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Abstracts of the 6th International Symposium on Predictive Oncology and Intervention Strategies. Paris, France, 9-12 February 2001. 第六届预测肿瘤学与干预策略国际研讨会摘要。2001年2月9日至12日，法国巴黎。
Cancer detection and prevention Pub Date : 2002-01-01
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引用次数: 0
Prognostic impact of Ets-1 overexpression in betel and tobacco related oral cancer. 槟榔和烟草相关口腔癌中 Ets-1 过表达的预后影响。
Cancer detection and prevention Pub Date : 2001-01-01
P Pande, S Soni, N Chakravarti, M Mathur, N K Shukla, R Ralhan
{"title":"Prognostic impact of Ets-1 overexpression in betel and tobacco related oral cancer.","authors":"P Pande, S Soni, N Chakravarti, M Mathur, N K Shukla, R Ralhan","doi":"","DOIUrl":"","url":null,"abstract":"<p><p>In oral squamous cell carcinoma (SCC), the presence of lymph node metastasis is one of the most important prognostic factors, correlating locoregional spread, recurrence, distant metastasis and survival. However, the biological factors implicated and the mechanisms underlying these events are not completely elucidated. We reported Ets-1 expression in oral SCC and proposed that it may serve as a plausible marker of invasive potential and lymph node metastasis. Herein, we investigate the prognostic significance of Ets-1 expression in oral cancer. One hundred oral SCCs assessed for Ets-1 expression showed significant association with tumor stage (P = .027), lymph node involvement (P < 10(-6)) and distant organ metastasis (P = .007). The transcription factor Ets-1 regulates the expression of several genes involved in extracellular matrix remodeling that may account for its association with lymph node and distant organ metastasis. Kaplan-Meier survival analysis entailed significant association of Ets-1 expression in oral SCCs with reduced disease free survival (P = .0041), suggesting its utility as a prognosticator for oral cancer. In conclusion, these findings underscore the role of Ets-1 in oral tumor invasion and metastasis and may thus account for its association with diminished disease free survival.</p>","PeriodicalId":9499,"journal":{"name":"Cancer detection and prevention","volume":"25 5","pages":"496-501"},"PeriodicalIF":0.0,"publicationDate":"2001-01-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"138797768","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
引用次数: 0
Association between apolipoprotein E genotypes and cancer risk in patients with acquired immunodeficiency syndrome. 载脂蛋白E基因型与获得性免疫缺陷综合征患者癌症风险的关系
Cancer detection and prevention Pub Date : 2000-01-01
K Liestøl, E A Kvittingen, H Rootwelt, O Dunlop, A K Goplen, J C Pedersen, S H Brorson, A L Børresen-Dale, B Myrvang, J Maehlen
{"title":"Association between apolipoprotein E genotypes and cancer risk in patients with acquired immunodeficiency syndrome.","authors":"K Liestøl,&nbsp;E A Kvittingen,&nbsp;H Rootwelt,&nbsp;O Dunlop,&nbsp;A K Goplen,&nbsp;J C Pedersen,&nbsp;S H Brorson,&nbsp;A L Børresen-Dale,&nbsp;B Myrvang,&nbsp;J Maehlen","doi":"","DOIUrl":"","url":null,"abstract":"<p><p>The apolipoprotein E (apoE) genotype was determined in 197 deceased acquired immunodeficiency syndrome (AIDS) patients treated at Ullevaal Hospital in Oslo, Norway. A full autopsy had been performed on all. Cancer had developed in 71 individuals, mainly lymphomas (46) and Kaposi's sarcomas (18). The apoE genotype distribution was consistent with Hardy-Weinberg equilibrium, and allele frequencies were in the typical Scandinavian range (6.9% apoE2; 75.6% apoE3; and 17.5% apoE4). Cancer cases had a significantly higher frequency of apoE4 alleles than noncancer cases (24.6% and 13.5%, respectively) and a lower frequency of apoE2 alleles (3.5% versus 8.7%). Background factors, such as survival from AIDS diagnosis, could not explain these differences. Our study thus indicates that apoE genotype affects the development of cancers among AIDS patients.</p>","PeriodicalId":9499,"journal":{"name":"Cancer detection and prevention","volume":"24 5","pages":"496-9"},"PeriodicalIF":0.0,"publicationDate":"2000-01-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"21953206","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
