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Acta neuropathologica. Supplementum最新文献

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Concurrent primary intracranial tumours of different histogenesis. 不同组织发生的并发原发性颅内肿瘤。
Acta neuropathologica. Supplementum Pub Date : 1981-01-01 DOI: 10.1007/978-3-642-81553-9_34
G C Andrioli, M Zuccarello, M Scanarini, D d'Avella
{"title":"Concurrent primary intracranial tumours of different histogenesis.","authors":"G C Andrioli,&nbsp;M Zuccarello,&nbsp;M Scanarini,&nbsp;D d'Avella","doi":"10.1007/978-3-642-81553-9_34","DOIUrl":"https://doi.org/10.1007/978-3-642-81553-9_34","url":null,"abstract":"<p><p>The occurrence of intracranial tumours of diverse histogenesis can be considered occasional. In a careful survey of the literature we have encountered 63 cases of concurrent intracranial tumours: we report five personal observations, too. This review raises the question as to whether the conjoint occurrence of two intracranial tumours is purely coincidental. The statistical analysis of the cases of concurrent intracranial tumours, and the comparison between the frequency found for each different tumour when it is solitary and when it is coincident in a same patient, suggests that a casual connection can be found between tumours of diverse histogenesis.</p>","PeriodicalId":75397,"journal":{"name":"Acta neuropathologica. Supplementum","volume":"7 ","pages":"111-5"},"PeriodicalIF":0.0,"publicationDate":"1981-01-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"17989766","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
引用次数: 22
Cerebral vessels in spontaneously hypertensive rats. 自发性高血压大鼠的脑血管。
Acta neuropathologica. Supplementum Pub Date : 1981-01-01 DOI: 10.1007/978-3-642-81553-9_105
C Nordborg, B B Johansson
{"title":"Cerebral vessels in spontaneously hypertensive rats.","authors":"C Nordborg,&nbsp;B B Johansson","doi":"10.1007/978-3-642-81553-9_105","DOIUrl":"https://doi.org/10.1007/978-3-642-81553-9_105","url":null,"abstract":"<p><p>Cerebral arterial vessels of 15- and 200-day-old spontaneously hypertensive rats and normotensive controls were studied concerning the ratio between media thickness and lumen radius. Media/radius ratio among larger vessels was significantly increased already in 15-day-old spontaneously hypertensive rats. Furthermore, the media cross section area and lumen radius was increased in the internal carotid arteries of these animals. The early vascular aberrations could be caused by the slight increase of blood pressure at this age or be due to other genetically determined mechanisms. In 200-day-old hypertensive rats a significantly increased media/radius ratio was seen in arterial vessels with a radius less than 80 microns when compared to local Wistar controls but only in the smallest arterioles (r less than 20 microns) when compared to Kyoto Wistar controls.</p>","PeriodicalId":75397,"journal":{"name":"Acta neuropathologica. Supplementum","volume":"7 ","pages":"369-71"},"PeriodicalIF":0.0,"publicationDate":"1981-01-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"17990142","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
引用次数: 24
Two cases of progressive multifocal leukoencephalopathy after renal transplantation. 肾移植术后进行性多灶性脑白质病2例。
Acta neuropathologica. Supplementum Pub Date : 1981-01-01 DOI: 10.1007/978-3-642-81553-9_57
M Reznik, J Halleux, E Urbain, R Mouchette, P Castermans, M Beaujean
{"title":"Two cases of progressive multifocal leukoencephalopathy after renal transplantation.","authors":"M Reznik,&nbsp;J Halleux,&nbsp;E Urbain,&nbsp;R Mouchette,&nbsp;P Castermans,&nbsp;M Beaujean","doi":"10.1007/978-3-642-81553-9_57","DOIUrl":"https://doi.org/10.1007/978-3-642-81553-9_57","url":null,"abstract":"<p><p>Progressive multifocal leukoencephalopathy (PML) occurred in two patients after kidney transplantation. Less than 2 years after such a transplantation associated with immunosuppressive chemotherapy a 54-year-old male developed polyneuropathy then clinical diffuse alteration of the central nervous system. He died three months later with the suspicion of hypertensive encephalopathy due to progressive renal