Advanced Science最新文献

{"title":"High-Performance Pure Water-Fed Anion Exchange Membrane Water Electrolysis with Patterned Membrane via Mechanical Stress and Hydration-Mediated Patterning Technique.","authors":"Yeonjae Lee, Sungjun Kim, Yoseph Shin, Yeram Shin, Seongmin Shin, Sanghyeok Lee, Minseop So, Tae-Ho Kim, Sehkyu Park, Jang Yong Lee, Segeun Jang","doi":"10.1002/advs.202409563","DOIUrl":"https://doi.org/10.1002/advs.202409563","url":null,"abstract":"<p><p>Despite rapid advancements in anion exchange membrane water electrolysis (AEMWE) technology, achieving pure water-fed AEMWE remains critical for system simplification and cost reduction. Under pure water-fed conditions, electrochemical reactions occur solely at active sites connected to ionic networks. This study introduces an eco-friendly patterning technique leveraging membrane swelling properties by applying mechanical stress during dehydration under fixed constraints. The method increases active sites by creating additional hydroxide ion pathways at the membrane-electrode interface, eliminating the need for additional ionomers in the electrode. This innovation facilitates ion conduction via locally shortened pathways. Membrane electrode assemblies (MEAs) with patterned commercial membranes demonstrated significantly improved performance and durability compared to MEAs with conventional catalyst-coated substrates and flat membranes under pure water-fed conditions. The universal applicability of this technique was confirmed using in-house fabricated anion exchange membranes, achieving exceptional current densities of 13.7 A cm^-2 at 2.0 V in 1.0 M potassium hydroxide (KOH) and 2.8 A cm^-2 at 2.0 V in pure water at 60 °C. Furthermore, the scalability of the technique was demonstrated through successful fabrication and operation of large-area cells. These findings highlight the potential of this patterning method to advance AEMWE technology, enabling practical applications under pure water-fed conditions.</p>","PeriodicalId":117,"journal":{"name":"Advanced Science","volume":" ","pages":"e2409563"},"PeriodicalIF":14.3,"publicationDate":"2024-12-16","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"142826633","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":1,"RegionCategory":"材料科学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}

{"title":"Physical One-Way Functions for Decentralized Consensus Via Proof of Physical Work.","authors":"Marvin Winkler, Catharina Peither, Simon Petrick, Lothar Seidemann, Holger Jelich, Frank Kleine Jäger, Jörn Müller-Quade, Alexander Colsmann, Hermann Nirschl, Frank Rhein","doi":"10.1002/advs.202409386","DOIUrl":"https://doi.org/10.1002/advs.202409386","url":null,"abstract":"<p><p>Decentralized consensus on the state of the Bitcoin blockchain is ensured by proof of work. It relies on digital one-way functions and is associated with an enormous environmental impact. This paper conceptualizes a physical one-way function that aims to transform a digital, electricity-consuming consensus mechanism into a physical process. Boundary conditions for the security requirements are established and discussed as well as experimentally investigated for a specific setup based on printing and optical analysis of pigment-carrier composites. In the context of the applied methods, this setup promises to be mathematically unclonable, steady, reproducible, collision resistant and non-invertible and illustrates the feasibility of a physical one-way function. Based on this, a framework for proof of physical work is conceptualized, which has the potential of a drastically lower CO₂ footprint. This work initiates a progressive, interdisciplinary field of research and demands further investigations with regards to alternative setups, security definitions and strategies for challenging them.</p>","PeriodicalId":117,"journal":{"name":"Advanced Science","volume":" ","pages":"e2409386"},"PeriodicalIF":14.3,"publicationDate":"2024-12-16","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"142833186","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":1,"RegionCategory":"材料科学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}

