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Adenomatoid tumor of testis. 睾丸腺瘤样肿瘤。
Clinical medicine. Pathology Pub Date : 2009-09-09 DOI: 10.4137/cpath.s3091
Waqas Amin, Anil V Parwani
{"title":"Adenomatoid tumor of testis.","authors":"Waqas Amin,&nbsp;Anil V Parwani","doi":"10.4137/cpath.s3091","DOIUrl":"https://doi.org/10.4137/cpath.s3091","url":null,"abstract":"<p><p>Adenomatoid tumors are responsible for 30% of all paratesticular masses. These are usually asymptomatic, slow growing masses. They are benign tumors comprising of cords and tubules of cuboidal to columnar cells with vacuolated cytoplasm and fibrous stroma. They are considered to be of mesothelial origin supported by histochemical studies and genetic analysis of Wilms tumor 1 gene expression. Excision biopsy is both diagnostic and therapeutic procedure. The main clinical consideration is accurate diagnosis preventing unnecessary orchiectomy. Diagnostic studies include serum tumor markers (negative alpha fetoprotein, beta HCG, LDH) ultrasonography (hypoechoic and homogenous appearance) and frozen section.</p>","PeriodicalId":89118,"journal":{"name":"Clinical medicine. Pathology","volume":"2 ","pages":"17-22"},"PeriodicalIF":0.0,"publicationDate":"2009-09-09","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://sci-hub-pdf.com/10.4137/cpath.s3091","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"29530720","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"OA","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
引用次数: 33
Expression of Placental Neurotrophin-3 (NT-3) in Physiological Pregnancy, Preeclampsia and Chorioamnionitis. 胎盘神经营养因子-3 (NT-3)在生理性妊娠、子痫前期和绒毛膜羊膜炎中的表达。
Clinical medicine. Pathology Pub Date : 2009-06-11 DOI: 10.4137/cpath.s2325
Alessandra Casciaro, Felice Arcuri, Rossella Occhini, M Stefania Toti, Claudio De Felice, Paolo Toti
{"title":"Expression of Placental Neurotrophin-3 (NT-3) in Physiological Pregnancy, Preeclampsia and Chorioamnionitis.","authors":"Alessandra Casciaro,&nbsp;Felice Arcuri,&nbsp;Rossella Occhini,&nbsp;M Stefania Toti,&nbsp;Claudio De Felice,&nbsp;Paolo Toti","doi":"10.4137/cpath.s2325","DOIUrl":"https://doi.org/10.4137/cpath.s2325","url":null,"abstract":"<p><p>Neurotrophic factors are a group of proteins that act as paracrine and autocrine growth factors. They are involved in the regulation of morphogenesis and development of several tissues. The present study aims to evaluate, for the first time, the expression of Neurotrophin-3 in the human placenta during normal pregnancy and in preeclampsia and chorioamnionitis. Neurotrophin-3 mRNA, assessed by RT-PCR analysis in six term placentas, were observed in all the specimens examined. Neurotrophin-3 protein expression and tissue distribution was evaluated by immunohistochemistry in placenta samples from uncomplicated first trimester (n = 5) and term (n = 5) pregnancies as well as in specimens from preeclampsia (n = 5) and chorioamnionitis (n = 5). In first trimester specimens, strong immunoreactivity was present in villous stromal cells, in the cyto- and syncytiotrophoblast, in decidua cells and in endometrial glands. Third trimester specimens showed prominent immunostaining in cyto- and syncytiotrophoblast cells, in decidua cells and in the amniotic membranes. Villous stromal cells were weakly stained. Similar protein localization was observed in placentas with preeclampsia and chorioamnionitis. In the latter, however, positive villous stromal cells increased in number and in staining intensity when compared with controls and preeclampsia (p < 0.001). The roles of Neurotrophin-3 in pregnancy are presently unknown. A regulatory function on placenta and foetal brain development and maternal inflammatory response may be hypothesized.</p>","PeriodicalId":89118,"journal":{"name":"Clinical medicine. Pathology","volume":"2 ","pages":"9-15"},"PeriodicalIF":0.0,"publicationDate":"2009-06-11","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://sci-hub-pdf.com/10.4137/cpath.s2325","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"29530251","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"OA","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
