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Stroke cases with apraxia of speech due to damage to the left frontal aslant tract 因左侧额叶斜束受损而出现语言障碍的中风病例
IF 0.4
Neurology and Clinical Neuroscience Pub Date : 2024-08-09 DOI: 10.1111/ncn3.12849
Masaharu Sawaki, Masahiko Yamamoto, Hiroyasu Yamamoto, Kenji Furukawa, Takahiro Yamauchi
{"title":"Stroke cases with apraxia of speech due to damage to the left frontal aslant tract","authors":"Masaharu Sawaki, Masahiko Yamamoto, Hiroyasu Yamamoto, Kenji Furukawa, Takahiro Yamauchi","doi":"10.1111/ncn3.12849","DOIUrl":"https://doi.org/10.1111/ncn3.12849","url":null,"abstract":"We encountered two acute stroke cases with apraxia of speech (AOS) due to left internal border‐zone infarction. Neither case had a lower precentral gyrus lesion in the left frontal lobe. The findings obtained confirmed the relationship between AOS with improvement and damage to the left frontal aslant tract (FAT) in stroke. FAT integrity has potential as a prognostic imaging biomarker of AOS. It is important to visualize the fiber connections of white matter using diffusion tensor tractography, which will predict the outcome of AOS.","PeriodicalId":19154,"journal":{"name":"Neurology and Clinical Neuroscience","volume":null,"pages":null},"PeriodicalIF":0.4,"publicationDate":"2024-08-09","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"141924267","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
引用次数: 0
Does Wilson's disease determine specific personality traits? Analysis of patients with the neurological form of the disease 威尔逊氏病是否决定了特定的人格特征？对神经型威尔森氏病患者的分析
IF 0.4
Neurology and Clinical Neuroscience Pub Date : 2024-07-26 DOI: 10.1111/ncn3.12847
J. Seniów, Magdalena Roessler-Górecka, A. Cudo, Anna Członkowska
{"title":"Does Wilson's disease determine specific personality traits? Analysis of patients with the neurological form of the disease","authors":"J. Seniów, Magdalena Roessler-Górecka, A. Cudo, Anna Członkowska","doi":"10.1111/ncn3.12847","DOIUrl":"https://doi.org/10.1111/ncn3.12847","url":null,"abstract":"In the autosomal recessive disorder of copper metabolism, Wilson's disease (WD), patients present hepatic, neurological, and psychiatric symptoms combined in different and unpredictable ways. Brain pathology primarily affects the basal ganglia and may cause movement, cognitive, affective, and behavioral disturbances. Personality traits have been rarely studied in the neurological form of the disease.To investigate whether patients with the neurological form of WD (WDn) have specific personality traits, especially those typical of frontobasal loop dysfunction.The personality of 29 adult WDn patients (without significant cognitive and psychiatric disorders) and 29 matched healthy controls was assessed using the Minnesota Multiphasic Personality Inventory (MMPI‐2).Compared to healthy individuals, patients with WDn scored higher on the following MMPI‐2 scales: hypochondriasis, physical malfunctioning, bizarre sensory experiences, health concerns, and general health concerns, with no differentiation between patients and controls on the remaining MMPI‐2 scales.Long‐term treated patients with WDn did not appear to have any specific personality traits reflecting unambiguously frontobasal psychopathology, if self‐report measurement is used. The more strongly exhibited features concerning their actual physical ailments and health anxiety, such as hypochondria, chronic fatigue, and poor psychophysical well‐being, could result from psychological reactions to a chronic disabling brain disease. Further study of personality traits and personality disorders is warranted in patients with clearly defined forms of WD, using various measurement tools. Psychopathological disorders in different forms of WD warrant further studies.","PeriodicalId":19154,"journal":{"name":"Neurology and Clinical Neuroscience","volume":null,"pages":null},"PeriodicalIF":0.4,"publicationDate":"2024-07-26","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"141798784","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
引用次数: 0
Pneumocephalus caused by Enterobacter cloacae meningoencephalitis following cerebral infarction 脑梗塞后由泄殖腔肠杆菌脑膜脑炎引起的脑积气
IF 0.4
Neurology and Clinical Neuroscience Pub Date : 2024-07-14 DOI: 10.1111/ncn3.12844
