{"title":"Pulseless (Takayasu) disease with ophthalmic manifestations.","authors":"J R Lewis, J S Glaser, N J Schatz, D G Hutson","doi":"","DOIUrl":"","url":null,"abstract":"<p><p>Pulseless disease (PD) is a rare disorder in which inflammation of the aorta and its major branches leads to stenosis or occlusion of these arteries. It mainly affects young Oriental women, who suffer chronic ischemic injury to tissues of the brain, orbits, upper limbs, myocardium, and kidneys. The ophthalmologic features of pulseless disease tend to be late manifestations, and can include ischemia of the retina, choroid, and anterior segment. The inflammatory process may be reversed in early stages with systemic corticosteroids, but, more frequently, significant arterial stenosis necessitates arterial bypass surgery. A 59-year-old Caucasian woman with stenosis of all four major cervical arteries presented with recurrent blurred vision, syncope, mental obtundation, and a remarkable funduscopic appearance due to bilateral orbital hypoperfusion. Her acute symptoms improved slightly on high-dose systemic corticosteroids, and then resolved completely following arterial bypass surgery.</p>","PeriodicalId":77200,"journal":{"name":"Journal of clinical neuro-ophthalmology","volume":"13 4","pages":"242-9"},"PeriodicalIF":0.0,"publicationDate":"1993-12-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"18903190","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
{"title":"Iron mydriasis. Pupillary paresis from occult intraocular foreign body.","authors":"M L Monteiro, J R Coppeto, J A Milani","doi":"","DOIUrl":"","url":null,"abstract":"<p><p>Two patients presented with unilateral dilated and poorly reactive pupils and were found to have a previously unsuspected intraocular iron foreign body. Both of them had normal vision and one had normal color of the iris so that the condition was missed by several physicians. The pupils reacted normally to pilocarpine 1% and also showed response to dilute (0.1%) pilocarpine. There was no response to phospholine iodide. These findings indicate that the mydriasis was due to a local siderotic parasympathetic neuropathy of the pupil.</p>","PeriodicalId":77200,"journal":{"name":"Journal of clinical neuro-ophthalmology","volume":"13 4","pages":"254-7"},"PeriodicalIF":0.0,"publicationDate":"1993-12-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"19105544","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
{"title":"Duane's syndrome with giant aneurysm of the vertebral basilar arterial junction.","authors":"P Hedera, R P Friedland","doi":"","DOIUrl":"","url":null,"abstract":"<p><p>A 36-year-old man who had signs of Duane's syndrome since birth developed symptoms of a posterior fossa mass. Angiography revealed a giant aneurysm in the area of the vertebral basilar junction. The association of these two anomalies provides support for the vascular origin of some cases of Duane's syndrome due to vascular hypofunction during the 4th and 5th weeks of embryogenesis.</p>","PeriodicalId":77200,"journal":{"name":"Journal of clinical neuro-ophthalmology","volume":"13 4","pages":"271-4"},"PeriodicalIF":0.0,"publicationDate":"1993-12-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"19105547","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
S L Galetta, L G Gray, E C Raps, R I Grossman, N J Schatz
{"title":"Unilateral ptosis and contralateral eyelid retraction from a thalamic-midbrain infarction. Magnetic resonance imaging correlation.","authors":"S L Galetta, L G Gray, E C Raps, R I Grossman, N J Schatz","doi":"","DOIUrl":"","url":null,"abstract":"<p><p>We report a patient with a third nerve palsy and contralateral eyelid retraction from a thalamic-midbrain infarction. Magnetic resonance imaging confirms that this unusual clinical combination, previously termed \"the plus-minus lid syndrome,\" results from a lesion in the region of the nucleus of the posterior commissure with extension to the third nerve fascicle.</p>","PeriodicalId":77200,"journal":{"name":"Journal of clinical neuro-ophthalmology","volume":"13 4","pages":"221-4"},"PeriodicalIF":0.0,"publicationDate":"1993-12-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"19106933","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
{"title":"Isolated palsy of the superior branch of the oculomotor nerve due to chronic erosive sphenoid sinusitis.","authors":"L Stefanis, S Przedborski","doi":"","DOIUrl":"","url":null,"abstract":"<p><p>We report one case with isolated unilateral superior branch oculomotor nerve palsy due to an erosive sphenoid sinusitis. Within 8 weeks after surgical drainage of the sphenoid sinusitis, the patient recovered fully from the superior branch oculomotor palsy. In view of the dramatic clinical improvement that followed surgery, we hypothesized a compression of the superior branch of the oculomotor nerve by the sphenoidal abscess. To our knowledge, this is the first reported case of a superior branch oculomotor nerve palsy related to an erosive sinusitis and cured by sinusotomy, a safe and simple surgical procedure. Thus, we believe that our observation bears some practical implications of clinical importance in the management of patients who present partial oculomotor nerve palsy.</p>","PeriodicalId":77200,"journal":{"name":"Journal of clinical neuro-ophthalmology","volume":"13 4","pages":"229-31"},"PeriodicalIF":0.0,"publicationDate":"1993-12-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"19105540","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
