The Australasian journal of dermatology最新文献

{"title":"Pityriasis rosea-like eruption revealing COVID-19.","authors":"Stefano Veraldi, Maurizio Romagnuolo, Valentina Benzecry","doi":"10.1111/ajd.13504","DOIUrl":"https://doi.org/10.1111/ajd.13504","url":null,"abstract":"Pityriasis rosea is an acute, exanthematous disease likely caused by human herpesvirus HHV-6 and/or HHV-7. It mainly affects young adults. We present two cases of pityriasis rosea-like eruptions associated with COVID-19 infection. A 26-year-old man was admitted with a diagnosis of pityriasis rosea. The patient was in good general health and was not on therapy with systemic drugs. He stated that the rash appeared some days earlier and was accompanied by pruritus. He also complained of mild headache, weakness and arthralgia. Dermatological examination revealed several erythematous-squamous papules and plaques located mainly on the upper limbs and trunk (Fig. 1). A diagnosis of pityriasis rosea-like eruption was made. Laboratory tests (including complete blood cell, liver and renal function, inflammatory markers, syphilis serology) showed only slight increase of erythrocyte sedimentation rate. Nasal swab was positive for SARS-CoV-2. Chest X-ray was normal. Cetirizine (10 mg/day) was prescribed. The patient was followed at his home. Skin lesions and other symptoms resolved within three weeks. A 21-year-old man was admitted because of the appearance of a single infiltrated, erythematous, oval lesion located on the left hip (Fig. 2a). Mycological examination was negative. A diagnosis of herald patch of pityriasis rosea was made. No treatment was suggested. One week later, the patient returned with a widespread erythematous-squamous rash on the chest and back, with lesions of different morphology and size (Fig. 2b). The patient complained of pruritus. Furthermore, the patient reported anorexia, abdominal pain, diarrhoea and weakness. We performed nasal swab for SARS-CoV-2, which was positive. All the other laboratory examinations were within normal ranges or negative. Also, this patient was followed at home and treated with cetirizine (10 mg/day). Both the rash and symptoms subsided within three weeks. Various cutaneous manifestations have been associated with COVID-19. In addition to our cases, one other case of a pityriasis rosea-like eruption has also been reported. The cases we have described show a temporal relationship between pityriasis rosea or pityriasis rosea-like eruption and COVID-19, but they do not prove causality.","PeriodicalId":243138,"journal":{"name":"The Australasian journal of dermatology","volume":" ","pages":"e333-e334"},"PeriodicalIF":2.0,"publicationDate":"2021-05-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://sci-hub-pdf.com/10.1111/ajd.13504","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"38624626","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"OA","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}

{"title":"Features of necrobiosis lipoidica in reflectance confocal microscopy.","authors":"Anna Pogorzelska-Antkowiak, Paola Corneli, Marina Agozzino","doi":"10.1111/ajd.13557","DOIUrl":"https://doi.org/10.1111/ajd.13557","url":null,"abstract":"1. Ruderman I, Toussaint ND, Hawley CM et al. The Australian Calciphylaxis Registry: reporting clinical features and outcomes of patients with calciphylaxis. Nephrol. Dial. Transplant. 2019; 1–8. 2. Rh W, Ld S, Md D et al. Calciphylaxis: natural history, risk factor analysis, and outcome. J. Am. Acad. Dermatol. 2006; 56(4): 569–79. 3. Strazzula L, Nigwekar SU, Steele D et al. Intralesional sodium thiosulfate for the treatment of calciphylaxis. JAMA Dermatol. 2013; 149(8): 946–9. 4. Wollina U, Helm C, Hansel G et al. Deep ulcer shaving combined with split-skin transplantation in distal calciphylaxis. Int. J. Low Extrem. Wounds 2008; 7(2): 102–7. 5. Acellular Fish Skin Graft Use for Diabetic Lower Extremity Wound Healing: A Retrospective Study of 58 Ulcerations and a Literature Review. Available at: https://www.woundsresearch.c om/article/acellular-fish-skin-graft-use-diabetic-lower-extre mity-wound-healing-retrospective-study-58. Accessed Aug 5, 2020. doi: 10.1111/ajd.13557","PeriodicalId":243138,"journal":{"name":"The Australasian journal of dermatology","volume":" ","pages":"e359-e361"},"PeriodicalIF":2.0,"publicationDate":"2021-05-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://sci-hub-pdf.com/10.1111/ajd.13557","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"25487935","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}

