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Psychiatria clinica最新文献

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Motor reactivity, pain threshold and effects of sleep deprivation in unipolar depressives. 运动反应，痛觉阈和睡眠剥夺对单极抑郁症的影响。
Psychiatria clinica Pub Date : 1981-01-01 DOI: 10.1159/000283911
G Bezzi, P Pinelli, P Tosca
{"title":"Motor reactivity, pain threshold and effects of sleep deprivation in unipolar depressives.","authors":"G Bezzi,&nbsp;P Pinelli,&nbsp;P Tosca","doi":"10.1159/000283911","DOIUrl":"https://doi.org/10.1159/000283911","url":null,"abstract":"<p><p>The authors studied the motor reactivity by means of serial choice reaction, electromyography of simple reactions, and defense reflexes in normal and in depressed patients. Significant differences were observed between normal and depressed patients, especially in those with retardation. The 'retarded' depressives exhibited higher pain threshold values, a lower number of motor reactions, and more prolonged reaction times. 2 normal and 4 depressed patients were submitted to the same tests after 1 night's total sleep deprivation. In normals negative effects were found in the levels of vigilance and endurance. In depressed patients, at variance with normals, the number of motor reactions increased, the reaction time decreased, and the rate of basal motor innervation improved, tending to normalization. On the contrary, pain threshold underwent the same type of changes as in controls. Sleep deprivation seemed to specifically reduce retardation phenomena in depressives, thus strongly improving motor reactivity.</p>","PeriodicalId":76382,"journal":{"name":"Psychiatria clinica","volume":"14 3","pages":"150-60"},"PeriodicalIF":0.0,"publicationDate":"1981-01-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://sci-hub-pdf.com/10.1159/000283911","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"18340860","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
引用次数: 17
The course of depression for different types of schizophrenia. 不同类型精神分裂症的抑郁病程。
Psychiatria clinica Pub Date : 1981-01-01 DOI: 10.1159/000283919
F Strian, R Heger, C Klicpera
{"title":"The course of depression for different types of schizophrenia.","authors":"F Strian,&nbsp;R Heger,&nbsp;C Klicpera","doi":"10.1159/000283919","DOIUrl":"https://doi.org/10.1159/000283919","url":null,"abstract":"<p><p>During 197 courses of treatment for schizophrenia, depressive mood was evaluated every other day with a self-rating mood scale. The different patterns of depression identified in an earlier study could be correlated with the diagnostic subgroups of schizophrenia in which the patients were classified. Hebephrenic affect disturbance appeared as temporal discontinuity of depressive mood during the courses of treatment. Patients with a paranoid type of schizophrenia were the least depressed and had the fewest fluctuations of mood. Although patients with a schizoaffective type of schizophrenia exhibited characteristic changes between manic and depressive mood, their mood within a manic or depressive period was more stable than that of any other subgroup. The differences in depressive course are discussed in terms of their symptomatological and diagnostic significance.</p>","PeriodicalId":76382,"journal":{"name":"Psychiatria clinica","volume":"14 4","pages":"205-14"},"PeriodicalIF":0.0,"publicationDate":"1981-01-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://sci-hub-pdf.com/10.1159/000283919","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"18360432","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
引用次数: 8
Psychophysiology of the reaction time experiment: a factor analytic approach. 反应时间实验的心理生理学:一种因子分析法。
Psychiatria clinica Pub Date : 1981-01-01 DOI: 10.1159/000283906
F Lolas
{"title":"Psychophysiology of the reaction time experiment: a factor analytic approach.","authors":"F Lolas","doi":"10.1159/000283906","DOIUrl":"https://doi.org/10.1159/000283906","url":null,"abstract":"<p><p>A principal component factor analysis with varimax rotation was applied to behavioral and physiological data from a group of 25 subjects comprising normals and depressives, in whom event-related slow brain potentials were recorded during performance of a fixed foreperiod, reaction time task. Five of the seven factors thus evinced were labelled 'activation', 'inhibition', 'resilience', 'depressivity-neuroticism' and 'extraversion', the remaining two factors being related to arousal in an unspecified manner. Although no clear-cut partitioning of variance into trait, state and task-related components was achieved, the contention is advanced that measurement of these factors and their patterning might contribute to psychophysiological diagnosis, provided they are substantiated by further research.</p>","PeriodicalId":76382,"journal":{"name":"Psychiatria clinica","volume":"14 2","pages":"88-95"},"PeriodicalIF":0.0,"publicationDate":"1981-01-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://sci-hub-pdf.com/10.1159/000283906","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"18268178","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
