":"10.5692/clinicalneurol.cn-002037","url":null,"abstract":"<p><p>Although disruption of basal ganglia loops due to reduced cortical blood flow has been postulated as a possible mechanism for chorea after cortical infarction, detailed studies have not been conducted. We report a case of cardiogenic cerebral embolism of the right frontal to insular cortex due to occlusion of the right M2 branch, followed by the appearance of chorea in the left upper limb on the next day, recanalization of the occluded vessel, and hyperperfusion detected in the same area of brain via single-photon emission computed tomography (SPECT). Interestingly, the blood flow to the right striatum, which was not infarcted, was increased; however, this was not observed after the disappearance of chorea. We speculated that hyperperfusion after cortical infarction affected the striatum, which resulted in the emergence of chorea. In addition to cortical infarction, increased cortical blood flow due to recanalization should be considered as a possible mechanism for chorea development due to cortical infarction.</p>","PeriodicalId":39292,"journal":{"name":"Clinical Neurology","volume":" ","pages":"290-293"},"PeriodicalIF":0.0,"publicationDate":"2025-04-25","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"143701830","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
引用次数: 0
[Symptomatic narcolepsy type 2 in a patient with AQP4-positive neuromyelitis optica spectrum disorder]. [aqp4阳性视神经脊髓炎谱系障碍患者的2型症状性发作]。
Clinical Neurology Pub Date : 2025-04-25 Epub Date: 2025-03-27 DOI: 10.5692/clinicalneurol.cn-002048
Takashi Yoshimura, Masakazu Wakai, Takashi Kanbayashi, Masahisa Katsuno
{"title":"[Symptomatic narcolepsy type 2 in a patient with AQP4-positive neuromyelitis optica spectrum disorder].","authors":"Takashi Yoshimura, Masakazu Wakai, Takashi Kanbayashi, Masahisa Katsuno","doi":"10.5692/clinicalneurol.cn-002048","DOIUrl":"10.5692/clinicalneurol.cn-002048","url":null,"abstract":"<p><p>A 63-year-old woman, previously diagnosed with multiple sclerosis (MS) and treated with IFNβ-1b, developed asymptomatic lesion expansion surrounding the thalamus and basal ganglia. However, an AQP4 autoantibody ELISA was negative. The patient was subsequently hospitalized due to drowsiness, lethargy, and difficulty in managing housework. During lumbar puncture, the patient lost consciousness. Electroencephalography (EEG) revealed rapid eye movements, and a multiple sleep latency test (MSLT) indicated narcolepsy. Cerebrospinal fluid (CSF) orexin levels were normal, and no cataplexy was observed. The patient was thus diagnosed with narcolepsy type 2. The patient tested positive for AQP4 antibody, and was diagnosed with neuromyelitis optica spectrum disorder (NMOSD). Sleep physiology testing is crucial because some patients with NMOSD do not exhibit hypothalamic lesions or decreased CSF orexin levels.</p>","PeriodicalId":39292,"journal":{"name":"Clinical Neurology","volume":" ","pages":"278-283"},"PeriodicalIF":0.0,"publicationDate":"2025-04-25","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"143732207","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
引用次数: 0
[Rapidly progressive muscle weakness in anti-myelin associated glycoprotein (MAG) neuropathy successfully treated with intravenous immunoglobulin]. 静脉注射免疫球蛋白成功治疗抗髓鞘相关糖蛋白(MAG)神经病变的快速进行性肌肉无力。
Clinical Neurology Pub Date : 2025-04-25 Epub Date: 2025-04-11 DOI: 10.5692/clinicalneurol.cn-002030
Mari Fukuda, Kazutoshi Ikeda, Kentaro Yamakawa, Yohei Takenobu, Akihiko Ozaki
{"title":"[Rapidly progressive muscle weakness in anti-myelin associated glycoprotein (MAG) neuropathy successfully treated with intravenous immunoglobulin].","authors":"Mari Fukuda, Kazutoshi Ikeda, Kentaro Yamakawa, Yohei Takenobu, Akihiko Ozaki","doi":"10.5692/clinicalneurol.cn-002030","DOIUrl":"10.5692/clinicalneurol.cn-002030","url":null,"abstract":"<p><p>Anti-myelin associated glycoprotein (MAG) neuropathy typically progresses slowly, but rare cases exhibit rapid deterioration. We report an 83-year-old man with a two-year history of paresthesia in both feet and recent-onset gait ataxia who developed rapidly progressive muscle weakness in all four limbs over several days. Nerve conduction studies and positive anti-MAG antibodies confirmed the diagnosis of anti-MAG neuropathy. The patient's muscle weakness improved with intravenous immunoglobulin (IVIg) therapy. This case highlights the existence of atypical anti-MAG neuropathy mimicking chronic inflammatory demyelinating polyradiculoneuropathy (CIDP) with a rapid deterioration course and suggests the potential efficacy of IVIg in such presentations.</p>","PeriodicalId":39292,"journal":{"name":"Clinical Neurology","volume":" ","pages":"267-272"},"PeriodicalIF":0.0,"publicationDate":"2025-04-25","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"143732203","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