{"title":"Two cases of granular C3 dermatosis with intraepidermal neutrophilic and eosinophilic infiltration possibly induced by anti-hypertensive drugs.","authors":"Takashi Hashimoto, Takahiko Tsunoda, Fumiko Sato, Kentaro Izumi, Wataru Nishie, Norito Ishii, Hua Qian, Xiaoguang Li, Daisuke Tsuruta","doi":"10.1111/ajd.13563","DOIUrl":"https://doi.org/10.1111/ajd.13563","url":null,"abstract":"with mucosal involvement, prognosis or duration of LABD. Our patient showed a complete response with dapsone, in keeping with the diagnosis of LABD. LABD cases, including those with IgG, IgM and C3 reactivity, responded rapidly to dapsone or sulfonamides. In summary, we report a rare adult case of LABD demonstrating typical clinical findings, with IgA, IgG and IgM reactivity, who responded rapidly to dapsone. We highlight the importance of LABD as a differential diagnosis in blistering dermatoses, even with the concomitant presence of IgG and IgM reactivity.","PeriodicalId":243138,"journal":{"name":"The Australasian journal of dermatology","volume":" ","pages":"e363-e365"},"PeriodicalIF":2.0,"publicationDate":"2021-05-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://sci-hub-pdf.com/10.1111/ajd.13563","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"25408030","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}

{"title":"Cutaneous reactive angiomatosis mimicking cellulitis in a patient with a renal transplant.","authors":"Harriet Bell, Natalie Poppito, Paul Jarrett","doi":"10.1111/ajd.13502","DOIUrl":"https://doi.org/10.1111/ajd.13502","url":null,"abstract":"Nie | Yun-Liu Chen | Zhao-Yang Wang | ZiGang Xu Department of Dermatology, Beijing Children’s Hospital, Capital Medical University, National Center for Children’s Health, Beijing, Department of Pharmacy, Beijing Children’s Hospital, Capital Medical University, National Center for Children’s Health, Beijing and Center of Clinical Epidemiology&Evidence-based Medicine, Beijing Children’s Hospital, Capital Medical University, National Center for Children’s Health, Beijing, China","PeriodicalId":243138,"journal":{"name":"The Australasian journal of dermatology","volume":" ","pages":"e331-e332"},"PeriodicalIF":2.0,"publicationDate":"2021-05-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://sci-hub-pdf.com/10.1111/ajd.13502","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"38572012","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}

{"title":"Refractory discoid lupus erythematosus responds to rituximab.","authors":"Blake P Mumford, Zahra Assarian, Shereen Oon, Alvin H Chong","doi":"10.1111/ajd.13514","DOIUrl":"https://doi.org/10.1111/ajd.13514","url":null,"abstract":"We present the case of a 56-year-old Caucasian man with longstanding, refractory discoid lupus erythematosus (DLE) with significant response to B-cell depletion therapy with rituximab. He was referred in 2012 with a chronic history of biopsy-proven DLE manifesting as erythematous scaly plaques and resultant scarring affecting the face, scalp, and neck. He was managed jointly with rheumatology but never displayed features of systemic lupus erythematosus (SLE). His antinuclear antibody titre was 1:160 with a speckled pattern; testing for renal disease, extractable nuclear antigen, double-stranded DNA and antiphospholipid antibodies was negative, and complement levels and inflammatory markers were normal. He had no other significant medical comorbidities but was an active cigarette smoker.","PeriodicalId":243138,"journal":{"name":"The Australasian journal of dermatology","volume":" ","pages":"e341-e343"},"PeriodicalIF":2.0,"publicationDate":"2021-05-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://sci-hub-pdf.com/10.1111/ajd.13514","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"38720016","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}

