{"title":"Responses to the views and commentary on \"Standard specification for transferring digital neurophysiological data between independent computer systems\" (ASTM E1467-92) (J Clin Neurophysiol 1993; 10:397)","authors":"P. Le Bars, N. Boutros","doi":"10.1097/00004691-199310000-00015","DOIUrl":"https://doi.org/10.1097/00004691-199310000-00015","url":null,"abstract":"","PeriodicalId":117726,"journal":{"name":"Journal of clinical neurophysiology : official publication of the American Electroencephalographic Society","volume":"297 1","pages":"0"},"PeriodicalIF":0.0,"publicationDate":"1993-10-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"115924187","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
{"title":"Standard specification for transferring digital neurophysiological data between independent computer systems (ASTM E1467-92)","authors":"J. Daube, P. Low, W. Litchy, F. Sharbrough","doi":"10.1097/00004691-199307000-00016","DOIUrl":"https://doi.org/10.1097/00004691-199307000-00016","url":null,"abstract":"","PeriodicalId":117726,"journal":{"name":"Journal of clinical neurophysiology : official publication of the American Electroencephalographic Society","volume":"11 1","pages":"0"},"PeriodicalIF":0.0,"publicationDate":"1993-07-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"126433285","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
{"title":"The UCLA Epilepsy Program: historical review 1960-1992.","authors":"P. Crandall, T. Babb","doi":"10.1097/00004691-199304000-00010","DOIUrl":"https://doi.org/10.1097/00004691-199304000-00010","url":null,"abstract":"","PeriodicalId":117726,"journal":{"name":"Journal of clinical neurophysiology : official publication of the American Electroencephalographic Society","volume":"4 1","pages":"0"},"PeriodicalIF":0.0,"publicationDate":"1993-04-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"127813625","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
{"title":"Evoked potentials in infants and children.","authors":"P. Kotagal","doi":"10.1097/00004691-199207010-00001","DOIUrl":"https://doi.org/10.1097/00004691-199207010-00001","url":null,"abstract":"","PeriodicalId":117726,"journal":{"name":"Journal of clinical neurophysiology : official publication of the American Electroencephalographic Society","volume":"293 1","pages":"0"},"PeriodicalIF":0.0,"publicationDate":"1992-07-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"133096708","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
{"title":"In memoriam Albert Melvin Grass. 1910-1992.","authors":"M. Henry","doi":"10.1097/00004691-199207010-00008","DOIUrl":"https://doi.org/10.1097/00004691-199207010-00008","url":null,"abstract":"","PeriodicalId":117726,"journal":{"name":"Journal of clinical neurophysiology : official publication of the American Electroencephalographic Society","volume":"1 1","pages":"0"},"PeriodicalIF":0.0,"publicationDate":"1992-07-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"129978942","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
{"title":"The American Electroencephalographic Society: the original cast of characters.","authors":"J. Knott, D. Lindsley, C. E. Henry","doi":"10.1097/00004691-199207010-00009","DOIUrl":"https://doi.org/10.1097/00004691-199207010-00009","url":null,"abstract":"","PeriodicalId":117726,"journal":{"name":"Journal of clinical neurophysiology : official publication of the American Electroencephalographic Society","volume":"13 1","pages":"0"},"PeriodicalIF":0.0,"publicationDate":"1992-07-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"131560875","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
{"title":"Magnetic stimulation of the nervous system.","authors":"J. D. Weissman, C. Epstein","doi":"10.1080/00029238.1992.11080401","DOIUrl":"https://doi.org/10.1080/00029238.1992.11080401","url":null,"abstract":"ABSTRACT.Magnetic stimulation is a painless and promising new technique for studying the nervous system. The basic theory and electronic circuits are straightforward. The magnetic field easily activates cerebral cortex and deep nerves that are inaccessible to conventional stimuli. Most applications thus far have been in the motor system. Thousand of patients with different neurological diseases have been studied clinically without adverse effects, and research applications are expanding.","PeriodicalId":117726,"journal":{"name":"Journal of clinical neurophysiology : official publication of the American Electroencephalographic Society","volume":"9 1","pages":"0"},"PeriodicalIF":0.0,"publicationDate":"1992-06-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"121567960","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
