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Statement of Retraction: Polyphyllin I, a lethal partner of Palbociclib, suppresses non-small cell lung cancer through activation of p21/CDK2/Rb pathway in vitro and in vivo. 撤回声明：Palbociclib的致命伴侣Polyphyllin I在体外和体内通过激活p21/CDK2/Rb通路抑制非小细胞肺癌。
IF 3.4 3区 生物学
Cell Cycle Pub Date : 2024-07-04 DOI: 10.1080/15384101.2024.2370721
{"title":"Statement of Retraction: Polyphyllin I, a lethal partner of Palbociclib, suppresses non-small cell lung cancer through activation of p21/CDK2/Rb pathway in vitro and in vivo.","authors":"","doi":"10.1080/15384101.2024.2370721","DOIUrl":"https://doi.org/10.1080/15384101.2024.2370721","url":null,"abstract":"","PeriodicalId":9686,"journal":{"name":"Cell Cycle","volume":null,"pages":null},"PeriodicalIF":3.4,"publicationDate":"2024-07-04","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"141533703","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":3,"RegionCategory":"生物学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
引用次数: 0
Statement of Retraction: Hsa-miR-186-3p suppresses colon cancer progression by inhibiting KRT18/MAPK signaling pathway. 撤回声明：Hsa-miR-186-3p通过抑制KRT18/MAPK信号通路抑制结肠癌的进展。
IF 3.4 3区 生物学
Cell Cycle Pub Date : 2024-07-04 DOI: 10.1080/15384101.2024.2370722
{"title":"Statement of Retraction: Hsa-miR-186-3p suppresses colon cancer progression by inhibiting KRT18/MAPK signaling pathway.","authors":"","doi":"10.1080/15384101.2024.2370722","DOIUrl":"https://doi.org/10.1080/15384101.2024.2370722","url":null,"abstract":"","PeriodicalId":9686,"journal":{"name":"Cell Cycle","volume":null,"pages":null},"PeriodicalIF":3.4,"publicationDate":"2024-07-04","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"141533696","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":3,"RegionCategory":"生物学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
引用次数: 0
Statement of Retraction: microRNA-192-5p is involved in nerve repair in rats with peripheral nerve injury by regulating XIAP. 撤回声明：microRNA-192-5p 通过调节 XIAP 参与周围神经损伤大鼠的神经修复。
IF 3.4 3区 生物学
Cell Cycle Pub Date : 2024-07-04 DOI: 10.1080/15384101.2024.2370711
{"title":"Statement of Retraction: microRNA-192-5p is involved in nerve repair in rats with peripheral nerve injury by regulating XIAP.","authors":"","doi":"10.1080/15384101.2024.2370711","DOIUrl":"https://doi.org/10.1080/15384101.2024.2370711","url":null,"abstract":"","PeriodicalId":9686,"journal":{"name":"Cell Cycle","volume":null,"pages":null},"PeriodicalIF":3.4,"publicationDate":"2024-07-04","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"141497163","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":3,"RegionCategory":"生物学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
引用次数: 0
Statement of Retraction: Knockdown of RNF183 suppressed proliferation of lung adenocarcinoma cells via inactivating the STAT3 signaling pathway. 撤回声明：敲除 RNF183 可通过使 STAT3 信号通路失活来抑制肺腺癌细胞的增殖。
IF 3.4 3区 生物学
Cell Cycle Pub Date : 2024-07-04 DOI: 10.1080/15384101.2024.2370723
{"title":"Statement of Retraction: Knockdown of RNF183 suppressed proliferation of lung adenocarcinoma cells via inactivating the STAT3 signaling pathway.","authors":"","doi":"10.1080/15384101.2024.2370723","DOIUrl":"https://doi.org/10.1080/15384101.2024.2370723","url":null,"abstract":"","PeriodicalId":9686,"journal":{"name":"Cell Cycle","volume":null,"pages":null},"PeriodicalIF":3.4,"publicationDate":"2024-07-04","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"141533698","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":3,"RegionCategory":"生物学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
引用次数: 0
Statement of Retraction: MicroRNA-203 restrains epithelial-mesenchymal transition, invasion and migration of papillary thyroid cancer by downregulating AKT3. 撤回声明：MicroRNA-203通过下调AKT3抑制甲状腺乳头状癌的上皮-间质转化、侵袭和迁移
IF 3.4 3区 生物学
Cell Cycle Pub Date : 2024-07-04 DOI: 10.1080/15384101.2024.2370714
{"title":"Statement of Retraction: MicroRNA-203 restrains epithelial-mesenchymal transition, invasion and migration of papillary thyroid cancer by downregulating AKT3.","authors":"","doi":"10.1080/15384101.2024.2370714","DOIUrl":"https://doi.org/10.1080/15384101.2024.2370714","url":null,"abstract":"","PeriodicalId":9686,"journal":{"name":"Cell Cycle","volume":null,"pages":null},"PeriodicalIF":3.4,"publicationDate":"2024-07-04","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"141533700","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":3,"RegionCategory":"生物学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
