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Sudden cardiac death caused by Kawasaki coronary artery vasculitis in a child with Hodgkin's lymphoma. Case report and literature review 一名霍奇金淋巴瘤患儿因川崎冠状动脉血管炎导致心脏猝死。病例报告和文献综述。
IF 2.3 4区 医学
Cardiovascular Pathology Pub Date : 2025-01-01 Epub Date: 2024-10-05 DOI: 10.1016/j.carpath.2024.107700
Cecilia Salzillo , Monica De Gaspari , Cristina Basso , Mariantonietta Francavilla , Francesco De Leonardis , Andrea Marzullo
{"title":"Sudden cardiac death caused by Kawasaki coronary artery vasculitis in a child with Hodgkin's lymphoma. Case report and literature review","authors":"Cecilia Salzillo ,&nbsp;Monica De Gaspari ,&nbsp;Cristina Basso ,&nbsp;Mariantonietta Francavilla ,&nbsp;Francesco De Leonardis ,&nbsp;Andrea Marzullo","doi":"10.1016/j.carpath.2024.107700","DOIUrl":"10.1016/j.carpath.2024.107700","url":null,"abstract":"<div><div>Coronary artery vasculitis is a rare pathological condition and is often a manifestation of systemic vasculitis, such as Polyarteritis Nodosa, Kawasaki Disease, Takayasu Arteritis, and Giant Cell Arteritis, with Kawasaki Disease being the most common cause in children.</div><div>We present the autopsy case of a 6-year-old boy with classic Hodgkin lymphoma who died of sudden cardiac death due to thrombosis caused by vasculitis, which exclusively affected the coronary arteries and was suggestive of Kawasaki Disease.</div><div>To further investigate the histological features of Kawasaki Disease across all age groups, we conducted a literature review using the search terms “Kawasaki AND vasculitis AND histopathology” and “Kawasaki vasculitis histopathology” in Scopus, Google Scholar, and PubMed, covering the period from 1967 to 2023.</div><div>The inclusion criteria were as follows: coronary histology (inflammation and/or aneurysm and/or thrombosis), postmortem studies, English language, free articles, all age groups, case reports, and case series.</div></div>","PeriodicalId":9451,"journal":{"name":"Cardiovascular Pathology","volume":"74 ","pages":"Article 107700"},"PeriodicalIF":2.3,"publicationDate":"2025-01-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"142388317","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":4,"RegionCategory":"医学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"OA","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
引用次数: 0
Sudden infant death syndrome “Gray Zone” in newborn with pneumonia 新生儿肺炎猝死综合征 "灰色地带"。
IF 2.3 4区 医学
Cardiovascular Pathology Pub Date : 2025-01-01 Epub Date: 2024-09-26 DOI: 10.1016/j.carpath.2024.107698
Tobia Tomasello , Beatrice Paradiso , Tommaso Rizzuti , Alessandro Del Gobbo , Lorenza Pugni , Giulia Ottaviani
{"title":"Sudden infant death syndrome “Gray Zone” in newborn with pneumonia","authors":"Tobia Tomasello ,&nbsp;Beatrice Paradiso ,&nbsp;Tommaso Rizzuti ,&nbsp;Alessandro Del Gobbo ,&nbsp;Lorenza Pugni ,&nbsp;Giulia Ottaviani","doi":"10.1016/j.carpath.2024.107698","DOIUrl":"10.1016/j.carpath.2024.107698","url":null,"abstract":"<div><div>Sudden infant death syndrome (SIDS) “gray zone” or borderline cases are those in which it is challenging to define whether the pathological findings are sufficiently severe to lead to death. We report a case of a 17-day old male newborn who came to our attention for unexplained death. A complete autopsy was performed, including close examination of the cardiac conduction system. Lungs presented diffuse alveolar damage and interstitial inflammation, the cardiac conduction system showed fetal dispersion, resorptive degeneration, junctional tissue islands and cartilaginous hypermetaplasia of the central fibrous body. The final cause of death was a “gray zone” SIDS. This case report will highlight the intersection of SIDS and pneumonia in newborns, exploring the challenges and controversies surrounding the diagnosis and management of this complex condition.</div></div>","PeriodicalId":9451,"journal":{"name":"Cardiovascular Pathology","volume":"74 ","pages":"Article 107698"},"PeriodicalIF":2.3,"publicationDate":"2025-01-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"142342177","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":4,"RegionCategory":"医学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
