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Epilepsy and Behavior Case Reports最新文献

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Improvement in anti-N-methyl-d-aspartate receptor antibody-mediated temporal lobe epilepsy with amygdala enlargement without immunotherapy 无免疫治疗的抗n -甲基-d-天冬氨酸受体抗体介导的颞叶癫痫伴杏仁核增大的改善
Epilepsy and Behavior Case Reports Pub Date : 2018-01-01 DOI: 10.1016/j.ebcr.2018.07.005
Go Taniguchi , Hitomi Fuse , Yumiko Okamura , Harushi Mori , Shinsuke Kondo , Kiyoto Kasai , Yukitoshi Takahashi , Keiko Tanaka
{"title":"Improvement in anti-N-methyl-d-aspartate receptor antibody-mediated temporal lobe epilepsy with amygdala enlargement without immunotherapy","authors":"Go Taniguchi ,&nbsp;Hitomi Fuse ,&nbsp;Yumiko Okamura ,&nbsp;Harushi Mori ,&nbsp;Shinsuke Kondo ,&nbsp;Kiyoto Kasai ,&nbsp;Yukitoshi Takahashi ,&nbsp;Keiko Tanaka","doi":"10.1016/j.ebcr.2018.07.005","DOIUrl":"10.1016/j.ebcr.2018.07.005","url":null,"abstract":"<div><p>Focal neuroinflammation is considered one of the hypotheses for the cause of temporal lobe epilepsy (TLE) with amygdala enlargement (AE). Here, we report a case involving an adult female patient with TLE-AE characterized by late-onset seizures and cognitive impairment. Anti-<em>N</em>-methyl-<span>d</span>-aspartate receptor (NMDAR) antibodies were detected in her cerebrospinal fluid. However, administration of appropriate anti-seizure drugs (ASD), without immunotherapy, improved TLE-AE associated with NMDAR antibodies. In the present case, two clinically significant observations were made: 1) anti-NMDAR antibody-mediated autoimmune processes may be associated with TLE-AE, and 2) appropriate administration of ASD alone can improve clinical symptoms in mild cases of autoimmune epilepsy.</p></div>","PeriodicalId":56365,"journal":{"name":"Epilepsy and Behavior Case Reports","volume":"10 ","pages":"Pages 96-98"},"PeriodicalIF":0.0,"publicationDate":"2018-01-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://sci-hub-pdf.com/10.1016/j.ebcr.2018.07.005","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"36459478","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"OA","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
引用次数: 1
Effect of plasmapheresis on serum levels of clobazam, levetiracetam and topiramate 血浆置换对氯巴唑、左乙拉西坦和托吡酯血清水平的影响
Epilepsy and Behavior Case Reports Pub Date : 2017-01-01 DOI: 10.1016/j.ebcr.2017.07.002
To Harmony Hau Man , Chang Richard Shek-kwan , Chan Angel On-kei , Chan Phoebe Wing Lam
{"title":"Effect of plasmapheresis on serum levels of clobazam, levetiracetam and topiramate","authors":"To Harmony Hau Man ,&nbsp;Chang Richard Shek-kwan ,&nbsp;Chan Angel On-kei ,&nbsp;Chan Phoebe Wing Lam","doi":"10.1016/j.ebcr.2017.07.002","DOIUrl":"10.1016/j.ebcr.2017.07.002","url":null,"abstract":"<div><p>A 27-year-old man with a diagnosis of new onset refractory status epilepticus (NORSE) was treated with five anti-seizure drugs (ASDs) including clobazam, levetiracetam and topiramate. He received plasma exchange (PE) for presumed autoimmune etiology. Serum ASD levels were serially monitored in two sessions. Levels of clobazam, levetiracetam and topiramate were significant reduced by PE. Serum clobazam level dropped down to at least 85% and 75% of the baseline during and after the procedure respectively; levetiracetam dropped down to 83% and 83%; and topiramate dropped to 86% and 79%. The results may imply a theoretical risk of breakthrough seizure during PE due to low ASD levels.</p></div>","PeriodicalId":56365,"journal":{"name":"Epilepsy and Behavior Case Reports","volume":"8 ","pages":"Pages 66-68"},"PeriodicalIF":0.0,"publicationDate":"2017-01-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://sci-hub-pdf.com/10.1016/j.ebcr.2017.07.002","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"35446142","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"OA","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
引用次数: 6
Paroxysmal dysphasia in a 68 year-old man: Enhancing the MRI spectrum! 68岁男性阵发性吞咽障碍:增强MRI频谱!
