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Intra- and extracranial circulatory changes in cigarette smokers assessed by ultrasound: transcranial Doppler (TCD) and duplex scanning investigation. 经颅多普勒(TCD)和双工扫描对吸烟者颅内外循环变化的评估。
Neurologija Pub Date : 1990-01-01
T Rundek, V Demarin, N Blazić-Cop, V Dordević
{"title":"Intra- and extracranial circulatory changes in cigarette smokers assessed by ultrasound: transcranial Doppler (TCD) and duplex scanning investigation.","authors":"T Rundek,&nbsp;V Demarin,&nbsp;N Blazić-Cop,&nbsp;V Dordević","doi":"","DOIUrl":"","url":null,"abstract":"<p><p>The group of 98 \"healthy smokers\" was analyzed in order to evaluate potential influence of cigarette smoking on intracranial blood flow disturbances and extracranial carotid intimal changes, performing transcranial Doppler (TCD) and Duplex scanning of the carotid arteries. Almost 70% of smokers analyzed in our study had some intracranial circulatory changes, predominantly in the vertebral arteries (35%). Half of smokers analyzed had some pathological intimal changes in the carotid arteries. The most frequent finding was wall thickening (23%) and then calcified (13%) and soft plaques (10%). Dose response relationship between smoking and atherosclerosis is also introduced. More than 30 cigarettes smoked per day can significantly influence plaque development and already 20 cigarettes smoked per day can cause significant intracranial blood flow disturbances. The majority of our investigated smokers were in high risk category for development of cerebrovascular disease and 5% were in a very high risk category. Cessation of cigarette smoking will eliminate it as a risk factor. A low dose-response relation and the development of tolerance produced by exposure to nicotine were also introduced.</p>","PeriodicalId":76210,"journal":{"name":"Neurologija","volume":"39 3","pages":"169-77"},"PeriodicalIF":0.0,"publicationDate":"1990-01-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"13441667","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
引用次数: 0
Gender differences in the associated complications among type-II diabetics with peripheral neuropathy. 2型糖尿病合并周围神经病变相关并发症的性别差异
Neurologija Pub Date : 1990-01-01
V R Kodali, V Seshaiah, T V Kumar
{"title":"Gender differences in the associated complications among type-II diabetics with peripheral neuropathy.","authors":"V R Kodali,&nbsp;V Seshaiah,&nbsp;T V Kumar","doi":"","DOIUrl":"","url":null,"abstract":"<p><p>Diabetic neuropathy is an intriguing problem both for the patient and the clinician, however the clinical characteristics of type-II (non insulin-dependent) diabetics with peripheral neuropathy with special reference to gender differences is not paid much attention. We studied peripheral neuropathy and related complications in 179 type-II diabetics. Peripheral neuropathy was noted in 46 of 111 men and in 46 of 68 women. The age group of subjects did not differ significantly. Duration of diabetes differed between the groups with (n = 46) and without (n = 65) peripheral neuropathy (expressed in years and as mean +/- S.E: 7.7 +/- 0.7 vs 5.5 +/- 0.7; t = 2.2; P less than 0.05) in men, whereas in women it was not significant. Higher proportion (62.5% of 24) of men with proteinuria had neuropathy. Similar findings in women were not different. We conclude i) a large percent (51.4%) of the type-II diabetics had peripheral neuropathy than realised ii) duration of diabetes and proteinuria are important risk factors associated with neuropathy especially in men and iii) the gender differences need further clinicopathologic evaluation.</p>","PeriodicalId":76210,"journal":{"name":"Neurologija","volume":"39 1","pages":"3-7"},"PeriodicalIF":0.0,"publicationDate":"1990-01-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"13416803","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
引用次数: 0
Medullomyoblastoma. 髓母细胞瘤。
Neurologija Pub Date : 1990-01-01
L Pazanin, D Jadro-Santel, Z Poljaković, K Zarković, V Hlavka
{"title":"Medullomyoblastoma.","authors":"L Pazanin,&nbsp;D Jadro-Santel,&nbsp;Z Poljaković,&nbsp;K Zarković,&nbsp;V Hlavka","doi":"","DOIUrl":"","url":null,"abstract":"<p><p>A case of medullomyoblastoma, arising in the cerebellar vermis of a five-year-old boy, is presented. The light and electron microscopic features of the tumor are described and compared to other published cases. Within typical medulloblastoma areas light microscopy revealed signs of neuroblastic differentiation thus indicating its neuroepithelial origin. No signs of glial differentiation were found. Myogenic tumor component formed irregular, distinctly separated islands although some intermingling of two cell types was observed at the borders between the two zones. Within the myogenic tumor component, electron microscopy revealed rhabdomyoblastic elements in different stages of differentiation, frequently forming small groups encompassed by the same basement membrane. No indication of a common host cell for two cell lines was observed. No mesenchymal elements other than muscle, and no indication of possible teratoid origin of this tumor were observed. Considering histogenesis of muscle elements within this neuroepithelial tumor, the origin from multipotential neural-crest-derived ectomesenchymal cells seems the most appropriate one.</p>","PeriodicalId":76210,"journal":{"name":"Neurologija","volume":"39 3","pages":"199-208"},"PeriodicalIF":0.0,"publicationDate":"1990-01-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"13440730","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
