Journal of clinical neuro-ophthalmology最新文献_第3页

Carotid artery disease in vascular ocular syndromes. 血管性眼综合征中的颈动脉疾病。

Journal of clinical neuro-ophthalmology Pub Date : 1993-09-01

M Müller, K Wessel, E Mehdorn, D Kömpf, C M Kessler

{"title":"Carotid artery disease in vascular ocular syndromes.","authors":"M Müller, K Wessel, E Mehdorn, D Kömpf, C M Kessler","doi":"","DOIUrl":"","url":null,"abstract":"We prospectively investigated 83 consecutive patients with vascular ocular syndromes: 19 suffered from amaurosis fugax attacks, 23 had occlusions of the central retinal artery or a branch retinal artery occlusion, 26 had a central retinal vein occlusion or a branch retinal vein occlusion, and another 15 exhibited an anterior ischemic optic neuropathy. In 5 patients bilateral symptoms occurred; thus a total of 88 eyes were affected. All patients underwent a neurological examination and ultrasound investigations of the carotid arteries, including continuous wave (cw)-Doppler-sonography and duplex ultrasound. Stenosis of more than 50% diameter reduction and occlusion of the internal carotid artery ipsilateral to the symptomatic eye were significantly more frequent in amaurosis fugax attacks and central or branch retinal artery occlusion than in central or branch retinal vein occlusion or anterior ischemic optic neuropathy (p < .025). Additionally, the analysis of plaque surface and echogenicity of the plaques on the affected side with a high-resolution duplex scan uncovered that ulcerated plaque surfaces and plaques with a heterogeneous echogenicity were found significantly more frequent in the internal carotid arteries of patients with amaurosis fugax attacks and central or branch retinal artery occlusions than in patients with anterior ischemic optic neuropathy (p < .04) or central and branch retinal vein occlusion (p < .025). We conclude that amaurosis fugax attacks and central retinal artery or branch retinal artery occlusions are due to arterio-arterial embolization from ulcerated and heterogeneous carotid artery plaques.(ABSTRACT TRUNCATED AT 250 WORDS)","PeriodicalId":77200,"journal":{"name":"Journal of clinical neuro-ophthalmology","volume":"13 3","pages":"175-80"},"PeriodicalIF":0.0,"publicationDate":"1993-09-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"19099689","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}

引用次数: 0

Delayed oculomotor palsy from focal subarachnoid hematoma. 局灶性蛛网膜下腔血肿引起的迟发性动眼性麻痹。

Journal of clinical neuro-ophthalmology Pub Date : 1993-09-01

T Y Shih

引用次数: 0

Pseudotumor cerebri secondary to dural sinus thrombosis in neurosarcoidosis. 神经结节病继发于硬脑膜窦血栓形成的假脑瘤。

Journal of clinical neuro-ophthalmology Pub Date : 1993-09-01

Y A Akova, T Kansu, S Duman

引用次数: 0

Do thalamic lesions really cause vertical gaze palsies? 丘脑病变真的会导致垂直凝视瘫痪吗?

Journal of clinical neuro-ophthalmology Pub Date : 1993-09-01

R M Siatkowski, N J Schatz, T P Sellitti, S L Galetta, J S Glaser

引用次数: 0

Amaurotic mydriasis. 黑蒙性瞳孔放大。

Journal of clinical neuro-ophthalmology Pub Date : 1993-09-01

F E Lepore

引用次数: 0

Ocular pulsation in neurofibromatosis. A clinical/neuroradiologic correlation. 神经纤维瘤病的眼搏动。临床/神经放射学相关性。

Journal of clinical neuro-ophthalmology Pub Date : 1993-09-01

J L Smith, B C Bowen

引用次数: 0

Spontaneous rupture of an intraorbital hydatid cyst. A rare cause of acute visual loss. 眼眶内包虫囊肿自发性破裂。一种罕见的急性视力丧失的病因。

Journal of clinical neuro-ophthalmology Pub Date : 1993-06-01

M M Ozek, M N Pamir, A Sav

引用次数: 0

Superior segmental optic hypoplasia in identical twins. 同卵双胞胎的上节段性视发育不全。

Journal of clinical neuro-ophthalmology Pub Date : 1993-06-01

M C Brodsky, G T Schroeder, R Ford

引用次数: 0

Cranial neuropathy heralding otherwise occult AIDS-related large cell lymphoma. 颅神经病变预示着隐匿的艾滋病相关大细胞淋巴瘤。