{"title":"Successful treatment of calciphylaxis in a renal transplant recipient with combination of intralesional sodium thiosulphate, intravenous sodium thiosulphate and fish skin graft.","authors":"See Wei Tan, Jiunn Wong, Terence Kee, Zi Teng Chai, Quan Yao Ho, Michelle Chan, Khong Yik Chew, Choon Chiat Oh","doi":"10.1111/ajd.13556","DOIUrl":"https://doi.org/10.1111/ajd.13556","url":null,"abstract":"Calciphylaxis is a rare small vessel vasculopathy with high mortality rate of 80%. Sepsis is the leading cause of the mortality. To date, there is still lack of consensus regarding the optimal approach to the treatment of calciphylaxis. We present a 42-year-old lady who developed calciphylaxis 12 years after kidney transplant with preserved graft function and normal mineral levels. Two months before her presentation, she was started on warfarin for upper limb deep vein thrombosis. Her warfarin was stopped. Intralesional (IL) and intravenous (IV) sodium thiosulphate (STS) were started concurrently. She initially showed improvement; however, the recovery was further complicated by wound infection (Figure 1). Culture-directed antibiotics was started, followed by surgical wound debridement. The infection was under controlled, and ulcer progression was arrested. Fish skin graft was transplanted with good uptake (Figure 2). She was able to ambulate again with walking aid at 90 days after surgery. Kidney transplant is a potential treatment for calciphylaxis, but there are reported cases of new-onset calciphylaxis after kidney transplant. This highlights the importance of high clinical suspicion for calciphylaxis in a kidney transplant recipient with preserved graft function. We summarised the case reports of calciphylaxis in kidney transplant recipients with preserved graft function (Table 1). Calciphylaxis that developed within 12 months after kidney transplant was associated with disrupted mineral balance. We identified 6 case reports of calciphylaxis which developed more than 12 months after the kidney transplant and found strong association between warfarin use and calciphylaxis development. Therefore, warfarin should be used judiciously in patients with ESRD. Timely intervention is crucial in the management of calciphylaxis. In our case, the concurrent administration of intravenous and intralesional STS, aggressive wound debridement, and the application of fish skin graft had yielded a good clinical outcome for calciphylaxis. STS enhances the solubility of calcium deposits and restores endothelial cell dysfunction through its antioxidant effect. Numerous studies have supported the use of either intravenous (IV) or intralesional (IL) STS in the treatment of calciphylaxis, but concurrent use of IV and IL STS is uncommon. The distribution of IV STS to the active ulcer is potentially affected by the calcified vessels. IL STS is a highly targeted delivery method to the ulcer bypassing the systemic distribution. We hypothesised IL STS could lead to rapid resolution of the calcium deposits and restoration of the endothelial function. This could potentially increase the subsequent absorption of the IV STS to the affected areas. Surgical debridement with split-skin transplantation has been shown to prevent deep infection and facilitate the ulcer healing. Fish skin graft contains high-concentration omega-3 polyunsaturated fatty acids, eicosapentaenoic acid a","PeriodicalId":243138,"journal":{"name":"The Australasian journal of dermatology","volume":" ","pages":"e358-e359"},"PeriodicalIF":2.0,"publicationDate":"2021-05-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://sci-hub-pdf.com/10.1111/ajd.13556","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"25342185","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}