引用次数: 0
Current concepts in the epigenetic regulation of cardiac fibrosis 心脏纤维化表观遗传调控的当前概念。
IF 2.3 4区 医学
Cardiovascular Pathology Pub Date : 2024-11-01 Epub Date: 2024-07-10 DOI: 10.1016/j.carpath.2024.107673
{"title":"Current concepts in the epigenetic regulation of cardiac fibrosis","authors":"","doi":"10.1016/j.carpath.2024.107673","DOIUrl":"10.1016/j.carpath.2024.107673","url":null,"abstract":"<div><p>Cardiac fibrosis is a significant driver of congestive heart failure, a syndrome that continues to affect a growing patient population globally. Cardiac fibrosis results from a constellation of complex processes at the transcription, receptor, and signaling axes levels. Various mediators and signaling cascades, such as the transformation growth factor-beta pathway, have been implicated in the pathophysiology of cardiac tissue fibrosis. Our understanding of these markers and pathways has improved in recent years as more advanced technologies and assays have been developed, allowing for better delineation of the crosstalk between specific factors. There is mounting evidence suggesting that epigenetic modulation plays a pivotal role in the progression of cardiac fibrosis. Transcriptional regulation of key pro- and antifibrotic pathways can accentuate or blunt the rate and extent of fibrosis at the tissue level. Exosomes, micro-RNAs, and long noncoding RNAs all belong to factors that can impact the epigenetic signature in cardiac fibrosis. Herein, we comprehensively review the latest literature about exosomes, their contents, and cardiac fibrosis. In doing so, we highlight the specific transcriptional factors with pro- or antifibrotic properties. We also assimilate the data supporting these mediators' potential utility as diagnostic or prognostic biomarkers. Finally, we offer insight into where further work can be done to fill existing gaps to translate preclinical findings better and improve clinical outcomes.</p></div>","PeriodicalId":9451,"journal":{"name":"Cardiovascular Pathology","volume":"73 ","pages":"Article 107673"},"PeriodicalIF":2.3,"publicationDate":"2024-11-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://www.sciencedirect.com/science/article/pii/S1054880724000693/pdfft?md5=360e02ff4c582ed754af921caf42764d&pid=1-s2.0-S1054880724000693-main.pdf","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"141598536","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":4,"RegionCategory":"医学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"OA","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
引用次数: 0
Recurrent cardiac tamponade following coronavirus disease 2019 mRNA vaccination: correspondence 2019 年冠状病毒病 mRNA 疫苗接种后复发的心脏填塞：通信。
IF 2.3 4区 医学
Cardiovascular Pathology Pub Date : 2024-11-01 Epub Date: 2024-06-24 DOI: 10.1016/j.carpath.2024.107672
{"title":"Recurrent cardiac tamponade following coronavirus disease 2019 mRNA vaccination: correspondence","authors":"","doi":"10.1016/j.carpath.2024.107672","DOIUrl":"10.1016/j.carpath.2024.107672","url":null,"abstract":"","PeriodicalId":9451,"journal":{"name":"Cardiovascular Pathology","volume":"73 ","pages":"Article 107672"},"PeriodicalIF":2.3,"publicationDate":"2024-11-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"141455499","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":4,"RegionCategory":"医学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
引用次数: 0
Ruptured pulmonary artery aneurysm with prior dissection and persistent ductus arteriosus: Autopsy case report 肺动脉动脉瘤破裂并伴有动脉夹层和持续性动脉导管未闭：尸检病例报告。
IF 2.3 4区 医学