Epilepsy and Behavior Case Reports Pub Date : 2017-01-01 DOI: 10.1016/j.ebcr.2017.08.005
Ugur Sener , William O. Tatum , Alfredo Quinones-Hinojosa , Deependra Mahato , Anteneh M. Feyissa
{"title":"Paroxysmal dysphasia in a 68 year-old man: Enhancing the MRI spectrum!","authors":"Ugur Sener ,&nbsp;William O. Tatum ,&nbsp;Alfredo Quinones-Hinojosa ,&nbsp;Deependra Mahato ,&nbsp;Anteneh M. Feyissa","doi":"10.1016/j.ebcr.2017.08.005","DOIUrl":"10.1016/j.ebcr.2017.08.005","url":null,"abstract":"<div><p>Brain tumor-related epilepsy is a common complication of primary and metastatic brain tumors with seizures often representing the first manifestation of the tumor. The size and location of the tumor can make detection of epileptiform discharges on scalp electroencephalogram and safe surgical resection challenging. We describe a case of a patient with glioblastoma multiforme presenting as dominant temporal lobe epilepsy. Seizures were manifest as episodes of speech arrest on a background of long-standing history of episodic speech difficulty and headache. In this case, recognizing a change in semiology allowed diagnosis of a high-grade glioma. Use of electrocorticography during surgical excision of the tumor guided safe maximal excision without damage to eloquent cortex and helped confirm the diagnosis of brain tumor-related epilepsy.</p></div>","PeriodicalId":56365,"journal":{"name":"Epilepsy and Behavior Case Reports","volume":"8 ","pages":"Pages 96-99"},"PeriodicalIF":0.0,"publicationDate":"2017-01-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://sci-hub-pdf.com/10.1016/j.ebcr.2017.08.005","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"35540865","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"OA","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
引用次数: 1
Extreme delta brush evolving into status epilepticus in a patient with anti-NMDA encephalitis 抗nmda脑炎患者的极端三角刷演变为癫痫持续状态
Epilepsy and Behavior Case Reports Pub Date : 2017-01-01 DOI: 10.1016/j.ebcr.2016.09.002
Aline Herlopian MD , Eric S Rosenthal MD , Catherine J Chu MD , Andrew J Cole MD, FRCP , Aaron F Struck MD
{"title":"Extreme delta brush evolving into status epilepticus in a patient with anti-NMDA encephalitis","authors":"Aline Herlopian MD ,&nbsp;Eric S Rosenthal MD ,&nbsp;Catherine J Chu MD ,&nbsp;Andrew J Cole MD, FRCP ,&nbsp;Aaron F Struck MD","doi":"10.1016/j.ebcr.2016.09.002","DOIUrl":"10.1016/j.ebcr.2016.09.002","url":null,"abstract":"<div><p>Extreme delta brush (EDB) is an EEG pattern unique to anti-NMDA encephalitis. It is correlated with seizures and status epilepticus in patients who have a prolonged course of illness. The etiology of the underlying association between EDB and seizures is not understood. We present a patient with anti-NMDA encephalitis who developed status epilepticus evolving from the high frequency activity of the extreme delta brush. This case demonstrates that EDB is not only a marker for a greater propensity for seizures but also directly implicated in seizure generation.</p></div>","PeriodicalId":56365,"journal":{"name":"Epilepsy and Behavior Case Reports","volume":"7 ","pages":"Pages 69-71"},"PeriodicalIF":0.0,"publicationDate":"2017-01-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://sci-hub-pdf.com/10.1016/j.ebcr.2016.09.002","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"35089732","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"OA","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
引用次数: 9
Non-hyperammonaemic valproate encephalopathy after 20 years of treatment 非高氨血症丙戊酸脑病治疗20年后
Epilepsy and Behavior Case Reports Pub Date : 2017-01-01 DOI: 10.1016/j.ebcr.2017.04.002
Elizabeth Caruana Galizia , Jeremy D. Isaacs , Hannah R. Cock
{"title":"Non-hyperammonaemic valproate encephalopathy after 20 years of treatment","authors":"Elizabeth Caruana Galizia ,&nbsp;Jeremy D. Isaacs ,&nbsp;Hannah R. Cock","doi":"10.1016/j.ebcr.2017.04.002","DOIUrl":"10.1016/j.ebcr.2017.04.002","url":null,"abstract":"<div><p>Sodium valproate is a commonly used antiseizure drug with broad indications for different seizuretypes and epilepsy syndromes. Well-recognised side effects include weight gain, tremor, dizziness, and unsteadiness. Non-hyperammonaemic parkinsonism, with or without cognitive impairment, is a rare adverse effect of sodium valproate. We present the case of a sixty year-old lady with a generalized seizure disorder, treated with phenytoin, valproate, lamotrigine and clonazepam. Following withdrawal of phenytoin she developed an akinetic-rigid syndrome, with ataxia and marked cognitive impairment. Extensive