{"title":"Mesenchymal Stem Cells Prevent SLC39A14-Dependent Hepatocyte Ferroptosis through Exosomal miR-16-5p in Liver Graft.","authors":"Zhizhao Deng, Weiqi Zeng, Yingxin Gao, Zhenyu Yang, Xinling Luo, Xianlong Li, Guoliang Sun, Erfeng Xiong, Fei Huang, Gangjian Luo, Ziqing Hei, Dongdong Yuan","doi":"10.1002/advs.202411380","DOIUrl":"https://doi.org/10.1002/advs.202411380","url":null,"abstract":"<p><p>Ischemia-reperfusion injury (IRI) is the leading cause of hepatic graft dysfunction, resulting from hepatocyte damage. Nevertheless, given the few specialized therapeutics available in hepatic IRI, additional mechanistic insights into hepatocyte damage are required. Here, the protein solute carrier family 39 member 14 (SLC39A14) is identified as a pro-ferroptosis target in hepatocytes of human liver allografts through single-cell RNA sequencing analysis. SLC39A14 knockdown significantly mitigated hepatic IRI by preventing hepatocyte ferroptosis in vivo and in vitro. Mechanistically, the inhibition of SLC39A14 suppressed non-transferrin-bound iron (NTBI) uptake by hepatocytes, thereby reducing iron overload and cell ferroptosis. Moreover, human bone marrow-derived mesenchymal stem cells (hBMSCs) are found to exhibit a notable therapeutic effect on hepatic IRI by downregulating SLC39A14 expression. Exosomes derived from hBMSCs delivered abundant miR-16-5p into hepatocytes, which post-transcriptionally suppressed the expression of SLC39A14 and reduced cell ferroptosis induced by hepatic IRI. In conclusion, SLC39A14 triggers hepatic IRI by mediating NTBI uptake into hepatocytes and inducing hepatocyte ferroptosis. Moreover, hBMSC-based therapy is promising to reverse this progression of hepatic IRI.</p>","PeriodicalId":117,"journal":{"name":"Advanced Science","volume":" ","pages":"e2411380"},"PeriodicalIF":14.3,"publicationDate":"2024-12-16","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"142833185","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":1,"RegionCategory":"材料科学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}

{"title":"Unveiling the Origin of the Strengthening Mechanism in a Novel Precious Metal Multi-Principal Elements Alloy.","authors":"Xuan Zhou, Hualong Ge, Kai Xiong, Junjie He, Shunmeng Zhang, Li Fu, Zhilong Tan, Xiaofei Wu, Xuming Li, Haijun Wu, Junmei Guo, Yong Mao","doi":"10.1002/advs.202410936","DOIUrl":"https://doi.org/10.1002/advs.202410936","url":null,"abstract":"<p><p>Precious metal electrical contact materials are pivotal in microelectronic devices due to their excellent chemical stability and electrical properties. Their practical application is hindered by the strength, contact resistance, and high cost. Multi-principal elements alloys (MPEAs) provide the possibility to develop cost-effective materials with enhanced mechanical properties. To address this, a novel precious metal MPEA, PdAgCuAuPtZn alloy, is designed, which exhibits significant solid solution strengthening and aging strengthening effects. The ultimate tensile strength increases from 747 MPa in the solution state to 1126 MPa in the aged state, while resistivity remains low. This study presents the first systematic investigation into the strengthening mechanisms of precious metal MPEAs using nanoindentation technology. These findings indicate that the aging strengthening of the alloy is attributed to spinodal decomposition (SD) and chemical short-range order (CSRO) in the matrix. Furthermore, the precipitation structure with Cu-rich and Ag-rich phases gradually replaces the matrix, primarily accounting for aging softening. Additionally, it is discovered that precipitation structure can be strengthened by CSRO formed in the Cu-rich phase, thus providing an innovative strengthening in PdAgCuAuPtZn alloy. These results will be beneficial to both deeply understanding the aging behaviors of PdAgCuAuPtZn alloys and designing high-performance precious metal MPEAs.</p>","PeriodicalId":117,"journal":{"name":"Advanced Science","volume":" ","pages":"e2410936"},"PeriodicalIF":14.3,"publicationDate":"2024-12-16","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"142833191","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":1,"RegionCategory":"材料科学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}

