The Egyptian heart journal : (EHJ) : official bulletin of the Egyptian Society of Cardiology最新文献

{"title":"Correction: Feasibility and safety of coronary catheter rolling technique (Jahanzeb technique) for radial artery catheterization on arterial perforation and radial artery patency.","authors":"Wahab Anwar, Ali Karim, Mubeena Javed, Poonam Kumari, Daya Kumari, F N U Karishma, Muhammad Salman, Hesham Naeem, Abida Perveen, Jahanzeb Malik","doi":"10.1186/s43044-026-00789-6","DOIUrl":"10.1186/s43044-026-00789-6","url":null,"abstract":"","PeriodicalId":74993,"journal":{"name":"The Egyptian heart journal : (EHJ) : official bulletin of the Egyptian Society of Cardiology","volume":"78 1","pages":""},"PeriodicalIF":0.0,"publicationDate":"2026-10-15","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://www.ncbi.nlm.nih.gov/pmc/articles/PMC13672724/pdf/","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"149198111","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"OA","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}

{"title":"Utilizing bedside VExUS and the S-prime velocity for real-time assessment of venous congestion on right ventricular function in heart failure.","authors":"Pramukh Hegde, Malvika P Prasad, Kingshuk Saha, Vimal Bhardwaj, Vikneswaran Gunaseelan, Nicolas Orozco, Arjun Alva","doi":"10.1186/s43044-026-00788-7","DOIUrl":"10.1186/s43044-026-00788-7","url":null,"abstract":"<p><strong>Background: </strong>Right ventricular (RV) function is a major determinant of outcomes in heart failure (HF), yet RV systolic performance and venous congestion (VC) are often evaluated separately. We investigated the relationship between tricuspid annular systolic velocity (S'), a tissue Doppler marker of RV systolic function and systemic VC measured by the Venous Excess Ultrasound (VExUS) score in patients with HF. In addition, RV-pulmonary artery (RV-PA) coupling parameters including TAPSE/PASP and S'/PASP were evaluated across VExUS grades.</p><p><strong>Methods: </strong>In this prospective, single-center observational study, 155 patients admitted with decompensated HF underwent standardized echocardiography, including VExUS assessment and S' measurement. Lung Ultrasound Score (LUS), RV-PA coupling indices, clinical characteristics, in-hospital mortality, and acute kidney injury (AKI) were also recorded.</p><p><strong>Results: </strong>The mean age was 57.5 years, with 68% of the cohort being males. A significant inverse correlation was observed between VExUS grade and S' velocity (p < 0.001). Patients with VExUS Grade 0 had higher S' velocities (median = 11.0 cm/s), while those with Grade 3 had the lowest (8.5 cm/s). Increasing VExUS grades were also associated with progressive worsening of TAPSE, TAPSE/PASP, and S'/PASP ratios, suggesting impaired RV-PA coupling with increasing venous congestion. Both higher VExUS grades and reduced S' were associated with more severe pulmonary congestion on LUS (p < 0.001 and p = 0.003, respectively). Although patients with higher VExUS grades and lower S' values trended towards worse outcomes, neither parameter significantly predicted in-hospital mortality or AKI.</p><p><strong>Conclusion: </strong>Systemic VC and RV systolic function are strongly and inversely related in HF. The combined use of VExUS and S' provides an integrated bedside assessment of hemodynamic status that may help guide decongestive therapy. Integration of RV-PA coupling parameters may further enhance bedside evaluation of hemodynamic compromise in HF patients. Additionally, S'/PASP and TAPSE/PASP demonstrated comparable trends across all HF groups, suggesting that S'/PASP may serve as a practical alternative marker of RV-PA coupling in heart failure patients. Larger studies are needed to determine their prognostic utility for predicting mortality and organ injury.</p><p><strong>Trial registration: </strong>This trial was registered with Clinical Trial Registry-India ( https://www.ctri.nic.in/ ), Trial Number - CTRI/2024/10/075219 on 14/10/2024.</p>","PeriodicalId":74993,"journal":{"name":"The Egyptian heart journal : (EHJ) : official bulletin of the Egyptian Society of Cardiology","volume":"78 1","pages":""},"PeriodicalIF":0.0,"publicationDate":"2026-10-06","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://www.ncbi.nlm.nih.gov/pmc/articles/PMC13642859/pdf/","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"149120007","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"OA","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}

