Journal of oral pathology最新文献_第3页

Fibrous histiocytomas of the oral and maxillofacial regions. 口腔和颌面区纤维组织细胞瘤。

Journal of oral pathology Pub Date : 1984-06-01 DOI: 10.1111/j.1600-0714.1984.tb01426.x

S H Thompson, M Shear

{"title":"Fibrous histiocytomas of the oral and maxillofacial regions.","authors":"S H Thompson, M Shear","doi":"10.1111/j.1600-0714.1984.tb01426.x","DOIUrl":"https://doi.org/10.1111/j.1600-0714.1984.tb01426.x","url":null,"abstract":"Fibrous histiocytomas of the oral and maxillofacial regions are rare. The present study analyzes a sample of 63 cases from the literature together with 7 personally observed cases. Age, sex, site, size, clinical behaviour, treatment and survival were evaluated and related to a histological classification based on that of Rosai (1981). Lesions were assessed for histological criteria which would place them in one of 2 groups: fibrous histiocytoma (FH) or pleomorphic fibrous histiocytoma (PFH). Within these 2 histological groups the cases were subdivided into clinically benign, aggressive or malignant lesions. Thirty-nine cases (56.0%) were classified as FH and 31 cases (44.0%) as PFH. The mean age of male patients with PFH was significantly higher than female patients with FH (t = 3.05; d.f. 37; p less than 0.0025). Lesions in the PFH group involved bone more frequently than those in the FH group (Yate's Chi2 = 16.66; d.f. 1; p less than 0.00025). Lesions involving bone for both histological groups were more likely to be aggressive or malignant than soft-tissue lesions (Yate's Chi2 = 29.9; d.f. 1; p less than 0.00025). Soft-tissue lesions were usually less than 5 cm in greatest diameter. Radiographic features of malignancy for lesions with bone involvement is of prognostic importance regardless of the histological appearance of the lesion. The majority of the lesions under study were from the deep tissues of the oral and maxillofacial regions.","PeriodicalId":16672,"journal":{"name":"Journal of oral pathology","volume":null,"pages":null},"PeriodicalIF":0.0,"publicationDate":"1984-06-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://sci-hub-pdf.com/10.1111/j.1600-0714.1984.tb01426.x","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"17390708","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}

引用次数: 0

Development of dinitrochlorobenzene contact hypersensitivity in rat submaxillary salivary gland. 大鼠颌下唾液腺二硝基氯苯接触性超敏反应的进展。

Journal of oral pathology Pub Date : 1984-02-01 DOI: 10.1111/j.1600-0714.1984.tb01396.x

A R Mohammad

{"title":"Development of dinitrochlorobenzene contact hypersensitivity in rat submaxillary salivary gland.","authors":"A R Mohammad","doi":"10.1111/j.1600-0714.1984.tb01396.x","DOIUrl":"https://doi.org/10.1111/j.1600-0714.1984.tb01396.x","url":null,"abstract":"Stimulation of cell-mediated immunity (CMI) by 2,4 dinitrochlorobenzene (DNCB) was reported to be effective in clinical regression of some carcinomas and precancerous lesions. This study investigated development of CMI in rat submaxillary salivary glands with dermal application of DNCB to provide a model for the study of immunotherapy in salivary gland neoplasia. Twenty rats received 0.02 ml of 0.5% DNCB in 4:1 acetone corn-oil applied with a glass rod to 3 cm2 of clipped ventral skin of the neck covering the submaxillary salivary gland on 2 successive days. Ten days later, the submaxillary glands were challenged with an injection of 0.02 ml of DNCB. Thirty-two h later the animals were killed and the glands examined grossly and microscopically. Thirty control animals were sensitized and challenged, (a) 10 with 0.5% DNCB, then vehicle only, (b) 10 with vehicle only, then 0.5% DNCB, and (c) 10 with vehicle both times. Gross examination of experimental and control glands disclosed moderate edema and congestion. Microscopically, experimental glands showed inflammatory changes including infiltration, vascular congestion and perivascular cuffing of lymphocytes. Regional lymph nodes showed proliferation of cortical germinal centers only. Control glands showed acute vascular congestion in group (a), and absence of microscopic features of delayed hypersensitivity.","PeriodicalId":16672,"journal":{"name":"Journal of oral pathology","volume":null,"pages":null},"PeriodicalIF":0.0,"publicationDate":"1984-02-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://sci-hub-pdf.com/10.1111/j.1600-0714.1984.tb01396.x","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"17481253","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}

引用次数: 0

Immunohistochemical demonstration of plasma protein in squamous epithelium of formalin-fixed, paraffin-embedded oral mucosa. 福尔马林固定、石蜡包埋口腔黏膜鳞状上皮中血浆蛋白的免疫组化分析。

Journal of oral pathology Pub Date : 1984-02-01 DOI: 10.1111/j.1600-0714.1984.tb01403.x

J Reibel

引用次数: 7

Glucocorticosteroids and oral medicine. 糖皮质激素和口服药物。

Journal of oral pathology Pub Date : 1984-02-01 DOI: 10.1111/j.1600-0714.1984.tb01395.x

A Pedersen, B Klausen

引用次数: 25

Experimental auto-allergic sialoadenitis in male rats. 实验性雄性大鼠自身变应性涎腺炎。

Journal of oral pathology Pub Date : 1984-02-01 DOI: 10.1111/j.1600-0714.1984.tb01401.x

D H Dean, R N Hiramoto

引用次数: 3

Plasma amylase after parotid traumatization or duct ligation of male rats. 雄性大鼠腮腺外伤或导管结扎后血浆淀粉酶的变化。