{"title":"Reversible Carrier Modulation in InP Nanolasers by Ionic Liquid Gating with Low Energy Consumption.","authors":"Chia-Hung Wu, Chi-Wen Chen, Hung-Jung Shen, Hsiang-Yu Chuang, Hark Hoe Tan, Chennupati Jagadish, Tien-Chang Lu, Satoshi Ishii, Kuo-Ping Chen","doi":"10.1002/advs.202412340","DOIUrl":"https://doi.org/10.1002/advs.202412340","url":null,"abstract":"<p><p>Nanoscale light sources are demanded vigorously due to rapid development in photonic integrated circuits (PICs). III-V semiconductor nanowire (NW) lasers have manifested themselves as indispensable components in this field, associated with their extremely compact footprint and ultra-high optical gain within the 1D cavity. In this study, the carrier concentrations of indium phosphide (InP) NWs are actively controlled to modify their emissive properties at room temperature. The InP NW lasers can achieve repetitive switching between photoluminescence (PL) and lasing with an extinction ratio of 22-fold by applying a gate voltage of 3 V using ionic liquid (IL) as a dielectric layer. IL brings forth ultra-high capacitance due to the nanometer-wide electric double layer (EDL) between interfaces, mapping out gating efficiency of ≈100-fold compared to the conventional bottom gate configurations. This IL-embedded nanolaser device can be a promising platform for the advanced integrated nanophotonic system.</p>","PeriodicalId":117,"journal":{"name":"Advanced Science","volume":" ","pages":"e2412340"},"PeriodicalIF":14.3,"publicationDate":"2024-12-16","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"142833120","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":1,"RegionCategory":"材料科学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}

{"title":"Hypoxia Microenvironment Preconditioning Attenuated Myocardial Ischemia-Reperfusion Injury via Stc1-Mediating Cardiomyocyte Self-Protection and Neutrophil Polarization.","authors":"Haoxiang Huang, Yifei Ruan, Chuling Li, Hao Zheng, Yating Tang, Yijin Chen, Fengling He, Yu Liu, Guangkai Wu, Zhenhua Li, Yuegang Wang, Yulin Liao, Jianping Bin, Yanmei Chen","doi":"10.1002/advs.202411880","DOIUrl":"https://doi.org/10.1002/advs.202411880","url":null,"abstract":"<p><p>Ischemic preconditioning (IPC) therapy application to attenuate myocardial ischemia-reperfusion (MI/R) injury in clinical practice remains challenging. The secretome, derived from hypoxia-preconditioned cardiomyocytes (SHPC), potentially mimics the IPC microenvironment and facilitates IPC clinical translation. This study aims to determine whether SHPC can be a feasible alternative to IPC for attenuating MI/R injury, and to identify the functional factor of SHPC. The ultrafiltration technique is applied to generate an SHPC formulation that is intramyocardially injected before reperfusion in a murine MI/R model. The effects of SHPC on cardiomyocyte apoptosis, pyroptosis, and neutrophil polarization are evaluated. Secretomics, neutralizing antibodies, and recombinant proteins are employed to identify the functional factor in SHPC. Co-immunoprecipitation assays, RNA sequencing, and site-directed mutagenesis are conducted to investigate the underlying mechanism. Additionally, a recombinant functional factor-encapsulated hydrogel is developed for intrapericardial injections (iPC). An intramyocardial SHPC injection in MI/R-injured mice strikingly reduces infarct size and the expression of cardiac injury biomarker while improving cardiac function. SHPC eliminated mitochondrial reactive oxygen species and triggered neutrophil polarization to reduce cardiomyocyte apoptosis/pyroptosis upon hypoxia/reoxygenation injury. Stanniocalcin 1 (Stc1) is identified as the functional factor in SHPC, mediating hypoxic microenvironment. Mechanistically, hypoxia-preconditioned cardiomyocytes secrete Stc1 into the microenvironment and activate calcium-sensing receptor (CaSR) that increases Stat3 phosphorylation at Ser727 via nitric oxide synthase 2 (NOS2)-mediated S-nitrosylation, thereby decreasing cardiomyocyte apoptosis/pyroptosis in an autocrine mechanism. Simultaneously, Stc1 facilitates cardiomyocyte-neutrophil crosstalk, thereby triggering neutrophil polarization to reduce inflammatory damage via the CaSR/NOS2/Stat3 axis in a paracrine mechanism. Pericardial delivery of a recombinant rStc1-encapsulated hydrogel has extended the therapeutic time window of rStc1, improving long-term cardiac function. The hypoxia microenvironment preconditioning, which mimicked by SHPC, attenuated MI/R injury via Stc1-mediated cardiomyocyte self-protection and neutrophil polarization. This study suggests that SHPC, with hypoxia preconditioning factor Stc1, represents a clinically feasible alternative to IPC for attenuating MI/R injury.</p>","PeriodicalId":117,"journal":{"name":"Advanced Science","volume":" ","pages":"e2411880"},"PeriodicalIF":14.3,"publicationDate":"2024-12-16","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"142826702","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":1,"RegionCategory":"材料科学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}

