Journal of veterinary emergency and critical care最新文献

{"title":"Issue Information - Prelim","authors":"","doi":"10.1111/vec.70037","DOIUrl":"https://doi.org/10.1111/vec.70037","url":null,"abstract":"","PeriodicalId":17603,"journal":{"name":"Journal of veterinary emergency and critical care","volume":"35 S2","pages":"S1-S2"},"PeriodicalIF":1.2,"publicationDate":"2025-10-06","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://onlinelibrary.wiley.com/doi/epdf/10.1111/vec.70037","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"145230671","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":3,"RegionCategory":"农林科学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"OA","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}

{"title":"Abstracts From the International Veterinary Emergency and Critical Care Symposium, and the European Veterinary Emergency and Critical Care Annual Congress 2025","authors":"","doi":"10.1111/vec.70030","DOIUrl":"https://doi.org/10.1111/vec.70030","url":null,"abstract":"","PeriodicalId":17603,"journal":{"name":"Journal of veterinary emergency and critical care","volume":"35 S2","pages":"S3-S45"},"PeriodicalIF":1.2,"publicationDate":"2025-10-06","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"145230672","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":3,"RegionCategory":"农林科学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}

{"title":"Issue Information - Prelim","authors":"","doi":"10.1111/vec.13398","DOIUrl":"https://doi.org/10.1111/vec.13398","url":null,"abstract":"","PeriodicalId":17603,"journal":{"name":"Journal of veterinary emergency and critical care","volume":"35 4","pages":"319-322"},"PeriodicalIF":1.2,"publicationDate":"2025-10-02","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://onlinelibrary.wiley.com/doi/epdf/10.1111/vec.13398","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"145204807","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":3,"RegionCategory":"农林科学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"OA","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}

{"title":"Clinical Applications of the ARDSVet (Acute Respiratory Distress Syndromes in Veterinary Medicine) Definitions in Small and Large Animal Patients","authors":"Tereza Stastny,&nbsp;Daniela Bedenice,&nbsp;JoAnn Slack,&nbsp;Anusha Balakrishnan,&nbsp;Deborah C. Silverstein","doi":"10.1111/vec.70015","DOIUrl":"10.1111/vec.70015","url":null,"abstract":"<div>\u0000 \u0000 \u0000 <section>\u0000 \u0000 <h3> Objective</h3>\u0000 \u0000 <p>To illustrate the use of the ARDSVet (Acute Respiratory Distress Syndromes in Veterinary Medicine) definitions in small and large animal patients using a case-based approach.</p>\u0000 </section>\u0000 \u0000 <section>\u0000 \u0000 <h3> Etiology</h3>\u0000 \u0000 <p>Acute respiratory distress syndrome (ARDS) in veterinary patients is triggered by a wide range of clinical insults. These include probable risk factors such as systemic inflammation, pancreatitis, and sepsis, as well as possible risk factors such as blood transfusions and ventilator-induced lung injury. These conditions may lead to diffuse alveolar damage and increased pulmonary capillary permeability.</p>\u0000 </section>\u0000 \u0000 <section>\u0000 \u0000 <h3> Diagnosis</h3>\u0000 \u0000 <p>ARDS remains challenging to diagnose, particularly in veterinary patients with variable resources. The updated ARDSVet definitions offer a structured framework based on five criteria: a known or suspected risk factor, onset of respiratory distress within 1 week, exclusion of cardiogenic edema and volume overload, thoracic imaging (including point-of-care ultrasound) demonstrating diffuse pulmonary infiltrates, and impaired oxygenation assessed by PaO₂/FiO₂ or SpO₂/FiO₂ ratios. Case vignettes highlight revised oxygenation thresholds, expanded use of point-of-care ultrasound, and the role of advanced respiratory support techniques.</p>\u0000 </section>\u0000 \u0000 <section>\u0000 \u0000 <h3> Therapy</h3>\u0000 \u0000 <p>ARDS treatment is primarily supportive, focusing on oxygen supplementation, high-flow nasal oxygen, and/or mechanical ventilation, along with management of the underlying cause. While ARDSVet does not offer formal treatment guidelines, case vignettes illustrate how supportive strategies may be adapted across disease stages without endorsing any specific therapeutic interventions.</p>\u0000 </section>\u0000 \u0000 <section>\u0000 \u0000 <h3> Prognosis</h3>\u0000 \u0000 <p>Prognosis in animals with ARDS is influenced by the severity of respiratory compromise, the underlying cause, and the timelines of appropriate interventions. The updated definitions will aid clinicians in early and timely recognition of ARDS, although further studies are needed to assess its impact on clinical outcomes.</p>\u0000 </section>\u0000 </div>","PeriodicalId":17603,"journal":{"name":"Journal of veterinary emergency and critical care","volume":"35 4","pages":"339-345"},"PeriodicalIF":1.2,"publicationDate":"2025-09-11","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://onlinelibrary.wiley.com/doi/epdf/10.1111/vec.70015","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"145034890","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":3,"RegionCategory":"农林科学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"OA","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}