{"title":"Neuronal excitability: voltage-dependent currents and synaptic transmission.","authors":"Paul Rutecki","doi":"10.1097/00004691-199204010-00003","DOIUrl":"https://doi.org/10.1097/00004691-199204010-00003","url":null,"abstract":"Neuronal membrane excitability and the synaptic connections among neurons produce behavior and cognition. The intracellular compartment of neurons is negatively charged relative to the extracellular space, and this charge, as well as current flow, is produced by ions. From the perspective of charged ions, the lipid bilayer of the neuronal membrane acts as a capacitor, and transmembrane glycoprotein pores or channels act as resistors. The open and closed states of ionic channels determine the membrane potential. At equilibrium, the lowest resistance or greatest permeability is for potassium, and the resting membrane potential is close to the equilibrium potential for potassium. When a channel is opened, permeable ions diffuse down their electrochemical gradients and the membrane potential is changed. Channels are gated (opened or closed) by voltage, neurotransmitters, and second messengers. The neuron integrates synaptic potentials produced by transmitter-gated channel activity and either generates a subthreshold potential, or a suprathreshold depolarization that generates an action potential or a burst of action potentials. Action potential generation is mediated by a large, brief sodium influx that is followed by activation of a voltage-dependent potassium eflux. The pattern of action potential firing is dependent on the interaction of a repertoire of voltage-dependent ion conductances. The action potential is the main signaling mechanism to activate synaptic transmission at axon terminals. Synaptic transmission is graded depending on the amount of calcium entering the presynaptic terminal. The number of action potentials, or the shape of the action potential, will determine the amount of calcium entering the terminal and the efficacy of synaptic transmission. Presynaptic ion channels may also be controlled by neurotransmitters or modulators and affect synaptic transmission by altering the amount of calcium influx.","PeriodicalId":117726,"journal":{"name":"Journal of clinical neurophysiology : official publication of the American Electroencephalographic Society","volume":"66 1","pages":"0"},"PeriodicalIF":0.0,"publicationDate":"1992-04-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"121961862","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
{"title":"Sudden unexpected death in epilepsy: a series from an epilepsy surgery program and speculation on the relationship to sudden cardiac death.","authors":"R. Dasheiff","doi":"10.1097/00004691-199104000-00010","DOIUrl":"https://doi.org/10.1097/00004691-199104000-00010","url":null,"abstract":"Sudden unexpected death represents a significant cause of mortality in people with epilepsy. It derives this significance not because it is the most frequent cause of death but because it is apparently a direct consequence of a seizure. The implication is that epilepsy is an inherently lethal disorder. Seven patients who were studied in an epilepsy surgery program died a sudden unexpected death. This incidence of sudden unexpected death was five times higher than the 1-2/1,000 per year reported in the general epilepsy population. Sudden unexpected death shares some of the characteristics associated with sudden cardiac death, which kills 300,000 people in the United States each year. A cardiac arrhythmia, usually ventricular fibrillation, is the most common terminal event for sudden cardiac death and is the leading candidate as the mechanism for sudden unexpected death. Despite this knowledge, little is known on how to identify a high-risk group of patients for sudden death or how these deaths might be prevented.","PeriodicalId":117726,"journal":{"name":"Journal of clinical neurophysiology : official publication of the American Electroencephalographic Society","volume":"65 1","pages":"0"},"PeriodicalIF":0.0,"publicationDate":"1991-04-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"132739994","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
{"title":"Episodic low-amplitude events: an under-recognized phenomenon in clinical electroencephalography.","authors":"A. Rae-Grant, C. Strapple, P. Barbour","doi":"10.1097/00004691-199104000-00008","DOIUrl":"https://doi.org/10.1097/00004691-199104000-00008","url":null,"abstract":"In a series of 20 EEGs from 15 patients, well-defined brief attenuations occurred interspersed among the background activity. These episodic low-amplitude events (ELAEs) typically lasted 0.5-4 s. They were hemispheric or bisynchronous and occurred in patients with coma of various etiologies, including status epilepticus. The episodes of attenuation were brief and no bursts of activity were present, distinguishing this finding from burst-suppression. Prognosis was poor in the patients with coma due to entities other than status epilepticus. In the setting of status epilepticus, the prognosis depended on the etiology. This pattern may be an ictal phenomenon, or a product of waveform simplification. ELAEs are a manifestation of seriously abnormal EEG activity and correlate with a 50% mortality.","PeriodicalId":117726,"journal":{"name":"Journal of clinical neurophysiology : official publication of the American Electroencephalographic Society","volume":"212 1","pages":"0"},"PeriodicalIF":0.0,"publicationDate":"1991-04-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"121718844","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}