引用次数: 0
Ginsenoside Rg3 attenuates neuroinflammation and hippocampal neuronal damage after traumatic brain injury in mice by inactivating the NF-kB pathway via SIRT1 activation 人参皂苷 Rg3 通过激活 SIRT1 使 NF-kB 通路失活，从而减轻小鼠脑外伤后的神经炎症和海马神经元损伤
IF 4.3 3区 生物学
Cell Cycle Pub Date : 2024-05-25 DOI: 10.1080/15384101.2024.2355008
Xi Liu, Jia Gu, Cheng Wang, Min Peng, Jilin Zhou, Xiyun Fei, Zhijun Zhong, Bo Li
{"title":"Ginsenoside Rg3 attenuates neuroinflammation and hippocampal neuronal damage after traumatic brain injury in mice by inactivating the NF-kB pathway via SIRT1 activation","authors":"Xi Liu, Jia Gu, Cheng Wang, Min Peng, Jilin Zhou, Xiyun Fei, Zhijun Zhong, Bo Li","doi":"10.1080/15384101.2024.2355008","DOIUrl":"https://doi.org/10.1080/15384101.2024.2355008","url":null,"abstract":"This investigation examined the potential of ginsenoside Rg3 in addressing traumatic brain injury (TBI). A TBI mouse model underwent treatment with ginsenoside Rg3 and nicotinamide (NAM). Neurologi...","PeriodicalId":9686,"journal":{"name":"Cell Cycle","volume":null,"pages":null},"PeriodicalIF":4.3,"publicationDate":"2024-05-25","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"141148042","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":3,"RegionCategory":"生物学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
引用次数: 0
Cell death in atherosclerosis 动脉粥样硬化中的细胞死亡
IF 4.3 3区 生物学
Cell Cycle Pub Date : 2024-04-27 DOI: 10.1080/15384101.2024.2344943
Dan Ni, Cai Lei, Minqi Liu, Jinfu Peng, Guanghui Yi, Zhongcheng Mo
{"title":"Cell death in atherosclerosis","authors":"Dan Ni, Cai Lei, Minqi Liu, Jinfu Peng, Guanghui Yi, Zhongcheng Mo","doi":"10.1080/15384101.2024.2344943","DOIUrl":"https://doi.org/10.1080/15384101.2024.2344943","url":null,"abstract":"A complex and evolutionary process that involves the buildup of lipids in the arterial wall and the invasion of inflammatory cells results in atherosclerosis. Cell death is a fundamental biological...","PeriodicalId":9686,"journal":{"name":"Cell Cycle","volume":null,"pages":null},"PeriodicalIF":4.3,"publicationDate":"2024-04-27","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"140811319","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":3,"RegionCategory":"生物学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
引用次数: 0
Osteopontin-driven partial epithelial-mesenchymal transition governs the development of middle ear cholesteatoma. 骨蛋白驱动的部分上皮-间充质转化控制着中耳胆脂瘤的发展。
IF 4.3 3区 生物学
Cell Cycle Pub Date : 2024-04-25 DOI: 10.1080/15384101.2024.2345481
Lingling Zeng, Li Xie, Jin Hu, Chao He, Aiguo Liu, Xiang Lu, Wen Zhou
{"title":"Osteopontin-driven partial epithelial-mesenchymal transition governs the development of middle ear cholesteatoma.","authors":"Lingling Zeng, Li Xie, Jin Hu, Chao He, Aiguo Liu, Xiang Lu, Wen Zhou","doi":"10.1080/15384101.2024.2345481","DOIUrl":"https://doi.org/10.1080/15384101.2024.2345481","url":null,"abstract":"Cholesteatoma is a common disease of the middle ear. Currently, surgical removal is the only treatment option and patients face a high risk of relapse. The molecular basis of cholesteatoma remains largely unknown. Here, we show that Osteopontin (OPN), a predominantly secreted protein, plays a crucial role in the development of middle ear cholesteatoma. Global transcriptome analysis revealed the loss of epithelial features and an enhanced immune response in human cholesteatoma tissues. Quantitative RT-PCR and immunohistochemical staining of middle ear cholesteatoma validated the reduced expression of epithelial markers, as well as the elevated expression of mesenchymal markers including Vimentin and Fibronectin, but not N-Cadherin, α-smooth muscle actin (α-SMA) or ferroptosis suppressor protein 1 (FSP1), indicating a partial epithelial-mesenchymal transition (EMT) state. Besides, the expression of OPN was significantly elevated in human cholesteatoma tissues. Treatment with OPN promoted cell proliferation, survival and migration and led to a partial EMT in immortalized human keratinocyte cells. Importantly, blockade of OPN signaling could remarkably improve the cholesteatoma-like symptoms in SD rats. Our mechanistic study demonstrated that the AKT-zinc finger E-box binding homeobox 2 (ZEB2) axis mediated the effects of OPN. Overall, these findings suggest that targeting the OPN signaling represents a promising strategy for the treatment of middle ear cholesteatoma.","PeriodicalId":9686,"journal":{"name":"Cell Cycle","volume":null,"pages":null},"PeriodicalIF":4.3,"publicationDate":"2024-04-25","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"140654193","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":3,"RegionCategory":"生物学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
