Clinical and Experimental Hypertension最新文献_第4页

Safety and efficacy of extravascular renal denervation using HyperQure™ renal denervation system in short-term swine model of hypertension. HyperQure™肾去神经系统对短期猪高血压模型血管外肾去神经的安全性和有效性

IF 1.5 4区医学

Clinical and Experimental Hypertension Pub Date : 2025-12-01 Epub Date: 2025-03-04 DOI: 10.1080/10641963.2025.2474520

Jang Hee Han, Yerzhan Sharapatov, Minh-Tung Do, Sang Youn Kim, Bogyeong Han, Eue-Keun Choi, Chang Wook Jeong

{"title":"Safety and efficacy of extravascular renal denervation using HyperQure™ renal denervation system in short-term swine model of hypertension.","authors":"Jang Hee Han, Yerzhan Sharapatov, Minh-Tung Do, Sang Youn Kim, Bogyeong Han, Eue-Keun Choi, Chang Wook Jeong","doi":"10.1080/10641963.2025.2474520","DOIUrl":"10.1080/10641963.2025.2474520","url":null,"abstract":"This study investigated the safety and efficacy of the HyperQure™ extravascular renal denervation (RDN) system in a swine model of mild hypertension. Ten female pigs were fed a 3% salt diet to induce hypertension and underwent either extravascular RDN using the HyperQure™ RDN system (n = 7) or a sham procedure (n = 3). Blood pressure (BP) was continuously monitored using implanted transmitters, and safety assessments were conducted via computed tomography angiography (CTA) at 28 days post-procedure. The primary endpoint was the change in systolic blood pressure (SBP) at four weeks, while secondary endpoints included changes in diastolic BP (DBP), mean arterial pressure (MAP), and histological evaluation of renal nerve and artery integrity. At four weeks, SBP decreased by 11.8 ± 5.2 mmHg in the RDN group compared to an increase of 6.4 ± 4.2 mmHg in controls, resulting in a mean difference of 18.2 mmHg (p < .05). Similar improvements were observed in DBP and MAP, with mean differences of 15.4 and 16.2 mmHg, respectively (both p < .05). CTA revealed no significant renal artery or intraperitoneal organ injury. Histological analysis confirmed effective nerve ablation, as evidenced by reduced tyrosine hydroxylase staining, without intimal damage. No postoperative complications were observed during the 28-day study period. These findings demonstrate the safety and efficacy of the HyperQure™ extravascular RDN system in reducing BP, providing a promising alternative for patients with resistant hypertension or those ineligible for intravascular RDN. Further clinical trials are warranted to validate these results.","PeriodicalId":10333,"journal":{"name":"Clinical and Experimental Hypertension","volume":"47 1","pages":"2474520"},"PeriodicalIF":1.5,"publicationDate":"2025-12-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"143556081","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":4,"RegionCategory":"医学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}

引用次数: 0

Changes and significance of high-density lipoprotein A-I, A-II, and serum amyloid levels in patients with chronic obstructive pulmonary disease complicated by coronary heart disease. 慢性阻塞性肺疾病合并冠心病患者高密度脂蛋白A-I、A-II和血清淀粉样蛋白水平的变化及意义

IF 1.5 4区医学

Clinical and Experimental Hypertension Pub Date : 2025-12-01 Epub Date: 2025-07-01 DOI: 10.1080/10641963.2025.2524105

Yao Tian, Hongru Liu, Yaoyong Wang

{"title":"Changes and significance of high-density lipoprotein A-I, A-II, and serum amyloid levels in patients with chronic obstructive pulmonary disease complicated by coronary heart disease.","authors":"Yao Tian, Hongru Liu, Yaoyong Wang","doi":"10.1080/10641963.2025.2524105","DOIUrl":"https://doi.org/10.1080/10641963.2025.2524105","url":null,"abstract":"Objective: To investigate the significance of blood lipids and pro-inflammatory in chronic obstructive pulmonary disease (COPD) patients in stable and acute exacerbations (AECOPD) with or without coronary heart disease (CAD).Methods: One hundred and sixty COPD patients were divided into four groups based on whether COPD was in the acute or stable phase and whether comorbid CAD: AECOPD without CAD group, AECOPD with CAD group, stable COPD without CAD group, and stable COPD with CAD group.Results: The levels of ApoA-I and ApoA-II in the AECOPD group and AECOPD with CAD group were significantly lower than in the stable COPD group, stable COPD with CAD group, and control group. The levels of SAA, TNF-α, and IL-6 was significantly higher in AECOPD group with CAD or without CAD compared with control group. SAA levels were significantly increased in stable COPD with CAD group compared with control group.Conclusion: The levels of ApoA-I and ApoA-II in the AECOPD without CAD group and AECOPD with CAD group were significantly lower than those in the stable COPD group, stable COPD with CAD group. The change of pro-inflammatory factor TNF-α and IL-6 levels would be an important reason. The SAA level was significantly increased in the AECOPD without CAD group and AECOPD with CAD group, which indicated that the changes in HDL-C components in this group may be an important reason for promoting the progress of CAD.","PeriodicalId":10333,"journal":{"name":"Clinical and Experimental Hypertension","volume":"47 1","pages":"2524105"},"PeriodicalIF":1.5,"publicationDate":"2025-12-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"144539169","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":4,"RegionCategory":"医学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}

