Headache Currents最新文献

{"title":"The Anatomy and Physiology of Pain Referral Patterns in Primary and Cervicogenic Headache Disorders","authors":"Anish Bahra \u0000 M.D., M.R.C.P.","doi":"10.1111/j.1743-5013.2005.20203.x","DOIUrl":"10.1111/j.1743-5013.2005.20203.x","url":null,"abstract":"","PeriodicalId":100600,"journal":{"name":"Headache Currents","volume":"2 2","pages":"49-52"},"PeriodicalIF":0.0,"publicationDate":"2005-03-11","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://sci-hub-pdf.com/10.1111/j.1743-5013.2005.20203.x","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"80041681","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}

{"title":"Distinguishing Primary Headache Disorders from Cervicogenic Headache: Clinical and Therapeutic Implications","authors":"Frederick R. Taylor \u0000 M.D.","doi":"10.1111/j.1743-5013.2005.20202.x","DOIUrl":"10.1111/j.1743-5013.2005.20202.x","url":null,"abstract":"","PeriodicalId":100600,"journal":{"name":"Headache Currents","volume":"2 2","pages":"37-41"},"PeriodicalIF":0.0,"publicationDate":"2005-03-11","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://sci-hub-pdf.com/10.1111/j.1743-5013.2005.20202.x","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"89129494","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}

{"title":"Anatomy and Physiology of Pain Referral Patterns in Primary and Cervicogenic Headache Disorders","authors":"T. Bartsch M.D.,&nbsp;P. J. Goadsby M.D., Ph.D.","doi":"10.1111/j.1743-5013.2005.20201.x","DOIUrl":"10.1111/j.1743-5013.2005.20201.x","url":null,"abstract":"<p> <i>The clinical presentation of pain in primary headache disorders, such as migraine and cervicogenic headache, shows that the trigeminal and cervical innervation territories frequently are not respected during an attack. Here we review evidence that trigeminal afferents innervating the meninges, and cervical afferents in the greater occipital nerve (GON), have synaptic convergent input onto the same second-order neurons in the trigeminocervical complex. Furthermore, dynamic changes of excitability in terms of a sensitization to noxious input may occur on the central neuron level. The possible role of segmental mechanisms in the spinal cord and of inhibitory projections from brainstem structures such as the periaqueductal gray (PAG) in pain processing is discussed in view of data using neurostimulation of peripheral nerves in pain modulation. Recent experimental animal and human studies have facilitated our understanding of the role of these basic mechanisms in the spread, referral, and maintenance of pain in headache.</i> </p>","PeriodicalId":100600,"journal":{"name":"Headache Currents","volume":"2 2","pages":"42-48"},"PeriodicalIF":0.0,"publicationDate":"2005-03-11","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://sci-hub-pdf.com/10.1111/j.1743-5013.2005.20201.x","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"79988772","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}

{"title":"Distinguishing Primary Headache Disorders from Cervicogenic Headache: Clinical and Therapeutic Implications","authors":"Nikolai Bogduk M.D., D.Sc.","doi":"10.1111/j.1743-5013.2005.20204.x","DOIUrl":"10.1111/j.1743-5013.2005.20204.x","url":null,"abstract":"<p> <i>The objectives of this article are to encapsulate the literature on the clinical aspects of cervicogenic headache, in order to help readers understand how cervicogenic headache can be distinguished from other primary headaches, and how it might be treated. Cervicogenic headache has evolved as a contentious and controversial entity. Tensions have developed concerning how it should be diagnosed, and if it at all exists as an entity. The literature is summarized and appraised in the form of a narrative review. Two conflicting approaches have been used to define cervicogenic headache. One has pursued the definition according to clinical features, but the validity of this approach has not been established. The other approach ignores clinical features and seeks instead to establish the diagnosis by demonstrating a cervical source of pain in the head. For that purpose diagnostic blocks have been used to pinpoint sources of pain in the upper cervical joints. Few treatments have been tested and validated for this condition. In conclusion, cervicogenic headache can be suspected but not diagnosed on the basis of clinical features. The diagnosis ultimately requires diagnostic blocks. Intra-articular steroids may be a useful treatment. Complete relief of headache can be achieved by radiofreqeuncy neurotomy in patients whose headache stems from the C2–3 zygapophysial joint.</i> </p>","PeriodicalId":100600,"journal":{"name":"Headache Currents","volume":"2 2","pages":"27-36"},"PeriodicalIF":0.0,"publicationDate":"2005-03-11","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://sci-hub-pdf.com/10.1111/j.1743-5013.2005.20204.x","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"91127739","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}