引用次数: 0
Genetic predisposition of BMP7 polymorphisms to lumbar disk herniation in the Chinese Han population. 中国汉族人群腰椎间盘突出症的 BMP7 多态性遗传易感性。
IF 4.3 3区 生物学
Cell Cycle Pub Date : 2024-04-23 DOI: 10.1080/15384101.2024.2342703
Kai Sun, Ruiyu Liu
{"title":"Genetic predisposition of BMP7 polymorphisms to lumbar disk herniation in the Chinese Han population.","authors":"Kai Sun, Ruiyu Liu","doi":"10.1080/15384101.2024.2342703","DOIUrl":"https://doi.org/10.1080/15384101.2024.2342703","url":null,"abstract":"Bone morphogenetic protein 7 (BMP7) can induce skeletal formation, promote the differentiation of chondrocytes and osteoblasts, and ameliorate intervertebral disc degeneration. The study was designed to evaluate the relationship of BMP7 variants to LDH risk in the Chinese Han population. BMP7 variants were genotyped with the Agena MassARRAY system among 690 LDH patients and 690 healthy controls. The odds ratio (OR) and 95% confidence interval (CI) were calculated by logistic regression. Multi-factor dimension reduction (MDR) (version 3.0.2) software was used to evaluate the effect of BMP7 variant-variant interaction on the susceptibility to LDH. Here, the risk-reducing association between rs230189 and LDH occurrence was found (p = 0.005, OR = 0.79). Specially, rs230189 was associated with decreased LDH risk in females (p = 0.001, OR = 0.60), elder group (p = 0.025, OR = 0.76), subjects with BMI < 24 kg/m2 (p = 0.027, OR = 0.48), nonsmokers (p = 0.001, OR = 0.66), and nondrinkers (p = 0.011, OR = 0.72). Moreover, rs1321862 might be the risk factor for LDH susceptibility among the participants with BMI < 24 kg/m2 (p = 0.024, OR = 1.84). MDR results displayed that rs230189 was the greatest attribution factor on LDH risk in the single-locus model, with an information gain of 0.44%. The present study demonstrated that BMP7 rs230189 g.55771443A>C may play a protective role against LDH risk. Our findings may help to understand the potential mechanism of BMP7 in LDH susceptibility.","PeriodicalId":9686,"journal":{"name":"Cell Cycle","volume":null,"pages":null},"PeriodicalIF":4.3,"publicationDate":"2024-04-23","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"140671322","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":3,"RegionCategory":"生物学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
引用次数: 0
Discovery of common molecular signatures and drug repurposing for COVID-19/Asthma comorbidity: ACE2 and multi-partite networks 发现COVID-19/哮喘合并症的共同分子特征和药物再利用：ACE2 和多方网络
IF 4.3 3区 生物学
Cell Cycle Pub Date : 2024-04-19 DOI: 10.1080/15384101.2024.2340859
Jiajun Xu, Raghad Abdulsalam Khaleel, Haider Kamil Zaidan, Ahmed Faisal Mutee, Khaled Fahmi Fawy, Anita Gehlot, Alaa Hashim Abbas, José Luis Arias Gonzáles, Ali H Amin, Maribel Carmen Ruiz-Balvin, Shima Imannezhad, Abolfazl Bahrami, Reza Akhavan-Sigari
{"title":"Discovery of common molecular signatures and drug repurposing for COVID-19/Asthma comorbidity: ACE2 and multi-partite networks","authors":"Jiajun Xu, Raghad Abdulsalam Khaleel, Haider Kamil Zaidan, Ahmed Faisal Mutee, Khaled Fahmi Fawy, Anita Gehlot, Alaa Hashim Abbas, José Luis Arias Gonzáles, Ali H Amin, Maribel Carmen Ruiz-Balvin, Shima Imannezhad, Abolfazl Bahrami, Reza Akhavan-Sigari","doi":"10.1080/15384101.2024.2340859","DOIUrl":"https://doi.org/10.1080/15384101.2024.2340859","url":null,"abstract":"Angiotensin-converting enzyme 2 (ACE2) is identified as the functional receptor for severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2), the causative agent of the ongoing global coronavir...","PeriodicalId":9686,"journal":{"name":"Cell Cycle","volume":null,"pages":null},"PeriodicalIF":4.3,"publicationDate":"2024-04-19","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"140626958","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":3,"RegionCategory":"生物学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
引用次数: 0
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