Cardiovascular Pathology Pub Date : 2024-11-01 Epub Date: 2024-08-10 DOI: 10.1016/j.carpath.2024.107684
Patcharanun Chulamanee , Pornpatsorn Siriwattanaskul , Vijarn Vachirawongsakorn , Sakda Sathirareuangchai
{"title":"Ruptured pulmonary artery aneurysm with prior dissection and persistent ductus arteriosus: Autopsy case report","authors":"Patcharanun Chulamanee ,&nbsp;Pornpatsorn Siriwattanaskul ,&nbsp;Vijarn Vachirawongsakorn ,&nbsp;Sakda Sathirareuangchai","doi":"10.1016/j.carpath.2024.107684","DOIUrl":"10.1016/j.carpath.2024.107684","url":null,"abstract":"<div><p>Cardiac tamponade from ruptured intrathoracic organs can lead to sudden cardiac death. In rare circumstances, the pulmonary artery can be the source of hemopericardium. We describe a case of a 62-year-old woman with no significant past medical history, who presented with sudden unexpected death. A forensic autopsy revealed 500 ml of hemopericardium. Further dissection demonstrated a saccular aneurysm in the pulmonary artery trunk, along with the evidence of prior dissection, i.e., neointimal layer. Persistent ductus arteriosus (PDA) was also present. Pulmonary artery aneurysms (PAA) are rare and often associated with congenital heart disease (CHD). PDA is the most common CHD related to PAA. Secondary pulmonary hypertension makes the pulmonary artery vulnerable to medial degeneration and increases the risk of dissection and rupture. Careful inspection of the great vessels and congenital anomalies are essential in the forensic autopsies for sudden death investigation.</p></div>","PeriodicalId":9451,"journal":{"name":"Cardiovascular Pathology","volume":"73 ","pages":"Article 107684"},"PeriodicalIF":2.3,"publicationDate":"2024-11-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"141970676","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":4,"RegionCategory":"医学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
引用次数: 0
Hamartoma of mature cardiac myocytes presenting as a polypoid epicardial tumor in the interatrial groove and with gene fusions by copy number anomalies of chromosome 7 成熟心肌细胞脂肪瘤（Hamartoma of Mature Cardiac Myocytes），表现为位于心房间沟的多形性心外膜肿瘤，并伴有 7 号染色体拷贝数异常的基因融合。
IF 2.3 4区 医学
Cardiovascular Pathology Pub Date : 2024-11-01 Epub Date: 2024-05-29 DOI: 10.1016/j.carpath.2024.107660
{"title":"Hamartoma of mature cardiac myocytes presenting as a polypoid epicardial tumor in the interatrial groove and with gene fusions by copy number anomalies of chromosome 7","authors":"","doi":"10.1016/j.carpath.2024.107660","DOIUrl":"10.1016/j.carpath.2024.107660","url":null,"abstract":"<div><p>Hamartoma of mature cardiac myocytes (HMCM) is an extremely rare cardiac tumor characterized by benign growth of differentiated mature striated cardiac myocytes, and usually involves the ventricular myocardium. We describe the case of a 15-year-old female who presented with a short history of atrial fibrillation and a polypoid epicardial tumor that was attached to the interatrial groove by a short pedicle. The resected specimen showed features consistent with HMCM. Although these tumors are not associated with any known molecular or cytogenetic abnormalities, we identified fusions transcripts along with complex copy number anomalies of chromosome 7.</p></div>","PeriodicalId":9451,"journal":{"name":"Cardiovascular Pathology","volume":"73 ","pages":"Article 107660"},"PeriodicalIF":2.3,"publicationDate":"2024-11-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"141183825","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":4,"RegionCategory":"医学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
引用次数: 0
COVER 3: Editorial Board 封面 3：编辑委员会
IF 2.3 4区 医学
Cardiovascular Pathology Pub Date : 2024-11-01 Epub Date: 2024-09-10 DOI: 10.1016/S1054-8807(24)00092-9
