Journal of Photochemistry and Photobiology最新文献

{"title":"Investigating mass transport and recombination as a function of structural variation in dye-sensitized solar cells employing indole fused heterocyclic organic sensitizers and cobalt electrolytes","authors":"Jayadev V ,&nbsp;Sourava C. Pradhan ,&nbsp;P.R. Nitha ,&nbsp;Jubi John ,&nbsp;K.N. Narayanan Unni ,&nbsp;Suraj Soman","doi":"10.1016/j.jpap.2023.100223","DOIUrl":"10.1016/j.jpap.2023.100223","url":null,"abstract":"<div><p>Alternate cobalt redox mediator based dye-sensitized solar cells (DSCs) are getting widespread attention taking advantage of their one-electron transfer mechanism compared to the conventional iodide/triiodide electrolyte. In the present study, we used indole fused heterocyclic organic sensitizers having indolo[3,2-<em>b</em>]indole as donor with three different π-spacers [(benzene (IID-1), thiophene (IID-2) and furan (IID-3)] along with cobalt bipyridine derivatives as redox mediators having different peripheral substituents {[Co(bpy)₃]^3+/2+, [Co(Me₂bpy)₃]^3+/2+, and [Co(t-Bu₂bpy)₃]^3+/2+}. A detailed investigation was carried out to understand the fundamental charge transfer processes and loss mechanism happening at the various interfaces as a function of structural variations in the present dye-electrolyte combinations. Among the investigated systems, higher performance was obtained for the association of furan substituted dye (IID-3) with [Co(t-Bu₂bpy)₃]^3+/2+ electrolyte. The importance of choosing the right combination of sensitizer and electrolyte is critical to realize higher performance in dye-sensitized solar cells particularly while employing organic dyes and alternate metal complex redox electrolytes which was systematically investigated in the present manuscript.</p></div>","PeriodicalId":375,"journal":{"name":"Journal of Photochemistry and Photobiology","volume":"19 ","pages":"Article 100223"},"PeriodicalIF":3.261,"publicationDate":"2023-12-07","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://www.sciencedirect.com/science/article/pii/S2666469023000647/pdfft?md5=63ec329b37502d8462933cd154b2ed96&pid=1-s2.0-S2666469023000647-main.pdf","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"138546730","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"OA","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}

{"title":"Rooting out ultraweak photon emission a-mung bean sprouts","authors":"Alasdair M. Mackenzie ,&nbsp;Holly E. Smith ,&nbsp;Rhys R. Mould ,&nbsp;Jimmy D. Bell ,&nbsp;Alistair V.W. Nunn ,&nbsp;Stanley W. Botchway","doi":"10.1016/j.jpap.2023.100224","DOIUrl":"10.1016/j.jpap.2023.100224","url":null,"abstract":"<div><p>It is well known that life has evolved to use and generate light, for instance, photosynthesis, vision and bioluminescence. What is less well known is that during normal metabolism, it can generate 1–100 photons s⁻¹ cm^–2 known as ultra-weak photon emission (UPE), biophoton emission or biological autoluminescence. The highest generation of these metabolic photons seem to occur during oxidative stress due to the generation and decay of reactive oxygen species (ROS), and their interaction with other components of the cell. To study this further, we have configured a sensitive detection system to study photon emission in germinating mung beans.</p><p>Here we investigated growing mung beans over 7 days at a constant temperature of 21 ± 1 °C in a light tight box, using dual top and bottom opposing photomultiplier tubes. Over this time period we showed that in total, mung beans grown from seeds generated an average of 5 ± 1 counts s⁻¹ above background. As the new bean stems grew, they showed a gradual linear increase in emission of up to 30 ± 1 counts s⁻¹, in agreement with previous literature. In addition to this “steady-state” emission we also observe delayed luminescence and drought-stress response emission previously observed in other species. Finally, we also observe episodic increased emission events of between 2 and 15 counts s⁻¹ for durations of around 3 h detected underneath the sample, and assign these to the growing of secondary roots.