Mayumi Senda, Nozomi Harada, Konoka Kanda, T. Kako, Makoto Urano, Mizuki Ito
{"title":"Pneumocephalus caused by Enterobacter cloacae meningoencephalitis following cerebral infarction","authors":"Mayumi Senda, Nozomi Harada, Konoka Kanda, T. Kako, Makoto Urano, Mizuki Ito","doi":"10.1111/ncn3.12844","DOIUrl":"https://doi.org/10.1111/ncn3.12844","url":null,"abstract":"Pneumocephalus develops due to various reasons; however, pneumocephalus caused by factors other than head trauma is rare, and a few cases of infections have been reported. We encountered a case of pneumocephalus caused by Enterobacter cloacae meningoencephalitis following cerebral infarction. Although we could not perform cerebrospinal fluid analysis and culture, E. cloacae was cultured from blood. We conducted an autopsy and confirmed meningoencephalitis in the cerebral infarction lesion. Thus, we considered that E. cloacae meningoencephalitis caused the pneumocephalus. Because bacterial meningoencephalitis is a treatable disease, we should strictly consider that meningoencephalitis due to bacteria such as E. cloacae causes pneumocephalus.","PeriodicalId":19154,"journal":{"name":"Neurology and Clinical Neuroscience","volume":null,"pages":null},"PeriodicalIF":0.4,"publicationDate":"2024-07-14","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"141649864","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
引用次数: 0
Post‐extrapontine myelinolysis mimicking multiple system atrophy 模仿多系统萎缩的外展神经髓鞘后溶解症
IF 0.4
Neurology and Clinical Neuroscience Pub Date : 2024-07-12 DOI: 10.1111/ncn3.12845
Yukiko Inamori, Ayano Matsuyoshi, Yusuke Nakazawa, Wataru Shiraishi
{"title":"Post‐extrapontine myelinolysis mimicking multiple system atrophy","authors":"Yukiko Inamori, Ayano Matsuyoshi, Yusuke Nakazawa, Wataru Shiraishi","doi":"10.1111/ncn3.12845","DOIUrl":"https://doi.org/10.1111/ncn3.12845","url":null,"abstract":"","PeriodicalId":19154,"journal":{"name":"Neurology and Clinical Neuroscience","volume":null,"pages":null},"PeriodicalIF":0.4,"publicationDate":"2024-07-12","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"141654420","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
引用次数: 0
Recurrent ischemic strokes and elevated CA125 levels in a patient with Meigs syndrome: A case report 一名梅格斯综合征患者的复发性缺血性中风和 CA125 水平升高：病例报告
IF 0.4
Neurology and Clinical Neuroscience Pub Date : 2024-07-05 DOI: 10.1111/ncn3.12843
Hisashi Takahashi, R. Yasuda, Misaki Kubota‐Hanya, Keisuke Kitani, Suguru Yamashita, H. Kuroboshi, Mizuki Honda, Tetsuya Imura, Tomoyuki Ohara, Tadashi Kimura
{"title":"Recurrent ischemic strokes and elevated CA125 levels in a patient with Meigs syndrome: A case report","authors":"Hisashi Takahashi, R. Yasuda, Misaki Kubota‐Hanya, Keisuke Kitani, Suguru Yamashita, H. Kuroboshi, Mizuki Honda, Tetsuya Imura, Tomoyuki Ohara, Tadashi Kimura","doi":"10.1111/ncn3.12843","DOIUrl":"https://doi.org/10.1111/ncn3.12843","url":null,"abstract":"A 72‐year‐old woman developed left hemiparesis, and her initial magnetic resonance imaging (MRI) showed multiple acute ischemic lesions. Although she was treated with antithrombotic therapies, follow‐up MRI showed a recurrent small ischemic stroke. Her serum CA125 level was markedly elevated, and abdominal computed tomography (CT) and MRI showed a pelvic mass lesion with pleural effusions and ascites, suggesting malignant ovarian tumor associated with Trousseau's syndrome. However, the surgical resection specimens were pathologically diagnosed as benign ovarian fibroma and serous cystadenoma, and she was finally diagnosed as having Meigs syndrome. This case suggested that Meigs syndrome with elevated CA125 levels might play a role in the development of recurrent ischemic strokes.","PeriodicalId":19154,"journal":{"name":"Neurology and Clinical Neuroscience","volume":null,"pages":null},"PeriodicalIF":0.4,"publicationDate":"2024-07-05","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"141676591","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
引用次数: 0
A patient with neuronal intranuclear inclusion disease developed encephalitis‐like symptoms after cerebral angiography 一名神经元核内包涵体病患者在脑血管造影术后出现脑炎样症状
IF 0.4
Neurology and Clinical Neuroscience Pub Date : 2024-06-14 DOI: 10.1111/ncn3.12839
