{"title":"Bedwetting at a health camp: lack of effect of dry-bed training.","authors":"E A Mitchell, M Sinclair","doi":"","DOIUrl":"","url":null,"abstract":"","PeriodicalId":75574,"journal":{"name":"Australian paediatric journal","volume":"22 3","pages":"228"},"PeriodicalIF":0.0,"publicationDate":"1986-08-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"14885231","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
{"title":"Follow up of premature and 'at risk' infants.","authors":"M O'Callaghan","doi":"","DOIUrl":"","url":null,"abstract":"","PeriodicalId":75574,"journal":{"name":"Australian paediatric journal","volume":"22 2","pages":"83-5"},"PeriodicalIF":0.0,"publicationDate":"1986-05-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"14848842","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
{"title":"Apnoea in the newborn infant.","authors":"D J Henderson-Smart, G Cohen","doi":"","DOIUrl":"","url":null,"abstract":"<p><p>Clinical apnoea in infants is defined as a pause in breathing of more than 20 s duration or a briefer pause associated with bradycardia. Such events are uncommon in term infants and usually are due to some precipitating factor. They occur more commonly in preterm infants and there is an exponential increase in incidence with decreasing gestational age at birth. Although insults such as infection or hypoxia can accentuate the problem, the major factor appears to be immaturity. Studies of brain-stem maturity using auditory evoked responses indicate that infants with immature responses have a higher incidence of apnoea. Brain-stem immaturity has also been implicated in histopathological studies of infants dying from Sudden Infant Death Syndrome (SIDS). In preterm infants, upper airway obstruction occurs commonly at the end of longer events and some infants have a predominantly obstructive pattern. These latter infants are often neurologically abnormal and have had prolonged endotracheal intubation. Both of there factors could be associated with disordered control of upper airway patency. This notion is supported by the finding of upper airway instability during nasal occlusion in these infants. A link with SIDS is uncertain, although preterm infants with chronic pulmonary insufficiency, usually following a prolonged intubation, are said to be at particularly high risk of dying suddenly and unexpectedly during infancy.</p>","PeriodicalId":75574,"journal":{"name":"Australian paediatric journal","volume":"22 Suppl 1 ","pages":"63-6"},"PeriodicalIF":0.0,"publicationDate":"1986-01-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"14661383","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
{"title":"Opioid peptides: a role in sudden infant death syndrome?","authors":"I C McMillen","doi":"","DOIUrl":"","url":null,"abstract":"<p><p>This brief review details some recent advances in the study of the development of the opioid peptides in the fetal central nervous system. The role of opioid peptides such as beta-endorphin and the enkephalins in the control of development of respiration in the newborn is discussed.</p>","PeriodicalId":75574,"journal":{"name":"Australian paediatric journal","volume":"22 Suppl 1 ","pages":"75-6"},"PeriodicalIF":0.0,"publicationDate":"1986-01-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"14082317","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
{"title":"Review of 'near-miss' sudden infant death syndrome and results of simplified pneumographic studies.","authors":"P M Rahilly","doi":"","DOIUrl":"","url":null,"abstract":"<p><p>Pneumographic studies were carried out on 92 infants suffering from 'near-miss' Sudden Infant Death Syndrome, 67 siblings of Sudden Infant Death Syndrome victims, and five infants of anxious parents. Medical investigations revealed a wide range of causes for episodes presenting as apnoea. The simple pneumographic studies had a 94% sensitivity and specificity for the prediction of future apnoeas.</p>","PeriodicalId":75574,"journal":{"name":"Australian paediatric journal","volume":"22 Suppl 1 ","pages":"53-4"},"PeriodicalIF":0.0,"publicationDate":"1986-01-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"14906966","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
{"title":"Changes in lung surfactant lipids associated with the sudden infant death syndrome.","authors":"R A Gibson, E J McMurchie","doi":"","DOIUrl":"","url":null,"abstract":"<p><p>The lipid composition of lung surfactant obtained by lung lavage at autopsy in 40 infants dying from Sudden Infant Death Syndrome (SIDS) was compared with that obtained from 12 infants dying from other causes (control group). Analysis of the total lipids extracted from the surfactant of the SIDS and the control groups showed no major differences in the proportion of the various phospholipids including the major phospholipid, phosphatidylcholine (PC). However, a significantly reduced proportion of the PC was in the form of the biologically active species, disaturated phosphatidylcholine (DSPC) (P less than 0.005). In surfactant from SIDS infants, mean DSPC amounted to 65.8% (s.e.m. = 1.6) compared with normal surfactant 77.4% (s.e.m. = 3.5). A functional consequence of such a reduction in DSPC content of lung surfactant of SIDS infants could be a greater degree of fluidity of surfactant, particularly at exhalation. Such a biophysical change in surfactant properties could have a profound influence on lung function and, perhaps in response to external or internal stimuli, could be a causative factor in sudden infant death.</p>","PeriodicalId":75574,"journal":{"name":"Australian paediatric journal","volume":"22 Suppl 1 ","pages":"77-80"},"PeriodicalIF":0.0,"publicationDate":"1986-01-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"14906969","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