{"title":"Visual loss due to progressive multifocal leukoencephalopathy in a heart transplant patient.","authors":"A R Lewis, L B Kline, N B Pinkard","doi":"","DOIUrl":"","url":null,"abstract":"<p><p>A 59-year-old male heart transplant recipient experienced progressive visual loss following routine cataract surgery with intraocular lens implantation. Cranial magnetic resonance (MR) imaging suggested and brain biopsy confirmed the cause to be progressive multifocal leukoencephalopathy (PML). The patient died 2 months following cataract surgery. This case illustrates that visual failure may be the initial manifestation of PML, and the ophthalmologist should be aware of this central demyelinating disorder when dealing with immunocompromised patients.</p>","PeriodicalId":77200,"journal":{"name":"Journal of clinical neuro-ophthalmology","volume":"13 4","pages":"237-41"},"PeriodicalIF":0.0,"publicationDate":"1993-12-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"19105542","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
{"title":"Consecutive oculomotor nerve palsy from a de novo cerebral aneurysm.","authors":"G G Striph","doi":"","DOIUrl":"","url":null,"abstract":"<p><p>A 26-year-old woman presented with a partial left oculomotor nerve palsy and an acute subarachnoid hemorrhage. Bilateral angiography showed a left posterior communicating artery aneurysm and a normal right anterior circulation. The aneurysm was successfully clipped. At 4 1/2 years later, she had an acute, nearly complete, right oculomotor nerve palsy. Repeat bilateral angiography showed a right posterior communicating artery aneurysm, in a previously angiographically normal region, and continued closure of the original left-sided aneurysm. New aneurysms may develop from angiographically normal locations. It is possible that certain patients are more susceptible than average to aneurysm formation or that aneurysm repair leads to a higher rate of new aneurysm occurrence. The previous cases of de novo aneurysms are summarized and the implications for repeat aneurysm screening, using current technology, are discussed.</p>","PeriodicalId":77200,"journal":{"name":"Journal of clinical neuro-ophthalmology","volume":"13 3","pages":"181-7"},"PeriodicalIF":0.0,"publicationDate":"1993-09-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"19099626","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
{"title":"Cysticercosis: unusual neuro-ophthalmologic signs.","authors":"J R Keane","doi":"","DOIUrl":"","url":null,"abstract":"<p><p>Papilledema and pretectal signs are the usual neuro-ophthalmological manifestations of cysticercosis. The following patients with severe posterior fossa involvement exhibited uncommon signs of bilateral fourth nerve palsy, facial myokymia, upbeat nystagmus, periodic alternating nystagmus, and rhythmic oculopalatal myoclonus.</p>","PeriodicalId":77200,"journal":{"name":"Journal of clinical neuro-ophthalmology","volume":"13 3","pages":"194-9"},"PeriodicalIF":0.0,"publicationDate":"1993-09-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"19099629","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
{"title":"Severe enophthalmos following intracranial decompression in a von Recklinghausen patient.","authors":"B G Haik, M Pohlod","doi":"","DOIUrl":"","url":null,"abstract":"<p><p>A patient with neurofibromatosis type I and absence of the sphenoid wing developed proptosis of the right eye secondary to expansion of the intracranial contents from an intracranial astrocytoma associated with cerebral edema and hydrocephalus. Marked enophthalmos developed following excision of the intracranial tumor and placement of a ventriculoperitoneal shunt. Absence of the sphenoid bone limited the orbit's ability to halt posterior refraction of the orbital contents when the intracranial volume decreased.</p>","PeriodicalId":77200,"journal":{"name":"Journal of clinical neuro-ophthalmology","volume":"13 3","pages":"171-4"},"PeriodicalIF":0.0,"publicationDate":"1993-09-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"19099688","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
V Preac-Mursic, H W Pfister, H Spiegel, R Burk, B Wilske, S Reinhardt, R Böhmer
{"title":"First isolation of Borrelia burgdorferi from an iris biopsy.","authors":"V Preac-Mursic, H W Pfister, H Spiegel, R Burk, B Wilske, S Reinhardt, R Böhmer","doi":"","DOIUrl":"","url":null,"abstract":"<p><p>The persistence of Borrelia burgdorferi in six patients is described. Borrelia burgdorferi has been cultivated from iris biopsy, skin biopsy, and cerebrospinal fluid also after antibiotic therapy for Lyme borreliosis. Lyme Serology: IgG antibodies to B. burgdorferi were positive, IgM negative in four patients; in two patients both IgM and IgG were negative. Antibiotic therapy may abrogate the antibody response to the infection as shown by our results. Patients may have subclinical or clinical disease without diagnostic antibody titers. Persistence of B. burgdorferi cannot be excluded when the serum is negative for antibodies against it.</p>","PeriodicalId":77200,"journal":{"name":"Journal of clinical neuro-ophthalmology","volume":"13 3","pages":"155-61; discussion 162"},"PeriodicalIF":0.0,"publicationDate":"1993-09-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"19099686","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}