Journal of chemical neuroanatomy最新文献

{"title":"Retraction notice to “Exercise ameliorates hippocampal damage induced by Wi-Fi radiation; A biochemical, histological, and immunohistochemical study” [J. Chem. Neuroanat. 129 (2023) 102252]","authors":"","doi":"10.1016/j.jchemneu.2024.102433","DOIUrl":"10.1016/j.jchemneu.2024.102433","url":null,"abstract":"","PeriodicalId":15324,"journal":{"name":"Journal of chemical neuroanatomy","volume":"139 ","pages":"Article 102433"},"PeriodicalIF":2.7,"publicationDate":"2024-09-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://www.sciencedirect.com/science/article/pii/S0891061824000474/pdfft?md5=e8a2f64d8f8d3b57ce8c7bfeb429c6ae&pid=1-s2.0-S0891061824000474-main.pdf","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"141293451","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":4,"RegionCategory":"医学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"OA","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}

{"title":"Retraction notice to aerobic exercise training reduces deep-frying oil-induced apoptosis of hippocampal tissue by reducing oxidative stress in male rats [J. Chem. Neuroanat. 133 (2023) 102328]","authors":"Sina Nikbin , Gita Fardad , Sara Yazdi , Marzieh Hosseini Bahman , Parvaneh Ettefagh , Fatemeh Khalegi , Mino Molaei , Kamal Azizbeigi , Myriam Guerra-Balic , Joel Montan , Mehdi Zargani , Mohammad Ali Azarbayjani","doi":"10.1016/j.jchemneu.2024.102447","DOIUrl":"10.1016/j.jchemneu.2024.102447","url":null,"abstract":"","PeriodicalId":15324,"journal":{"name":"Journal of chemical neuroanatomy","volume":"139 ","pages":"Article 102447"},"PeriodicalIF":2.7,"publicationDate":"2024-09-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://www.sciencedirect.com/science/article/pii/S0891061824000632/pdfft?md5=ce62f06f34a8ff1e7f6aa12d30e61c97&pid=1-s2.0-S0891061824000632-main.pdf","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"141758933","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":4,"RegionCategory":"医学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"OA","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}

{"title":"Retraction notice to “Anti-inflammatory, anticholinesterase, antioxidant, and memory enhancement potential of Phyllanthus amarus in potassium-dichromate induced neurotoxicity of male Wistar rats” [J. Chem. Neuroanat. 132 (2023) 102308]","authors":"Kingsley Afoke Iteire , Tolulope Judah Gbayisomore , Olalekan Marvelous Olatuyi","doi":"10.1016/j.jchemneu.2024.102446","DOIUrl":"10.1016/j.jchemneu.2024.102446","url":null,"abstract":"","PeriodicalId":15324,"journal":{"name":"Journal of chemical neuroanatomy","volume":"139 ","pages":"Article 102446"},"PeriodicalIF":2.7,"publicationDate":"2024-09-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://www.sciencedirect.com/science/article/pii/S0891061824000620/pdfft?md5=1eebf137d4540ae8e781a9ba197312ad&pid=1-s2.0-S0891061824000620-main.pdf","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"141758931","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":4,"RegionCategory":"医学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"OA","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}

{"title":"Retraction notice to “Neuroprotective potential of Ginkgo biloba on alteration of rat cerebellum following prenatal exposure to cyclophosphamide” J. Chem. Neuroanat. 130 (2023) 102268","authors":"","doi":"10.1016/j.jchemneu.2024.102434","DOIUrl":"10.1016/j.jchemneu.2024.102434","url":null,"abstract":"","PeriodicalId":15324,"journal":{"name":"Journal of chemical neuroanatomy","volume":"139 ","pages":"Article 102434"},"PeriodicalIF":2.7,"publicationDate":"2024-09-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://www.sciencedirect.com/science/article/pii/S0891061824000486/pdfft?md5=89ffdf67e82bf66e050afff09069e87e&pid=1-s2.0-S0891061824000486-main.pdf","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"141293452","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":4,"RegionCategory":"医学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"OA","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}