</p><p>We then induce secondary root formation using aqueous solutions of growth hormones hydrogen peroxide (H₂O₂, 167 µM) or 3-indole acetic acid (IAA, 0.5 µM) for watering. Both hormones show prolonged increase in emission above steady-state, over days 3–5 with at least 3 times the number of secondary roots formed compared with water alone. We also observed a significant peak increase in photon emission (474 and 1738 cps vs. 28 and 55 cps for water alone) for the H₂O₂ which we attribute to direct ROS reaction emission as confirmed by measurement on dead plants.</p><p>Altogether we have expanded upon and demonstrated an instrument and biological system for reliably producing and measuring intrinsic metabolic photons, first observed 100 years ago by Alexander Gurwitsch.</p></div>","PeriodicalId":375,"journal":{"name":"Journal of Photochemistry and Photobiology","volume":"19 ","pages":"Article 100224"},"PeriodicalIF":3.261,"publicationDate":"2023-12-05","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://www.sciencedirect.com/science/article/pii/S2666469023000659/pdfft?md5=beeaf5a8eac4676da665966029c2d799&pid=1-s2.0-S2666469023000659-main.pdf","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"138519651","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"OA","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}

{"title":"The phenomenon of phototoxicity and long-term risks of commonly prescribed and structurally diverse drugs","authors":"Anna E. Davis ,&nbsp;Gabrielle E. Kennelley ,&nbsp;Tatiana Amaye-Obu ,&nbsp;Peter F. Jowdy ,&nbsp;Sarah Ghadersohi ,&nbsp;Mehr Nasir-Moin ,&nbsp;Gyorgy Paragh ,&nbsp;Harvey A. Berman ,&nbsp;Wendy J. Huss","doi":"10.1016/j.jpap.2023.100221","DOIUrl":"10.1016/j.jpap.2023.100221","url":null,"abstract":"<div><p>Photosensitivity to structurally diverse drugs is a common but under-reported adverse cutaneous reaction and can be classified as phototoxic or photoallergic. Phototoxic reactions occur when the skin is exposed to sunlight after administering topical or systemic medications that exhibit photosensitizing activity. These reactions depend on the dose of medication, degree of exposure to ultraviolet light, type of ultraviolet light, and sufficient skin distribution volume. Accurate prediction of the incidence and phototoxic response severity is challenging due to a paucity of literature, suggesting that phototoxicity may be more frequent than reported. This paper reports an extensive literature review on phototoxic drugs; the review employed pre-determined search criteria that included meta-analyses, systematic reviews, literature reviews, and case reports freely available in full text. Additional reports were identified from reference sections that contributed to the understanding of phototoxicity. The following drugs and/or drug classes are discussed: amiodarone, voriconazole, chlorpromazine, doxycycline, fluoroquinolones, hydrochlorothiazide, nonsteroidal anti-inflammatory drugs, and vemurafenib. In reviewing phototoxic skin reactions, this review highlights drug molecular structures, their reactive pathways, and, as there is a growing association between photosensitizing drugs and the increasing incidence of skin cancer, the consequential long-term implications of photocarcinogenesis.</p></div>","PeriodicalId":375,"journal":{"name":"Journal of Photochemistry and Photobiology","volume":"19 ","pages":"Article 100221"},"PeriodicalIF":3.261,"publicationDate":"2023-12-05","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://www.sciencedirect.com/science/article/pii/S2666469023000623/pdfft?md5=ee655310aeb220d34333663dc2c8134b&pid=1-s2.0-S2666469023000623-main.pdf","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"138546988","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"OA","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}