{"title":"Inadvertent Insertion of a Nasoenteric Feeding Tube Into the Brain of an Anesthetized Persian Cat","authors":"Federico Gianolli,&nbsp;Lea Carisch,&nbsp;Katrin Melanie Beckmann,&nbsp;Annette Patricia Nora Kutter,&nbsp;Meike Hammer","doi":"10.1111/vec.70018","DOIUrl":"10.1111/vec.70018","url":null,"abstract":"<div>\u0000 \u0000 \u0000 <section>\u0000 \u0000 <h3> Objective</h3>\u0000 \u0000 <p>To describe the inadvertent insertion of a tube intended for nasogastric placement into the brain of an anesthetized brachycephalic cat.</p>\u0000 </section>\u0000 \u0000 <section>\u0000 \u0000 <h3> Case Summary</h3>\u0000 \u0000 <p>A 7-year-old Persian cat underwent a cholecystectomy for emergency surgical management of biliary obstruction, with postoperative placement of a nasogastric tube planned for nutritional support. General anesthesia with spontaneous breathing was administered, and the surgical procedure was performed without adverse events. At the end of the surgical procedure, while the anesthetized cat was in dorsal recumbency, a 5-French polyurethane nasogastric tube with a stylet was inserted into the right nostril. Immediately after insertion, the patient experienced apnea and required hemodynamic and ventilatory support. Because the tube could not be palpated in the stomach, apnea persisted, and the patient ultimately failed to regain consciousness, a CNS lesion was suspected, and magnetic resonance imaging of the head was performed. Lesions consistent with nasogastric tube insertion into the brain were diagnosed, and because the prognosis was believed to be grave, the cat was euthanized.</p>\u0000 </section>\u0000 \u0000 <section>\u0000 \u0000 <h3> New or Unique Information Provided</h3>\u0000 \u0000 <p>Few reports of nasoenteric tube insertion into the brain of humans are available. To our knowledge, this is the first report of a nasoenteric tube insertion into the brain of a dog or cat.</p>\u0000 </section>\u0000 </div>","PeriodicalId":17603,"journal":{"name":"Journal of veterinary emergency and critical care","volume":"35 4","pages":"430-435"},"PeriodicalIF":1.2,"publicationDate":"2025-08-30","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"144981539","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":3,"RegionCategory":"农林科学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}