引用次数: 0

High-salt diets provoke phosphorus absorption from the small intestine in mice. 高盐饮食刺激小鼠小肠对磷的吸收。

IF 1.5 4区医学

Clinical and Experimental Hypertension Pub Date : 2025-12-01 Epub Date: 2025-02-25 DOI: 10.1080/10641963.2025.2472066

Mingxin Yang, Huaizhou You, Li Ni, Jianping Mao, Jing Chen

{"title":"High-salt diets provoke phosphorus absorption from the small intestine in mice.","authors":"Mingxin Yang, Huaizhou You, Li Ni, Jianping Mao, Jing Chen","doi":"10.1080/10641963.2025.2472066","DOIUrl":"10.1080/10641963.2025.2472066","url":null,"abstract":"Background: Recent studies indicate that tenapanor, an inhibitor of sodium/proton exchanger-3 (NHE3), diminishes intestinal phosphorus (Pi) absorption. Given NHE3's pivotal role in sodium (Na+) metabolism, there is a suspected functional link between Na+ and Pi metabolism. High-salt diets (HSD) have been demonstrated to disrupt calcium (Ca2+) metabolism. Since Ca2+ and Pi share analogous metabolic pathways, it is yet to be determined whether HSD also impacts Pi metabolism.Methods: Male C57 mice were randomly assigned to three groups: a standard diet group, HSD groups for 1 week (HSD-1w) and 4 weeks (HSD-4w). Throughout the study, dietary intake and water consumption were monitored using metabolic cages, and urine and feces were collected. Blood pressure was measured using a noninvasive tail vein sphygmomanometer. Upon completion of the intervention, mice were euthanized under anesthesia for blood collection, and intestinal and renal tissues were harvested for molecular analysis.Results: Although plasma Pi levels were comparable between HSD groups and the control group, HSD groups exhibited increased urinary Pi excretion and decreased fecal Pi excretion. The HSD-4w group displayed elevated parathyroid hormone levels, reduced fibroblast growth factor 23 levels, and higher renal Cyp27b1 mRNA expression. The expression of sodium-dependent phosphate transporter 2b (Npt2b) and NHE3 was elevated in the intestine of HSD mice.Conclusion: HSD disrupts Pi metabolism by enhancing urinary Pi excretion and altering hormonal levels. The decrease in fecal Pi excretion, coupled with the upregulation of intestinal Pi transporter expression, suggests that HSD promotes intestinal Pi absorption in mice.","PeriodicalId":10333,"journal":{"name":"Clinical and Experimental Hypertension","volume":"47 1","pages":"2472066"},"PeriodicalIF":1.5,"publicationDate":"2025-12-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"143491121","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":4,"RegionCategory":"医学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}

引用次数: 0

Effect of oral potassium supplementation on urinary potassium excretion and its diagnostic value for primary aldosteronism. 口服补钾对尿钾排泄的影响及其对原发性醛固酮增多症的诊断价值。

IF 1.5 4区医学

Clinical and Experimental Hypertension Pub Date : 2025-12-01 Epub Date: 2025-01-23 DOI: 10.1080/10641963.2025.2457768