引用次数: 0
Dietary calcium and growth modulation of human colon cancer cells: role of the extracellular calcium-sensing receptor. 膳食钙和人结肠癌细胞的生长调节:细胞外钙感应受体的作用。
Cancer detection and prevention Pub Date : 2000-01-01
E Kállay, E Bajna, F Wrba, S Kriwanek, M Peterlik, H S Cross
{"title":"Dietary calcium and growth modulation of human colon cancer cells: role of the extracellular calcium-sensing receptor.","authors":"E Kállay,&nbsp;E Bajna,&nbsp;F Wrba,&nbsp;S Kriwanek,&nbsp;M Peterlik,&nbsp;H S Cross","doi":"","DOIUrl":"","url":null,"abstract":"<p><p>Using the human colon adenocarcinoma-derived cell line Caco-2, we investigated the possible role of the Ca2+-sensing receptor (CaR) in mediating effects of extracellular Ca2+ on cellular proliferation. Caco-2 cells respond to low ambient [Ca2+]o by activation of the protein kinase C-signaling pathway, leading to upregulation of c-myc mRNA expression and thereby, finally, to alleviation from the G1/S phase control of the cell cycle. This proliferative response can be reverted by activation of the CaR either through raising [Ca2+]o or, respectively, by using the CaR agonist Gd3+ as a substitute for Ca2+. The inhibitory effect of [Ca2+]o on cell replication exhibits saturation kinetics (IC50 = 0.045 mM), indicating the existence of a highly sensitive CaR operating at low ambient [Ca2+]o. Specific immunostaining revealed the presence of CaR-positive cells in the crypt epithelium of normal human colonic mucosa as well as in glandular (i.e., differentiated structures) of carcinomatous lesions. This could provide a rationale for use of calcium supplements for intervention in early phases of colon tumorigenesis.</p>","PeriodicalId":9499,"journal":{"name":"Cancer detection and prevention","volume":"24 2","pages":"127-36"},"PeriodicalIF":0.0,"publicationDate":"2000-01-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"21756612","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
引用次数: 0
Determination of p53-mediated transactivational ability in radiation-treated cervical cancer. 放射治疗宫颈癌中p53介导的交互激活能力的测定。
Cancer detection and prevention Pub Date : 2000-01-01
A Oka, Y Harima, K Harima, V V Ostapenko, Y Tanaka, T Ohnishi, S Sawada
{"title":"Determination of p53-mediated transactivational ability in radiation-treated cervical cancer.","authors":"A Oka,&nbsp;Y Harima,&nbsp;K Harima,&nbsp;V V Ostapenko,&nbsp;Y Tanaka,&nbsp;T Ohnishi,&nbsp;S Sawada","doi":"","DOIUrl":"","url":null,"abstract":"<p><p>To establish a new predictor of human cervical cancer radioresponse, we investigated the transactivational ability of p53 gene in tumor tissue for use as a marker of both pretreatment and postirradiation levels of mRNA of its downstream gene, WAF1. A total of 38 wild-type p53-bearing patients with histologically proved uterine cervical cancer were treated with definitive radiotherapy. Their p53 status was investigated using a single-strand conformation polymorphism analysis, and human papilloma virus 16, 18, 33, and 58 E6 was determined by polymerase chain reaction in pretreatment biopsy specimens. WAF1 mRNA was estimated by reverse transcriptase-polymerase chain reaction in both pretreatment specimens and those obtained after the administration of 10.8 Gy. Undetectable or low pretreatment levels of WAF1 mRNA were associated with complete response in the majority of cases, whereas only a few patients with a high pretreatment WAF1 level responded to treatment (P = .03). The increase in the postirradiation level of WAF1 mRNA positively correlated with better treatment response and long survival (P = .02). Although the human papilloma virus infection did not change the radiation response directly, it decreased the inducibility of WAF1. Consequently, the lower inducibility of WAF1 resulted in a poor treatment response. This is the first clinical report showing that the transactivational ability of p53 may be a determinant of the efficacy of cervical cancer radiotherapy.</p>","PeriodicalId":9499,"journal":{"name":"Cancer detection and prevention","volume":"24 3","pages":"275-82"},"PeriodicalIF":0.0,"publicationDate":"2000-01-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"21810856","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