引用次数: 21
Treated choroidal melanoma with late metastases to the contralateral orbit. 治疗晚期转移到对侧眼眶的脉络膜黑色素瘤。
Clinical medicine. Pathology Pub Date : 2009-04-03 DOI: 10.4137/cpath.s767
Sonia George, Carole A Cooke, Gerald F Mc Ginnity, Steve White, Laksmi Venkatraman
{"title":"Treated choroidal melanoma with late metastases to the contralateral orbit.","authors":"Sonia George,&nbsp;Carole A Cooke,&nbsp;Gerald F Mc Ginnity,&nbsp;Steve White,&nbsp;Laksmi Venkatraman","doi":"10.4137/cpath.s767","DOIUrl":"https://doi.org/10.4137/cpath.s767","url":null,"abstract":"<p><p>Choroidal melanoma is the commonest adult primary intraocular tumour,1 and usual sites of secondary spread are to liver, bone and lung. Although delayed recurrence of ipsilateral orbital melanoma is well documented, metastasis to the contralateral orbit is a rarely encountered phenomenon. We describe a case of metastatic spread to the contralateral orbit in a patient 12 years after proton beam radiotherapy of choroidal melanoma.</p>","PeriodicalId":89118,"journal":{"name":"Clinical medicine. Pathology","volume":"2 ","pages":"5-8"},"PeriodicalIF":0.0,"publicationDate":"2009-04-03","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://sci-hub-pdf.com/10.4137/cpath.s767","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"29530250","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"OA","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
引用次数: 32
Pharmacotherapeutic options for visceral leishmaniasis-current scenario. 内脏利什曼病的药物治疗选择-目前的情况。
Clinical medicine. Pathology Pub Date : 2009-01-23
Krishna Pandey, Prabhat Kumar Sinha, Vidyanand Ravi Das, Sanjiva Bimal, Shubhankar K Singh, Pradeep Das
{"title":"Pharmacotherapeutic options for visceral leishmaniasis-current scenario.","authors":"Krishna Pandey,&nbsp;Prabhat Kumar Sinha,&nbsp;Vidyanand Ravi Das,&nbsp;Sanjiva Bimal,&nbsp;Shubhankar K Singh,&nbsp;Pradeep Das","doi":"","DOIUrl":"","url":null,"abstract":"<p><p>Visceral leishmaniasis (VL) or Kala-azar is a protozoal disease, which was previously regarded as one of the most neglected tropical diseases. Management of this disease is quite difficult, because it is said to affect the poorest of the poor. Previously Sodium Stibogluconate (SSG) was regarded as the gold standard treatment for VL. But due to the increasing unresponsiveness, to this drug various other drugs were tried and are still being tried. Pentamidine is very toxic and has been discarded of late. Amphotericin B and its lipid formulations are very effective but require hospital admission and monitoring. Oral drugs like Miltefosine have already been launched. An amino glycoside Paromomycin and another oral drug Sitamaquine are in the pipe line. Interferon gamma has been used with discouraging results.</p>","PeriodicalId":89118,"journal":{"name":"Clinical medicine. Pathology","volume":"2 ","pages":"1-4"},"PeriodicalIF":0.0,"publicationDate":"2009-01-23","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2990237/pdf/","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"29530249","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"OA","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
引用次数: 0
Pharmacotherapeutic Options for Visceral Leishmaniasis—Current Scenario 内脏利什曼病的药物治疗选择-目前的情况
Clinical medicine. Pathology Pub Date : 2009-01-01 DOI: 10.4137/CPath.S821
K. Pandey, P. Sinha, V. R. Das, S. Bimal, Shubhankar K. Singh, P. Das