{"title":"DNA lesions triggered by visible light in skin cells: In the search for comprehensive sun protection","authors":"Paulo Newton Tonolli ,&nbsp;Orlando Chiarelli-Neto ,&nbsp;Maurício S. Baptista","doi":"10.1016/j.jpap.2023.100217","DOIUrl":"https://doi.org/10.1016/j.jpap.2023.100217","url":null,"abstract":"<div><p>Skin cells present many endogenous photosensitizers (ePS) that interact with light, generating oxidizing species, causing molecular damage in proteins, lipids, and nucleic acids, and consequently triggering cellular and organelle malfunction. Several cell lines with terminal differentiation are susceptible to accumulating non-digestible pigments, such as lipofuscin or melanin-lipofuscin. Besides being hallmarks of aging, both pigments can work as photosensitizers, increasing and expanding the toxicity of sunlight to the range of visible light (VL, 400–700 nm). In here we review the literature to describe the mechanisms by which the photosensitized oxidation reactions induced by VL cause DNA damage. We aim to provide the mechanistic background needed to improve the current strategies of photoprotection.</p></div>","PeriodicalId":375,"journal":{"name":"Journal of Photochemistry and Photobiology","volume":"18 ","pages":"Article 100217"},"PeriodicalIF":3.261,"publicationDate":"2023-12-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://www.sciencedirect.com/science/article/pii/S2666469023000581/pdfft?md5=1e55efd127a6fe0916787d8c796651fc&pid=1-s2.0-S2666469023000581-main.pdf","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"138471688","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"OA","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}

{"title":"Synthesis of pyrenocycloalkenes by using [2 + 2] photocycloaddition to pyrene and Diels–Alder reaction","authors":"Hajime Maeda,&nbsp;Masashi Maeda,&nbsp;Masahito Segi","doi":"10.1016/j.jpap.2023.100218","DOIUrl":"10.1016/j.jpap.2023.100218","url":null,"abstract":"<div><p>Photoreaction of pyrene <strong>1</strong> with methyl cinnamate <strong>15a</strong> gave a photocycloadduct <strong>16a</strong> at 4,5-position of pyrene stereoselectively. Oxidation of <strong>16a</strong> using DDQ yielded a pyrenocyclobutene derivative <strong>18a</strong>. Functional group conversion of the ester moiety of <strong>18a</strong> resulted in the synthesis of carboxylic acid <strong>19</strong>, alcohols <strong>20</strong> and <strong>21</strong>, sulfonate <strong>23</strong>, and methylenecyclobutene <strong>24</strong>. Diels–Alder reactions of <strong>18a</strong> with electron-deficient alkenes or alkynes <strong>25a–f</strong> afforded cycloadducts, pyrenocyclohexenes <strong>26a–c</strong> and pyrenocyclohexadienes <strong>26e, f</strong> stereoselectively in good yields. Thermal reactions of pyrenocyclobutene-linked electron-deficient alkenes <strong>22a, b</strong> produced 4-benzyl-5-alkenylpyrenes <strong>29a, b</strong> via ring cleavage followed by 1,5-hydrogen transfer.</p></div>","PeriodicalId":375,"journal":{"name":"Journal of Photochemistry and Photobiology","volume":"19 ","pages":"Article 100218"},"PeriodicalIF":3.261,"publicationDate":"2023-11-29","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://www.sciencedirect.com/science/article/pii/S2666469023000593/pdfft?md5=8445c1f0e21178936b14c6167b3ab83e&pid=1-s2.0-S2666469023000593-main.pdf","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"138519633","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"OA","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}

{"title":"Cell and tissue-based models for evaluating the cutaneous impact of visible light","authors":"Anthony Brown,&nbsp;Carles Trullas,&nbsp;Eric Jourdan","doi":"10.1016/j.jpap.2023.100216","DOIUrl":"https://doi.org/10.1016/j.jpap.2023.100216","url":null,"abstract":"<div><p>Owing to its low energy, visible light (VIS) was previously considered to have no photobiological effects and research was focused on the ultraviolet (UV) end of the solar spectrum. However, the discovery that exposure of skin to VIS leads to clinical changes in skin reminiscent of those of UV led to a reassessment of its effects. Driving our understanding have been cell and tissue-based models that permit a thorough dissection of the molecular events in skin cells following exposure to specific wavelengths and intensities of VIS. Here we explore how these models have been used to understand the cutaneous impact of VIS and identify substances that protect skin from its damaging effects.