failure. A 45-year-old female had a kidney transplantation first rapidly complicated by Listeria monocytogenes meningoencephalitis. She was cured from this disease and had a satisfactory social rehabilitation during two years. Afterwards, she suffered various neurological troubles, including epilepsy, that were attributed to combined renal failure and developing hydrocephalus. One year after the onset of these neurological symptoms, the grafted kidney was removed and chemotherapy was discontinued. She died three months later. Both patients had typical PML with eosinophilic intranuclear inclusions in presumptive oligodendroglial cells. By electron microscopy, performed on formalin fixed brain tissue, round particles (40-50 nm) could be recognized in some glial cell nuclei. These two cases are confronted with the four published observations of PML following organ transplantation.</p>","PeriodicalId":75397,"journal":{"name":"Acta neuropathologica. Supplementum","volume":"7 ","pages":"189-91"},"PeriodicalIF":0.0,"publicationDate":"1981-01-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"18059817","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
引用次数: 8
Antigenicity of galactocerebroside in experimental allergic demyelinating diseases. 半乳糖脑苷在实验性变应性脱髓鞘疾病中的抗原性。
Acta neuropathologica. Supplementum Pub Date : 1981-01-01 DOI: 10.1007/978-3-642-81553-9_49
T Yonezawa, M Hasegawa, N Arizona, H Okabe
{"title":"Antigenicity of galactocerebroside in experimental allergic demyelinating diseases.","authors":"T Yonezawa,&nbsp;M Hasegawa,&nbsp;N Arizona,&nbsp;H Okabe","doi":"10.1007/978-3-642-81553-9_49","DOIUrl":"https://doi.org/10.1007/978-3-642-81553-9_49","url":null,"abstract":"<p><p>Monkeys, rabbits, guinea pigs, and rats were inoculated with GC, carrier protein and complete Freund's adjuvant. Clinical and pathological changes were obtained in monkeys and rabbits, but not, to date, in guinea pigs and rats. In rabbits, alterations were restricted to the PNS tissue, distributed in spinal roots, ganglia, and peripheral nerves. The lesions were characterized by perivenous myelin breakdown and accumulations of macrophages. The response in monkeys, also restricted to PNS, was mainly an axonal degeneration. Myelin breakdown was interpreted as secondary to the axonal damage. Axonal reactions were observed in root ganglion and anterior horn cells, being accompanied by ascending degeneration in the posterior columns of the spinal cord. Demyelinating antiserum was present in all rabbits, but absent in all monkeys. Myelination inhibiting factor was also positive in all rabbits but not in monkeys. From these findings, it can be concluded that the antigenicity of the GC is not a generalized response in all animals. The lesions produced by GC in monkeys seem to be degenerative rather than allergic.</p>","PeriodicalId":75397,"journal":{"name":"Acta neuropathologica. Supplementum","volume":"7 ","pages":"162-4"},"PeriodicalIF":0.0,"publicationDate":"1981-01-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"17836587","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
引用次数: 6
Rigid spine syndrome: histological examinations of male and female cases. 脊柱僵硬综合征:男性和女性病例的组织学检查。
Acta neuropathologica. Supplementum Pub Date : 1981-01-01 DOI: 10.1007/978-3-642-81553-9_95
J M Mussini, F Gray, J J Hauw, A M Piette, A Prost
{"title":"Rigid spine syndrome: histological examinations of male and female cases.","authors":"J M Mussini,&nbsp;F Gray,&nbsp;J J Hauw,&nbsp;A M Piette,&nbsp;A Prost","doi":"10.1007/978-3-642-81553-9_95","DOIUrl":"https://doi.org/10.1007/978-3-642-81553-9_95","url":null,"abstract":"<p><p>Histological findings in the limb-muscle of two new cases of Rigid Spine Syndrome are described. The first male case exhibited a disproportion in fiber type with predominance of the type II fibers with a slight increase of large fibers. Some of the type I fibers were slightly atrophic. No other consistent abnormalities could be seen, excepted thickness of vessel basal lamina. The second female case had a relatively distinct muscular pattern. Necrotizing phenomenon were mild without fibrosis. Type I predominance were obvious. Atrophic and hypertrophic fibers could be seen in both types populations, with a relatively higher rate in type II. These two additional cases and those of the literature showed varied non specific histology, without any histo-chemical nor ultrastructural characteristic pattern.</p>","PeriodicalId":75397,"journal":{"name":"Acta neuropathologica. Supplementum","volume":"7 ","pages":"331-3"},"PeriodicalIF":0.0,"publicationDate":"1981-01-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"17511263","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