{"title":"Unraveling cardiac amyloidosis in Egypt: diagnostic challenges and disease outcomes.","authors":"Rasha Mohamed Abayazeed, Ingy Etman, Mahmoud Hassanein, Hagar Mahmoud Kamal, Mohamed Elsakhawy, Ahmed Abdelaaty, Maram Allam, Nouran Soror, Ashraf Elghandour, Mohamed H Elbahoty, Sara Salama, Osama Mohammed Refai, Noha Elkholy","doi":"10.1186/s43044-026-00784-x","DOIUrl":"10.1186/s43044-026-00784-x","url":null,"abstract":"<p><strong>Background: </strong>Cardiac amyloidosis is an infiltrative cardiomyopathy caused by extracellular deposition of insoluble amyloid fibrils. Despite the growing global recognition of cardiac amyloidosis and the recent development of targeted therapeutic strategies for specific amyloidosis subtypes, demographic and epidemiological data regarding cardiac amyloidosis in Egypt remain limited. The aim of the study was to describe the demographic and clinical characteristics of cardiac amyloidosis in Egyptian patients; and to identify the diagnostic challenges associated with cardiac amyloidosis and the disease-related outcomes.</p><p><strong>Results: </strong>Among 84 patients initially evaluated for suspected cardiac amyloidosis at tertiary care facility in Egypt, 70 were confirmed to have the disease. The mean interval from symptom onset to diagnosis was 11.86 months. AL amyloidosis was the most common subtype, followed by AA amyloidosis. ATTR amyloidosis accounted for only 4.3% of cases, while the specific amyloidosis subtype could not be identified in 21.4% of patients. New complications developed in 21.4% of patients, with renal complications being the most common. The study observed a high mortality rate, with 48.6% of patients dying during the study period.</p><p><strong>Conclusions: </strong>This tertiary care center study of cardiac amyloidosis in Egyptian patients demonstrated a substantial diagnostic delay and poor clinical outcomes. In our study cohort, the most common subtype was AL amyloidosis and nearly half of the study population died during the study period, highlighting the advanced stage of disease at presentation and its grave prognosis. These findings underscore the need for greater clinical awareness, streamlined diagnostic pathways and improved access to comprehensive diagnostic modalities to ensure timely diagnosis and optimal patient care.</p>","PeriodicalId":74993,"journal":{"name":"The Egyptian heart journal : (EHJ) : official bulletin of the Egyptian Society of Cardiology","volume":"78 1","pages":""},"PeriodicalIF":0.0,"publicationDate":"2026-09-29","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://www.ncbi.nlm.nih.gov/pmc/articles/PMC13624237/pdf/","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"149083192","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"OA","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}

{"title":"Congenital muscular ventricular septal aneurysm coexisting with a pericardial cyst, a case report.","authors":"Qian Mou, Jin-Jing Li, Xue Li, Qiang Chen","doi":"10.1186/s43044-026-00786-9","DOIUrl":"10.1186/s43044-026-00786-9","url":null,"abstract":"<p><strong>Background: </strong>Muscular ventricular septal aneurysm (VSA) is commonly caused by myocardial infarction, myocarditis or trauma, but it may occasionally be congenital. For congenital VSA, the diagnosis is challenging, and the therapeutic strategy and prognosis remain uncertain.