{"title":"On the long-term development of shinkeishitsu-neurotics treated by Morita therapy. A statistical quantitative analysis.","authors":"T Suzuki, H Kataoka, O Karasawa","doi":"10.1159/000283934","DOIUrl":"https://doi.org/10.1159/000283934","url":null,"abstract":"We considered the long-term development of shinkeishitsu-type neurotics (hypochondriacal neurosis: 154 cases; anxiety neurosis: 122 cases; obsessive neurosis: 436 cases; depressive neurosis: 123 cases) treated by Morita therapy by using the Life Table analysis. We obtained the following results: (i) at least 90% of the patients improved greatly within at most 8 years after admission; (ii) the mean time required for the patients to improve greatly are 38.0 months in hypochondriacal neurosis, 34.8 months in anxiety neurosis, 50.0 months in obsessive neurosis, and 47.4 months in depressive neurosis.","PeriodicalId":76382,"journal":{"name":"Psychiatria clinica","volume":"15 3","pages":"145-52"},"PeriodicalIF":0.0,"publicationDate":"1982-01-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://sci-hub-pdf.com/10.1159/000283934","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"18148128","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
{"title":"Neurotic symptoms and major depressive illness.","authors":"G A Fava","doi":"10.1159/000283942","DOIUrl":"https://doi.org/10.1159/000283942","url":null,"abstract":"<p><p>Neurotic symptoms as measured by self-rating scales were studied in 40 patients suffering from a major depressive disorder and 40 control subjects matched for sociodemographic variables. The depressive patients reported significantly more symptoms (global neuroticism, anxiety, somatization, and hostility) than controls. The more severe were the depressive self-rated symptoms, the more intense was the neurotic symptomatology. A principal component analysis was carried out in depressives and controls. In both groups a first general factor reflecting severity was obtained.</p>","PeriodicalId":76382,"journal":{"name":"Psychiatria clinica","volume":"15 4","pages":"231-8"},"PeriodicalIF":0.0,"publicationDate":"1982-01-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://sci-hub-pdf.com/10.1159/000283942","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"18191543","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
P Tosca, L Fenoglio, F Zerbi, A Romani, G Bezzi, E Ferrari, C Canepari
{"title":"Neuroendocrinological aspects of depression and symptomatological picture.","authors":"P Tosca, L Fenoglio, F Zerbi, A Romani, G Bezzi, E Ferrari, C Canepari","doi":"10.1159/000283935","DOIUrl":"https://doi.org/10.1159/000283935","url":null,"abstract":"<p><p>Cortisol and prolactin circadian rhythms were determined in a group of depressed hospitalized women who were submitted the next day to the dexamethasone suppression test. The depressed were classified as endogenous and non-endogenous according to symptomatological criteria. Some significant differences were found between the two groups when compared to control subjects in cortisol as well as prolactin biorhythms, and the cortisol suppression percentages were significantly lower in the endogenous than in the non-endogenous. This difference is abolished by benzodiazepine treatment.</p>","PeriodicalId":76382,"journal":{"name":"Psychiatria clinica","volume":"15 3","pages":"153-9"},"PeriodicalIF":0.0,"publicationDate":"1982-01-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://sci-hub-pdf.com/10.1159/000283935","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"18148129","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
{"title":"[Under what conditions can further course research still be expected to produce new knowledge on endogenous psychoses?].","authors":"P Berner","doi":"","DOIUrl":"","url":null,"abstract":"<p><p>Exhaustive follow-up studies of schizophrenia from Zürich, Bonn and Lausanne have produced strikingly similar results, despite considerable methodological differences. These 'classical' modes of differentiating the endogenous psychoses are critically analyzed. In contrast, the application of the operational research criteria recently developed by American authors results in a narrowing of the concept of schizophrenia and a proportional broadening of the cyclothymic disorders. Use of the Viennese research criteria, the definition of an 'axial syndrome' of schizophrenia, has the identical nosological effect. While the American diagnosis is phenomenological and essentially 'atheoretical', the Viennese view is based on the hypothesis of a core disorder. Further comparative polydiagnostic studies are called for in order to determine the relative value of these various research instruments.</p>","PeriodicalId":76382,"journal":{"name":"Psychiatria clinica","volume":"15 3","pages":"97-123"},"PeriodicalIF":0.0,"publicationDate":"1982-01-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"18151405","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
{"title":"Genetic markers for schizophrenic subgroups.","authors":"V Lange","doi":"10.1159/000283933","DOIUrl":"https://doi.org/10.1159/000283933","url":null,"abstract":"<p><p>By the study of hereditary serum protein markers in psychotic patients and normal controls, a surplus of Gc 1-1 (p less than 0.01) and transferrin B variants (p less than 0.0027) has been established in schizophrenias. Affective psychoses are characterized by an excess of the haptoglobin (Hp) serum type 2-2 (p less than 0.001). These general statements have to be modified in regard to the clinical and psychopathological subdivision beyond the traditional classification into two major groups of endogenous mental disease. Using Leonhard's criteria, the prevalence of Gc 1-1 is restricted to the systematic schizophrenias reaching its highest value in hebephrenias, which are followed by paraphrenic and catatonic forms in this trait. In contrast to this, periodical catatonia and affective paraphrenia, classified as subgroups of the unsystematic schizophrenias, have Gc 1-1 frequencies like healthy controls. On the other hand, the Hp 2-2 value is not increased in the systematic schizophrenias, but it displays a relative overplus in the unsystematic forms. Concerning the Hp 2-2 and Gc 1-1 frequencies a certain similarity can be observed between affective paraphrenia and the paranoid psychoses with late onset, it they are characterized by a cyclic axis syndrome as described by the Vienna school. The cycloid psychoses are marked by an extreme surplus of Hp 2-2 (p less than 0.001) and an overweight of Gc 1-1 (p less than 0.05). Probably the Gc and Hp alleles play a role as risk factors or accidental effectors in the multifactorial genetic systems responsible for the biological background of psychoses. For both serum systems a selective interaction is discussed considering the vitamin D transport by the Gc proteins with the relation to neuronal consolidation and the possible influence of Hp 2-2 on transport and receptor functions.</p>","PeriodicalId":76382,"journal":{"name":"Psychiatria clinica","volume":"15 3","pages":"133-44"},"PeriodicalIF":0.0,"publicationDate":"1982-01-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://sci-hub-pdf.com/10.1159/000283933","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"17247505","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