引用次数: 7
Ionic shifts in myelinated nerve fibers during early stages of Wallerian degeneration. 沃勒氏变性早期有髓神经纤维中的离子移位。
Acta neuropathologica. Supplementum Pub Date : 1981-01-01 DOI: 10.1007/978-3-642-81553-9_10
W Schlote, H Wolburg, M F Wendt-Gallitelli
{"title":"Ionic shifts in myelinated nerve fibers during early stages of Wallerian degeneration.","authors":"W Schlote,&nbsp;H Wolburg,&nbsp;M F Wendt-Gallitelli","doi":"10.1007/978-3-642-81553-9_10","DOIUrl":"https://doi.org/10.1007/978-3-642-81553-9_10","url":null,"abstract":"<p><p>The distribution and relative mass fraction of the diffusible ions Na, K, Cl and Ca were determined by X-ray microanalysis in the axons of rat sciatic nerve 18 h and 36 h after crush and in control nerves. The investigations were performed in freeze-dried ultrathin cryosections after shock freezing of the nerves in liquified propane. 18 h after crush, no definite alteration of the ions were found compared to the control nerves. In electron micrographs of routinely processed nerves, no ultrastructural changes were seen. 36 h after crush, two types of ionic imbalance were found, the first characterized by decreased K and slightly increased Na in the axon, the second by concurrently increasing axonal Na and Cl, accompanied, in some fibers, by accumulating Ca. These types of ionic imbalance presumably represent two stages of axolemmal permeability alteration corresponding to early structural changes of axoplasm in electron micrographs of routinely processed nerves at that time.</p>","PeriodicalId":75397,"journal":{"name":"Acta neuropathologica. Supplementum","volume":"7 ","pages":"31-5"},"PeriodicalIF":0.0,"publicationDate":"1981-01-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"17229567","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
引用次数: 10
The fine structure of cells in astrocytomas of various grades of malignancy. 不同恶性程度星形细胞瘤细胞的精细结构。
Acta neuropathologica. Supplementum Pub Date : 1981-01-01 DOI: 10.1007/978-3-642-81553-9_27
G Ebhardt, J Cervós-Navarro
{"title":"The fine structure of cells in astrocytomas of various grades of malignancy.","authors":"G Ebhardt,&nbsp;J Cervós-Navarro","doi":"10.1007/978-3-642-81553-9_27","DOIUrl":"https://doi.org/10.1007/978-3-642-81553-9_27","url":null,"abstract":"<p><p>87 gliomas were studied electron microscopically. 9 tumors were excluded from the series as mixed gliomas. The ultrastructure of the cell cytoplasm was considered particularly relevant for the present study. According to the electron density and arrangement of organelles and cytofilaments 4 basic cell types I (Ia), II, III, and IV are defined. An attempt is made to scatch the development of immature glial cells in slowly grown tumors as well as their dedifferentiation in 2 cell types V and VI in malignant growths.</p>","PeriodicalId":75397,"journal":{"name":"Acta neuropathologica. Supplementum","volume":"7 ","pages":"88-90"},"PeriodicalIF":0.0,"publicationDate":"1981-01-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"17323462","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
引用次数: 3
"Thalamic" dementia in herpes encephalitis: clinico-pathological report. 疱疹性脑炎的“丘脑”痴呆:临床病理报告。
Acta neuropathologica. Supplementum Pub Date : 1981-01-01 DOI: 10.1007/978-3-642-81553-9_47
G Pilleri, V Pietrini, F Tagliavini, G Trabattoni, A Lechi