</p><p><strong>Case presentation: </strong>A 32-year-old man was admitted following a detection of left ventricular aneurysm on echocardiography during a routine health checkup. The patient was asymptomatic, the echocardiography showed an aneurysm in the midventricular septum associated with regional hypokinesis, accompanied by left ventricular dilation and reduced left ventricular ejection fraction (LVEF) of 46%. Coronary computed tomography angiography and cardiac magnetic resonance localized the muscular VSA and revealed a cyst at the anteroinferior margin of the pericardium, further coronary angiography was unremarkable. Laboratory findings such as cardiac markers, rheumatologic, immunologic and inflammatory markers were within normal limits. The diagnosis of congenital muscular VSA coexisting with a pericardial cyst was considered. Pharmacological treatment for heart failure was initiated to prevent further reduction in LVEF. At the 3-month and 1-year follow-ups the patient remained asymptomatic, and echocardiography demonstrated the LVEF increased from 50% to 57%.</p><p><strong>Conclusion: </strong>Congenital muscular VSA is an uncommon cardiac abnormality that may be accompanied by impaired cardiac function. Treatment for heart failure could improve the cardiac function.</p>","PeriodicalId":74993,"journal":{"name":"The Egyptian heart journal : (EHJ) : official bulletin of the Egyptian Society of Cardiology","volume":"78 1","pages":""},"PeriodicalIF":0.0,"publicationDate":"2026-09-28","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://www.ncbi.nlm.nih.gov/pmc/articles/PMC13620056/pdf/","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"149076950","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"OA","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}

{"title":"A rare congenital triad: ebsteinoid tricuspid valve, giant right atrial aneurysm, and atrial septal defect.","authors":"Uma Devi Karuru, Sunitha Arumulla, Neusha Doddi, Rama Kumari Nuthalapati","doi":"10.1186/s43044-026-00787-8","DOIUrl":"10.1186/s43044-026-00787-8","url":null,"abstract":"<p><strong>Background: </strong>Ebstein anomaly is a rare congenital malformation of the tricuspid valve, accounting for less than 1% of all congenital heart diseases. Ebsteinoid anomaly is an uncommon morphological variant characterized by abnormal tricuspid valve leaflet morphology without the classical apical displacement of the septal leaflet seen in Ebstein anomaly. The coexistence of an Ebsteinoid anomaly, giant right atrial aneurysm (RAA), and atrial septal defect (ASD) is exceptionally rare.</p><p><strong>Case presentation: </strong>A 15-year-old girl presented with progressive exertional dyspnoea (New York Heart Association [NYHA] class II) and intermittent palpitations. Chest radiography demonstrated marked cardiomegaly. Transthoracic echocardiography revealed a giant right atrium, severe low-pressure tricuspid regurgitation, an ostium secundum ASD, and an Ebsteinoid tricuspid valve with an elongated anterior leaflet and tethered septal leaflet, without apical displacement or right ventricular atrialization. Four-dimensional echocardiography and cardiac computed tomography confirmed the anatomical findings. Cardiac catheterization demonstrated pulmonary artery pressures approximately one-third of systemic pressure, confirming operability. The patient underwent successful tricuspid valve repair, ASD closure, and right atrial reduction atrioplasty. At one-year follow-up, she remained asymptomatic (NYHA class I) with preserved right ventricular function and only trivial residual tricuspid regurgitation.</p><p><strong>Conclusion: </strong>This case describes an exceptionally rare combination of Ebsteinoid tricuspid valve anomaly, giant right atrial aneurysm, and an ostium secundum ASD. Multimodality imaging was essential for accurate anatomical delineation and surgical planning. Early surgical correction resulted in excellent clinical and echocardiographic outcomes, underscoring the importance of timely intervention in complex congenital tricuspid valve anomalies.</p>","PeriodicalId":74993,"journal":{"name":"The Egyptian heart journal : (EHJ) : official bulletin of the Egyptian Society of Cardiology","volume":"78 1","pages":""},"PeriodicalIF":0.0,"publicationDate":"2026-09-27","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://www.ncbi.nlm.nih.gov/pmc/articles/PMC13616891/pdf/","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"149070732","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"OA","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}

