{"title":"Complete Right Bundle Branch Block in Arrhythmogenic Cardiomyopathy: Similarities to Brugada Syndrome","authors":"Peters Stefan","doi":"10.23937/2378-2951/1410137","DOIUrl":"https://doi.org/10.23937/2378-2951/1410137","url":null,"abstract":"Background: In arrhythmogenic cardiomyopathy complete right bundle branch block do appear. In rare cases complete right bundle branch block with Brugada-like presentation. Method: In 24 patients with typical arrhythmogenic cardiomyopathy ECG showed complete right bundle branch block. In 17 cases atypical RBBB with low voltage were present, in 7 cases typical rSR’ RBBB occurred. Results: Brugada-like ECG could be excluded in 6 cases of typical complete rSR’ right bundle branch block, only in one case Brugada ECG with coved-type ST segment elevation, sharp upstroke and symmetrical T-wave inversion was present. Conclusion: In typical arrhythmogenic cardiomyopathy atypical low voltage and typical rSR’ right bundle branch block appear. Only in one case with typical arrhythmogenic cardiomyopathy the ECG resembles classical Brugada ECG. In right bundle branch block both-arrhythmogenic cardiomyopathy and Brugada-like syndrome-could be presented as an overlap syndrome.","PeriodicalId":112011,"journal":{"name":"International Journal of Clinical Cardiology","volume":"88 1","pages":"0"},"PeriodicalIF":0.0,"publicationDate":"2019-03-31","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"123811309","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
{"title":"Ventricular Fibrillation in Arrhythmogenic Cardiomyopathy: Four Strategies to Effective Risk Management","authors":"S. Peters","doi":"10.23937/2378-2951/1410141","DOIUrl":"https://doi.org/10.23937/2378-2951/1410141","url":null,"abstract":"This case report combines four high risk markers for the development of ventricular fibrillation in Brugada syndrome at the early stage of arrhythmogenic cardiomyopathy with prehistologic abnormalities. In many cases ventricular fibrillation can be the first manifestation of arrhythmogenic cardiomyopathy as a pure electrical disease and later on manifest arrhythmogenic disease develops. It takes about 5 years to recurrent ventricular fibrillation according to a recent study [5]. In this period of time it is not an option to explant the ICD for missing arrhythmic events.","PeriodicalId":112011,"journal":{"name":"International Journal of Clinical Cardiology","volume":"45 1","pages":"0"},"PeriodicalIF":0.0,"publicationDate":"2019-03-25","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"127005526","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
{"title":"A Rare Coronary Anomaly: Congenital Absence of the Left Circumflex Artery","authors":"Robert Tonks, Rajiv B Sharma, Raj Baljepally","doi":"10.23937/2378-2951/1410156","DOIUrl":"https://doi.org/10.23937/2378-2951/1410156","url":null,"abstract":"Congenital anomalies of the coronary vasculature are usually asymptomatic and rare with an incidence of less than 1% and there are few cases reported of congenital absence of the left circumflex artery. It is important to recognize this anomaly and define the anatomy by left heart catheterization or coronary angiography by computerized tomography when considering percutaneous intervention or coronary artery bypass surgery on patients without a left circumflex artery.","PeriodicalId":112011,"journal":{"name":"International Journal of Clinical Cardiology","volume":"1 1","pages":"0"},"PeriodicalIF":0.0,"publicationDate":"2019-03-23","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"125401349","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
{"title":"Effects of Bisoprolol and Flecainide in an Elderly Patient with Paroxysmal Supraventricular Tachycardia and Atrial Fibrillation in Wolff-Parkinson-White Syndrome","authors":"Kwak Jae-Jin, Doh Joon Hyung, Lee Sung Yun","doi":"10.23937/2378-2951/1410138","DOIUrl":"https://doi.org/10.23937/2378-2951/1410138","url":null,"abstract":"Wolff-Parkinson-White (WPW) syndrome can develop paroxysmal supraventricular tachycardia (PSVT) or/and paroxysmal atrial fibrillation (AF). AF in WPW syndrome is a potentially life-threatening arrhythmia. Radiofrequency catheter ablation is recommended in patients with AF in WPW syndrome. Atrioventricular nodal blockers can precipitate ventricular fibrillation and should not be used. We present patients with PSVT and AF in WPW syndrome who was treated with bisoprolol and flecainide.","PeriodicalId":112011,"journal":{"name":"International Journal of Clinical Cardiology","volume":"18 1","pages":"0"},"PeriodicalIF":0.0,"publicationDate":"2019-02-28","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"132530883","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