investigation failed to identify a cause. Serum ammonia and valproate levels were normal. Hypothesizing this might be valproate encephalopathy, valproate was rapidly substituted with levetiracetam. Her severe motor symptoms resolved within two weeks and cognitive impairment markedly improved. Valproate-induced encephalopathy, with or without hyperammonaemia and liver toxicity are typically recognizable for their temporal relation between the start of therapy with valproate and emergence of the clinical syndrome. Reversible disorders of motor function and cognition attributable to valproate are well described, but few cases have been reported presenting years after starting treatment. Given the insidious progression, delayed onset, lack of association with drug levels or presence of hyperammonaemia, a high index of suspicion is needed to make the diagnosis.</p></div>","PeriodicalId":56365,"journal":{"name":"Epilepsy and Behavior Case Reports","volume":"8 ","pages":"Pages 9-11"},"PeriodicalIF":0.0,"publicationDate":"2017-01-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://sci-hub-pdf.com/10.1016/j.ebcr.2017.04.002","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"35118568","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"OA","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
引用次数: 9
A major miss in prognostication after cardiac arrest: Burst suppression and brain healing 心脏骤停后预测的主要失误:突发抑制和脑愈合
Epilepsy and Behavior Case Reports Pub Date : 2017-01-01 DOI: 10.1016/j.ebcr.2016.09.004
Danielle A. Becker , Nicholas D. Schiff , Lance B. Becker , Manisha G Holmes , Joseph J. Fins , James M. Horowitz , Orrin Devinsky
{"title":"A major miss in prognostication after cardiac arrest: Burst suppression and brain healing","authors":"Danielle A. Becker ,&nbsp;Nicholas D. Schiff ,&nbsp;Lance B. Becker ,&nbsp;Manisha G Holmes ,&nbsp;Joseph J. Fins ,&nbsp;James M. Horowitz ,&nbsp;Orrin Devinsky","doi":"10.1016/j.ebcr.2016.09.004","DOIUrl":"10.1016/j.ebcr.2016.09.004","url":null,"abstract":"<div><p>We report a case with therapeutic hypothermia after cardiac arrest where meaningful recovery far exceeded anticipated negative endpoints following cardiac arrest with loss of brainstem reflexes and subsequent status epilepticus. This man survived and recovered after an out-of-hospital cardiac arrest followed by a 6-week coma with absent motor responses and 5<!--> <!-->weeks of burst suppression. Standard criteria suggested no chance of recovery. His recovery may relate to the effect of burst-suppression on EEG to rescue neurons near neuronal cell death. Further research to understand the mechanisms of therapeutic hypothermia and late restoration of neuronal functional capacity may improve prediction and aid end-of-life decisions after cardiac arrest.</p></div>","PeriodicalId":56365,"journal":{"name":"Epilepsy and Behavior Case Reports","volume":"7 ","pages":"Pages 1-5"},"PeriodicalIF":0.0,"publicationDate":"2017-01-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://sci-hub-pdf.com/10.1016/j.ebcr.2016.09.004","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"74044055","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"OA","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
引用次数: 13
Non ictal onset zone: A window to ictal dynamics 非发病区:发病动态的窗口
Epilepsy and Behavior Case Reports Pub Date : 2017-01-01 DOI: 10.1016/j.ebcr.2017.10.003
Pegah Afra , Sara J. Hanrahan , Spencer Sterling Kellis , Paul House
{"title":"Non ictal onset zone: A window to ictal dynamics","authors":"Pegah Afra ,&nbsp;Sara J. Hanrahan ,&nbsp;Spencer Sterling Kellis ,&nbsp;Paul House","doi":"10.1016/j.ebcr.2017.10.003","DOIUrl":"10.1016/j.ebcr.2017.10.003","url":null,"abstract":"<div><p>The focal and network concepts of epilepsy present different aspects of electroclinical phenomenon of seizures. Here, we present a 23-year-old man undergoing surgical evaluation with left fronto-temporal electrocorticography (ECoG) and microelectrode-array (MEA) in the middle temporal gyrus (MTG). We compare action-potential (AP) and local field potentials (LFP) recorded from MEA with ECoG. Seizure onset in the mesial-temporal lobe was characterized by changes in the pattern of AP-firing without clear changes in LFP or ECoG in MTG. This suggests simultaneous analysis of neuronal activity in differing spatial scales and frequency ranges provide complementary insights into how focal and network neurophysiological activity contribute to ictal activity.</p></div>","PeriodicalId":56365,"journal":{"name":"Epilepsy and Behavior Case Reports","volume":"8 ","pages":"Pages 123-127"},"PeriodicalIF":0.0,"publicationDate":"2017-01-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://sci-hub-pdf.com/10.1016/j.ebcr.2017.10.003","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"35223096","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"OA","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