{"title":"Wearable Temperature Sensor Enhanced Volatilomics Technique for Swift and Convenient Detection of Latrogenic Botulism.","authors":"Xiaoyang Li, Yufei Yan, Chenyi Hu, Jing Wang, Jinlin Wang, Hao Yang, Daxiang Cui, Wenwen Xin, Shan Gao, Han Jin","doi":"10.1002/advs.202411738","DOIUrl":"https://doi.org/10.1002/advs.202411738","url":null,"abstract":"<p><p>Accurately assessing potential side effects following botulinum neurotoxin (BoNT) injection remains a formidable challenge. To address this issue, an innovative approach is developed that combines a wearable temperature sensor with a sophisticated volatilomics technique, aimed at facilitating the rapid and convenient prediction of potential physical discomfort related to latrogenic botulism. The investigation identifies five volatile organic compounds (VOCs)-acetone, styrene, ethanol, 2-pentanone, and n-butano-as promising markers indicative of BoNT poisoning. Specifically, a handheld breath analyzer, featuring a yttrium stabilized zirconia (YSZ)-based gas sensor array, alongside a wearable temperature sensor integrated with a bio-compatible methacrylated gelatin (GelMA) sensing film, are developed to simultaneously monitor breath signal variations and body temperature fluctuations. Preliminary animal testing validates the effectiveness of the integrated approach, achieving an accuracy exceeding 91.2% in early detection of physical discomfort associated with BoNT poisoning. These promising findings represent a significant advancement towards the early identification of BoNT-related issues, enabling timely intervention and improved management strategies.</p>","PeriodicalId":117,"journal":{"name":"Advanced Science","volume":" ","pages":"e2411738"},"PeriodicalIF":14.3,"publicationDate":"2024-12-16","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"142826782","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":1,"RegionCategory":"材料科学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}

{"title":"SREBP1c-Mediated Transcriptional Repression of YME1L1 Contributes to Acute Kidney Injury by Inducing Mitochondrial Dysfunction in Tubular Epithelial Cells.","authors":"Wang Xin, Jie Zhou, Yuzhu Peng, Shuiqin Gong, Wenhao Liao, Yaqin Wang, Xixin Huang, Yang Mao, Mengying Yao, Shaozong Qin, Jiachuan Xiong, Yan Li, Qigang Lan, Yinghui Huang, Jinghong Zhao","doi":"10.1002/advs.202412233","DOIUrl":"https://doi.org/10.1002/advs.202412233","url":null,"abstract":"<p><p>Acute kidney injury (AKI) is a prevalent clinical syndrome with high morbidity and mortality. Accumulating studies suggest mitochondrial dysfunction as the typical characteristics and key process of AKI, but the underlying mechanism remains elusive. The YME1-like 1 (YME1L1) ATPase, an inner mitochondrial membrane protein, is screened and identified to be downregulated in renal tubular epithelial cells of various mouse models and patients of AKI. Dramatically, restoration of YME1L1 expression significantly alleviates cisplatin-induced AKI and subsequent chronic kidney disease (CKD) through attenuating mitochondrial dysfunction via maintaining optic atrophy 1 (OPA1)-mediated mitochondrial energy metabolism homeostasis. Mechanistically, the upregulated expression of sterol regulatory element binding transcription factor 1c (SREBP1c) is demonstrated to be responsible for cisplatin-mediated transcriptional inhibition of YME1L1 via directly binding to its promoter region. Moreover, cisplatin-induced methyltransferase-like 3 (METTL3)-mediated m6A modification enhances SREBP1c mRNA stability, thereby upregulating its expression. Notably, both depletion of SREBP1c and renal tubule-specific overexpression of YME1L1 markedly ameliorate cisplatin-induced AKI and its transition to CKD. Taken together, these findings suggest that METTL3-mediated SREBP1c upregulation contributes to AKI and its progression to CKD through disrupting mitochondrial energy metabolism via transcriptionally suppressing YME1L1. Targeting the SREBP1c/YME1L1 signaling may serve as a novel therapeutic strategy against AKI.</p>","PeriodicalId":117,"journal":{"name":"Advanced Science","volume":" ","pages":"e2412233"},"PeriodicalIF":14.3,"publicationDate":"2024-12-16","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"142833135","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":1,"RegionCategory":"材料科学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}

