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Clinical and experimental dialysis and apheresis最新文献

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The role of urinary tract infection in chronic renal failure. 尿路感染在慢性肾衰竭中的作用。
Clinical and experimental dialysis and apheresis Pub Date : 1981-01-01 DOI: 10.3109/08860228109076013
R B Freeman
{"title":"The role of urinary tract infection in chronic renal failure.","authors":"R B Freeman","doi":"10.3109/08860228109076013","DOIUrl":"https://doi.org/10.3109/08860228109076013","url":null,"abstract":"<p><p>The importance of bacterial infection as a major cause of progressive renal failure has become less prominent as long-term studies have failed to show progressive renal disease in bacteriuric humans. Functional or anatomic abnormalities of the urinary tract are necessary to perpetuate infection and cause renal scars and renal failure. In children, the most common abnormality is reflux. Sterile reflux that extends into the renal collecting ducts may cause scars previously called atrophic pyelonephritis. This entity is now referred to as reflux nephropathy. Other predisposing factors may lead to end-stage disease in a small proportion of bacteriuric patients. The most common are obstructive uropathy and calculus disease. Bacteriuria is difficult to eradicate in maintenance hemodialysis patients and may require bilateral nephrectomy. In transplant recipients, bacteriuria is common and has been associated with rejection and loss of allograft.</p>","PeriodicalId":79208,"journal":{"name":"Clinical and experimental dialysis and apheresis","volume":null,"pages":null},"PeriodicalIF":0.0,"publicationDate":"1981-01-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://sci-hub-pdf.com/10.3109/08860228109076013","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"18084622","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
引用次数: 8
Recovery from acute renal failure. 从急性肾功能衰竭恢复。
Clinical and experimental dialysis and apheresis Pub Date : 1981-01-01 DOI: 10.3109/08860228109076011
C M Kjellstrand, C Gornick, T Davin
{"title":"Recovery from acute renal failure.","authors":"C M Kjellstrand,&nbsp;C Gornick,&nbsp;T Davin","doi":"10.3109/08860228109076011","DOIUrl":"https://doi.org/10.3109/08860228109076011","url":null,"abstract":"<p><p>Acute tubular necrosis is the most common cause of acute renal failure making up two-thirds of such cases. Mortality is best correlated to basic disease. Surgery, particularly in the abdomen, carries an unusually sinister prognosis. The influence of age on outcome is controversial. Intensified dialysis, early reoperations, hyperalimentation, and possibly continuous dialysis and antibiotic barrage deserves close investigation as tools of improving survival. Almost all surviving patients recover renal function within 30 days and beyond two months recovery almost never occurs. Approximately 3% of the patients initially suspected of having acute tubular necrosis will need chronic hemodialysis indefinitely or have a transplant to regain renal function. The older patient seems to be more susceptible to this problem. Delayed recovery and chronic renal failure is unusual. High dose loop diuretic therapy and hyperalimentation with intravenous amino acids may shorten the time for recovery, although considerable controversy exists.</p>","PeriodicalId":79208,"journal":{"name":"Clinical and experimental dialysis and apheresis","volume":null,"pages":null},"PeriodicalIF":0.0,"publicationDate":"1981-01-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://sci-hub-pdf.com/10.3109/08860228109076011","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"18344671","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
引用次数: 90
Disturbance of serum lipid metabolism in acute renal failure. 急性肾功能衰竭患者血清脂质代谢紊乱。
Clinical and experimental dialysis and apheresis Pub Date : 1981-01-01 DOI: 10.3109/08860228109076027
F Ogata, Y Hirasawa, S Takahashi, M Tajiri, K Kanno, T Deguchi, T Nakamura
{"title":"Disturbance of serum lipid metabolism in acute renal failure.","authors":"F Ogata,&nbsp;Y Hirasawa,&nbsp;S Takahashi,&nbsp;M Tajiri,&nbsp;K Kanno,&nbsp;T Deguchi,&nbsp;T Nakamura","doi":"10.3109/08860228109076027","DOIUrl":"https://doi.org/10.3109/08860228109076027","url":null,"abstract":"<p><p>Serial studies of serum lipids were performed on five patients with acute renal failure (ARF) due to five different causes (Of five patients one did not achieve complete recovery.). There were striking alterations in serum lipid levels at the period of oliguria in all patients, characterized by an increase in triglycerides (TG) and an extreme decrease in HDL-cholesterol (HDL-C). These conditions gradually returned to normal as the patients improved. The restoration to normal of the altered lipid levels were preceded by normalization of serum creatinine (S-Cr) and followed by creatinine clearance (Ccr). These fluctuational patterns of the lipid levels in the course of illness were observed similarly in all patients who recovered, despite the difference in the cause of their diseases. Improvement of the lipid metabolism was not observed in the one patient who did not recover. These results suggest that the alteration in lipid metabolism of ARF is due to renal impairment and not related to uremic state per se.</p>","PeriodicalId":79208,"journal":{"name":"Clinical and experimental dialysis and apheresis","volume":null,"pages":null},"PeriodicalIF":0.0,"publicationDate":"1981-01-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://sci-hub-pdf.com/10.3109/08860228109076027","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"18352571","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
引用次数: 4
In vitro simulations of peritoneal dialysis: a systematic approach to demonstrating isonatric contraction with sodium dextran sulfate. 腹膜透析的体外模拟:一种系统的方法来证明与葡聚糖硫酸钠等钠收缩。
Clinical and experimental dialysis and apheresis Pub Date : 1981-01-01 DOI: 10.3109/08860228109076023