{"title":"A Practical Approach to Intracranial Hypertension","authors":"Deborah I. Friedman M.D.","doi":"10.1111/j.1743-5013.2005.20101.x","DOIUrl":"10.1111/j.1743-5013.2005.20101.x","url":null,"abstract":"","PeriodicalId":100600,"journal":{"name":"Headache Currents","volume":"2 1","pages":"1-10"},"PeriodicalIF":0.0,"publicationDate":"2005-01-17","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://sci-hub-pdf.com/10.1111/j.1743-5013.2005.20101.x","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"84493065","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}

{"title":"Spontaneous CSF Leaks—Underlying Connective Tissue Disorder","authors":"Jerry W. Swanson \u0000 M.D.","doi":"10.1111/j.1743-5013.2005.20104.x","DOIUrl":"10.1111/j.1743-5013.2005.20104.x","url":null,"abstract":"","PeriodicalId":100600,"journal":{"name":"Headache Currents","volume":"2 1","pages":"25-26"},"PeriodicalIF":0.0,"publicationDate":"2005-01-17","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://sci-hub-pdf.com/10.1111/j.1743-5013.2005.20104.x","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"104892589","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}

{"title":"Spontaneous Intracranial Hypotension Spontaneous CSF Leaks","authors":"Bahram Mokri M.D.","doi":"10.1111/j.1743-5013.2005.20102.x","DOIUrl":"10.1111/j.1743-5013.2005.20102.x","url":null,"abstract":"<p> <i>MR imaging has truly revolutionized the diagnosis of spontaneous intracranial hypotension (SIH). A substantially larger number of patients are now diagnosed and a broader clinical and imaging spectrum of the disorder is recognized. It is now realized that SIH nearly always results from spontaneous CSF leaks. The majority of these leaks occur at the level of the spine and only rarely at the skull base. Considerable variability exists in clinical manifestations, including the headaches. Although the typical headache is an orthostatic headache, not all headaches in CSF leaks are orthostatic and not all orthostatic headaches are caused by intracranial hypotension or CSF leaks. Furthermore, imaging and CSF findings also reveal considerable variability, including patients who may display CSF opening pressures that are consistently within normal limits or head MRIs that may not show abnormal pachymeningeal enhancement. The core pathogenetic factor is decreased CSF volume (CSF hypovolemia) as the independent variable, while CSF pressures, clinical manifestations, and MRI abnormalities are variable and dependent on loss of CSF volume. Epidural blood patch (EBP) has emerged as the treatment of choice for patients who fail initial conservative measures. However, response to EBP in spontaneous leaks is far less impressive than in post-lumbar puncture headaches. In spontaneous leaks, the anatomy of the leak is frequently complex and quite different from a simple hole or rent as might be seen in post-lumbar puncture headaches. These two entities should not be equated. At least a significant minority of the patients with spontaneous CSF leaks have pre-existing dural weakness, likely based on a disorder of connective tissue matrix. There is also a considerable variability in the rate of leakage of CSF in these patients. This can create diagnostic challenges and novel diagnostic techniques are evolving to address the fast- and slow-flow leaks. A large majority of the patients make excellent recovery spontaneously, with conservative measures, with epidural injections, or surgery. A small minority, however, continue to remain symptomatic. Subdural hematomas may complicate CSF leaks, may become symptomatic, and create therapeutic challenges but, fortunately, uncommonly. Rarely, cerebral venous thrombosis may develop. Sometimes, after treatment of spontaneous CSF leaks, rebound intracranial hypertension may occur which is likely self-limiting.</i> </p>","PeriodicalId":100600,"journal":{"name":"Headache Currents","volume":"2 1","pages":"11-22"},"PeriodicalIF":0.0,"publicationDate":"2005-01-17","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://sci-hub-pdf.com/10.1111/j.1743-5013.2005.20102.x","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"127274275","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}