{"title":"The pathology of cot death.","authors":"J L Emery","doi":"","DOIUrl":"","url":null,"abstract":"<p><p>Based on personal investigation of more than 2000 unexpected infant deaths it appears that 'cot deaths' do not form a pathological entity. This concept is supported by detailed pathological studies undertaken in a multicentre study of postperinatal mortality in the United Kingdom. Comprehensive necropsies were performed by paediatric pathologists on 519 infants including 293 whose death certificates indicated cot death. These 519 infants were examined for abnormalities of growth and development, metabolism and immunological status. Together with any evidence of atopy, and of old or recent tissue damage, a summation of disease present was obtained. Examined in this way the author believes that an explanation can be given for almost as many unexpected deaths as for deaths that occur in hospital, that is, are expected.</p>","PeriodicalId":75574,"journal":{"name":"Australian paediatric journal","volume":"22 Suppl 1 ","pages":"9-12"},"PeriodicalIF":0.0,"publicationDate":"1986-01-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"14906970","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
{"title":"'Near-miss' or 'near-myth' for sudden infant death syndrome? Clinical observations on 57 infants.","authors":"H Simpson, U M MacFadyen, J Y Paton","doi":"","DOIUrl":"","url":null,"abstract":"<p><p>A clinical approach to infants who may have 'nearly missed' Sudden Infant Death Syndrome (SIDS) is suggested as appropriate whether or not the eventual conclusion is that there is no connection between these surviving infants and those who die of SIDS. A non-restrictive investigative approach has been applied to 57 infants with episodes causing similar levels of alarm and recourse to medical aid but not exclusively having documented apnoea or need for cardiopulmonary resuscitation. Similarities among patients in past history, recent minor symptoms, modes of presentation and positive investigations support the concept that they may show a common response to a variety of pathophysiologic stresses. Epidemiological similarities to SIDS cases support the suggestion that SIDS may be the end-stage inadequate response to such stresses. Future studies should address the reasons for 'inappropriate' collapse, its treatment and prevention and be alert to any resultant effect upon the incidence of SIDS or further evidence of a link between the conditions.</p>","PeriodicalId":75574,"journal":{"name":"Australian paediatric journal","volume":"22 Suppl 1 ","pages":"47-51"},"PeriodicalIF":0.0,"publicationDate":"1986-01-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"14906964","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
{"title":"Scientific research into the sudden infant death syndrome: an introduction to the First Rotary Health Research Fund Conference.","authors":"A L Williams","doi":"","DOIUrl":"","url":null,"abstract":"","PeriodicalId":75574,"journal":{"name":"Australian paediatric journal","volume":"22 Suppl 1 ","pages":"3-4"},"PeriodicalIF":0.0,"publicationDate":"1986-01-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"14908478","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
{"title":"State-related physiological changes and risk for the sudden infant death syndrome.","authors":"R M Harper","doi":"","DOIUrl":"","url":null,"abstract":"<p><p>Sleep-states alter the influences exerted by suprapontine structures on respiratory and cardiac patterning. These influences consist of normal modulation exerted by descending cortical and limbic structures, and include hypothalamic temperature control. Rapid eye movement sleep dissociates hypothalamic activity from temperature control and nearly abolishes blood pressure rises and respiratory pattern changes induced by stimulation of suprapontine regions. Sleep-states also regulate the occurrence of aberrant cardiorespiratory influences from epileptiform activity manifested in suprapontine structures by altering the threshold for seizure incidence. Thus, sleep may pose particular states of vulnerability for the Sudden Infant Death Syndrome (SIDS), since both temperature influences on respiration and aberrant suprapontine epileptiform activity may be factors underlying the mechanism of failure in SIDS.</p>","PeriodicalId":75574,"journal":{"name":"Australian paediatric journal","volume":"22 Suppl 1 ","pages":"55-8"},"PeriodicalIF":0.0,"publicationDate":"1986-01-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"14906967","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}