{"title":"Sex disparities in STEMI care: pre-hospital delay and acute heart failure.","authors":"Artur Dziewierz, Barbara Zdzierak, Tomasz Rakowski","doi":"10.1186/s43044-026-00785-w","DOIUrl":"10.1186/s43044-026-00785-w","url":null,"abstract":"","PeriodicalId":74993,"journal":{"name":"The Egyptian heart journal : (EHJ) : official bulletin of the Egyptian Society of Cardiology","volume":"78 1","pages":""},"PeriodicalIF":0.0,"publicationDate":"2026-09-22","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://www.ncbi.nlm.nih.gov/pmc/articles/PMC13598034/pdf/","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"149027148","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"OA","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}

{"title":"Early administration of ivabradine in patients with acute anterior ST-elevation myocardial infarction: a prospective, randomized, open-label study on in-hospital and short-term major adverse cardiac events and left ventricular function.","authors":"Omar Mohammed Al Hawwari, Mohamed Elsayed Zahran, Khaled Mohamed Said Othman, Hazem Mansour","doi":"10.1186/s43044-026-00782-z","DOIUrl":"10.1186/s43044-026-00782-z","url":null,"abstract":"<p><strong>Background: </strong>Elevated heart rate (HR) in acute anterior ST-elevation myocardial infarction (STEMI) increases myocardial oxygen demand and limits perfusion, worsening outcomes. Ivabradine selectively reduces HR without negative inotropy. This open-label randomized controlled study investigates the efficacy and safety of early ivabradine administration in patients with acute anterior STEMI.</p><p><strong>Methods: </strong>This randomized controlled prospective study included 300 patients with acute anterior STEMI (sinus rhythm, HR > 70 bpm) at Ain Shams University hospitals. Patients were randomized 1:1 to receive either standard beta-blocker therapy plus ivabradine (Ivabradine group, n = 150) or standard beta-blocker therapy alone (Control group, n = 150). Echocardiographic assessors and clinical event adjudicators were blinded to treatment allocation. Primary outcomes were changes in left ventricular ejection fraction (LVEF), left ventricular end-diastolic volume (LVEDV), left ventricular end-systolic volume (LVESV), HR, cardiac biomarkers (CK-MB, Troponin), and the incidence of major adverse cardiac events (MACE) at 3-month follow-up. Procedural variables (door-to-balloon time, final TIMI flow, myocardial blush grade, no-reflow phenomenon), Killip class, arrhythmic events, and guideline-directed medical therapy use were documented.</p><p><strong>Results: </strong>Baseline demographic, angiographic, and medical therapy characteristics were similar between groups. At 3 months, the Ivabradine group demonstrated a significantly greater improvement in LVEF compared to controls (mean ± SD: 51.96 ± 6.09% vs. 49.79 ± 4.47%, p < 0.001). LVEDV significantly decreased in the Ivabradine group (from 101.44 ± 13.84 mL to 83.65 ± 8.76 mL) but increased in the control group (from 100.49 ± 12.73 mL to 120.67 ± 10.15 mL) (p < 0.001 for final LVEDV between groups). Final HR was significantly lower in the Ivabradine group (62.31 ± 1.79 vs. 76.13 ± 3.54 bpm, p < 0.001). The incidence of heart failure hospitalization (1.3% vs. 11.3%, p < 0.001) and total MACE (12.7% vs. 32.0%, p < 0.001) were significantly reduced in the Ivabradine group. No significant differences were observed in no-reflow rates (12.0% vs. 14.7%, p = 0.491) or ventricular arrhythmias (2.7% vs. 4.0%, p = 0.523) between groups.</p><p><strong>Conclusion: </strong>Early administration of ivabradine in patients with acute anterior STEMI and elevated HR without cardiogenic shock was associated with better heart-rate control and favorable short-term echocardiographic outcomes, while the clinical outcome findings require confirmation in prospectively registered, adequately powered multicenter trials. These findings apply to Killip class I-III patients; Killip class IV patients (cardiogenic shock) were excluded from this study.</p>","PeriodicalId":74993,"journal":{"name":"The Egyptian heart journal : (EHJ) : official bulletin of the Egyptian Society of Cardiology","volume":"78 1","pages":""},"PeriodicalIF":0.0,"publicationDate":"2026-09-22","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://www.ncbi.nlm.nih.gov/pmc/articles/PMC13597995/pdf/","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"149027025","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"OA","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}