Shingo Koide, Shintaro Tsuboguchi, Shingo Koide, Itaru Ninomiya, Taiki Saito, T. Ishiguro, E. Saji, Yo Higuchi, Takeshi Ikeuchi, Makoto Oishi, Masato Kanazawa, Osamu Onodera
{"title":"A patient with neuronal intranuclear inclusion disease developed encephalitis‐like symptoms after cerebral angiography","authors":"Shingo Koide, Shintaro Tsuboguchi, Shingo Koide, Itaru Ninomiya, Taiki Saito, T. Ishiguro, E. Saji, Yo Higuchi, Takeshi Ikeuchi, Makoto Oishi, Masato Kanazawa, Osamu Onodera","doi":"10.1111/ncn3.12839","DOIUrl":"https://doi.org/10.1111/ncn3.12839","url":null,"abstract":"Patients with neuronal intranuclear inclusion disease (NIID) can present with encephalitis‐like symptoms such as recurrent paroxysmal fever and unconsciousness. To date, no specific triggers for these symptoms have been reported. In our case, an 78‐year‐old woman became unconscious and developed fever after cerebral angiography. The patient had experienced four episodes of unconsciousness and fever in the past 7 years. Postangiography, she immediately became unconscious and developed fever. No vascular abnormalities were found and magnetic resonance imaging of the brain revealed expanding white matter lesions and hyperintense lesions along the corticomedullary junction. Genetic analysis revealed an abnormal GGC repeat expansion in NOTCH2NLC. Thus, we diagnosed the patient with NIID. We suggest that cerebral angiography is a possible trigger for encephalitis‐like symptoms in NIID.","PeriodicalId":19154,"journal":{"name":"Neurology and Clinical Neuroscience","volume":null,"pages":null},"PeriodicalIF":0.4,"publicationDate":"2024-06-14","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"141345109","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
引用次数: 0
A case of SBMA presenting with myasthenic syndrome limited to a dropped head 一例因头下垂而出现肌无力综合征的 SBMA 患者
IF 0.4
Neurology and Clinical Neuroscience Pub Date : 2024-06-12 DOI: 10.1111/ncn3.12831
Hiroyuki Sumikura, Naoki Hatayama, Tomohiro Fujioka, Nozomi Nagashima, Yuki Shimada, I. Fukasaka, Manabu Sakaguchi
{"title":"A case of SBMA presenting with myasthenic syndrome limited to a dropped head","authors":"Hiroyuki Sumikura, Naoki Hatayama, Tomohiro Fujioka, Nozomi Nagashima, Yuki Shimada, I. Fukasaka, Manabu Sakaguchi","doi":"10.1111/ncn3.12831","DOIUrl":"https://doi.org/10.1111/ncn3.12831","url":null,"abstract":"A 76‐year‐old man with spinal and bulbar muscular atrophy (SBMA) developed an acute course of the dropped head with diurnal fluctuations that worsened in the evening. He was diagnosed with myasthenic syndrome based on a decreased response to repeated stimulation, as well as increased jitter and blocking on single‐fiber electromyography, indicating disturbed neuromuscular transmission. Dropped head symptom was successfully alleviated and maintained by immunotherapy. Although limited reports are available on SBMA cases accompanied by myasthenic syndrome and on the clinical impact of impaired neuromuscular transmission on SBMA, myasthenic syndrome may be a common condition and even a part of the phenotypic spectrum of SBMA. To our knowledge, this is the first case of SBMA comorbid with myasthenic syndrome limited to the dropped head.","PeriodicalId":19154,"journal":{"name":"Neurology and Clinical Neuroscience","volume":null,"pages":null},"PeriodicalIF":0.4,"publicationDate":"2024-06-12","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"141354249","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
引用次数: 0
A good therapeutic response to evocalcet for dropped head due to primary hyperparathyroidism: A case report 原发性甲状旁腺功能亢进症导致的头颅下垂对 evocalcet 的治疗反应良好：病例报告
IF 0.4
Neurology and Clinical Neuroscience Pub Date : 2024-06-11 DOI: 10.1111/ncn3.12837
Hiroyuki Sumikura, Hiromitsu Miyakawa, Takahiro Tomoda, Takuma Sato, Mai Ito, Yuki Shimada, Yoshiyasu Ueda, Manabu Sakaguchi