{"title":"Sudden colour enhancement of congenital speckled lentiginous naevus after partial resection.","authors":"Honami Nishio-Tatemichi, Emi Sato, Yoshitsugu Shibayama, Kaori Koga, Shinichi Imafuku","doi":"10.1111/ajd.13552","DOIUrl":"https://doi.org/10.1111/ajd.13552","url":null,"abstract":"A 17-year-old girl was referred to our hospital for excision of a congenital pale brown macule with multiple dark brown spots on her left buttock (Fig. 1a). Serial resections were planned, and one-third of the macule was excised along its long axis. Histopathological examination revealed hyperpigmentation and scattered melanocytes in the basal layer without nest formation (Fig. 2a). A diagnosis of speckled lentiginous naevus was established. About 2 months after the first resection, she noticed marked darkening of the entire macule. When she revisited us for additional resection 3 months after the first surgery, the entire macule had become homogeneously black and slightly elevated (Fig. 1b). Dermatoscopy showed starburst-like streaks of pigment around the edge. The whole lesion was completely resected to exclude malignancy. Histopathologically, melanocytic cells were proliferating with forming nests in the basal layer of the epidermis and appendage epithelium. The cells contained large amounts of melanin, and the nuclei were large and uniform (Fig. 2b). There were no malignant findings. We diagnosed the lesion as a recurrent melanocytic naevus occurring on speckled lentiginous naevus. Speckled lentiginous naevus generally refers to a congenital round tan patch containing multiple small dark brown spots. The area of the tan patch contains scattered lentigo-like melanocytes in the basal layer, while dark brown spots are junctional or compound naevi; however, some reports have stated that naevus cells were also present in the tan patch. A recurrent melanocytic naevus is induced by proliferation of remaining melanocytes after partial incomplete resection of a melanocytic naevus. A recurrent naevus has uncommon features such as starburst pattern by dermatoscopy, and the histopathological examination sometimes reveals mild to moderate atypia known as ‘pseudomelanoma’, which make the naevus difficult to differentiate from malignant melanoma. In a recurrent naevus, melanocytes are activated by several growth factors possibly released from fibroblasts and keratinocytes during wound healing. The same phenomenon may have occurred in our case; however, our case is distinctive in that the whole pigmented area, not only the area around the scar, became blackish and formed numerous nests. We speculate that growing nests near the scar can influence the adjacent melanocytes and gradually spread to neighbouring cells, which eventually results in the whole macule turning black. A retrospective study of recurrent pigmentation after resection showed that 61.3% of lesions were recurrent nevi and that 38.8% were melanomas. In our case, malignant melanoma was ruled out because the pigmentation was very homogenous and did not extend beyond the original lesion, no atypical cells were found in the histopathologic examination, and the time to recurrence was short. Our case indicates that the tan patch of a congenital speckled lentiginous naevus can potentially form nests i","PeriodicalId":243138,"journal":{"name":"The Australasian journal of dermatology","volume":" ","pages":"e355-e356"},"PeriodicalIF":2.0,"publicationDate":"2021-05-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://sci-hub-pdf.com/10.1111/ajd.13552","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"25325496","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}

{"title":"Two cases of yellow urticaria revealing acute lithiasic biliary disease.","authors":"Marianela Ackerman, Eric Esteve, Pascal Potier, Antoine Finon","doi":"10.1111/ajd.13496","DOIUrl":"https://doi.org/10.1111/ajd.13496","url":null,"abstract":"described it as an acanthoma with features of porokeratosis, predominantly in men, on their extremeties. It is clinically described as either solitary or multiple hyperkeratotic plaques (often verrucous) or a nodule with mild pruritus. Absence of a central area of epidermal atrophy is a clinically important feature in distinguishing porokeratoma from porokeratosis. Porokeratoma is characterised by prominent multiple and confluent cornoid lamellae throughout the lesion in histopathological examination, whereas the cornoid lamellae is only located at the periphery in classical porokeratosis. The cause of porokeratoma has not been elucidated in which no family history of porokeratoma has been reported as yet. Although dermal amyloid deposits have been observed in several cases of porokeratosis, this is the first report of it occurring in porokeratoma. The causal link between the porokeratosis and the amyloidosis remains unclear. Possible treatments could include complete excision of the lesion, cryotherapy, laser or oral acitretin. It has been reported that porokeratotic cases can have malignant potential, particularly in chronic large lesions. Regular monitoring and follow-up is therefore recommended.","PeriodicalId":243138,"journal":{"name":"The Australasian journal of dermatology","volume":" ","pages":"e326-e327"},"PeriodicalIF":2.0,"publicationDate":"2021-05-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://sci-hub-pdf.com/10.1111/ajd.13496","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"38720015","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}