{"title":"Pharmacotherapeutic Options for Visceral Leishmaniasis—Current Scenario","authors":"K. Pandey, P. Sinha, V. R. Das, S. Bimal, Shubhankar K. Singh, P. Das","doi":"10.4137/CPath.S821","DOIUrl":"https://doi.org/10.4137/CPath.S821","url":null,"abstract":"Visceral leishmaniasis (VL) or Kala-azar is a protozoal disease, which was previously regarded as one of the most neglected tropical diseases. Management of this disease is quite difficult, because it is said to affect the poorest of the poor. Previously Sodium Stibogluconate (SSG) was regarded as the gold standard treatment for VL. But due to the increasing unresponsiveness, to this drug various other drugs were tried and are still being tried. Pentamidine is very toxic and has been discarded of late. Amphotericin B and its lipid formulations are very effective but require hospital admission and monitoring. Oral drugs like Miltefosine have already been launched. An amino glycoside Paromomycin and another oral drug Sitamaquine are in the pipe line. Interferon gamma has been used with discouraging results.","PeriodicalId":89118,"journal":{"name":"Clinical medicine. Pathology","volume":"112 1","pages":"1 - 4"},"PeriodicalIF":0.0,"publicationDate":"2009-01-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"79577324","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
引用次数: 9
Combination of immunohistochemistry and ploidy analysis to assist histopathological diagnosis of molar diseases. 结合免疫组织化学和倍体分析协助磨牙疾病的组织病理学诊断。
Clinical medicine. Pathology Pub Date : 2008-01-01 Epub Date: 2008-03-19 DOI: 10.4137/cpath.s601
M C Osterheld, L Caron, P Chaubert, K Meagher-Villemure
{"title":"Combination of immunohistochemistry and ploidy analysis to assist histopathological diagnosis of molar diseases.","authors":"M C Osterheld,&nbsp;L Caron,&nbsp;P Chaubert,&nbsp;K Meagher-Villemure","doi":"10.4137/cpath.s601","DOIUrl":"https://doi.org/10.4137/cpath.s601","url":null,"abstract":"<p><strong>Background: </strong>Differential diagnosis between hydropic abortion, partial mole and complete mole is still a challenge for pathologists but really important for patient management.</p><p><strong>Material and method: </strong>In this study, we have evaluated 111 products of conception from the first trimester. Histological analysis was made according to the main diagnostic histopathological features described in the literature and the cases were categorized in hydropic abortus (HA), partial mole (PM) and complete mole (CM). Immunohistochemistry was performed using monoclonal antibody against p57(kip) protein a putative paternally imprinted inhibitor gene and DNA ploidy was analysed in all cases by image cytometry.</p><p><strong>Results: </strong>All 23 HAs presented a diploid DNA content and were p57(kip2) positive. From the 28 CMs, 12 cases (43%) were diploid and 16 cases (57%) were tetraploid but no expression of p57(kip2) was found with positive internal controls. From the 60 PMs, 58 cases were positive for p57(kip2) expression and 53 cases (88%) were triploid, 6 cases (10%) tetraploid and 1 case (2%) diploid.</p><p><strong>Conclusion: </strong>This study on 111 cases of early pregnancies confirms the usefulness of immunohistochemistry and cytometry but demonstrates the importance of the combination of both techniques to assist histology for the best reliable diagnosis.</p>","PeriodicalId":89118,"journal":{"name":"Clinical medicine. Pathology","volume":"1 ","pages":"61-7"},"PeriodicalIF":0.0,"publicationDate":"2008-01-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://sci-hub-pdf.com/10.4137/cpath.s601","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"30107813","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"OA","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
引用次数: 10
Sclerosing 'mucinous' blue nevus: a clinical simulator of dermatofibroma. 硬化“粘液”蓝色痣:皮肤纤维瘤的临床模拟器。
Clinical medicine. Pathology Pub Date : 2008-01-01 Epub Date: 2008-03-19 DOI: 10.4137/cpath.s508