引用次数: 0
[Distal latency in motor neuron disease]. [运动神经元疾病的远端潜伏期]。
Neurologija Pub Date : 1990-01-01
B Bedenić, D Filipusić
{"title":"[Distal latency in motor neuron disease].","authors":"B Bedenić,&nbsp;D Filipusić","doi":"","DOIUrl":"","url":null,"abstract":"<p><p>In our investigation 24 patients suffering from spinal amyotrophy and 11 from amyotrophic lateral sclerosis were analyzed in order to determine the influence of these diseases on teh conduction velocity of the distal motor and sensory fibers. Both diseases belong to motor neuron diseases and are of unknown etiology. The conduction velocity was measured by using electroneurographic methods. Distal motor latency is expressed as a quotient of terminal latency that is obtained by dividing the distance between the stimulation and the registration point by the latency of response and was measured in cm/ms. It represents the conduction velocity of the distal motor fibers. Its sensory equivalent is the sensory conduction velocity S0-S1. In our work the mean values of distal motor and sensory latencies were counted and the correlation between them was examined. We also counted the percentage of the patients with the pathological values of these parameters. The results indicate that the \"motor neurone disease\" affects the conduction velocity of the distal motor and sensory fibers. The distal sensory latency was prolonged in higher percentage than the motor latency. Between motor and sensory latencies no correlation was found.</p>","PeriodicalId":76210,"journal":{"name":"Neurologija","volume":"39 4","pages":"273-84"},"PeriodicalIF":0.0,"publicationDate":"1990-01-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"13440735","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
引用次数: 0
Electroencephalographic findings in coma caused by fatal chloroquine poisoning. 致命氯喹中毒致昏迷的脑电图表现。
Neurologija Pub Date : 1990-01-01
Z Martinović, M Sasić
{"title":"Electroencephalographic findings in coma caused by fatal chloroquine poisoning.","authors":"Z Martinović,&nbsp;M Sasić","doi":"","DOIUrl":"","url":null,"abstract":"<p><p>A 19-year-old patient in deep anoxic coma after cardiopulmonary arrest due to suicidal acute overdosage of chloroquine was resuscitated at the intensive care unit. An EEG starting about 12 hours later and lasting for 93 minutes showed a rapid succession of diffuse ectopic rhythms, diffuse slow output, generalized periodic slow waves in paroxysmal episodes which preceded burst suppression patterns and definite establishment of electrocerebral silence. All these EEG patterns indicating a poor prognosis were registered about three hours before the patient died during irreversible ventricular fibrillation.</p>","PeriodicalId":76210,"journal":{"name":"Neurologija","volume":"39 4","pages":"299-304"},"PeriodicalIF":0.0,"publicationDate":"1990-01-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"13440736","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
引用次数: 0
30th international Neuropsychiatric Symposium. Pula, 10th-16th June 1990. Abstracts. 第30届国际神经精神病学研讨会。普拉，1990年6月10日至16日。摘要。
Neurologija Pub Date : 1990-01-01
{"title":"30th international Neuropsychiatric Symposium. Pula, 10th-16th June 1990. Abstracts.","authors":"","doi":"","DOIUrl":"","url":null,"abstract":"","PeriodicalId":76210,"journal":{"name":"Neurologija","volume":"39 Suppl 2 ","pages":"1-123"},"PeriodicalIF":0.0,"publicationDate":"1990-01-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"13134324","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
引用次数: 0
Haemodynamic changes in patients with subarachnoid haemorrhage assessed by Tc99m labeled albumin microspheres. 用Tc99m标记白蛋白微球评价蛛网膜下腔出血患者的血流动力学变化。
Neurologija Pub Date : 1990-01-01
M Bajc, D Bozicević, N Topuzović, M Besić, D Ivancević
{"title":"Haemodynamic changes in patients with subarachnoid haemorrhage assessed by Tc99m labeled albumin microspheres.","authors":"M Bajc,&nbsp;D Bozicević,&nbsp;N Topuzović,&nbsp;M Besić,&nbsp;D Ivancević","doi":"","DOIUrl":"","url":null,"abstract":"<p><p>The aim of the present study was the investigation of haemodynamic changes in the brain in patients after subarachnoid haemorrhage (SAH) using Tc 99 m labelled albumin microspheres (LMS). Twenty-nine patients after SAH (13 in the acute and 16 in the chronic stage of the disease) and four neurologically healthy subjects were examined. LMS were injected in the carotid during angiography and brain and lung scintigrams were taken using gamma camera. In the control subjects a diffuse and uniform distribution of the activity in the brain was found. In the patients with acute SAH \"patchy\", nonhomogenous distribution of the activity was found, ranging up to the regional absence of perfusion. This pattern was observed in the chronic stage as well, but less obvious than in the acute. In all the patients the evident trapping of LMS in the lungs was demonstrated confirming brain shunts, absent in the control group. By measuring the activity in the brain and lungs after intracarotid injection of LMS, haemodynamic changes in the patients after SAH were proved and the existence of brain shunts was demonstrated. It seems that shunts are functional anastomoses and important protective mechanism, opening when cerebral haemodynamics is altered.