Journal of clinical neuro-ophthalmology Pub Date : 1993-06-01

J R Berger, M Flaster, N Schatz, D Droller, P Benedetto, R Poblete, M J Post

{"title":"Cranial neuropathy heralding otherwise occult AIDS-related large cell lymphoma.","authors":"J R Berger, M Flaster, N Schatz, D Droller, P Benedetto, R Poblete, M J Post","doi":"","DOIUrl":"","url":null,"abstract":"Three HIV-infected patients developed cranial neuropathy as the initial manifestation of an AIDS-related large cell lymphoma. All were homosexual men known to be HIV seropositive for 3 to 4.5 years. At the time of presentation for neurological disease, the CD4 T-lymphocyte count was < 400 cells/mm3 in each. Initial manifestations were retro-orbital headache and oculomotor nerve palsy in two and an abducens nerve palsy in the other. Repeatedly negative CSF cytologies and recovery of the cranial neuropathy obscured the diagnosis. These patients illustrate that cranial neuropathy with HIV infection may herald the presence of an occult large cell lymphoma. Spontaneous or corticosteroid-associated improvement of the cranial neuropathy, absence of abnormalities on brain imaging studies, and negative CSF cytologies do not exclude this diagnosis. We suggest that a diligent and repeated search for lymphoma be considered in HIV-infected patients presenting with cranial neuropathy, including repeated CSF examinations, MRI of brain and spine (T1 and T2) with and without gadolinium enhancement, chest and abdominal CT scans, and bone marrow biopsy.","PeriodicalId":77200,"journal":{"name":"Journal of clinical neuro-ophthalmology","volume":"13 2","pages":"113-8"},"PeriodicalIF":0.0,"publicationDate":"1993-06-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"19094232","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}

引用次数: 0

Acute zonal occult outer retinopathy. Donders Lecture: The Netherlands Ophthalmological Society, Maastricht, Holland, June 19, 1992. 急性区域性隐蔽性外视网膜病变。Donders讲座:荷兰眼科学会，马斯特里赫特，荷兰，1992年6月19日。

Journal of clinical neuro-ophthalmology Pub Date : 1993-06-01

J D Gass

{"title":"Acute zonal occult outer retinopathy. Donders Lecture: The Netherlands Ophthalmological Society, Maastricht, Holland, June 19, 1992.","authors":"J D Gass","doi":"","DOIUrl":"","url":null,"abstract":"Purpose: This report describes 13 patients, predominantly young women, with a syndrome characterized by rapid loss of one or more large zones of outer retinal function, photopsia, minimal funduscopic changes, and electroretinographic abnormalities affecting one or both eyes. All patients on follow-up examination had persistent visual field defects, and most had chronic photopsia and zones of pigment epithelial atrophy. Evidence is presented that these patients probably represent part of the spectrum of a single disorder that includes the multiple evanescent white-dot syndrome (MEWDS), acute idiopathic blind-spot-enlargement syndrome (AIBSES), acute macular neuroretinopathy (AMN), and the pseudo-presumed ocular histoplasmosis syndrome (P-POHS).Methods: The medical records of these 13 patients and 2 additional patients, who developed, in addition to the features of this syndrome, funduscopic changes typical of MEWDS, AMN, and P-POHS, were reviewed and follow-up obtained.Results: These patients had extensive unrewarding medical and neurological investigations because of suspected diagnoses, including central nervous system disorders, cancer-associated retinopathy, retinal vasculitis, diffuse unilateral subacute neuroretinitis, and tapetoretinal degenerations. Although most patients retained good visual acuity, all had permanent visual field loss that in some cases was severe. The cause of the disorder was not determined. No effective treatment was found.Conclusions: Acute visual loss and photopsia in these patients is probably caused by damage to large zones of the outer retina that appears unaffected ophthalmoscopically. Electroretinography is important in early diagnosis. Future investigations probably will reveal further evidence linking this disorder to MEWDS, AIBSES, AMN, and P-POHS.","PeriodicalId":77200,"journal":{"name":"Journal of clinical neuro-ophthalmology","volume":"13 2","pages":"79-97"},"PeriodicalIF":0.0,"publicationDate":"1993-06-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"19326731","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}

引用次数: 0