Claudia-S Vetter-Kauczok, Jürgen-C Becker, Eva-B Bröcker
{"title":"Sclerosing 'mucinous' blue nevus: a clinical simulator of dermatofibroma.","authors":"Claudia-S Vetter-Kauczok,&nbsp;Jürgen-C Becker,&nbsp;Eva-B Bröcker","doi":"10.4137/cpath.s508","DOIUrl":"https://doi.org/10.4137/cpath.s508","url":null,"abstract":"","PeriodicalId":89118,"journal":{"name":"Clinical medicine. Pathology","volume":"1 ","pages":"15-6"},"PeriodicalIF":0.0,"publicationDate":"2008-01-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://sci-hub-pdf.com/10.4137/cpath.s508","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"30107807","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"OA","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
引用次数: 2
Tumorigenic effects of tamoxifen on the female genital tract. 他莫昔芬对女性生殖道的致瘤作用。
Clinical medicine. Pathology Pub Date : 2008-01-01 Epub Date: 2008-03-01 DOI: 10.4137/cpath.s487
Kaei Nasu, Noriyuki Takai, Masakazu Nishida, Hisashi Narahara
{"title":"Tumorigenic effects of tamoxifen on the female genital tract.","authors":"Kaei Nasu, Noriyuki Takai, Masakazu Nishida, Hisashi Narahara","doi":"10.4137/cpath.s487","DOIUrl":"10.4137/cpath.s487","url":null,"abstract":"<p><p>Tamoxifen is widely used for endocrine treatment and breast cancer prevention. It acts as both an estrogen antagonist in breast tissue and an estrogen agonist in the female lower genital tract. Tamoxifen causes severe gynecologic side effects, such as endometrial cancer. This review focuses on the effects of prolonged tamoxifen treatment on the human female genital tract and considers its tumorigenicity in the gynecologic organs through clinical data analysis. Tamoxifen is associated with an increased incidence of benign endometrial lesions such as polyps and hyperplasia and a two- to four-fold increased risk of endometrial cancer in postmenopausal patients. Moreover, the incidence of functional ovarian cysts is significantly high in premenopausal tamoxifen users. To prevent tamoxifen from having severe side effects in gynecologic organs, frequent gynecological examination should be performed for both premenopausal and postmenopausal patients with breast cancer who are treated with this drug.</p>","PeriodicalId":89118,"journal":{"name":"Clinical medicine. Pathology","volume":"1 ","pages":"17-34"},"PeriodicalIF":0.0,"publicationDate":"2008-01-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3160006/pdf/","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"30107809","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"OA","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
引用次数: 0
Progesterone and Estrogen Receptors in Neurofibromas of Patients with NF1. NF1神经纤维瘤患者的孕激素和雌激素受体。
Clinical medicine. Pathology Pub Date : 2008-01-01 Epub Date: 2008-09-15 DOI: 10.4137/cpath.s1002
Mauro Geller, Spyros G E Mezitis, Fabio Pereira Nunes, Marcia G Ribeiro, Alexandra Prufer de Q C Araújo, Marcello D Bronstein, Rodrigo Siqueira-Batista, Andréia Patrícia Gomes, Lisa Oliveira, Karin Soares Gonçalves Cunha
{"title":"Progesterone and Estrogen Receptors in Neurofibromas of Patients with NF1.","authors":"Mauro Geller,&nbsp;Spyros G E Mezitis,&nbsp;Fabio Pereira Nunes,&nbsp;Marcia G Ribeiro,&nbsp;Alexandra Prufer de Q C Araújo,&nbsp;Marcello D Bronstein,&nbsp;Rodrigo Siqueira-Batista,&nbsp;Andréia Patrícia Gomes,&nbsp;Lisa Oliveira,&nbsp;Karin Soares Gonçalves Cunha","doi":"10.4137/cpath.s1002","DOIUrl":"https://doi.org/10.4137/cpath.s1002","url":null,"abstract":"<p><p>Neurofibromatosis type 1 (NF1) or von Recklinghausen disease is a genetic disorder affecting the growth of cells in nervous system. One of the most remarkable characteristics of this disease is the development of benign tumors of the nervous system (neurofibromas).The purpose of this study was to test tissue samples taken from neurofibromas and plexiform neurofibromas