{"title":"Favorable maternal and neonatal outcomes in probable Eisenmenger physiology with suspected DORV: a case report and literature review.","authors":"Mohamed Mukhtar Mohamed, Mehmet Necmeddin Sutaşır, Khadija Yusuf Ali, Osman Farah Dahir, Nurto Abdulkadir Said, Tuba Doğan, Sharmarke Hassan Ali, Nihat Müjdat Hökenek","doi":"10.1186/s43044-026-00781-0","DOIUrl":"10.1186/s43044-026-00781-0","url":null,"abstract":"<p><strong>Background: </strong>Pregnancy in Eisenmenger syndrome is associated with very high maternal and fetal risk. Complex congenital anatomy further complicates diagnosis and peripartum management, particularly where advanced imaging and pulmonary hypertension therapies are limited. Published evidence specifically addressing pregnancy with uncorrected DORV and Eisenmenger physiology remains sparse.</p><p><strong>Case presentation: </strong>A 25-year-old primigravida at 34 weeks' gestation presented with progressive dyspnoea and generalized oedema. The available record documented a heart rate of 130 beats/min, respiratory rate of 25 breaths/min, oxygen saturation of 80% despite face-mask oxygen, a loud second heart sound, and a systolic murmur. Echocardiography showed large ventricular and atrial septal defects with colour Doppler flow, severe tricuspid regurgitation, and an estimated systolic pulmonary artery pressure of 85 mmHg. In the setting of marked hypoxaemia, these findings supported probable Eisenmenger physiology, although shunt direction and pulmonary vascular resistance were not documented. The aorta appeared to arise predominantly from the right ventricle, supporting suspected DORV, but the origin of both great arteries, VSD-great-artery relationship, and DORV subtype could not be defined. Following cardiology and obstetric assessment, labour was induced and vaginal delivery occurred four hours later. The liveborn neonate weighed 2.7 kg, had an initial Apgar score of 5, and was admitted to neonatal intensive care for stabilisation and monitoring. Postpartum haemoglobin fell to 7 g/dL and two units of packed red cells were transfused. The mother was monitored in intensive care for three days, transferred to cardiology on day 6, and discharged in stable condition on day 7.</p><p><strong>Conclusion: </strong>This report documents maternal survival through induced vaginal delivery and the first postpartum week, with a live birth, despite late recognition of probable Eisenmenger physiology and suspected complex congenital heart disease. The report does not establish the safety of vaginal delivery in Eisenmenger syndrome, and incomplete haemodynamic, anatomical, anaesthetic, and neonatal data limit causal interpretation.</p>","PeriodicalId":74993,"journal":{"name":"The Egyptian heart journal : (EHJ) : official bulletin of the Egyptian Society of Cardiology","volume":"78 1","pages":""},"PeriodicalIF":0.0,"publicationDate":"2026-09-20","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://www.ncbi.nlm.nih.gov/pmc/articles/PMC13590458/pdf/","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"149009467","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"OA","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}

{"title":"An integrated overview of acute heart failure: a narrative review of pathophysiological mechanisms and contributing factors.","authors":"Mohammadjavad Sotoudeheian, Mohammad Moradi, Mohammad Pirhayati, Seyed-Mohamad-Sadegh Mirahmadi, Reza Azarbad, Mohammad Sedigh Dakkali, Mehdi Taghizadeh, Hamidreza Pazoki Toroudi","doi":"10.1186/s43044-026-00780-1","DOIUrl":"10.1186/s43044-026-00780-1","url":null,"abstract":"<p><strong>Background: </strong>Acute heart failure (AHF) is a significant cardiovascular syndrome characterized by either new-onset heart failure (HF) or acutely decompensated heart failure (ADHF), typically presenting with systemic and pulmonary congestion. The European Society of Cardiology has reinforced the importance of early guideline-directed medical therapy (GDMT) initiation and individualized decongestive strategies. This article explores current knowledge of AHF pathophysiology. The simplified hemodynamic paradigm previously used to explain AHF has been replaced over the past few decades by a more complex network of interacting mechanisms.