Stevanovic Ksenija, Trailović Ranko, Kukic Biljana, D. Marko, Cvetković Slobodan, D. Lazar
{"title":"Carotid Sinus Syndrome in a Patient with Symptomatic Abdominal Aortic Aneurysm - A Case Report","authors":"Stevanovic Ksenija, Trailović Ranko, Kukic Biljana, D. Marko, Cvetković Slobodan, D. Lazar","doi":"10.23937/2378-2951/1410130","DOIUrl":"https://doi.org/10.23937/2378-2951/1410130","url":null,"abstract":"Carotid sinus syndrome (CSS) is defined as syncope with reproduction of symptoms during 10s of carotid sinus massage. The etiology of this condition is unknown, patients are usually symptomatic and present with episodes of syncope. Due to the associated marked sinus bradycardia, sinus arrest or severe hypotension, carotid sinus syndrome may be life-threatening. In patients without previous symptoms, CSS must be promptly recognized and treated, otherwise may lead to catastrophic consequences. If symptoms of CSS appear for the first time during urgent symptomatic abdominal aortic aneurysm surgery consequences might be devastating. Herein, we describe a case of successful management of a patient with carotid sinus syndrome who has undergone urgent symptomatic abdominal aortic aneurysm open repair.","PeriodicalId":112011,"journal":{"name":"International Journal of Clinical Cardiology","volume":"1 1","pages":"0"},"PeriodicalIF":0.0,"publicationDate":"2018-12-31","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"127239657","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
Lee Sung Yun, Kim, Sang-wook, Lee Jin Bae, Shin Eun-Seok, Doh Joon Hyung, Hong Young Joon, Kwak Jae-Jin, Kim Hyung Yoon
{"title":"IVUS Findings of Drug Eluting Balloon Failure in the Treatment of In-Stent Restenosis","authors":"Lee Sung Yun, Kim, Sang-wook, Lee Jin Bae, Shin Eun-Seok, Doh Joon Hyung, Hong Young Joon, Kwak Jae-Jin, Kim Hyung Yoon","doi":"10.23937/2378-2951/1410133","DOIUrl":"https://doi.org/10.23937/2378-2951/1410133","url":null,"abstract":"Background and objective: The objective of this study is to investigate pre-intervention IVUS characteristics of instent restenosis (ISR) lesion correlated with PEB failure. Materials and methods: We performed pre-intervention IVUS for 58 patients with ISR and treated with PEB angioplasty. The PEB failure was defined as death, myocardial infarction and symptom driven revascularization. IVUS images were analyzed at 6 locations: Proximal and distal vessel segment within 3 mm from stent edge, proximal and distal stent edge, lesion site at minimal lumen area and minimal stent area. Results: Among 58 patients treated with 58 PEB (3.0 ± 0.4 mm by 21.9 ± 4.8 mm), PEB failure were developed at 8 patients (13.8%) during 0.81 years of follow-up. ISR of drug eluting stent comprised large proportion (86.2%) among index procedure. There was no significant difference in clinical presentation and used stents at index procedure between PEB failure and non-failure group. In PEB failure group, neointimal area (4.0 ± 0.7 vs. 2.9 ± 0.8 mm2, p = 0.05) and neointimal hyperplasia (%) (59.4 ± 12.0 vs. 46.0 ± 24.9%, p = 0.05) were significant greater than non-failure group. Stent under-expansion was found in 24 ISR lesions (48.0%) of non-failure group, whereas none was detected in PEB failure group. Conclusion: PEB failure for ISR lesion could be associated with prominent neointimal hyperplasia on normally extended stent. Future studies are warranted to investigate another RESEaRcH aRticLE","PeriodicalId":112011,"journal":{"name":"International Journal of Clinical Cardiology","volume":"16 1","pages":"0"},"PeriodicalIF":0.0,"publicationDate":"2018-12-31","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"127675186","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