{"title":"Comparison of Two Lung Ultrasound Protocols for Identification and Distribution of B-, I-, and Z-Lines in Clinically Healthy Cats","authors":"Hugo K. J. Swanstein,&nbsp;Rebecca Langhorn,&nbsp;Kris Gommeren,&nbsp;Søren Boysen","doi":"10.1111/vec.70011","DOIUrl":"10.1111/vec.70011","url":null,"abstract":"<div>\u0000 \u0000 \u0000 <section>\u0000 \u0000 <h3> Objective</h3>\u0000 \u0000 <p>To investigate the frequency and distribution of B-, I-, and Z-lines in clinically healthy cats and to determine whether there is a difference in frequency and distribution between two different lung ultrasound protocols.</p>\u0000 </section>\u0000 \u0000 <section>\u0000 \u0000 <h3> Design</h3>\u0000 \u0000 <p>Prospective cross-sectional study from June to September 2022.</p>\u0000 </section>\u0000 \u0000 <section>\u0000 \u0000 <h3> Setting</h3>\u0000 \u0000 <p>Companion animal referral hospital.</p>\u0000 </section>\u0000 \u0000 <section>\u0000 \u0000 <h3> Animals</h3>\u0000 \u0000 <p>Sixty-four cats determined to be healthy based on history and physical examination.</p>\u0000 </section>\u0000 \u0000 <section>\u0000 \u0000 <h3> Interventions</h3>\u0000 \u0000 <p>All cats had point-of-care lung ultrasound performed with frequency of B-, I-, and Z-lines recorded. Findings of a modified Calgary pleura and lung ultrasound (PLUS) protocol (19 sites recorded) and a veterinary Brief Lung Ultrasound Exam (VetBLUE) protocol (nine sites), both including the subxiphoid site, were compared.</p>\u0000 </section>\u0000 \u0000 <section>\u0000 \u0000 <h3> Measurements and Main Results</h3>\u0000 \u0000 <p>B-lines were identified in 14%–27% of cats depending on the protocol used, with a median of &lt;3 B-lines total per cat, &lt;2 B-lines per hemithorax, and ≤2 positive B-line sites per hemithorax, regardless of the protocol used. The total number of B-, I-, or Z-lines identified was significantly higher for the modified Calgary PLUS compared with VetBLUE. The number of cats with ≥1 B- and I-lines was significantly higher with modified Calgary PLUS compared with VetBLUE, with 17 (26.6%) and 42 (65.6%) versus nine (14.1%) and 34 (53.1%) cats identified, respectively. Z-lines were identified in 62 (96.9%) cats regardless of the protocol used. Three (4.7%) cats had ≥1 B-line at the subxiphoid view.</p>\u0000 </section>\u0000 \u0000 <section>\u0000 \u0000 <h3> Conclusions</h3>\u0000 \u0000 <p>B-lines were detected in &lt;30%, I-lines in &gt;50%, and Z-lines in &gt;95% of clinically healthy cats regardless of protocol used. B-lines were rarely detected at the subxiphoid view. A significant difference between protocols was found when comparing the percentage of cats with B- and I-lines.</p>\u0000 </section>\u0000 </div>","PeriodicalId":17603,"journal":{"name":"Journal of veterinary emergency and critical care","volume":"35 4","pages":"368-377"},"PeriodicalIF":1.2,"publicationDate":"2025-08-30","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"144981402","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":3,"RegionCategory":"农林科学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}