Ke Zhang, Xiyun Deng, Zhuoran Li, Chen Yi, Jianqiong Kong, Yunhong Wang

{"title":"Effect of oral potassium supplementation on urinary potassium excretion and its diagnostic value for primary aldosteronism.","authors":"Ke Zhang, Xiyun Deng, Zhuoran Li, Chen Yi, Jianqiong Kong, Yunhong Wang","doi":"10.1080/10641963.2025.2457768","DOIUrl":"10.1080/10641963.2025.2457768","url":null,"abstract":"Objectives: We aimed to: (1) explore the effect of oral potassium supplementation on urinary potassium excretion, and (2) evaluate the value of urinary potassium-related indicators in distinguishing primary aldosteronism (PA) from non-PA patients.Design and methods: A prospective study of 20 patients with hypertension and hypokalemia caused by renal potassium loss between November 2023 and April 2024 was conducted. Demographic features, 24-hour urine collection before and after potassium supplementation were all collected.Results: The patients had a mean age of 49.38 years and 70% were male. Following a median potassium supplement dose of 8.50 g, serum potassium increased from 3.25 to 3.90 mmol/L (p < .001), and 24-hour urinary potassium (24 h UK) rose from 41.40 to 59.75 mmol/24 h (p = .004). After supplementation, 20% of patients had decreased 24 h UK, while 25%, 25%, and 40% showed increases of 0-10, 10-20, and > 20 mmol/24 h. Urinary-to-serum potassium ratio (USR) decreased in 40% of patients, while it increased by 0-5, 5-10, and > 10 L/24 h in 25%, 25%, and 10% of patients, respectively. Both 24 h UK and USR after repletion predicted PA with moderate-to-high accuracy (AUC = 0.808 for both). The optimal cutoff of 24 h UK and USR after supplementation were 51 mmol/24 h and 17.43 L/24 h. The AUC for 24 h USR and 24 h UK before repletion in predicting PA were 0.788 and 0.652, respectively.Conclusions: Urinary potassium does not increase proportionally with serum potassium levels or the oral potassium dose, showing individual variability. Post-supplementation urinary potassium has greater diagnostic value for distinguishing PA than pre-supplementation indicators.","PeriodicalId":10333,"journal":{"name":"Clinical and Experimental Hypertension","volume":"47 1","pages":"2457768"},"PeriodicalIF":1.5,"publicationDate":"2025-12-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"143021643","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":4,"RegionCategory":"医学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}

引用次数: 0

Parathyroid hormone and vitamin D modulate nocturnal blood pressure dipping: a retrospective cohort study in primary hypertension. 甲状旁腺激素和维生素D调节夜间血压下降：原发性高血压的回顾性队列研究。

IF 1.5 4区医学

Clinical and Experimental Hypertension Pub Date : 2025-12-01 Epub Date: 2025-05-27 DOI: 10.1080/10641963.2025.2512130

Yixin Xu, Changjiang Deng, Xiaohui Liang

{"title":"Parathyroid hormone and vitamin D modulate nocturnal blood pressure dipping: a retrospective cohort study in primary hypertension.","authors":"Yixin Xu, Changjiang Deng, Xiaohui Liang","doi":"10.1080/10641963.2025.2512130","DOIUrl":"https://doi.org/10.1080/10641963.2025.2512130","url":null,"abstract":"Background: Non-dipper hypertension, defined by reduced nocturnal blood pressure (BP) decline, increases cardiovascular risk. Calcium-phosphate metabolism (parathyroid hormone [PTH], vitamin D [25(OH)D]) may influence circadian BP rhythms, but their independent roles remain unclear.Objective: To investigate associations between PTH, 25(OH)D, and nocturnal BP dipping in primary hypertension, addressing prior inconsistencies.Methods: This retrospective cohort included 585 hypertensive adults stratified by nocturnal systolic BP dipping (dippers [≥10% decline, n = 250]; non-dippers [<10%, n = 335]) using 24-hour ambulatory monitoring. Serum PTH, 25(OH)D, renal/metabolic parameters, and vascular indices were analyzed. Multivariate linear regression assessed associations with dipping, adjusted for age, sex, renal function, and arterial stiffness.Results: Non-dippers showed elevated PTH (median: 5.23 vs. 4.70 pmol/L, p = .011) and lower 25(OH)D (30.89 vs. 36.79 nmol/L, p < .001). Adjusted models confirmed PTH (β=-0.21, 95% CI:-0.34--0.08; p = .002) and 25(OH)D (β = 0.03, 95% CI:0.01-0.04; p < .001) as associated factors of dipping. Although statistically significant, these correlations were modest and suggested possible trends rather than strong causal relationships. Age/sex effects on BP patterns became nonsignificant after adjusting for calcium-phosphate markers.Conclusion: Calcium-phosphate dysregulation (elevated PTH, low vitamin D) independently contributes to non-dipping, suggesting biomarker-guided approaches for circadian BP control. Demographic disparities in dipping may reflect underlying mineral metabolism disturbances. Prospective trials should explore calcium-modulating therapies (e.g. vitamin D supplementation) in non-dipper hypertension.","PeriodicalId":10333,"journal":{"name":"Clinical and Experimental Hypertension","volume":"47 1","pages":"2512130"},"PeriodicalIF":1.5,"publicationDate":"2025-12-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"144149518","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":4,"RegionCategory":"医学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}