引用次数: 0
A new concept of tumor promotion by tumor necrosis factor-alpha, and cancer preventive agents (-)-epigallocatechin gallate and green tea--a review. 肿瘤坏死因子- α和抗癌药物(-)-表没食子儿茶素没食子酸酯和绿茶促进肿瘤的新概念综述。
Cancer detection and prevention Pub Date : 2000-01-01
H Fujiki, M Suganuma, S Okabe, E Sueoka, K Suga, K Imai, K Nakachi
{"title":"A new concept of tumor promotion by tumor necrosis factor-alpha, and cancer preventive agents (-)-epigallocatechin gallate and green tea--a review.","authors":"H Fujiki,&nbsp;M Suganuma,&nbsp;S Okabe,&nbsp;E Sueoka,&nbsp;K Suga,&nbsp;K Imai,&nbsp;K Nakachi","doi":"","DOIUrl":"","url":null,"abstract":"<p><p>The study of tumor promotion in rodent carcinogenesis using chemical tumor promoters has revealed various tumor promotion pathways, such as the 12-O-tetradecanoylphorbol-13-acetate (TPA) pathway mediated through activation of protein kinase C, and the okadaic acid pathway mediated through inhibition of protein phosphatases 1 and 2A (PP-1 and PP-2A). We previously demonstrated that application of TPA and okadaic acid induced tumor necrosis factor-alpha (TNF-alpha) gene expression in mouse skin, but that tautomycin, which is an inhibitor of PP-1 and PP-2A and not a tumor promoter on mouse skin, did not. Moreover, we found that TNF-alpha stimulated transformation of BALB/3T3 cells initiated with 3-methylcholanthrene 1,000 times stronger than did TPA (Cancer Res. 53, 1982-1985, 1993). This evidence demonstrates a link between the okadaic acid pathway and the endogenous tumor promotion pathway of TNF-alpha. Recently we presented the first evidence that tumor promotion in TNF-alpha(-/-) mice was significantly depressed compared with TNF-alpha(+/+) mice. Thus, in human carcinogenesis, we think that TNF-alpha and other inflammatory cytokines in preneoplastic lesion stimulate tumor promotion and progression of initiated cells as well as premalignant cells. The first part of this paper reports on this TNF-alpha tumor promotion pathway. In the second part, we report a promising screening method for cancer preventive agents, based on evidence that pretreatment with agents such as tamoxifen, sulindac, 1alpha, 25-(OH)2 vitamin D3, quercetin, caffeic acid phenethyl ester, and (-)-epigallocatechin gallate (EGCG) commonly inhibited TNF-alpha release from BALB/3T3 cells induced by okadaic acid. EGCG, the main constituent of Japanese green tea, and green tea itself are acknowledged cancer preventives in Japan, and this paper presents evidence of their effectiveness in both a high-risk group and the general population.</p>","PeriodicalId":9499,"journal":{"name":"Cancer detection and prevention","volume":"24 1","pages":"91-9"},"PeriodicalIF":0.0,"publicationDate":"2000-01-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"21607789","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
引用次数: 0
Interleukin 18 induces a synergistic enhancement of interferon gamma production in mixed murine spleen cell-tumor cell cultures: role of endogenous interleukin 12. 白细胞介素18诱导混合小鼠脾细胞-肿瘤细胞培养中干扰素γ产生的协同增强:内源性白细胞介素12的作用。
Cancer detection and prevention Pub Date : 2000-01-01
M J Micallef, T Tanimoto, K Kohno, H Ikegami, M Kurimoto