</p></div>","PeriodicalId":375,"journal":{"name":"Journal of Photochemistry and Photobiology","volume":"19 ","pages":"Article 100216"},"PeriodicalIF":3.261,"publicationDate":"2023-11-28","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://www.sciencedirect.com/science/article/pii/S266646902300057X/pdfft?md5=517fef5109525633a1535a1535e6aa0c&pid=1-s2.0-S266646902300057X-main.pdf","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"138484372","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"OA","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}

{"title":"Light-emitting diode (LED) photobiomodulation exerts anti-inflammatory action in murine thioglycolate-elicited macrophages stimulated by Bothrops jararacussu venom and by isolated PLA2s","authors":"Valdison P. Reis ,&nbsp;Alex A. Ferreira e Ferreira ,&nbsp;Sulamita da S. Setúbal ,&nbsp;Hallison M. Santana ,&nbsp;Milena D.S. Silva ,&nbsp;Carolina P. da Silva ,&nbsp;Neriane M. Nery ,&nbsp;Charles Nunes Boeno ,&nbsp;Mauro V. Paloschi ,&nbsp;Andreimar M. Soares ,&nbsp;Stella R. Zamuner ,&nbsp;Juliana P. Zuliani","doi":"10.1016/j.jpap.2023.100214","DOIUrl":"https://doi.org/10.1016/j.jpap.2023.100214","url":null,"abstract":"<div><p>Although the treatment currently recommended for snakebite accidents is serum therapy using antivenom, a need for adjunctive therapy associated with serum therapy for treating the local effects caused by snakebites is an effort of the WHO to reduce local signals and symptoms. Photobiomodulation with laser or LED therapy is one of the primary examples of adjuvant therapy to serum therapy to lessen these local effects caused by snakebite envenoming. For this purpose, the project aims to study the action of photobiomodulation with LED therapy in isolated thioglycolate-elicited macrophages stimulated with <em>Bothrops jararacussu</em> venom (BjV) and isolated bothropstoxins BthTX-I and BthTX-II focusing on cell dead mechanism such as necrosis and apoptosis, mitochondrial membrane potential, and cytokines [Interleukin (IL)-1β, IL-10, IL-6], and [tumor necrosis factor (TNF)-α] and lipid mediator [prostaglandin (PG)E₂] liberation. Briefly, thioglycollate-elicited macrophages were harvested from Swiss male mice incubated with BjV or BthTXs irradiated or not with LED, and the following parameters were analyzed: necrosis and apoptosis, mitochondrial membrane potential, cytokines, and lipid mediator liberation. Herein, results showed that LED therapy was able to decrease necrosis cell death, caspase-3 activity, and TNF-α liberation. In addition, LED therapy induces mitochondrial membrane potential and modulates gene expression of lipid mediators. In conclusion, the data of this study support the use of phototherapy as an adjuvant therapeutical approach in combination with serum therapy to mitigate the local effects resulting from snakebite envenoming.</p></div>","PeriodicalId":375,"journal":{"name":"Journal of Photochemistry and Photobiology","volume":"19 ","pages":"Article 100214"},"PeriodicalIF":3.261,"publicationDate":"2023-11-27","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://www.sciencedirect.com/science/article/pii/S2666469023000556/pdfft?md5=02063ef578c294e14082566b508125e0&pid=1-s2.0-S2666469023000556-main.pdf","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"138472689","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"OA","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}