引用次数: 0
[Is neurofilament light chain useful as a disease progression marker for ATTRv amyloidosis? A literature review]. 神经丝轻链作为ATTRv淀粉样变的疾病进展标志物有用吗?文献综述]。
Clinical Neurology Pub Date : 2025-04-25 Epub Date: 2025-03-22 DOI: 10.5692/clinicalneurol.cn-002063
Yoshiki Sekijima, Mitsuharu Ueda, Kentaro Takahashi, Hiroaki Kitaoka
{"title":"[Is neurofilament light chain useful as a disease progression marker for ATTRv amyloidosis? A literature review].","authors":"Yoshiki Sekijima, Mitsuharu Ueda, Kentaro Takahashi, Hiroaki Kitaoka","doi":"10.5692/clinicalneurol.cn-002063","DOIUrl":"10.5692/clinicalneurol.cn-002063","url":null,"abstract":"<p><p>ATTRv amyloidosis is an autosomal-dominant disorder characterized by mutations in the transthyretin (TTR) gene, systemic deposition of transthyretin amyloid fibrils, and progressive polyneuropathy. Current scoring systems developed for ATTRv amyloidosis to measure the severity of polyneuropathy are not sufficiently sensitive or are difficult to implement in daily practice. Results of phase 3 trials for oligonucleotide therapeutics and real-world evidence have shown that neurofilament light chain (NfL), a key structural component of axons, is a reliable blood biomarker for assessing disease progression and treatment response in patients with ATTRv amyloidosis with polyneuropathy. Because blood NfL levels can be affected by factors such as age, body mass index (BMI), and renal function, its significance in patient monitoring needs to be assessed carefully while considering the clinical characteristics of each patient.</p>","PeriodicalId":39292,"journal":{"name":"Clinical Neurology","volume":" ","pages":"251-259"},"PeriodicalIF":0.0,"publicationDate":"2025-04-25","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"143701831","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
引用次数: 0
[Varicella zoster infection as a risk factor for dementia: a scoping review]. [水痘带状疱疹感染是痴呆的危险因素:一项范围综述]。
Clinical Neurology Pub Date : 2025-03-27 Epub Date: 2025-02-22 DOI: 10.5692/clinicalneurol.cn-002047
Yasuko Mori, Yoya Ono, Takayoshi Shimohata
{"title":"[Varicella zoster infection as a risk factor for dementia: a scoping review].","authors":"Yasuko Mori, Yoya Ono, Takayoshi Shimohata","doi":"10.5692/clinicalneurol.cn-002047","DOIUrl":"10.5692/clinicalneurol.cn-002047","url":null,"abstract":"<p><p>A scoping review analyzing 21 selected publications was conducted to determine \"whether the varicella zoster virus (VZV) is a risk factor for dementia\". One meta-analysis concluded that VZV infection increases dementia risk, while another meta-analysis contradicts this finding. Several reports have shown an increased risk of dementia associated with VZV infections of the eyes and central nervous system. Additionally, a third meta-analysis reported that VZV vaccination reduces dementia. Therefore, VZV infection may be a risk factor for dementia. However, most of the reviewed articles were retrospective cohort studies, which limits the strength of conclusions that can be drawn. To provide more robust evidence, prospective cohort studies and clinical trials are needed to evaluate the impact of VZV itself, as well as the effectiveness of vaccines and antiviral therapies.</p>","PeriodicalId":39292,"journal":{"name":"Clinical Neurology","volume":" ","pages":"191-196"},"PeriodicalIF":0.0,"publicationDate":"2025-03-27","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"143494039","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
引用次数: 0
[A case of possible multifocal motor neuropathy with suspected posterior interosseous nerve palsy]. [疑似多局灶性运动神经病伴后骨间神经麻痹1例]。
Clinical Neurology Pub Date : 2025-03-27 Epub Date: 2025-02-22 DOI: 10.5692/clinicalneurol.cn-002053
Koichi Tanaka, Akihiro Kitamura, Ryutaro Nakamura, Isamu Yamakawa, Tomoya Terashima, Makoto Urushitani
{"title":"[A case of possible multifocal motor neuropathy with suspected posterior interosseous nerve palsy].","authors":"Koichi Tanaka, Akihiro Kitamura, Ryutaro Nakamura, Isamu Yamakawa, Tomoya Terashima, Makoto Urushitani","doi":"10.5692/clinicalneurol.cn-002053","DOIUrl":"10.5692/clinicalneurol.cn-002053","url":null,"abstract":"<p><p>We present the case of a 33-year-old man with progressive weakness in the left wrist and finger extensors. Initially, posterior interosseous nerve (PIN) palsy was suspected. However, nerve conduction studies detected conduction block and ultrasonography demonstrated swelling between the elbow and spiral groove in the left radial nerve. Anti-GM1 IgM antibodies were positive, leading to a diagnosis of possible multifocal motor neuropathy (MMN). Although MMN usually represents multifocal pure motor neuropathies, the involvement of a single nerve allows for diagnosis of possible MMN. PIN palsy of indeterminate etiology should prompt consideration, underscoring the utility of ultrasonography in the diagnostic process.</p>","PeriodicalId":39292,"journal":{"name":"Clinical Neurology","volume":" ","pages":"230-235"},"PeriodicalIF":0.0,"publicationDate":"2025-03-27","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"143494028","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