{"title":"Retraction notice to the effects of myricitrin and chebulinic acid on the rat hippocampusexposed to gamma radiation: A stereological, histochemical andbiochemical study J. Chem. Neuroanat. Volume 132, October 2023, 102305","authors":"Uzun Sümeyye Gümüs , Berrin Zuhal Altunkaynak , ınsu Alkan Is","doi":"10.1016/j.jchemneu.2024.102444","DOIUrl":"10.1016/j.jchemneu.2024.102444","url":null,"abstract":"","PeriodicalId":15324,"journal":{"name":"Journal of chemical neuroanatomy","volume":"139 ","pages":"Article 102444"},"PeriodicalIF":2.7,"publicationDate":"2024-09-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://www.sciencedirect.com/science/article/pii/S0891061824000590/pdfft?md5=fbf2492899b8ce1782878b84b228487c&pid=1-s2.0-S0891061824000590-main.pdf","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"141758934","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":4,"RegionCategory":"医学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"OA","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}

{"title":"Retraction notice to “TGN020 application against aquaporin 4 improved multiple sclerosis by inhibiting astrocytes, microglia, and NLRP3 inflammasome in a cuprizone mouse model” [J. Chem. Neuroanat. 132 (2023) 102306]","authors":"Yousef Mohamadi , Maryam Borhani-Haghighi","doi":"10.1016/j.jchemneu.2024.102445","DOIUrl":"10.1016/j.jchemneu.2024.102445","url":null,"abstract":"","PeriodicalId":15324,"journal":{"name":"Journal of chemical neuroanatomy","volume":"139 ","pages":"Article 102445"},"PeriodicalIF":2.7,"publicationDate":"2024-09-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://www.sciencedirect.com/science/article/pii/S0891061824000607/pdfft?md5=923416bb3c9518d9433511555b1f76ed&pid=1-s2.0-S0891061824000607-main.pdf","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"141758932","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":4,"RegionCategory":"医学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"OA","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}

{"title":"Impacts of SARS-CoV-2 on brain renin angiotensin system related signaling and its subsequent complications on brain: A theoretical perspective","authors":"Negar Etebar ,&nbsp;Saghi Naderpour ,&nbsp;Setareh Akbari ,&nbsp;Alireza Zali ,&nbsp;Meisam Akhlaghdoust ,&nbsp;Seyed Mojtaba Daghighi ,&nbsp;Matin Baghani ,&nbsp;Seyed Hootan Hamidi ,&nbsp;Milad Rahimzadegan","doi":"10.1016/j.jchemneu.2024.102423","DOIUrl":"10.1016/j.jchemneu.2024.102423","url":null,"abstract":"<div><p>Cellular ACE2 (cACE2), a vital component of the renin–angiotensin system (RAS), possesses catalytic activity to maintain AngII and Ang 1–7 balance, which is necessary to prevent harmful effects of AngII/AT2R and promote protective pathways of Ang (1−7)/MasR and Ang (1–7)/AT2R. Hemostasis of the brain–RAS is essential for maintaining normal central nervous system (CNS) function. Severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) is a viral disease that causes multi-organ dysfunction. SARS-CoV-2 mainly uses cACE2 to enter the cells and cause its downregulation. This, in turn, prevents the conversion of Ang II to Ang (1−7) and disrupts the normal balance of brain–RAS. Brain–RAS disturbances give rise to one of the pathological pathways in which SARS-CoV-2 suppresses neuroprotective pathways and induces inflammatory cytokines and reactive oxygen species. Finally, these impairments lead to neuroinflammation, neuronal injury, and neurological complications. In conclusion, the influence of RAS on various processes within the brain has significant implications for the neurological manifestations associated with COVID-19. These effects include sensory disturbances, such as olfactory and gustatory dysfunctions, as well as cerebrovascular and brain stem-related disorders, all of which are intertwined with disruptions in the RAS homeostasis of the brain.</p></div>","PeriodicalId":15324,"journal":{"name":"Journal of chemical neuroanatomy","volume":"138 ","pages":"Article 102423"},"PeriodicalIF":2.8,"publicationDate":"2024-07-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"140855320","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":4,"RegionCategory":"医学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}