{"title":"The Profile of an Adult with Congenital Heart Disease","authors":"Araujo John Jairo","doi":"10.23937/2378-2951/1410131","DOIUrl":"https://doi.org/10.23937/2378-2951/1410131","url":null,"abstract":"Congenital heart disease is the most frequent malformation. In the most recent data has a prevalence of thirteen cases per 1,000 children and six cases per 1,000 adults. Great strides in pediatric cardiovascular surgery have increased the number of survivors, especially in adulthood. Nowadays more adults are living with congenital heart disease than children, particularly in developed countries, and more than two thirds fall into the categories of moderate and high complexity defects. This growing population called: Grown up congenital heart, requires super-specialized healthcare, the profile of the new cardiovascular patient differs from the traditional adult with heart disease, the first paradigm that should be broken is to understand that cardiovascular surgery is restorative (it substantially improves the hemodynamic condition), but is not 100% curative. Surgical and survival successes have turned the pediatric patient with congenital heart disease into an adult postoperative patient with residua, sequela and complications. The adult's postoperative heart is not a natural heart, it is a transformed, \"neo-formed\", heart. They will have relapses and hospitalizations due decompensation to congenital heart disease. A new specialist profile is necessary. Training in adult congenital heart disease is valid beginning from both a pediatric and an adult cardiology environment. The special needs of this population are very costly for health systems. Recognize what are the main causes of decompensation is mandatory and finally adequate care decreases prolonged hospital stays and health system cost.","PeriodicalId":112011,"journal":{"name":"International Journal of Clinical Cardiology","volume":"9 1","pages":"0"},"PeriodicalIF":0.0,"publicationDate":"2018-12-31","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"124188585","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
{"title":"Cardiac Arrest from Life-Threatening Massive Pulmonary Emboli: A Rare Survival Post Thrombolysis","authors":"Taylor M Gregory, P. David, B. Michael, A. Haidar","doi":"10.23937/2378-2951/1410129","DOIUrl":"https://doi.org/10.23937/2378-2951/1410129","url":null,"abstract":"","PeriodicalId":112011,"journal":{"name":"International Journal of Clinical Cardiology","volume":"56 1","pages":"0"},"PeriodicalIF":0.0,"publicationDate":"2018-12-31","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"113959966","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
{"title":"Neurologic and Renal Outcomes of Elective Proximal Aortic Repair and Current Cannulation Trends","authors":"I. Sahin, Eygi Bortecin","doi":"10.23937/2378-2951/1410128","DOIUrl":"https://doi.org/10.23937/2378-2951/1410128","url":null,"abstract":"Background: We aimed to determine factors influencing neurologic dysfunction and acute kidney injury (AKI) in patients undergoing elective aortic aneurysm repair and we also investigated them according to cannulation sites. Methods: From January 2012 to April 2018, 112 patients received proximal aortic repair. We used aortic arch, axillary and femoral artery cannulation. Antegrade cerebral protection and total circulatory arrest were used. Results: We detected 6 (5.4%) transient and 6 (5.4%) permanent neurologic dysfunction. Peripheral arterial diseases, total bypass time, transfusion and AKI showed correlation with neurologic dysfunction. Peripheral arterial diseases and AKI were independent risk factors (p < 0.05). AKI was reported in 38 patients. Postoperative revision, cross-clamp time, total bypass time and total circulatory arrest showed correlation with AKI (p < 0.05). Patients with neurologic dysfunction (66%) and patients with AKI (63%) had more femoral cannulation. Conclusion: Proximal aortic repair can be achieved with low morbidity and mortality using appropriate cerebral protection and cannulation technique. Neurologic dysfunction and AKI are important factors for aortic surgery and femoral cannulation has the highest risk for neurologic dysfunction and AKI.","PeriodicalId":112011,"journal":{"name":"International Journal of Clinical Cardiology","volume":"188 1","pages":"0"},"PeriodicalIF":0.0,"publicationDate":"2018-12-31","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"132349501","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}