{"title":"A good therapeutic response to evocalcet for dropped head due to primary hyperparathyroidism: A case report","authors":"Hiroyuki Sumikura, Hiromitsu Miyakawa, Takahiro Tomoda, Takuma Sato, Mai Ito, Yuki Shimada, Yoshiyasu Ueda, Manabu Sakaguchi","doi":"10.1111/ncn3.12837","DOIUrl":"https://doi.org/10.1111/ncn3.12837","url":null,"abstract":"An 83‐year‐old woman presented with an acute course of dropped head syndrome (DHS) with muscle weakness localized to the neck. Serum calcium and intact parathyroid hormone (iPTH) levels were elevated to 12.2 mg/dL and 173 pg/mL, respectively, and 99mTc‐MIBI scintigraphy showed a hyperfunctioning parathyroid gland. Electromyography revealed myopathic features in the posterior neck extensor muscles. Primary hyperparathyroidism (PHPT)‐associated myopathy was diagnosed as the cause of the DHS. Oral administration of evocalcet ameliorated DHS and normalized the elevated serum calcium levels, whereas iPTH levels remained elevated. Subsequently, parathyroidectomy reduced the iPTH level to normal, and DHS did not flare‐up. Cases of PHPT with DHS as the initial symptom are extremely rare, and to our knowledge, this is the first report of remission of DHS with evocalcet. Therefore, serum iPTH levels may not necessarily correlate with muscle weakness.","PeriodicalId":19154,"journal":{"name":"Neurology and Clinical Neuroscience","volume":null,"pages":null},"PeriodicalIF":0.4,"publicationDate":"2024-06-11","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"141360454","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
引用次数: 0
Definition of refractory migraine and their evolution 难治性偏头痛的定义及其演变
IF 0.4
Neurology and Clinical Neuroscience Pub Date : 2024-06-07 DOI: 10.1111/ncn3.12835
S. Kikui, D. Danno, Takao Takeshima
{"title":"Definition of refractory migraine and their evolution","authors":"S. Kikui, D. Danno, Takao Takeshima","doi":"10.1111/ncn3.12835","DOIUrl":"https://doi.org/10.1111/ncn3.12835","url":null,"abstract":"The term ‘refractory migraine’ (RM) is commonly used in clinical settings; however, it is not recognized in the International Classification of Headache Disorders, third edition. A growing need for a shared definition of refractoriness has been highlighted by a multidisciplinary expert group. Although definitions for RM currently exist, the key parameters for the definition of refractoriness (e.g., unresponsiveness to treatment, high frequency, severe disability, or all of these features) remain contentious. Thus, a consensus on the definition of RM is crucial.Calcitonin gene‐related peptide (CGRP) is a neuropeptide that plays an important role in migraine pathophysiology and is a target for migraine preventive therapies. Monoclonal antibodies targeting the CGRP (i.e., galcanezumab, fremanezumab, and eptinezumab) and its receptor (erenumab) have shown consistent efficacy for migraine prophylaxis with excellent safety profiles. Their effect on refractory cases has also been reported, offering promise to the many patients who have not found relief with existing treatments. Therefore, we anticipate a paradigm shift in migraine treatment.Following the widespread use of monoclonal antibodies targeting the CGRP and its receptor, the European Headache Federation proposed a definition for two subsets of difficult‐to‐treat migraine—resistant and refractory migraine—that considers both the frequency and disability caused by single and frequent attacks.We expect that this definition will help resolve previous conflicts that have limited the use of earlier definitions.","PeriodicalId":19154,"journal":{"name":"Neurology and Clinical Neuroscience","volume":null,"pages":null},"PeriodicalIF":0.4,"publicationDate":"2024-06-07","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"141373771","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
引用次数: 0
Encephalitis mediated by the olfactory pathway can occur even during predominance of the Omicron mutant strain 嗅觉通路介导的脑炎即使在奥米克龙突变株占优势时也会发生
IF 0.4
Neurology and Clinical Neuroscience Pub Date : 2024-06-07 DOI: 10.1111/ncn3.12836
Kazuki Iwata, N. Yoshikura, Akio Kimura, T. Shimohata
{"title":"Encephalitis mediated by the olfactory pathway can occur even during predominance of the Omicron mutant strain","authors":"Kazuki Iwata, N. Yoshikura, Akio Kimura, T. Shimohata","doi":"10.1111/ncn3.12836","DOIUrl":"https://doi.org/10.1111/ncn3.12836","url":null,"abstract":"","PeriodicalId":19154,"journal":{"name":"Neurology and Clinical Neuroscience","volume":null,"pages":null},"PeriodicalIF":0.4,"publicationDate":"2024-06-07","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"141373855","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
引用次数: 0
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