</p><p><strong>Main text: </strong>The clinical presentation of AHF reflects a heterogeneous combination of cardiac abnormalities and systemic pathophysiological processes rather than a single disease entity. Despite overlapping clinical presentations, AHF arises from diverse underlying conditions and pathophysiological mechanisms. Left ventricular systolic or diastolic dysfunction elevates filling pressures, contributing to increased preload and afterload, which in turn promotes pulmonary congestion. End-organ dysfunction may result from the combined effects of venous congestion, impaired perfusion, and neurohormonal and inflammatory activation. Despite advances, limited progress in acute management may contribute to persistently poor outcomes in AHF patients.</p><p><strong>Conclusion: </strong>Initial management of AHF commonly includes intravenous diuretics for patients with congestion, while vasodilators and other therapies are selected according to congestion status, perfusion, blood pressure, hemodynamic profile, clinical phenotype, and the predominant underlying pathophysiological mechanism. This review examines pathophysiological processes contributing to AHF onset and progression, highlighting clinical implications and therapeutic targets.</p>","PeriodicalId":74993,"journal":{"name":"The Egyptian heart journal : (EHJ) : official bulletin of the Egyptian Society of Cardiology","volume":"78 1","pages":""},"PeriodicalIF":0.0,"publicationDate":"2026-09-18","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://www.ncbi.nlm.nih.gov/pmc/articles/PMC13589015/pdf/","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"149003108","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"OA","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}

{"title":"Acute aortic dissection masquerading as large bowel obstruction: a case report.","authors":"Olarinde Jeffrey Oluwademilade Nee Ogunmola, Azemobor Osigbeme, Henry Omagbeitse Abiyere, Eunice Oluremi Olabinri, Tolulope Fasanmi Kolawole","doi":"10.1186/s43044-026-00775-y","DOIUrl":"10.1186/s43044-026-00775-y","url":null,"abstract":"<p><strong>Background: </strong>Acute aortic dissection (AD) is a life-threatening disorder. A significant diagnostic pitfall is its ability to mimic an acute surgical abdomen, particularly in Stanford type B dissections. The aim of this report is to underscore the lifesaving step of appropriate imaging to probe the vascular integrity of the aorta before surgical intervention in a hypertensive patient that presented with classic features of large bowel acute intestinal obstruction.</p><p><strong>Case presentation: </strong>A 50-year-old hypertensive male presented with a one-week history of constipation, abdominal distension, and colicky pain, initially diagnosed as large bowel obstruction. Cardiologist review was requested due to persistent severe hypertension. Further evaluation with a contrast-enhanced CT imaging revealed an extensive Stanford type B (DeBakey type IIIb) aortic dissection involving the arch and abdominal aorta. The patient was managed conservatively with parenteral labetalol and opioid analgesics before transfer to a facility of his choice.</p><p><strong>Conclusion: </strong>In the setting of acute abdomen and severe hypertension, imaging to probe the vascular integrity of the aorta is essential to avoid life-threatening, inappropriate surgical interventions. The primary \"take-away\" lesson is the need for high suspicion of AD in hypertensive patients with abdominal symptoms.</p>","PeriodicalId":74993,"journal":{"name":"The Egyptian heart journal : (EHJ) : official bulletin of the Egyptian Society of Cardiology","volume":"78 1","pages":""},"PeriodicalIF":0.0,"publicationDate":"2026-09-17","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://www.ncbi.nlm.nih.gov/pmc/articles/PMC13586086/pdf/","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"148993150","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"OA","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}