{"title":"A review of photobiomodulation on PI3K/AKT/mTOR in wound healing","authors":"Patricia Kasowanjete,&nbsp;Sathish Sundar Dhilip Kumar,&nbsp;Nicolette N. Houreld","doi":"10.1016/j.jpap.2023.100215","DOIUrl":"https://doi.org/10.1016/j.jpap.2023.100215","url":null,"abstract":"<div><p>Wound healing involves a series of cellular and molecular processes to heal injured tissue. Growth factors such as vascular endothelial growth factor (VEGF), and signalling pathways such as phosphatidylinositol 3-kinase, protein kinase B, and mammalian target of rapamycin (PI3K/AKT/mTOR) are essential in wound healing. VEGF is linked to intracellular signalling pathways including PI3K/AKT/mTOR, which controls cell growth, metabolism, proliferation, apoptosis, and protein synthesis. During photobiomodulation (PBM), low-level light in the visible red and near-infrared (NIR) spectrum is employed to promote healing, and reduce pain, inflammation, and oedema. Several studies demonstrate that PBM enhances cellular survival, proliferation, migration, and viability in vitro<em>,</em> however, the exact cellular and molecular mechanisms responsible for these benefits have not yet been identified. The aim of this review is to explore the effects of PBM on the PI3K/AKT/mTOR signalling pathway in wound healing.</p></div>","PeriodicalId":375,"journal":{"name":"Journal of Photochemistry and Photobiology","volume":"19 ","pages":"Article 100215"},"PeriodicalIF":3.261,"publicationDate":"2023-11-25","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://www.sciencedirect.com/science/article/pii/S2666469023000568/pdfft?md5=9ed078789ce41d6b57eccdb98dd37ee6&pid=1-s2.0-S2666469023000568-main.pdf","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"138480219","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"OA","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}

{"title":"Use of reconstructed skin model to assess the photoprotection afforded by three sunscreen products having different SPF values against DNA lesions and cellular alterations","authors":"D. Lelièvre ,&nbsp;F. Canivet ,&nbsp;F. Thillou ,&nbsp;C. Tricaud ,&nbsp;C. Le Floc'h ,&nbsp;F. Bernerd","doi":"10.1016/j.jpap.2023.100213","DOIUrl":"https://doi.org/10.1016/j.jpap.2023.100213","url":null,"abstract":"<div><h3>Introduction</h3><p>Both UVB and UVA rays induce biological damages in the epidermis and the dermis that contribute to photo-carcinogenesis and photoaging. In the present study, the photoprotective effect of 2 ISO standard sunscreens, P3 (Sun Protection Factor [SPF]15) and P6 (SPF40) and of an SPF50+ labeled commercial sunscreen product was tested in reconstructed skin tissues exposed to increasing doses of UV Solar Simulated Radiation (UV-SSR). UV-induced damages were evaluated using several biological markers, including DNA lesions in the presence or absence of sunscreen protection.</p></div><div><h3>Method</h3><p>T-Skin™ model samples (EPISKIN), composed of a fibroblast-populated dermal equivalent and a fully differentiated epidermis, were protected with the test sunscreens (1.3 mg/cm² topically applied on molded polymethyl methacrylate plate) before being exposed to increasing UV doses (0 – 2.5 – 5 - 25 – 40 J/cm²). Twenty-four hours after exposure, tissues with and without sunscreen protection, were analyzed for skin viability and morphology, DNA lesions (cyclobutane pyrimidine dimer) and inflammatory mediator quantification. Results were compared to untreated exposed tissues using a Wilcoxon non-parametric test.</p></div><div><h3>Results</h3><p>For untreated tissues, UV-SSR exposure induced a dose-dependent decrease in epidermal and dermal viabilities, an increase in release of proinflammatory cytokines and matrix metalloproteinases and were associated with morphological damages at doses as low as 5 J/cm². DNA lesions were even detected at the lowest dose of 2.5 J/cm², and their number increased with the UV-SSR dose. In the samples protected with sunscreens, these abnormalities were partially or totally prevented with P6 providing a better protection compared to P3, and the SPF50+ sunscreen showing a trend for better protection than P6, for example against DNA damage.