{"title":"Management of a Nondeflating Foley Urinary Catheter Balloon by Transrectal Centesis in a Dog","authors":"Ciaran C. O'Carroll,&nbsp;Meagan A. Walker,&nbsp;Katie L. Hoddinott","doi":"10.1111/vec.70022","DOIUrl":"10.1111/vec.70022","url":null,"abstract":"<div>\u0000 \u0000 \u0000 <section>\u0000 \u0000 <h3> Objective</h3>\u0000 \u0000 <p>To describe the management of a nondeflating urinary catheter balloon via transrectal centesis in a dog and to propose an algorithm for the nonsurgical management of this adverse event in dogs.</p>\u0000 </section>\u0000 \u0000 <section>\u0000 \u0000 <h3> Case Summary</h3>\u0000 \u0000 <p>A mature neutered male crossbreed dog presented with a &lt;24-h history of acute paraplegia secondary to a T13–L1 hydrated nucleus pulposus extrusion. During placement of an indwelling urinary catheter, the Foley balloon reservoir was inadvertently inflated in the proximal urethra. Initial attempts to deflate the balloon through aspiration of the inflation channel, transection of the inflation channel proximal to the inflation valve, and insertion of the stylet into the inflation channel to remove potential obstructions were unsuccessful. The balloon reservoir was successfully deflated by transrectal centesis using a 22-gauge needle. No short- or long-term complications were associated with this procedure.</p>\u0000 </section>\u0000 \u0000 <section>\u0000 \u0000 <h3> New or Unique Information Provided</h3>\u0000 \u0000 <p>Nondeflation is a relatively infrequent complication of urinary catheterization using a Foley catheter. Causes include faulty valve mechanisms, obstruction of the inflation channel, or crystallization of fluid within the balloon. This report describes the use of transrectal centesis to deflate a balloon reservoir lodged in the proximal urethra of a male dog. This technique has not previously been reported. An algorithm for the nonsurgical management of nondeflating Foley catheters in dogs is also proposed.</p>\u0000 </section>\u0000 </div>","PeriodicalId":17603,"journal":{"name":"Journal of veterinary emergency and critical care","volume":"35 4","pages":"425-429"},"PeriodicalIF":1.2,"publicationDate":"2025-08-30","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"144981488","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":3,"RegionCategory":"农林科学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}

{"title":"Refractory Hypoglycemia in a Dog With Severe Zinc Intoxication","authors":"John Vietti,&nbsp;Jennifer Herring,&nbsp;Shaunita Sharpe,&nbsp;Barb Gertz","doi":"10.1111/vec.70024","DOIUrl":"10.1111/vec.70024","url":null,"abstract":"<div>\u0000 \u0000 \u0000 <section>\u0000 \u0000 <h3> Objective</h3>\u0000 \u0000 <p>To describe hypoglycemia secondary to zinc toxicosis in a dog.</p>\u0000 </section>\u0000 \u0000 <section>\u0000 \u0000 <h3> Case Summary</h3>\u0000 \u0000 <p>A 3-year-old spayed female Pug weighing 9.0 kg presented with lethargy and labored breathing. Diagnostic tests revealed hypoglycemia, regenerative anemia, azotemia, increased liver values, and metallic objects in the stomach. The dog was treated with IV dextrose but remained refractory to therapy for the first 5 h. A serum zinc concentration was 32.28 µg/mL (reference interval, 0.8–1.8 µg/mL). The dog was treated supportively, and the blood glucose concentration responded within hours of endoscopic removal of the metallic foreign bodies.</p>\u0000 </section>\u0000 \u0000 <section>\u0000 \u0000 <h3> New or Unique Information Provided</h3>\u0000 \u0000 <p>This case demonstrates refractory hypoglycemia associated with zinc toxicosis, which has not been previously reported in the literature.</p>\u0000 </section>\u0000 </div>","PeriodicalId":17603,"journal":{"name":"Journal of veterinary emergency and critical care","volume":"35 4","pages":"449-454"},"PeriodicalIF":1.2,"publicationDate":"2025-08-30","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"144981524","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":3,"RegionCategory":"农林科学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}