</p>","PeriodicalId":76210,"journal":{"name":"Neurologija","volume":"39 1","pages":"19-27"},"PeriodicalIF":0.0,"publicationDate":"1990-01-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"13416801","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
引用次数: 0
Primary trigeminal neuralgia (algophoric deafferentation hypersensitivity syndrome). 原发性三叉神经痛(algophoric de传入神经过敏综合征)。
Neurologija Pub Date : 1990-01-01
N Zurak
{"title":"Primary trigeminal neuralgia (algophoric deafferentation hypersensitivity syndrome).","authors":"N Zurak","doi":"","DOIUrl":"","url":null,"abstract":"<p><p>In the article the author presents his theory of the pathogenesis of primary trigeminal neuralgia, explaining the occurrence of this mysterious disease by algophoric deafferentation hypersensitivity. Tooth extraction is the sole cause of algophoric deafferentation hypersensitivity, which, culminating in epileptiform discharges of the trigeminal nociceptive pathway neurons, leads to clinical features of characteristic neuralgic paroxysms. Trigger mechanism is explained by ephaptic transmission between the broken fibers for phasic pain of the tooth pulp and neighbouring fibers of epicritic and proprioceptive sensitivity. The typical occurrence of periods with remission of neuralgic paroxysms the author explains by his original theory of biorhythms neogenesis with the involvement of two antagonistic neural subsystems (nociceptive and antinociceptive system). The concept is based on indisputable clinical, anatomical, pathoanatomical, experimental and pharmacologic facts, which the author quotes as the contribution to his theory.</p>","PeriodicalId":76210,"journal":{"name":"Neurologija","volume":"39 2","pages":"75-90"},"PeriodicalIF":0.0,"publicationDate":"1990-01-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"13418753","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
引用次数: 0
The treatment of nocturnal enuresis by acupuncture. 针刺治疗夜间遗尿症。
Neurologija Pub Date : 1990-01-01
M Roje-Starcević
{"title":"The treatment of nocturnal enuresis by acupuncture.","authors":"M Roje-Starcević","doi":"","DOIUrl":"","url":null,"abstract":"<p><p>The etiology of enuresis is not completely explained. It is assumed that it might be a psychosomatic disorder caused by psychological and urological predispositions combined with unfavorable environmental factors. Thirty-seven patients of both sexes (mean age 8 years) were included in the acupuncture treatment. They had not shown any improvement after psychotherapy. During the observation period of 6 months (180 days) after the acupuncture treatment, the statistical decrease of enuresis was evident (2.9), thus confirming that acupuncture represents a new possibility of treatment of patients with enuresis.</p>","PeriodicalId":76210,"journal":{"name":"Neurologija","volume":"39 3","pages":"179-84"},"PeriodicalIF":0.0,"publicationDate":"1990-01-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"13441668","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
引用次数: 0
Multiple sclerosis in Istria, Yugoslavia. 南斯拉夫伊斯特拉的多发性硬化症。
Neurologija Pub Date : 1989-01-01
E Materljan, J Sepcić, L Antonelli, D Sepić-Grahovac
{"title":"Multiple sclerosis in Istria, Yugoslavia.","authors":"E Materljan,&nbsp;J Sepcić,&nbsp;L Antonelli,&nbsp;D Sepić-Grahovac","doi":"","DOIUrl":"","url":null,"abstract":"<p><p>An epidemiological research of multiple sclerosis (MS) in Istria, Yugoslavia, was made in the period of 1980-1981. After examining all the sources of health care information, 125 potential MS patients were found in the investigated area. According to the diagnostic criteria by Schumacher et al., 47 affected were recognized and accepted as clinically definite MS patients. The MS prevalence rate in Istria on March 31st, 1981 amounted to 25.0/10(5) inhabitants (CI: 19.9-38.9). Such rates classify Istria in the middle between the medium and high risk zones for the disease in Europe and in the world. The onset age of MS in Istria was about 30 years, the female/male sex ratio was 2.13. The average duration of MS in Istria up to the prevalence day was 16.5 years. The average annual incidence rate was 1.5/10(5) inhabitants.</p>","PeriodicalId":76210,"journal":{"name":"Neurologija","volume":"38 3","pages":"201-12"},"PeriodicalIF":0.0,"publicationDate":"1989-01-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"13842218","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
引用次数: 0
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