{"title":"AI-Guided Inverse Design and Discovery of Recyclable Vitrimeric Polymers.","authors":"Yiwen Zheng, Prakash Thakolkaran, Agni K Biswal, Jake A Smith, Ziheng Lu, Shuxin Zheng, Bichlien H Nguyen, Siddhant Kumar, Aniruddh Vashisth","doi":"10.1002/advs.202411385","DOIUrl":"https://doi.org/10.1002/advs.202411385","url":null,"abstract":"<p><p>Vitrimer is a new, exciting class of sustainable polymers with healing abilities due to their dynamic covalent adaptive networks. However, a limited choice of constituent molecules restricts their property space and potential applications. To overcome this challenge, an innovative approach coupling molecular dynamics (MD) simulations and a novel graph variational autoencoder (VAE) model for inverse design of vitrimer chemistries with desired glass transition temperature (T_g) is presented. The first diverse vitrimer dataset of one million chemistries is curated and T_g for 8,424 of them is calculated by high-throughput MD simulations calibrated by a Gaussian process model. The proposed VAE employs dual graph encoders and a latent dimension overlapping scheme which allows for individual representation of multi-component vitrimers. High accuracy and efficiency of the framework are demonstrated by discovering novel vitrimers with desirable T_g beyond the training regime. To validate the effectiveness of the framework in experiments, vitrimer chemistries are generated with a target T_g = 323 K. By incorporating chemical intuition, a novel vitrimer with T_g of 311-317 K is synthesized, experimentally demonstrating healability and flowability. The proposed framework offers an exciting tool for polymer chemists to design and synthesize novel, sustainable polymers for various applications.</p>","PeriodicalId":117,"journal":{"name":"Advanced Science","volume":" ","pages":"e2411385"},"PeriodicalIF":14.3,"publicationDate":"2024-12-16","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"142833233","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":1,"RegionCategory":"材料科学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}

{"title":"DNMT3a Downregulation Ttriggered Upregulation of GABA_A Receptor in the mPFC Promotes Paclitaxel-Induced Pain and Anxiety in Male Mice.","authors":"Lixia Tian, Xu-Hui Li, Yu-Long Zhao, Hui-Yuan Yi, Xue-Ru Liu, Rongrong Yao, Xue-Mei Hou, Xuan Zhu, Fu-Quan Huo, Tao Chen, Lingli Liang","doi":"10.1002/advs.202407387","DOIUrl":"https://doi.org/10.1002/advs.202407387","url":null,"abstract":"<p><p>Chemotherapeutic agents, such as paclitaxel (PTX), induce neuroplastic changes and alter gene expression in the prefrontal cortex (PFC), which may be associated with chemotherapy-induced pain and negative emotions. Notably, DNA methylation undergoes adaptive changes in neurological disorders, emerging as a promising target for neuromodulation. In this study, systemic administration of PTX leads to a decrease in the expression of the DNA methyltransferase DNMT3a, while concurrently upregulating the expression of Gabrb1 mRNA and its encoded GABA_ARβ1 protein in the medial PFC (mPFC) of male mice. Overexpression of DNMT3a in the mPFC alleviates PTX-induced pain hypersensitivity, and anxiety-like behavior in these mice. Additionally, it reverses the PTX-induced increase in inhibitory synaptic transmission in the pyramidal neurons of the mPFC. Mechanistically, the upregulation of GABA_ARβ1 in the mPFC is linked to the reduced expression of DNMT3a and DNA hypomethylation at the promoter region of the Gabrb1 gene. Furthermore, a long-term diet rich in methyl donors alleviates PTX-induced pain hypersensitivity and anxiety-like behavior in mice. These findings suggest that the DNMT3a-mediated upregulation of GABA_ARβ1 in the mPFC contributes to PTX-induced neuropathic pain and anxiety, highlighting DNA methylation-dependent epigenetic regulation as a potential therapeutic target for addressing chemotherapy-induced cortical dysfunction.</p>","PeriodicalId":117,"journal":{"name":"Advanced Science","volume":" ","pages":"e2407387"},"PeriodicalIF":14.3,"publicationDate":"2024-12-16","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"142826696","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":1,"RegionCategory":"材料科学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}