{"title":"Hypermagnesemia in a Cat Secondary to Suspected Intoxication","authors":"Jackson A. Griffith,&nbsp;Melissa R. Santonocito,&nbsp;Katie D. Mauro","doi":"10.1111/vec.70021","DOIUrl":"10.1111/vec.70021","url":null,"abstract":"<div>\u0000 \u0000 \u0000 <section>\u0000 \u0000 <h3> Objective</h3>\u0000 \u0000 <p>To describe the management of a unique case of hypermagnesemia in a cat.</p>\u0000 </section>\u0000 \u0000 <section>\u0000 \u0000 <h3> Case Summary</h3>\u0000 \u0000 <p>An 8-year-old spayed female domestic shorthair cat was presented for acute-onset stupor, hypothermia, and bradycardia. Overtly elevated ionized (&gt;1.5 mmol/L [&gt;3.6 mg/dL]; reference interval: 0.4–0.8 mmol/L [0.9–1.9 mg/dL]) and total magnesium (3.0 mmol/L [7.3 mg/dL]; reference interval: 0.74–1.0 mmol/L [1.8–2.5 mg/dL]) concentrations were identified. A thorough history uncovered the long-term at-home administration of an anxiolytic supplement containing magnesium stearate as an inactive ingredient. No other sources of nondietary magnesium were identified. A diagnosis of magnesium toxicosis was reached. Secondarily, an acute kidney injury developed. After 10 days of hospitalization, the cat was successfully discharged with normalized kidney and electrolyte values.</p>\u0000 </section>\u0000 \u0000 <section>\u0000 \u0000 <h3> New or Unique Information Provided</h3>\u0000 \u0000 <p>Few case reports regarding magnesium toxicosis and its management exist in the veterinary literature. A rare case of hypermagnesemia is presented in this report, highlighting the importance of a complete laboratory workup that includes an evaluation of magnesium levels in previously healthy animals that present with otherwise unexplained clinical signs.</p>\u0000 </section>\u0000 </div>","PeriodicalId":17603,"journal":{"name":"Journal of veterinary emergency and critical care","volume":"35 4","pages":"442-448"},"PeriodicalIF":1.2,"publicationDate":"2025-08-30","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://onlinelibrary.wiley.com/doi/epdf/10.1111/vec.70021","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"144981527","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":3,"RegionCategory":"农林科学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"OA","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}

{"title":"Asymmetric Multifocal Neurological Signs in a Dog With Eunatremic, Eukalemic Hypoadrenocorticism With Severe Hypoglycemia","authors":"Cameron A. D. Morris,&nbsp;Rebekah E. Donaldson","doi":"10.1111/vec.70019","DOIUrl":"10.1111/vec.70019","url":null,"abstract":"<div>\u0000 \u0000 \u0000 <section>\u0000 \u0000 <h3> Objective</h3>\u0000 \u0000 <p>To describe the clinical presentation of a dog with eunatremic, eukalemic hypoadrenocorticism (EEH) with multifocal, asymmetrical neurological signs.</p>\u0000 </section>\u0000 \u0000 <section>\u0000 \u0000 <h3> Case Summary</h3>\u0000 \u0000 <p>A 7-year-old neutered male miniature Poodle presented for collapse, vomiting, and acute onset of neurological signs. The dog was obtunded and nonambulatory, with a right-sided head tilt and turn with rotary nystagmus. No seizure activity was observed by the owner. The patient was in hypovolemic shock. Hypoglycemia, mild azotemia, and metabolic acidosis characterized by hyperlactatemia were identified. Magnetic resonance imaging of the brain and CSF analysis performed within 24 h of presentation were unremarkable. ACTH stimulation testing confirmed hypoadrenocorticism. Neurological signs failed to significantly improve despite systemic stabilization, glucose supplementation, and dexamethasone administration until approximately 48 h following admission. A mild proprioceptive ataxia was present at discharge approximately 84 h following admission and had resolved by the time of initial recheck 7 days following discharge.</p>\u0000 </section>\u0000 \u0000 <section>\u0000 \u0000 <h3> New Information Provided</h3>\u0000 \u0000 <p>EEH can cause neurological signs with forebrain localization that are typically symmetrical due to the global nature of the injury. To the authors’ knowledge, this is the first report of a dog with EEH presenting with severe, asymmetric, and multifocal neurological signs.</p>\u0000 </section>\u0000 </div>","PeriodicalId":17603,"journal":{"name":"Journal of veterinary emergency and critical care","volume":"35 4","pages":"455-460"},"PeriodicalIF":1.2,"publicationDate":"2025-08-30","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"144981393","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":3,"RegionCategory":"农林科学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}