{"title":"\"Thalamic\" dementia in herpes encephalitis: clinico-pathological report.","authors":"G Pilleri,&nbsp;V Pietrini,&nbsp;F Tagliavini,&nbsp;G Trabattoni,&nbsp;A Lechi","doi":"10.1007/978-3-642-81553-9_47","DOIUrl":"https://doi.org/10.1007/978-3-642-81553-9_47","url":null,"abstract":"<p><p>Herpes zoster (HZ) primary affections of the CNS are rare and, in most of the reported patients, are representing variously extended forms of ascending myelitis. Our examination concerns a man who at the age of 37 developed apathy after a feverish episode with iridocyclitis. Six months later an ophthalmic HZ was diagnosed and thenceforth the patient showed a dementia with Korsakow's syndrome, apathy and a right hemipalsy, and diplopia appeared; the later symptoms remitted after steroid therapy. Post-mortem examination revealed a slowly progressive encephalitis with symmetrical impairment of the anterior ventral, medial, and centrum medianum of the thalamus. The HZ origin of the lesions and the relation between their site and the peculiar form of dementia, to be ascribed to the \"thalamic\" ones, are discussed. A vasculitis process can be hypothesized considering both the symmetrical localisation and the microscopical aspects of the lesions.</p>","PeriodicalId":75397,"journal":{"name":"Acta neuropathologica. Supplementum","volume":"7 ","pages":"156-9"},"PeriodicalIF":0.0,"publicationDate":"1981-01-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"18020625","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
引用次数: 8
Experimental hexachlorophene encephalopathy in mice and baboons: light and electron microscopic study. 小鼠和狒狒的实验性六氯酚脑病:光镜和电镜研究。
Acta neuropathologica. Supplementum Pub Date : 1981-01-01 DOI: 10.1007/978-3-642-81553-9_12
M F Tripier, M Berard, M Toga, G Martin-Bouyer, R Le Breton, J Garat
{"title":"Experimental hexachlorophene encephalopathy in mice and baboons: light and electron microscopic study.","authors":"M F Tripier,&nbsp;M Berard,&nbsp;M Toga,&nbsp;G Martin-Bouyer,&nbsp;R Le Breton,&nbsp;J Garat","doi":"10.1007/978-3-642-81553-9_12","DOIUrl":"https://doi.org/10.1007/978-3-642-81553-9_12","url":null,"abstract":"<p><p>An experimental study on acute Hexachlorophene (HCP) neurotoxicity is reported in mice and baboons: - by light microscopy, a severe spongiform lesion of the central nervous system is localized in the white matter without myelin breakdown or cellular reaction; - by electron microscopy, the myelin alteration is characterized by the presence of vacuolation of \"splitting\" in the intralamellar spaces of compact sheaths; myelinated axons are occasionally involved. The changes described are discussed according to various reports on HCP neurotoxicity in humans and experimental animals. The effects of this chemical agent on the central nervous system is related to the percentage of HCP in talcum powder or solution for topical use. The toxicity of very low dosage level is demonstrated in baboons. Therefore HCP use cannot be recommended for young infants.</p>","PeriodicalId":75397,"journal":{"name":"Acta neuropathologica. Supplementum","volume":"7 ","pages":"40-3"},"PeriodicalIF":0.0,"publicationDate":"1981-01-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"17986099","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
引用次数: 4
Symmetric central thalamic necrosis in experimental thiamine deficient encephalopathy. 实验性硫胺素缺乏性脑病的对称性丘脑中央坏死。
Acta neuropathologica. Supplementum Pub Date : 1981-01-01 DOI: 10.1007/978-3-642-81553-9_14
M Papp, M Tárczy, A Takáts, A Auguszt, S Komoly, I Tulok
{"title":"Symmetric central thalamic necrosis in experimental thiamine deficient encephalopathy.","authors":"M Papp,&nbsp;M Tárczy,&nbsp;A Takáts,&nbsp;A Auguszt,&nbsp;S Komoly,&nbsp;I Tulok","doi":"10.1007/978-3-642-81553-9_14","DOIUrl":"https://doi.org/10.1007/978-3-642-81553-9_14","url":null,"abstract":"<p><p>Besides the classical lesions, symmetric necrosis was found in the thalamus of pyrithiamine treated rats. The barrier systems of the two areas differ already in healthy animals and behave differently during the illness.</p>","PeriodicalId":75397,"journal":{"name":"Acta neuropathologica. Supplementum","volume":"7 ","pages":"48-9"},"PeriodicalIF":0.0,"publicationDate":"1981-01-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"17986103","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
引用次数: 10
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