{"title":"Sinovenous Stenosis and Idiopathic Intracranial Hypertension: Chicken versus Egg Revisited","authors":"Jessica Crowder \u0000 M.D.\u0000 ,&nbsp;John F. Rothrock \u0000 M.D.","doi":"10.1111/j.1743-5013.2005.20103.x","DOIUrl":"10.1111/j.1743-5013.2005.20103.x","url":null,"abstract":"","PeriodicalId":100600,"journal":{"name":"Headache Currents","volume":"2 1","pages":"23-24"},"PeriodicalIF":0.0,"publicationDate":"2005-01-17","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://sci-hub-pdf.com/10.1111/j.1743-5013.2005.20103.x","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"77368019","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}

{"title":"Clinical Science: Headache Induced by Acute Medication Overuse","authors":"Fred D. Sheftell \u0000 M.D.\u0000 ,&nbsp;Marcelo Bigal \u0000 M.D., Ph.D.","doi":"10.1111/j.1743-5013.2004.10109.x","DOIUrl":"10.1111/j.1743-5013.2004.10109.x","url":null,"abstract":"","PeriodicalId":100600,"journal":{"name":"Headache Currents","volume":"1 3","pages":"64-68"},"PeriodicalIF":0.0,"publicationDate":"2004-10-28","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://sci-hub-pdf.com/10.1111/j.1743-5013.2004.10109.x","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"76422903","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}

{"title":"Basic Science: Mechanisms of Medication Overuse Headache","authors":"Ian D. Meng,&nbsp;Frank Porreca","doi":"10.1111/j.1743-5013.2004.10110.x","DOIUrl":"10.1111/j.1743-5013.2004.10110.x","url":null,"abstract":"<p> <i>Headache caused by overuse of symptomatic medications to treat headache represents a challenging clinical problem. Recent studies exploring both the mechanisms of migraine headache and neuroplastic changes following sustained exposure to analgesic drugs suggest insights regarding the pathophysiology of medication overuse headache. In this review, changes that occur following chronic morphine that may be particularly relevant to the induction of medication overuse headache will be discussed. Peripherally, these changes include increased expression of calcitonin gene-related peptide (CGRP) in primary afferent neurons. Centrally, they include increased descending facilitation from the rostral ventromedial medulla and increased excitatory neurotransmission at the level of the dorsal horn. Interestingly, many of these same changes, including increased CGRP levels and peripheral and central sensitization, are apparent in inflammatory pain states. Recent revelations into the mechanisms of migraine headache suggest that neurogenic inflammation leads to both peripheral and central sensitization in the migraine sufferer. Furthermore, CGRP plays a prominent role in initiating vasodilation of the intracranial blood vessels and subsequent headache. Given the many parallels between the effects of chronic morphine exposure and processes that occur during migraine, it is evident that overuse of symptomatic medications to treat headache could lead to worsening of symptoms. In animals, the neural adaptations that occur after sustained morphine exposure are manifested behaviorally as an increased sensitivity to both tactile and thermal stimulation. In the headache sufferer, these adaptations could lead to medication overuse headache.</i> </p>","PeriodicalId":100600,"journal":{"name":"Headache Currents","volume":"1 3","pages":"47-54"},"PeriodicalIF":0.0,"publicationDate":"2004-10-28","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://sci-hub-pdf.com/10.1111/j.1743-5013.2004.10110.x","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"88999878","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}