引用次数: 2
[HLA, schizophrenias and arthropathies]. HLA、精神分裂症和关节病。
Psychiatria clinica Pub Date : 1981-01-01
W F Gattaz, S Kasper, R Ewald, H Beckmann
{"title":"[HLA, schizophrenias and arthropathies].","authors":"W F Gattaz,&nbsp;S Kasper,&nbsp;R Ewald,&nbsp;H Beckmann","doi":"","DOIUrl":"","url":null,"abstract":"<p><p>Significantly more individuals with human leucocyte antigens (HLA) A9 and B27 have been identified in the group of chronic paranoid schizophrenics with early onset of the disease. It is known that individuals with HLA B27 have a markedly increased risk to fall ill from arthropathies (i.e. Bechterew's disease). Generally, it seems extremely rare that arthropathies and schizophrenia occur together in the same person. In 16 chronic paranoid schizophrenics with HLA B27 no form of arthropathy and in 288 arthropathic patients no case of schizophrenia could be detected (evidenced in a psychiatric case register). Furthermore in 131 arthropathic patients with HLA B27 no psychiatric disease (except one feeble-minded and one with alcohol problems) could be identified. On the other hand, in the group of arthropathic patients without HLA B27 the incidence of psychiatric diseases was 5 times higher than in the group with HLA B27 and so comparable to the morbidity of the normal population. It is conceivable that HLA B27 is a 'genetic marker' for arthropathy as well as for a defined subgroup of schizophrenia. These data agree with the hypothesis that schizophrenia and arthropathies are mutually exclusive in one individual.</p>","PeriodicalId":76382,"journal":{"name":"Psychiatria clinica","volume":"14 1","pages":"49-55"},"PeriodicalIF":0.0,"publicationDate":"1981-01-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"18023005","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
引用次数: 0
[Recurrence prophylaxis of affective diseases with lithium and their results]. [锂对情感性疾病复发的预防作用及其结果]。
Psychiatria clinica Pub Date : 1981-01-01
W Felber
{"title":"[Recurrence prophylaxis of affective diseases with lithium and their results].","authors":"W Felber","doi":"","DOIUrl":"","url":null,"abstract":"","PeriodicalId":76382,"journal":{"name":"Psychiatria clinica","volume":"14 3","pages":"161-6"},"PeriodicalIF":0.0,"publicationDate":"1981-01-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"18082844","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
引用次数: 0
[Genetic markers in delusional diseases in middle age]. [中年妄想症的遗传标记]
Psychiatria clinica Pub Date : 1981-01-01
V Lange
{"title":"[Genetic markers in delusional diseases in middle age].","authors":"V Lange","doi":"","DOIUrl":"","url":null,"abstract":"<p><p>In the distribution of their serum groups the totality of 48 patients with paranoid psychoses of late onset is characterized by failure of the well-known markers of manic-depressive or schizophrenic illness. However, when differentiated psychopathologically, the haptoglobin (Hp) type 2-2 as a risk factor of cyclothymic psychoses is found to be significantly overrepresented in the patient group with a cyclic axis syndrome. On the other hand, an excess of Hp 1-1 can be observed and statistically confirmed in comparison with the population level among the patients offering a schizophrenic axis syndrome or resting unclassified. Possibly, the Hp serum groups play a role in the bioregulation of emotionality. The 'noncyclic' group does not show the marker traits of process psychoses, but in this sampling there is, in contrast to the 'cyclics,' a higher concentration of the immunoglobulin G serum level on the statistical border line, which perhaps is caused by destructive factors. The results correspond with the special state of the examined psychoses basing on biological determinants of different origin.</p>","PeriodicalId":76382,"journal":{"name":"Psychiatria clinica","volume":"14 1","pages":"23-34"},"PeriodicalIF":0.0,"publicationDate":"1981-01-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"17993394","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
引用次数: 0
Delusional misidentification syndromes and cerebral 'dysrhythmia'. 妄想性误诊综合征和脑性“心律失常”。
Psychiatria clinica Pub Date : 1981-01-01 DOI: 10.1159/000283922
G N Christodoulou, S Malliara-Loulakaki
{"title":"Delusional misidentification syndromes and cerebral 'dysrhythmia'.","authors":"G N Christodoulou,&nbsp;S Malliara-Loulakaki","doi":"10.1159/000283922","DOIUrl":"https://doi.org/10.1159/000283922","url":null,"abstract":"<p><p>Electroencephalographic investigation of 21 patients with delusional misidentification syndromes (11 with the syndrome of Capgras, 7 with the syndrome of Frégoli, 2 with the syndrome of intermetamorphosis and 1 with the syndrome of subjective doubles) revealed a high abundance and severity of abnormalities. The findings are discussed within the framework of a possible organic (and more specifically dysrhythmic) contribution to the pathogenesis of these syndromes.</p>","PeriodicalId":76382,"journal":{"name":"Psychiatria clinica","volume":"14 4","pages":"245-51"},"PeriodicalIF":0.0,"publicationDate":"1981-01-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://sci-hub-pdf.com/10.1159/000283922","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"18359229","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