of NF1 patients for the presence of estrogen and progesterone receptors. We used previously collected samples from patients registered in the database of the Centro Nacional de Neurofibromatose (CNNF-Brazil). Samples from twenty-five patients in the database presenting plexiform neurofibromas (N1 group) and 25 samples from the same database from patients presenting neurofibromas (N2 group) were tested.We observed positive staining for progesterone receptors in 13 of the neurofibroma samples and 19 of the plexiform neurofibroma samples. Among the neurofibroma samples, we observed one sample with positive estrogen receptor staining, but none of the plexiform neurofibroma samples showed positive staining. We suggest further studies to investigate in greater depth possible hormonal influences on the development and growth of neurofibromas and plexiform neurofibromas in NF1.</p>","PeriodicalId":89118,"journal":{"name":"Clinical medicine. Pathology","volume":"1 ","pages":"93-7"},"PeriodicalIF":0.0,"publicationDate":"2008-01-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://sci-hub-pdf.com/10.4137/cpath.s1002","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"30106636","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"OA","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
引用次数: 19
Pathology-dependent histological changes of the left stellate Ganglia: a cadaveric study. 左侧星状神经节病理依赖性组织学改变:尸体研究。
Clinical medicine. Pathology Pub Date : 2008-01-01 Epub Date: 2008-10-30 DOI: 10.4137/cpath.s979
Salvatore Docimo, Carmen Piccolo, Daniel Van Arsdale, David E Elkowitz
{"title":"Pathology-dependent histological changes of the left stellate Ganglia: a cadaveric study.","authors":"Salvatore Docimo,&nbsp;Carmen Piccolo,&nbsp;Daniel Van Arsdale,&nbsp;David E Elkowitz","doi":"10.4137/cpath.s979","DOIUrl":"https://doi.org/10.4137/cpath.s979","url":null,"abstract":"<p><p>Sympathetic hyperinnervation due to nerve sprouting generated by the left stellate ganglion has been noted following cardiopulmonary disease processes. Sympathetic hyperinnervation seems to be limited to cardiopulmonary diseases in the experimental and clinical settings. However, histological changes of the left stellate ganglion following cardiopulmonary diseases in humans have vet to be observed. This study intends to investigate the histological changes of cadaveric sympathetic nervous tissue of left stellate ganglia (n = 32) and their relationship to noted pathology. Our study found fibrotic changes of the left stellate ganglion are not significantly dependent upon pathological processes, however, changes in the number of nerve cell bodies seems to be pathology dependent. A relationship between respiratory (mean = 33.3; P = 0.023) and cardiovascular pathologies (mean = 29.6; P = 0.199) and an increase in nerve cell bodies of the left stellate ganglion was noted when compared to other pathologies (mean = 25.7). The link between cardiopulmonary disease and sympathetic hyperinnervation may be the increase in the number of nerve cell bodies of the left stellate ganglion. Our results are clinically significant considering sympathetic hyperinnervation is associated with arrythmogenesis and an increase in morbidity and mortality in patients with pulmonary disease. Such findings may warrant investigation into the use of ganglion blockade in cardiopulmonary diseases.</p>","PeriodicalId":89118,"journal":{"name":"Clinical medicine. Pathology","volume":"1 ","pages":"105-13"},"PeriodicalIF":0.0,"publicationDate":"2008-01-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://sci-hub-pdf.com/10.4137/cpath.s979","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"30106638","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"OA","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
引用次数: 8
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