{"title":"Role reversal: when doctors become patients.","authors":"E L Edelstein, L Baider","doi":"10.1159/000283938","DOIUrl":"https://doi.org/10.1159/000283938","url":null,"abstract":"<p><p>We intend to present a personal view of the social behavior of doctors in the role of patients. The hypotheses presented here have been formed through personal experience and the accumulation of reports from and about medical colleagues who have been patients. Our purpose is twofold: (a) to describe the mechanisms of doctors becoming patients and (b) to show the difficulties of this role reversal. We tried to show the complex interactions of role changes in both partners, doctor and doctor-patient, because of differences in role identity, in rigidity or flexibility of coping behavior and the myth and beliefs surrounding the very concept of the medical profession.</p>","PeriodicalId":76382,"journal":{"name":"Psychiatria clinica","volume":"15 4","pages":"177-83"},"PeriodicalIF":0.0,"publicationDate":"1982-01-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://sci-hub-pdf.com/10.1159/000283938","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"18191540","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
{"title":"[Anorexia nervosa and schizophrenia in a pair of monozygotic twins].","authors":"R Steinberg","doi":"","DOIUrl":"","url":null,"abstract":"<p><p>The case report describes two 20-year-old monozygotic twin sisters who have been suffering from the symptoms of primary anorexia nervosa for the last 10 years. Usually the disease manifested itself in such a manner that the twin who was currently in the mother's favour showed no signs of anorexia, whereas the other was severely afflicted. Occasionally, however, both girls suffered from the same anorectic symptoms at the same time. For the moment the condition of one of the twins seems to have become stable. Up to now the other twin has suffered three times from schizophrenic episodes which seem to be closely connected with the problems of anorexia. The most recent severe catatonic state, in particular, appeared to be a gradual transition from anorectic behaviour to psychotic experience so that the close relationship between primary anorexia and schizophrenic disturbances, as discussed in literature, becomes increasingly evident.</p>","PeriodicalId":76382,"journal":{"name":"Psychiatria clinica","volume":"15 4","pages":"184-205"},"PeriodicalIF":0.0,"publicationDate":"1982-01-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"17941157","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
{"title":"Familially accumulated schizophrenia (folie à cinq) in association with Stein-Leventhal syndrome.","authors":"B Pethö, I Karczag, A Czeizel","doi":"10.1159/000283940","DOIUrl":"https://doi.org/10.1159/000283940","url":null,"abstract":"<p><p>The authors describe the psychosis of both parents and their three daughters. The psychosis of the father and three daughters corresponds to the paranoid schizophrenias (more specifically, the affective paraphrenias), while the mother's psychosis is largely the result of induction. All 3 daughters suffer from surgically confirmed Stein-Leventhal syndrome. Such a close association of the two diseases cannot be by chance.</p>","PeriodicalId":76382,"journal":{"name":"Psychiatria clinica","volume":"15 4","pages":"206-11"},"PeriodicalIF":0.0,"publicationDate":"1982-01-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://sci-hub-pdf.com/10.1159/000283940","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"18191541","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
{"title":"[Diet triggered depression as well as its treatment: a behaviorally oriented group therapy concept].","authors":"G Sachs, D Nutzinger, H G Zapotoczky","doi":"","DOIUrl":"","url":null,"abstract":"<p><p>Preliminary results of a behavior-oriented therapeutic concept for adipose females, who can be called a risk group because of their psychic instability, are presented. 6 adipose females, who at the beginning of the therapy showed a partial endomorphic cyclothymic axial syndrome according to Berner, participated in group therapy. Half of the patients were additionally supported with antidepressants. The results show that those patients with additional medicamental treatment did profit far more from the therapeutic supply than did the others in all examined parameters: they were better motivated to stay with the group, became socially more competent, and lost more weight.</p>","PeriodicalId":76382,"journal":{"name":"Psychiatria clinica","volume":"15 3","pages":"160-72"},"PeriodicalIF":0.0,"publicationDate":"1982-01-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"18151404","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
{"title":"[Brain organic melancholia].","authors":"A Marneros","doi":"","DOIUrl":"","url":null,"abstract":"<p><p>We describe the syndrome of 'organic melancholia', i.e. the form of melancholia which in addition to the endogenous symptoms also shows organic psychopathological symptoms and is connected with brain damage. We found considerable differences between organic and cryptogenic melancholia: the development of organic melancholia has a regular course which can be distinguished into four stages: (1) the asthenic-hypochondriac, (2) the purely depressive, (3) the organically depressive and (4) the purely organic stage. The outcome of organic melancholia - a severe organic psychodeficiency, often combined with neurological symptoms, and the irreversible destruction of the professional life of the patient - is one of the more important differences distinguishing the organic and the cryptogenous type. Our findings are based on long-term observations of 30 patients.</p>","PeriodicalId":76382,"journal":{"name":"Psychiatria clinica","volume":"15 4","pages":"212-30"},"PeriodicalIF":0.0,"publicationDate":"1982-01-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"18191542","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}