{"title":"Extranodal NK/T cell lymphoma, nasal type, mimicking a bullous pyoderma gangrenosum.","authors":"Sheng-Hsiang Ma, Han-Nan Liu, Yi-Hsin Ho","doi":"10.1111/ajd.13541","DOIUrl":"https://doi.org/10.1111/ajd.13541","url":null,"abstract":"1. Imbern on-Moya A, Sidro M, Malvehy J et al. Negative mapleleaf-like areas: a new clue for basal cell carcinoma margin recognition. Br. J. Dermatol. 2016; 175: 818–20. 2. Rold an-Mar ın R, Leal-Osuna S, Lammoglia-Ordiales L et al. Infundibulocystic basal cell carcinoma: dermoscopic findings and histologic correlation. Dermatol. Pract. Concept. 2014; 4: 51–4. 3. Kawasaki Y, Ansai SI, Fujimoto K et al. A case of infundibulocystic basal cell carcinoma clinically mimicking a melanocytic nevus associated with epidermal cysts. J. Nippon. Med. Sch. 2018; 85: 228–30. 4. Arakawa A, Yatsushiro H, Hasegawa Y et al. Ber-EP4 immunoreactivity in infundibulocystic basal cell carcinoma. J. Dermatol. 2014; 41: 565–7. 5. Minagawa A. Dermoscopy-pathology relationship in seborrheic keratosis. J. Dermatol. 2017; 44: 518–24. doi: 10.1111/ajd.13541","PeriodicalId":243138,"journal":{"name":"The Australasian journal of dermatology","volume":" ","pages":"e351-e353"},"PeriodicalIF":2.0,"publicationDate":"2021-05-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://sci-hub-pdf.com/10.1111/ajd.13541","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"25321750","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}

{"title":"Aplasia cutis congenita with dermal melanocytosis.","authors":"Asmahane Souissi, Imene Ben Lagha, Marouane Mama, Ines Chelly, Slim Haouet, Mourad Mokni","doi":"10.1111/ajd.13505","DOIUrl":"https://doi.org/10.1111/ajd.13505","url":null,"abstract":"Aplasia cutis congenita is a rare disorder in which a localised area of skin is absent at birth. It most commonly affects the scalp. Two types of aplasia cutis congenita are distinguished: nonmembranous and membranous. A 10-year-old girl with no medical history presented with an area of congenital alopecia. On physical examination, a greyish-bluish to brown-coloured alopecic atrophic patch, measuring 40 mm 9 20 mm of diameter, was observed on the vertex. There was no hair collar sign (Fig. 1a). Her parents described the appearance of the lesion at birth, as a roundish erosion covered by a thin transparent membrane that gradually increased in size and turned brown. Trichoscopy showed an alopecic plaque, of a grey-blue colour, brownish at the periphery and white in the centre with some telangiectatic vessels. An absence of follicular openings and a lack of skin appendages were noticed. In the margin, hair bulbs with dark pigmented proximal ends were visible through the translucent epidermis (Fig. 1b). Because of the uneven and irregular pigmentation, a diagnostic biopsy was performed to rule out an unusual melanoma. The patient and the parent consent were obtained. Histopathological examination revealed flattening of the epidermis with loss of the rete ridges and a lack of appendages. Elongated dendritic cells containing melanin granules were scattered in the dermis (Fig. 2a,b). These dermal cells were positive for HMB-45 on immunohistochemical staining, consistent with the presence of melanocytes in the dermis (Fig. 2c). All these findings led to the diagnosis of membranous scalp aplasia cutis congenita associated with dermal melanocytosis. Because of parental pressure for a therapeutic solution, the cicatricial plaque was completely removed. Membranous scalp aplasia cutis congenita presents as a small ovoid defect covered by a thin translucent membrane. The distribution of lesions along the fusion lines and the frequent association with hair collar sign suggest that such lesions may represent a forme fruste of neural tube defect. In our case, it was associated with dermal melanocytosis. This association is very rare. During normal embryonic development, melanoblasts originate from embryonic neural crest cells. After the closure of the neural tube, they migrate from the neural crest and differentiate into melanocytes. Their migration, proliferation and differentiation into melanocytes are dependent on mediators produced by cells of the ectoderm, dorsal neural tube and keratinocytes such as the family of glycoproteins Wnt. Therefore, a preceding ectodermal fusion defect may affect the migration and proliferation of melanoblasts by the activation of Wnt signalling pathway due to the lack of antagonist signalling molecules produced by neighbouring cells. Thus, dermal melanocytes fail to reach the epidermis during migration and persist in the dermis. In addition, the presence of glial cells in the dermis, suggests that the glial tissue containing mela","PeriodicalId":243138,"journal":{"name":"The Australasian journal of dermatology","volume":" ","pages":"e335-e336"},"PeriodicalIF":2.0,"publicationDate":"2021-05-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://sci-hub-pdf.com/10.1111/ajd.13505","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"25334180","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}