{"title":"Chondroitin sulfate proteoglycans mRNA expression and degradation in the zebra finch following traumatic brain injury","authors":"Adam Talwalkar ,&nbsp;Gage Haden ,&nbsp;Kelli A. Duncan","doi":"10.1016/j.jchemneu.2024.102418","DOIUrl":"https://doi.org/10.1016/j.jchemneu.2024.102418","url":null,"abstract":"<div><p>Traumatic brain injury (TBI) is one of the leading causes of fatality and disability worldwide. From minutes to months following damage, injury can result in a complex pathophysiology that can lead to temporary or permanent deficits including an array of neurodegenerative symptoms. These changes can include behavioral dysregulation, memory dysfunctions, and mood changes including depression. The nature and severity of impairments resulting from TBIs vary widely given the range of injury type, location, and extent of brain tissue involved. In response to the injury, the brain induces structural and functional changes to promote repair and minimize injury size. Despite its high prevalence, effective treatment strategies for TBI are limited. PNNs are part of the neuronal extracellular matrix (ECM) that mediate synaptic stabilization in the adult brain and thus neuroplasticity. They are associated mostly with inhibitory GABAergic interneurons and are thought to be responsible for maintaining the excitatory/inhibitory balance of the brain. The major structural components of PNNs include multiple chondroitin sulfate proteoglycans (CSPGs) as well as other structural proteins. Here we examine the effects of injury on CSPG expression, specifically around the changes in the side change moieties. To investigate CSPG expression following injury, adult male and female zebra finches received either a bilateral penetrating, or no injury and qPCR analysis and immunohistochemistry for components of the CSPGs were examined at 1- or 7-days post-injury. Next, to determine if CSPGs and thus PNNs should be a target for therapeutic intervention, CSPG side chains were degraded at the time of injury with chondroitinase ABC (ChABC) CSPGs moieties were examined. Additionally, GABA receptor mRNA and aromatase mRNA expression was quantified following CSPG degradation as they have been implicated in neuronal survival and neurogenesis. Our data indicate the CSPG moieties change following injury, potentially allowing for a brief period of synaptic reorganization, and that treatments that target CSPG side chains are successful in further targeting this brief critical period by decreasing GABA mRNA receptor expression, but also decreasing aromatase expression.</p></div>","PeriodicalId":15324,"journal":{"name":"Journal of chemical neuroanatomy","volume":"138 ","pages":"Article 102418"},"PeriodicalIF":2.8,"publicationDate":"2024-07-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"140644648","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":4,"RegionCategory":"医学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}

{"title":"Herbert Major on the insula: An early depiction of von Economo neurones?","authors":"Andrew J. Larner ,&nbsp;Lazaros C. Triarhou","doi":"10.1016/j.jchemneu.2024.102435","DOIUrl":"10.1016/j.jchemneu.2024.102435","url":null,"abstract":"<div><p>Herbert Major (1850–1921) undertook histopathological studies of human and non-human primate brains at the West Riding Lunatic Asylum in Wakefield, England, during the 1870s. Two of his papers specifically investigated the structure of the island of Reil, or insula, “with the view of ascertaining its exact structure”. In addition to describing and illustrating its lamination as six-layered, Major also identified “spindle-shaped” cells in the lower layers of human brains, but not in non-human primates. His written description, including measurements of cell body size, and illustration are suggestive that these were the neurones later described in the frontoinsular and anterior cingulate cortex by Constantin von Economo and Georg N. Koskinas and which were subsequently given the eponym “von Economo neurones”. von Economo noted that this special neuronal type had been previously seen by <span>Betz (1881)</span>, <span>Hammarberg (1895)</span>, and <span>Ramón y Cajal (1899-1904)</span>, but he did not mention Major’s works. Major also ascribed linguistic functions to the insula. Hence, with respect to both anatomical and physiological features, Major may have pre-empted the findings of later research on this structure.</p></div>","PeriodicalId":15324,"journal":{"name":"Journal of chemical neuroanatomy","volume":"138 ","pages":"Article 102435"},"PeriodicalIF":2.8,"publicationDate":"2024-07-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"141186535","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":4,"RegionCategory":"医学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}

{"title":"Corrigendum to “Therapeutic effects of melatonin-treated bone marrow mesenchymal stem cells (BMSC) in a rat model of Alzheimer's disease” [J. Chem. Neuroanat. 108 (2020) 101804]","authors":"Mahdi Ramezani ,&nbsp;Alireza Komaki ,&nbsp;Nasrin Hashemi-Firouzi ,&nbsp;Keywan Mortezaee ,&nbsp;Nafiseh Faraji ,&nbsp;Zoleikha Golipoor","doi":"10.1016/j.jchemneu.2024.102425","DOIUrl":"https://doi.org/10.1016/j.jchemneu.2024.102425","url":null,"abstract":"","PeriodicalId":15324,"journal":{"name":"Journal of chemical neuroanatomy","volume":"138 ","pages":"Article 102425"},"PeriodicalIF":2.8,"publicationDate":"2024-07-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://www.sciencedirect.com/science/article/pii/S0891061824000383/pdfft?md5=363c5efe21862cbd4c7c2415be428b1c&pid=1-s2.0-S0891061824000383-main.pdf","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"140644649","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":4,"RegionCategory":"医学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"OA","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}