A D Harding, K D Nolph
{"title":"In vitro simulations of peritoneal dialysis: a systematic approach to demonstrating isonatric contraction with sodium dextran sulfate.","authors":"A D Harding,&nbsp;K D Nolph","doi":"10.3109/08860228109076023","DOIUrl":"https://doi.org/10.3109/08860228109076023","url":null,"abstract":"<p><p>Two sodium dextran sulfates (DS) were investigated as possible osmotic agents for use in peritoneal dialysis using an in vitro model. Initial studies with a medium molecular weight DS (Mn = 4.000) showed that sodium clearances were higher than water clearances and that both are linear functions of the concentration of polymer. In another series of studies using a low molecular weight DS (Mn = 5,000), it was found that sodium clearances decrease linearly as sodium chloride (NaCL) is added to the initial dialysate solution with only a slight increase in ultrafiltration. By plotting sodium clearances and water clearances as functions of added NaCl at constant polymer concentration, the point at which sodium clearance and water clearances are equal can be determined and, at that point, isonatric contraction can be demonstrated in the vitro model.</p>","PeriodicalId":79208,"journal":{"name":"Clinical and experimental dialysis and apheresis","volume":null,"pages":null},"PeriodicalIF":0.0,"publicationDate":"1981-01-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://sci-hub-pdf.com/10.3109/08860228109076023","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"17239602","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
引用次数: 0
Echocardiographic detection of cardiac effects of arterio-venous dialysis fistula. 超声心动图检测动静脉透析瘘对心脏的影响。
Clinical and experimental dialysis and apheresis Pub Date : 1981-01-01 DOI: 10.3109/08860228109076018
A Dal Canton, S Maione, D Russo, C Teti, C Serino, R Gallo, V E Andreucci
{"title":"Echocardiographic detection of cardiac effects of arterio-venous dialysis fistula.","authors":"A Dal Canton,&nbsp;S Maione,&nbsp;D Russo,&nbsp;C Teti,&nbsp;C Serino,&nbsp;R Gallo,&nbsp;V E Andreucci","doi":"10.3109/08860228109076018","DOIUrl":"https://doi.org/10.3109/08860228109076018","url":null,"abstract":"<p><p>The effects of the dialysis fistula were evaluated by echocardiography in 10 patients with terminal renal failure. Echocardiography was performed the day before creation of the fistula (E0) and was then repeated 24 hours after surgery (E1), the day before the first dialysis (E2), the day following the fourth dialysis (E3) and, finally, after 6 months of RDT (E4). Left ventricular internal diameter was measured both in diastole (LVIDd) and in systole (LVIDs), allowing calculation of ventricular diameter percent shortening (LVID%), of ventricular diastolic (EDV) and systolic volume (ESV). In E1 the fistula caused a significant increase in LVIDd, accounting for a rise also in EDV. This ventricular dilation persisted in E2, but was abolished in E3 and in E4. LVIDs, ESV, and LVID% were unmodified throughout the study. these results show that the dialysis fistula causes an immediate rise in cardiac pre-load, which is rapidly reversed by RDT.</p>","PeriodicalId":79208,"journal":{"name":"Clinical and experimental dialysis and apheresis","volume":null,"pages":null},"PeriodicalIF":0.0,"publicationDate":"1981-01-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://sci-hub-pdf.com/10.3109/08860228109076018","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"18345584","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
引用次数: 5
Septic and aseptic olecranon bursitis in patients on maintenance hemodialysis. 维持血液透析患者的脓毒性和无菌性鹰嘴滑囊炎。
Clinical and experimental dialysis and apheresis Pub Date : 1981-01-01 DOI: 10.3109/08860228109076032
V K Jain, R V Cestero, J Baum
{"title":"Septic and aseptic olecranon bursitis in patients on maintenance hemodialysis.","authors":"V K Jain,&nbsp;R V Cestero,&nbsp;J Baum","doi":"10.3109/08860228109076032","DOIUrl":"https://doi.org/10.3109/08860228109076032","url":null,"abstract":"<p><p>Over a period of four years, seven of 110 maintenance hemodialysis patients developed olecranon bursitis. In all patients it appeared in the arm on which AV access was constructed but one patient also had bilateral bursitis. None of the patients gave a history of trauma or gout. Despite similar clinical presentation, culture of bursal aspirate demonstrated septic bursitis in two patients. (One diabetic, the other on steroids). Sustained pressure on the olecranon bursa during maintenance hemodialysis and/or milk trauma in the anticoagulated state may be responsible for the lesion. Infection can occur in susceptible individuals despite no visible skin infection. Local steroid instillation and patient education to avoid putting pressure on bursa during dialysis was effective therapy in the aseptic group. Multiple aspirations and antibiotics were necessary in the septic group.</p>","PeriodicalId":79208,"journal":{"name":"Clinical and experimental dialysis and apheresis","volume":null,"pages":null},"PeriodicalIF":0.0,"publicationDate":"1981-01-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://sci-hub-pdf.com/10.3109/08860228109076032","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"18359120","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
引用次数: 5
Fourth Rogosin Kidney Center Symposium. Prevention of renal failure. 第四届罗戈辛肾脏中心研讨会。预防肾衰竭。
Clinical and experimental dialysis and apheresis Pub Date : 1981-01-01
{"title":"Fourth Rogosin Kidney Center Symposium. Prevention of renal failure.","authors":"","doi":"","DOIUrl":"","url":null,"abstract":"","PeriodicalId":79208,"journal":{"name":"Clinical and experimental dialysis and apheresis","volume":null,"pages":null},"PeriodicalIF":0.0,"publicationDate":"1981-01-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"18344669","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
引用次数: 0
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