{"title":"Interleukin 18 induces a synergistic enhancement of interferon gamma production in mixed murine spleen cell-tumor cell cultures: role of endogenous interleukin 12.","authors":"M J Micallef,&nbsp;T Tanimoto,&nbsp;K Kohno,&nbsp;H Ikegami,&nbsp;M Kurimoto","doi":"","DOIUrl":"","url":null,"abstract":"<p><p>Interleukin 18 (IL-18) reportedly synergizes with IL-12 and IL-10 for interferon gamma (IFN-gamma) synthesis and natural killer (NK) cell activity, respectively. Here we show that IL-18 alone induces low level IFN-gamma production by unstimulated Balb/c mouse spleen cells, but production is enhanced synergistically in cocultures of spleen cells and allogeneic living or fixed Yac-1 cells. Spleen cells could be primed with IL-18 prior to coculture with Yac-1 cells for IFN-gamma production, which also was observed in cocultures containing either syngeneic or xenogeneic tumor cells. IFN-gamma production in stimulated cocultures was abrogated almost completely by anti-IL-12 antibody and was unrelated to spleen cell lytic activity. IL-10 moderately inhibited IFN-gamma production induced by IL-18. Therefore, in spleen cell and tumor cell cocultures exposed to IL-18, high levels of IFN-gamma are produced by the spleen cells arising from a synergistic interaction between the exogenous IL-18 and endogenous IL-12; however, this activity is unrelated to the spleen cell lytic activity.</p>","PeriodicalId":9499,"journal":{"name":"Cancer detection and prevention","volume":"24 3","pages":"234-43"},"PeriodicalIF":0.0,"publicationDate":"2000-01-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"21809683","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
引用次数: 0
Induced apoptosis and necrosis by 2-methylfuranonaphthoquinone in human cervical cancer HeLa cells. 2-甲基呋喃醌诱导人宫颈癌HeLa细胞凋亡和坏死。
Cancer detection and prevention Pub Date : 2000-01-01
J Pan, E Simamura, J Koyama, H Shimada, K I Hirai
{"title":"Induced apoptosis and necrosis by 2-methylfuranonaphthoquinone in human cervical cancer HeLa cells.","authors":"J Pan,&nbsp;E Simamura,&nbsp;J Koyama,&nbsp;H Shimada,&nbsp;K I Hirai","doi":"","DOIUrl":"","url":null,"abstract":"<p><p>2-Methylnaphtho[2,3-b]furan-4,9-dione (FNQ3) has been reported to be more cytotoxic to human malignant tumor cell lines than are the corresponding normal epithelial cells. Therefore, we examined the dose response of FNQ3 against human cervical cancer HeLa cells in culture. When 1.25 mg/ml FNQ3 was applied, apoptosis was induced, as determined by an immunohistochemical staining of fragmented genome DNA and cell profiles. Significant inhibition of Bcl-2 oncogene protein expression by the same concentration of FNQ3 also was demonstrated by an immunohistochemical staining method to visualize the expressed cells and Western blot in polyacrylamide gel electrophoresis. Flow-cytometric spectra showed S-phase arrest in cell cycles and the appearance of sub-G1 phase consistent with apoptosis. On the other hand, concentrations of 5 microg/ml or more of FNQ3 induced necrosis. These results show that FNQ3 may act as an antitumor agent to induce apoptosis by affecting Bcl-2 expression and cell cycles, or necrosis as the result of primary mitochondrial injuries.</p>","PeriodicalId":9499,"journal":{"name":"Cancer detection and prevention","volume":"24 3","pages":"266-74"},"PeriodicalIF":0.0,"publicationDate":"2000-01-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"21810855","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
引用次数: 0
Nutrient intake and esophageal cancer in the Caspian littoral of Iran: a case-control study. 伊朗里海沿岸营养摄入与食道癌:一项病例对照研究
Cancer detection and prevention Pub Date : 2000-01-01
F Siassi, Z Pouransari, P Ghadirian