引用次数: 0

The role of immune mechanisms in hypertension and advances in immunomodulatory research. 免疫机制在高血压中的作用及免疫调节研究进展。

IF 3.5 4区医学

Clinical and Experimental Hypertension Pub Date : 2025-12-01 Epub Date: 2025-07-28 DOI: 10.1080/10641963.2025.2535328

XiaoLi Chen, Ling Sun, Ke Xuan, Aifen Zong

{"title":"The role of immune mechanisms in hypertension and advances in immunomodulatory research.","authors":"XiaoLi Chen, Ling Sun, Ke Xuan, Aifen Zong","doi":"10.1080/10641963.2025.2535328","DOIUrl":"10.1080/10641963.2025.2535328","url":null,"abstract":"Hypertension is one of the major risk factors for cardiovascular diseases and end-organ damage worldwide. Traditionally, it has been linked to factors such as genetics, diet, and lifestyle. However, increasing research indicates that the immune system also plays an important role in the initiation and progression of hypertension. In the initiation phase, pro-hypertensive stimuli including chronic sympathetic nervous system (SNS), high salt intake and imbalanced gut microbes all influence the function of immune cells, contributing to the development of elevated blood pressure. As hypertension progresses, many immune cells, such as macrophages, dendritic cells, NK cells, B and T lymphocytes, accumulate in organs such as the heart, blood vessels, and kidneys. These cells release effector cytokines and reactive oxygen species, exacerbating tissue damage and organ dysfunction. Meanwhile, numerous studies suggest that intervening in immune inflammatory processes has the potential to regulate blood pressure and prevent damage to vital organs. This review aims to explore the role of immune regulation in various stages of hypertension and the progress of research on immune interventions for hypertension treatment, with the aim of providing new perspectives and approaches for future research and treatment of hypertension.","PeriodicalId":10333,"journal":{"name":"Clinical and Experimental Hypertension","volume":"47 1","pages":"2535328"},"PeriodicalIF":3.5,"publicationDate":"2025-12-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"144728403","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":4,"RegionCategory":"医学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}

引用次数: 0

Correction. 修正。

IF 3.5 4区医学

Clinical and Experimental Hypertension Pub Date : 2025-08-14 Epub Date: 2025-08-21 DOI: 10.1080/10641963.2025.2548715

引用次数: 0

Chicoric acid advanced PAQR3 ubiquitination to ameliorate ferroptosis in diabetes nephropathy through the relieving of the interaction between PAQR3 and P110α pathway. 菊苣酸通过缓解PAQR3与P110α通路之间的相互作用，促进PAQR3泛素化，从而改善糖尿病肾病的铁变态反应。

IF 12.3 4区医学

Clinical and Experimental Hypertension Pub Date : 2024-12-31 Epub Date: 2024-03-25 DOI: 10.1080/10641963.2024.2326021