引用次数: 0
Vertical gaze palsy due to medication error 因用药错误导致垂直凝视麻痹
Epilepsy and Behavior Case Reports Pub Date : 2017-01-01 DOI: 10.1016/j.ebcr.2017.04.005
Shishir Rao , Meghan Harper-Shankie , Rajkumar Agarwal
{"title":"Vertical gaze palsy due to medication error","authors":"Shishir Rao ,&nbsp;Meghan Harper-Shankie ,&nbsp;Rajkumar Agarwal","doi":"10.1016/j.ebcr.2017.04.005","DOIUrl":"10.1016/j.ebcr.2017.04.005","url":null,"abstract":"<div><p>We present a teenage boy with recent onset of seizures, who was erroneously treated with a large dose of an antiseizure medication as a result of drug mix-up. The ensuing drug toxicity caused vertical gaze palsy, an unusual manifestation related to overdose of the agent. Timely recognition of the error and discontinuation of the drug resulted in complete recovery to baseline.</p></div>","PeriodicalId":56365,"journal":{"name":"Epilepsy and Behavior Case Reports","volume":"8 ","pages":"Pages 33-34"},"PeriodicalIF":0.0,"publicationDate":"2017-01-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://sci-hub-pdf.com/10.1016/j.ebcr.2017.04.005","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"35309839","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"OA","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
引用次数: 1
Effect of adjunctive perampanel on the quality of sleep and daytime somnolence in patients with epilepsy 辅助安眠药对癫痫患者睡眠质量及白天嗜睡的影响
Epilepsy and Behavior Case Reports Pub Date : 2017-01-01 DOI: 10.1016/j.ebcr.2016.10.002
Montserrat González-Cuevas , Odile Romero , Manuel Toledo , Manuel Quintana , Roser Cambrodí , Estevo Santamarina , Maria José Jurado , Alex Ferrer , Xavier Salas-Puig
{"title":"Effect of adjunctive perampanel on the quality of sleep and daytime somnolence in patients with epilepsy","authors":"Montserrat González-Cuevas ,&nbsp;Odile Romero ,&nbsp;Manuel Toledo ,&nbsp;Manuel Quintana ,&nbsp;Roser Cambrodí ,&nbsp;Estevo Santamarina ,&nbsp;Maria José Jurado ,&nbsp;Alex Ferrer ,&nbsp;Xavier Salas-Puig","doi":"10.1016/j.ebcr.2016.10.002","DOIUrl":"10.1016/j.ebcr.2016.10.002","url":null,"abstract":"<div><p>This prospective uncontrolled study evaluated the effect of low-dose adjunctive perampanel therapy (4<!--> <!-->mg/day for 3<!--> <!-->months) on the sleep-wake cycle and daytime somnolence in adult patients (n<!--> <!-->=<!--> <!-->10) with focal seizures. A &gt;<!--> <!-->50% reduction in the number of seizures was reported in 80% of the study patients; treatment had no significant effect on any sleep parameters as evident by the Maintenance of Wakefulness Test, Pittsburgh Sleep Quality Index and Epworth Sleepiness Scale scores. Two patients reported dizziness with treatment. In conclusion, low-dose perampanel may improve seizure control without affecting the sleep characteristics or daytime somnolence in patients with epilepsy.</p></div>","PeriodicalId":56365,"journal":{"name":"Epilepsy and Behavior Case Reports","volume":"7 ","pages":"Pages 13-15"},"PeriodicalIF":0.0,"publicationDate":"2017-01-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://sci-hub-pdf.com/10.1016/j.ebcr.2016.10.002","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"73799850","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"OA","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
引用次数: 17
Localization of ictal pouting in frontal lobe epilepsy: A case report 额叶癫痫发作时噘嘴的定位1例
Epilepsy and Behavior Case Reports Pub Date : 2017-01-01 DOI: 10.1016/j.ebcr.2017.04.003
Guray Koc , Semai Bek , Zeki Gokcil
{"title":"Localization of ictal pouting in frontal lobe epilepsy: A case report","authors":"Guray Koc ,&nbsp;Semai Bek ,&nbsp;Zeki Gokcil","doi":"10.1016/j.ebcr.2017.04.003","DOIUrl":"10.1016/j.ebcr.2017.04.003","url":null,"abstract":"","PeriodicalId":56365,"journal":{"name":"Epilepsy and Behavior Case Reports","volume":"8 ","pages":"Pages 27-30"},"PeriodicalIF":0.0,"publicationDate":"2017-01-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://sci-hub-pdf.com/10.1016/j.ebcr.2017.04.003","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"35223093","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"OA","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
引用次数: 7
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