Journal of oral pathology Pub Date : 1984-02-01 DOI: 10.1111/j.1600-0714.1984.tb01402.x

D H Dean, R N Hiramoto

引用次数: 2

Distribution of keratin and laminin in ameloblastoma. Comparison with developing tooth and epidermoid carcinoma. 成釉细胞瘤中角蛋白和层粘连蛋白的分布。发展中的牙齿癌与表皮样癌的比较。

Journal of oral pathology Pub Date : 1984-02-01 DOI: 10.1111/j.1600-0714.1984.tb01404.x

I Thesleff, P Ekblom

{"title":"Distribution of keratin and laminin in ameloblastoma. Comparison with developing tooth and epidermoid carcinoma.","authors":"I Thesleff, P Ekblom","doi":"10.1111/j.1600-0714.1984.tb01404.x","DOIUrl":"https://doi.org/10.1111/j.1600-0714.1984.tb01404.x","url":null,"abstract":"The nature of the tumor cells in 5 cases of ameloblastomas was studied by immunohistochemistry, and the findings were compared with developing mouse and human teeth as well as with 5 cases of carcinomas in the oral region. The antigens investigated were keratin, an intracellular cytoskeletal protein typical of epithelial cells, and laminin, an extracellular matrix protein found in basement membranes. Our results show that keratin is expressed by all types of epithelial cells in ameloblastomas as well as in the epidermoid carcinomas, and developing teeth. The epithelial, keratin-positive tumor islands in the ameloblastomas were surrounded by a continuous line of laminin, in a pattern similar to that seen in developing tooth. Laminin was seen also around the epidermoid carcinomas but large areas devoid of laminin were constantly seen between the stroma and the neoplastic epithelium. This indicates a lack of proper basement membrane formation by the malignant epidermoid carcinomas. This may be due either to a diminished production or an increased degradation of basement membrane proteins by the carcinoma cells. Our results are in line with suggestions that ameloblastomas are derived from odontogenic epithelial cells. Immunostaining for keratin does not distinguish between carcinomas and the ameloblastomas. However, visualization of basement membrane proteins such as laminin can apparently be used in the differential diagnosis between ameloblastomas and carcinomas.","PeriodicalId":16672,"journal":{"name":"Journal of oral pathology","volume":null,"pages":null},"PeriodicalIF":0.0,"publicationDate":"1984-02-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://sci-hub-pdf.com/10.1111/j.1600-0714.1984.tb01404.x","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"17263798","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}

引用次数: 56

Rheological properties of saliva substitutes containing mucin, carboxymethylcellulose or polyethylenoxide. 含粘蛋白、羧甲基纤维素或聚环氧乙烷的唾液替代品的流变性能。

Journal of oral pathology Pub Date : 1984-02-01 DOI: 10.1111/j.1600-0714.1984.tb01397.x

A Vissink, H A Waterman, E J s-Gravenmade, A K Panders, A Vermey

引用次数: 112

Immunocytochemical demonstration of intermediate filaments in a granular cell ameloblastoma. 颗粒细胞成釉细胞瘤中中间纤维的免疫细胞化学显示。

Journal of oral pathology Pub Date : 1984-02-01 DOI: 10.1111/j.1600-0714.1984.tb01398.x

P C de Wilde, P J Slootweg, H Müller, A Kant, O Moesker, P Vooijs, F C Ramaekers

引用次数: 32

Effects of vitamin A deficiency on rat incisor formation. 维生素A缺乏对大鼠切牙形成的影响。

Journal of oral pathology Pub Date : 1984-02-01 DOI: 10.1111/j.1600-0714.1984.tb01399.x

J T Punyasingh, S Hoffman, S S Harris, J M Navia

{"title":"Effects of vitamin A deficiency on rat incisor formation.","authors":"J T Punyasingh, S Hoffman, S S Harris, J M Navia","doi":"10.1111/j.1600-0714.1984.tb01399.x","DOIUrl":"https://doi.org/10.1111/j.1600-0714.1984.tb01399.x","url":null,"abstract":"Vitamin A deficiency (A-) is known to cause morphologic changes in tooth structures. However, its effects on glycosaminoglycan (GAG) distribution in dental pulp, and the role of retinoic acid (RA) in altering these effects are not clearly defined. Tissue changes induced by vitamin A deficiency and RA administration were evaluated histologically in incisors of rats fed on one of 3 different diets: a) vitamin A sufficient (A+); b) vitamin A deficient (A-); and c) vitamin A deficient supplemented with retinoic acid (A-/RA). Four weeks after the onset of vitamin A deficiency, all rats were killed and their 4 continuously erupting incisors evaluated histologically. A- rats had altered dentine and pulp with disrupted histodifferentiation of pulpal mesenchymal cells to normal odontoblasts. The frequency of these abnormalities in dentine and pulp was lower in A-/RA rats. The enamel organ was unremarkable in the 4-week deficient period. Using special stains, we noted that pulpal GAG accumulation in A- and A-/RA rats was limited to the lingual area, while in A+ rats, GAG were distributed throughout. These data suggest that vitamin A deficiency affects histodifferentiation of pulpal mesenchymal cells to odontoblasts, as well as GAG distribution in pulp. RA administration reduces the A- changes and therefore, appears to have some activity in dentinogenesis.","PeriodicalId":16672,"journal":{"name":"Journal of oral pathology","volume":null,"pages":null},"PeriodicalIF":0.0,"publicationDate":"1984-02-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://sci-hub-pdf.com/10.1111/j.1600-0714.1984.tb01399.x","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"17481256","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}

引用次数: 26