引用次数: 33
Cerebrospinal fluid amine metabolites in delirium tremens. 震颤性谵妄的脑脊液胺代谢物。
Psychiatria clinica Pub Date : 1981-01-01 DOI: 10.1159/000283913
C M Banki, G Molnar
{"title":"Cerebrospinal fluid amine metabolites in delirium tremens.","authors":"C M Banki,&nbsp;G Molnar","doi":"10.1159/000283913","DOIUrl":"https://doi.org/10.1159/000283913","url":null,"abstract":"<p><p>Cerebrospinal fluid (CSF) 5-hydroxyindoleacetic acid (5HIAA), homovanillic acid (HVA) and tryptophan (TRY) were measured in 14 male alcohol-dependent patients with delirium tremens. Lumbar punctures were performed immediately after admission following a standardized psychiatric examination and symptom rating in a drug-free state. Results were compared with a control group consisting of 32 neurological patients with only peripheral disorders, excluding spinal processes and abnormal routine CSF findings. All three substances were significantly increased in delirium tremens; 5HIAA showed the most marked and TRY the least pronounced increase. The statistical correction for age, height and body weight did not decrease but somewhat increased the differences. Duration of alcohol abuse did not account for the observed metabolic changes; severity of delirium tremens, however, correlated significantly with the 5HIAA and to a lesser degree with the HVA level. The further analysis revealed a differential relationship of the amine metabolite concentrations to some prominent symptoms: agitation was significantly dependent only on the HVA level while disorientation and hallucination seemed to be determined mostly by the serotonin metabolite 5HIAA in the CSF. TRY concentration proved to be unrelated to either global severity or any of these symptoms.</p>","PeriodicalId":76382,"journal":{"name":"Psychiatria clinica","volume":"14 3","pages":"167-77"},"PeriodicalIF":0.0,"publicationDate":"1981-01-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://sci-hub-pdf.com/10.1159/000283913","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"17239245","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
引用次数: 8
Epidemic faintness: a psychophysiological investigation. 流行性昏厥:一项心理生理学调查。
Psychiatria clinica Pub Date : 1981-01-01 DOI: 10.1159/000283920
J C Boulougouris, A D Rabavilas, C N Stefanis, N Vaidakis, D G Tabouratzis
{"title":"Epidemic faintness: a psychophysiological investigation.","authors":"J C Boulougouris,&nbsp;A D Rabavilas,&nbsp;C N Stefanis,&nbsp;N Vaidakis,&nbsp;D G Tabouratzis","doi":"10.1159/000283920","DOIUrl":"https://doi.org/10.1159/000283920","url":null,"abstract":"<p><p>A psychophysiological assessment was carried out in 16 telephone operators, who fainted during an epidemic, and in 16 control subjects. Heart rate, skin conductance level, number of spontaneous fluctuations and pulse volume were measured at rest and during periods of auditory and visual stimulation. All subjects completed rating scales on anxiety, depression, anxiety experienced during experimental procedure and personality inventories. Analysis of data support the notion that the fainting episodes represent a form of transitory anxiety attacks in response to environmental stress and are not related either to hysteria or to anxiety state.</p>","PeriodicalId":76382,"journal":{"name":"Psychiatria clinica","volume":"14 4","pages":"215-25"},"PeriodicalIF":0.0,"publicationDate":"1981-01-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://sci-hub-pdf.com/10.1159/000283920","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"18360433","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
引用次数: 8
[Attempt at a classification of manic-depressive disease course a communications pathology principle]. [基于交流病理学原理的躁狂抑郁症病程分类的尝试]。
Psychiatria clinica Pub Date : 1981-01-01
B Mitterauer
{"title":"[Attempt at a classification of manic-depressive disease course a communications pathology principle].","authors":"B Mitterauer","doi":"","DOIUrl":"","url":null,"abstract":"<p><p>Delusion, attempted suicide and suicide are represented as phenomena of pathological communication by means of system theory. On this basis the longitudinal observation and treatment of 158 syntonically delusive manic depressives is reported and classified. Here five typical courses of illness can be formulated in terms of communication pathology. The consequences which follow directly from this seem to be of particular interest for therapeutic measures in social psychiatry.</p>","PeriodicalId":76382,"journal":{"name":"Psychiatria clinica","volume":"14 1","pages":"35-42"},"PeriodicalIF":0.0,"publicationDate":"1981-01-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"18281582","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
引用次数: 0
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