Weiwei Zhang, Yong Liu, Jiajun Zhou, Teng Qiu, Haitang Xie, Zhichen Pu

{"title":"Chicoric acid advanced PAQR3 ubiquitination to ameliorate ferroptosis in diabetes nephropathy through the relieving of the interaction between PAQR3 and P110α pathway.","authors":"Weiwei Zhang, Yong Liu, Jiajun Zhou, Teng Qiu, Haitang Xie, Zhichen Pu","doi":"10.1080/10641963.2024.2326021","DOIUrl":"10.1080/10641963.2024.2326021","url":null,"abstract":"Purpose: This study aimed to examine the impact of CA on DN and elucidate its underlying molecular mechanisms of inflammation.Methods: We fed C57BL/6 mice injected with streptozotocin to induce diabetes. In addition, we stimulated NRK-52E cells with 20 mmol/L d-glucose to mimic the diabetic condition.Results: Our findings demonstrated that CA effectively reduced blood glucose levels, and improved DN in mice models. Additionally, CA reduced kidney injury and inflammation in both mice models and in vitro models. CA decreased high glucose-induced ferroptosis of NRK-52E cells by inducing GSH/GPX4 axis. Conversely, the ferroptosis activator or the PI3K inhibitor reversed positive effects of CA on DN in both mice and in vitro models. CA suppressed PAQR3 expression in DN models to promote PI3K/AKT activity. The PAQR3 activator reduced the positive effects of CA on DN in vitro models. Moreover, CA directly targeted the PAQR3 protein to enhance the ubiquitination of the PAQR3 protein.Conclusion: Overall, our study has uncovered that CA promotes the ubiquitination of PAQR3, leading to the attenuation of ferroptosis in DN. This effect is achieved through the activation of the PI3K/AKT signaling pathways by disrupting the interaction between PAQR3 and the P110α pathway. These findings highlight the potential of CA as a viable therapeutic option for the prevention of DN and other forms of diabetes.","PeriodicalId":10333,"journal":{"name":"Clinical and Experimental Hypertension","volume":"46 1","pages":"2326021"},"PeriodicalIF":12.3,"publicationDate":"2024-12-31","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"140206410","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":4,"RegionCategory":"医学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}

引用次数: 0

Analysis of the current status and associated risk factors of cognitive function in Tibetan hypertensive patients at various altitudes. 不同海拔地区藏族高血压患者认知功能现状及相关风险因素分析。

IF 1.5 4区医学

Clinical and Experimental Hypertension Pub Date : 2024-12-31 Epub Date: 2024-08-27 DOI: 10.1080/10641963.2024.2393331

Long Yin, Xiaoming Zhang, Huijuan Zhang, Ruizhen Li, Jing Zeng, Kaixuan Dong, Yi Wang, Xinghui Li

{"title":"Analysis of the current status and associated risk factors of cognitive function in Tibetan hypertensive patients at various altitudes.","authors":"Long Yin, Xiaoming Zhang, Huijuan Zhang, Ruizhen Li, Jing Zeng, Kaixuan Dong, Yi Wang, Xinghui Li","doi":"10.1080/10641963.2024.2393331","DOIUrl":"https://doi.org/10.1080/10641963.2024.2393331","url":null,"abstract":"Objective: This study aims to explore the current cognitive status and identify risk factors associated with cognitive function in Tibetan hypertensive patients living at various altitudes.Methods: The Simple Mental Status Scale (MMSE) was used to evaluate the cognitive function of 611 Tibetan hypertensive patients at various altitudes in Gannan Tibetan Autonomous Prefecture. Afterward, we conducted an analysis to identify the factors influencing their cognitive function.Results: The study found that the prevalence of cognitive dysfunction was 22.3%, with a higher incidence at high altitude (group D 29.0%) compared to low altitude (group A 16.0%). The study conducted a binary logistic regression analysis to identify the risk factors for cognitive dysfunction. The analysis revealed that altitude, age, body mass index, marital status, education, income level, and blood pressure control level were all significant risk factors. After controlling for age, body mass index, marital status, educational level, income level, and blood pressure control level, the risk of developing cognitive dysfunction was 2.773 times higher (p < .05) for individuals in group C at high altitude and 2.381 times higher (p < .05) for individuals in group D at high altitude compared to those in group A at low altitude.Conclusions: Altitude plays a role in the development of cognitive dysfunction in hypertensive patients. Tibetan hypertensive patients living at high altitudes may be at a higher risk of cognitive dysfunction compared to those living at lower altitudes. Therefore, interventions should be targeted to prevent or mitigate potential cognitive impairment.","PeriodicalId":10333,"journal":{"name":"Clinical and Experimental Hypertension","volume":"46 1","pages":"2393331"},"PeriodicalIF":1.5,"publicationDate":"2024-12-31","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"142079423","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":4,"RegionCategory":"医学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}

引用次数: 0

Research hotspots and trends regarding microRNAs in hypertension: a bibliometric analysis. 高血压微RNA的研究热点和趋势：文献计量分析。

IF 12.3 4区医学

Clinical and Experimental Hypertension Pub Date : 2024-12-31 Epub Date: 2024-01-17 DOI: 10.1080/10641963.2024.2304017

Yu Sun, Qingxin Shang

引用次数: 0