{"title":"Nutrient intake and esophageal cancer in the Caspian littoral of Iran: a case-control study.","authors":"F Siassi,&nbsp;Z Pouransari,&nbsp;P Ghadirian","doi":"","DOIUrl":"","url":null,"abstract":"<p><p>The purpose of this study was to investigate the possible contribution of different dietary nutrients in the development of esophageal cancer (EC) in the Caspian littoral of Iran. Forty-one cases and 145 members of their households were matched for age and gender with 40 non-blood-relative controls and 130 members of their households for their nutrient intake. A standard 24-hour dietary recall questionnaire was used to estimate the daily intake of energy, protein, P, Fe, Na, K, vitamins C and A, thiamin, riboflavin, and niacin. Dietary nutrient deficiency was defined as less than 75% of the World Health Organization human nutritional requirements, except for P, Na, and K, for which the United States Recommended Dietary Allowances were followed. The results indicate the following: (1) The mean daily intake of all nutrients, except for riboflavin, was significantly lower in cases than in control subjects (P < .05); (2) with the exception of protein, riboflavin, and phosphorus, significant correlation was observed between the pattern of nutrient intake and health status of the study subjects (P < .05); and (3) dietary deficiency of niacin and phosphorus was associated significantly with the risk of EC development among case and control households (P < .01-.001), indicating that persons living in case households with dietary deficiencies of these nutrients have more than twice the risk of developing EC tumors than those living in control households. In conclusion, apparently some nutrients, such as P and niacin, may play a role in the etiology of esophageal cancer, and the status of these nutrients may be used eventually as an epidemiologic predictive marker for EC in the Caspian littoral of Iran and perhaps other regions.</p>","PeriodicalId":9499,"journal":{"name":"Cancer detection and prevention","volume":"24 3","pages":"295-303"},"PeriodicalIF":0.0,"publicationDate":"2000-01-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"21810859","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
引用次数: 0
A case-control study of toenail selenium and cancer of the breast, colon, and prostate. 趾甲硒与乳腺癌、结肠癌和前列腺癌的病例对照研究。
Cancer detection and prevention Pub Date : 2000-01-01
P Ghadirian, P Maisonneuve, C Perret, G Kennedy, P Boyle, D Krewski, A Lacroix
{"title":"A case-control study of toenail selenium and cancer of the breast, colon, and prostate.","authors":"P Ghadirian,&nbsp;P Maisonneuve,&nbsp;C Perret,&nbsp;G Kennedy,&nbsp;P Boyle,&nbsp;D Krewski,&nbsp;A Lacroix","doi":"","DOIUrl":"","url":null,"abstract":"<p><p>To study the possible role of dietary and supplementary selenium intake in the etiology of cancer, we carried out a case-control study of breast, colon, and prostate cancer in Montreal between 1989 and 1993. In this study, we were able to interview a total of 1,048 incidence cases of colon (402), breast (414) and prostate (232) cancer subjects and 688 population-based controls matched for age and gender. Of these, a total of 501 cancer cases and 202 controls produced toenail samples for their selenium concentrations, which were determined by neutron activation analysis. We found no association between toenail selenium and breast cancer (odds ratio [OR], 0.72; 95% confidence interval [CI], 0.4-1.31) or prostate cancer (OR, 1.14; 95% CI, 0.46-2.83), though we did observe a statistically significant inverse association between toenail selenium level and the risk of colon cancer for both genders combined (OR, 0.42; 95% CI, 0.19-0.93; P = .009) and for female subjects (P = .050). We also found that nonsmoker case and control subjects had higher selenium in their toenail samples. This could be due either to the nature of tobacco, which reduces selenium absorption, or to smokers' consumption of certain foods containing less selenium. Further epidemiologic studies are required to clarify the role of selenium in the etiology of certain cancers.</p>","PeriodicalId":9499,"journal":{"name":"Cancer detection and prevention","volume":"24 4","pages":"305-13"},"PeriodicalIF":0.0,"publicationDate":"2000-01-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"21886237","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
引用次数: 0
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