</p></div><div><h3>Conclusions</h3><p>This study demonstrates that photoprotective effects of different sunscreens can be discriminated and ranked on reconstructed skin tissues (T-Skin™ model) exposed to UV-SSR. Showing significant differences between the reference products P3 and P6 in line with their respective SPF values, such study allows the evaluation of epidermal and dermal damages at the tissue, cellular and molecular levels. It thus opens the way to a new model of integrated assessment of sunscreens. In line with its labeled 50+ SPF, the commercial test product confirmed its improved protection especially on DNA damage prevention.</p></div>","PeriodicalId":375,"journal":{"name":"Journal of Photochemistry and Photobiology","volume":"19 ","pages":"Article 100213"},"PeriodicalIF":3.261,"publicationDate":"2023-11-25","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://www.sciencedirect.com/science/article/pii/S2666469023000544/pdfft?md5=e9553983022c06745e3365bffb3b0059&pid=1-s2.0-S2666469023000544-main.pdf","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"138480218","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"OA","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}

{"title":"Synthesis of CuInS2 nanoparticles and application in the photocatalytic degradation of tetracycline","authors":"Damian C. Onwudiwe ,&nbsp;Opeyemi A. Oyewo ,&nbsp;Naledi H. Seheri ,&nbsp;Mathato P. Motaung ,&nbsp;Seshibe S Makgato ,&nbsp;Sarah C. Motshekga","doi":"10.1016/j.jpap.2023.100212","DOIUrl":"https://doi.org/10.1016/j.jpap.2023.100212","url":null,"abstract":"<div><p>A lot of effort has been given to the development of nontoxic ternary semiconductor nanoparticles that could act as photocatalyst NIR-I (750–850 nm) or NIR-II (1000–1400 nm) optical windows. This is due to their good stability, high optical absorption coefficient, and desirable band gap that absorbs well within the solar spectrum. CuInS₂ is one of the ternary sulphide semiconductors, which has been considered to be a highly promising photocatalyst. The properties are attributed to its high optical absorption coefficient. In this study, copper indium sulphide (CuInS₂) nanoparticles were synthesized by a microwave irradiation route using copper(II) bis (<em>N</em>-methyl-<em>N</em>-ethanol dithiocarbamate) and In(III) tris (<em>N</em>-methyl-<em>N</em>-ethanol dithiocarbamate) as a precursor complexes. The copper(II) complex was varied in two different ratios (3:1 and 2:1) to determine the best synthesis regime. Then, the effect of the varying ratios on the crystalline structure, morphology, and optical properties of the CuInS₂ was studied by using X-ray diffraction (XRD), scanning and transmission electron microscopy (SEM and TEM), and absorption spectroscopy. The microscopic analyses revealed that the CuInS₂ nanoparticles have similar spherical grain-like shapes whose sizes range between 10.3–50.1 nm. The increase in the concentration of copper(II) complex also altered the band gap energy, given 2.87 and 1.61 eV for CuInS₂(3:1) and CuInS₂(2:1) respectively. The photocatalytic activities of the nanoparticles were determined for the degradation of Tetracycline (TC) under visible light irradiation. The effects of process parameters such as photocatalyst dosage and initial concentration of TC were investigated to establish the optimal performance of the CuInS₂ nanoparticles. The experimental data showed a higher TC degradation percentage for CuInS₂(2:1) (95 %) compared to CuInS₂(3:1) (90 %), indicating its high potential as a photocatalyst for the degradation of TC in aqueous solution.</p></div>","PeriodicalId":375,"journal":{"name":"Journal of Photochemistry and Photobiology","volume":"18 ","pages":"Article 100212"},"PeriodicalIF":3.261,"publicationDate":"2023-10-21","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://www.sciencedirect.com/science/article/pii/S2666469023000532/pdfft?md5=fd411b7b3b403f9ed489bdd1e73f60c4&pid=1-s2.0-S2666469023000532-main.pdf","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"92042283","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"OA","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}