引用次数: 0
Undiagnosed headaches in the emergency department: clinical characteristics and outcomes. 急诊科未确诊的头痛:临床特征和结果
Clinical Neurology Pub Date : 2025-03-27 Epub Date: 2025-02-22 DOI: 10.5692/clinicalneurol.cn-002052
Ryo Sasaki, Yoshiaki Takahashi, Mizuki Morimoto, Kazuhiro Sasaki, Nobutoshi Morimoto
{"title":"Undiagnosed headaches in the emergency department: clinical characteristics and outcomes.","authors":"Ryo Sasaki, Yoshiaki Takahashi, Mizuki Morimoto, Kazuhiro Sasaki, Nobutoshi Morimoto","doi":"10.5692/clinicalneurol.cn-002052","DOIUrl":"10.5692/clinicalneurol.cn-002052","url":null,"abstract":"<p><p>Headaches are a common complaint in the emergency department (ED). Understanding the characteristics and outcomes of headaches, especially in undiagnosed patients, is important for improving headache care in the ED. We conducted a retrospective study of 171 headache patients at the ED of Kagawa Prefectural Central Hospital, with a follow-up for the primary and undiagnosed headache groups via telephone to assess long-term outcomes. Primary, secondary, and undiagnosed headaches accounted for 15.2%, 58.4%, and 26.3% of cases, respectively. All life-threatening secondary headaches were successfully excluded with imaging tests performed on 73.7% of cases, despite the low rate of treatment in the ED (18.1%). Among the undiagnosed headache cases, the recurrence of severe headaches was low (11.9%), though awareness of chronic headaches was high (47.6%). Emergency physicians should be aware of the possibility that patients at an ED presenting an undiagnosed headache may include chronic headache patients. Seamless collaboration between the ED and headache specialists is needed to manage undiagnosed headaches in the ED.</p>","PeriodicalId":39292,"journal":{"name":"Clinical Neurology","volume":" ","pages":"197-202"},"PeriodicalIF":0.0,"publicationDate":"2025-03-27","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"143494042","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
引用次数: 0
[A case of anti-synthetase syndrome: negative ELISA/immunoblot, but positive RNA immunoprecipitation with multiple antibodies]. 【抗合成酶综合征1例:ELISA/免疫印迹阴性,RNA免疫沉淀阳性,多抗体】。
Clinical Neurology Pub Date : 2025-03-27 Epub Date: 2025-02-27 DOI: 10.5692/clinicalneurol.cn-002009
Saya Ikeda, Takahiro Shimizu, Tadashi Adachi, Shigeakai Suzuki, Ichizo Nishino, Ritsuko Hanajima
{"title":"[A case of anti-synthetase syndrome: negative ELISA/immunoblot, but positive RNA immunoprecipitation with multiple antibodies].","authors":"Saya Ikeda, Takahiro Shimizu, Tadashi Adachi, Shigeakai Suzuki, Ichizo Nishino, Ritsuko Hanajima","doi":"10.5692/clinicalneurol.cn-002009","DOIUrl":"10.5692/clinicalneurol.cn-002009","url":null,"abstract":"<p><p>A 68-year-old woman presented with a one-month history of polyarthritis and fever, followed by myalgia and muscle weakness involving the proximal limbs, neck, trunk, and distal upper extremities. Myositis was suspected based on an elevated serum CK level. Chest CT revealed interstitial pneumonia. Commercial laboratory tests, including ELISA and immunoblot, were positive for anti-Ro-52 antibodies but negative for anti-aminoacyl transfer RNA synthetase (ARS) antibodies. However, muscle pathology revealed perifascicular necrosis and perimysial pathology, strongly suggesting anti-synthetase syndrome (ASS). Further investigation using RNA immunoprecipitation (RIP) assay identified positive anti-EJ antibodies, leading to the diagnosis of ASS. In addition, anti-Ku and anti-U1 RNP antibodies were co-positive. These findings highlight that, even when ELISA and immunoblot were negative for specific antibodies, RIP assay is necessary when ASS is suspected based on myopathologic findings.</p>","PeriodicalId":39292,"journal":{"name":"Clinical Neurology","volume":" ","pages":"211-217"},"PeriodicalIF":0.0,"publicationDate":"2025-03-27","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"143516932","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
引用次数: 0
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