{"title":"COVER 3: Editorial Board","authors":"","doi":"10.1016/S1054-8807(24)00092-9","DOIUrl":"10.1016/S1054-8807(24)00092-9","url":null,"abstract":"","PeriodicalId":9451,"journal":{"name":"Cardiovascular Pathology","volume":"73 ","pages":"Article 107696"},"PeriodicalIF":2.3,"publicationDate":"2024-11-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://www.sciencedirect.com/science/article/pii/S1054880724000929/pdfft?md5=996b192e13ec7f524b13e94e46520691&pid=1-s2.0-S1054880724000929-main.pdf","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"142163057","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":4,"RegionCategory":"医学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"OA","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
引用次数: 0
Doxorubicin-related cardiotoxicity: review of fundamental pathways of cardiovascular system injury 多柔比星相关心脏毒性：心血管系统损伤的基本途径回顾。
IF 2.3 4区 医学
Cardiovascular Pathology Pub Date : 2024-11-01 Epub Date: 2024-08-05 DOI: 10.1016/j.carpath.2024.107683
Ashot Avagimyan , Nana Pogosova , Lev Kakturskiy , Mohammad Sheibani , Abhiram Challa , Eugenia Kogan , Federica Fogacci , Liudmila Mikhaleva , Rositsa Vandysheva , Marianna Yakubovskaya , Andrea Faggiano , Stefano Carugo , Olga Urazova , Behnaz Jahanbin , Ekaterina Lesovaya , Srujana Polana , Kirill Kirsanov , Yasar Sattar , Artem Trofimenko , Tatiana Demura , Nizal Sarrafzadegan
{"title":"Doxorubicin-related cardiotoxicity: review of fundamental pathways of cardiovascular system injury","authors":"Ashot Avagimyan ,&nbsp;Nana Pogosova ,&nbsp;Lev Kakturskiy ,&nbsp;Mohammad Sheibani ,&nbsp;Abhiram Challa ,&nbsp;Eugenia Kogan ,&nbsp;Federica Fogacci ,&nbsp;Liudmila Mikhaleva ,&nbsp;Rositsa Vandysheva ,&nbsp;Marianna Yakubovskaya ,&nbsp;Andrea Faggiano ,&nbsp;Stefano Carugo ,&nbsp;Olga Urazova ,&nbsp;Behnaz Jahanbin ,&nbsp;Ekaterina Lesovaya ,&nbsp;Srujana Polana ,&nbsp;Kirill Kirsanov ,&nbsp;Yasar Sattar ,&nbsp;Artem Trofimenko ,&nbsp;Tatiana Demura ,&nbsp;Nizal Sarrafzadegan","doi":"10.1016/j.carpath.2024.107683","DOIUrl":"10.1016/j.carpath.2024.107683","url":null,"abstract":"<div><p>Over the years, advancements in the field of oncology have made remarkable strides in enhancing the efficacy of medical care for patients with cancer. These modernizations have resulted in prolonged survival and improved the quality of life for these patients. However, this progress has also been accompanied by escalation in mortality rates associated with anthracycline chemotherapy.</p><p>Anthracyclines, which are known for their potent antitumor properties, are notorious for their substantial cardiotoxic potential. Remarkably, even after 6 decades of research, a conclusive solution to protect the cardiovascular system against doxorubicin-induced damage has not yet been established. A comprehensive understanding of the pathophysiological processes driving cardiotoxicity combined with targeted research is crucial for developing innovative cardioprotective strategies.</p><p>This review seeks to explain the mechanisms responsible for structural and functional alterations in doxorubicin-induced cardiomyopathy.</p></div>","PeriodicalId":9451,"journal":{"name":"Cardiovascular Pathology","volume":"73 ","pages":"Article 107683"},"PeriodicalIF":2.3,"publicationDate":"2024-11-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"141901039","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":4,"RegionCategory":"医学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
引用次数: 0
Dilated cardiomyopathy due to a novel combination of TTN and BAG3 genetic variants: From acute heart failure to subclinical phenotypes 由 TTN 和 BAG3 基因变异新组合引起的扩张型心肌病：从急性心力衰竭到亚临床表型。
IF 2.3 4区 医学
Cardiovascular Pathology Pub Date : 2024-11-01 Epub Date: 2024-07-25 DOI: 10.1016/j.carpath.2024.107675
Irene Bottillo , Carla Giordano , Maria Pia Ciccone , Maria Gemma Pignataro , Fiammetta Albi , Gabriella Parisi , Daniela Formicola , Simona Grotta , Federico Ranocchi , Maria Valeria Giuli , Saula Checquolo , Laura Masuelli , Federica Re , Silvia Majore , Giulia d'Amati , Paola Grammatico
{"title":"Dilated cardiomyopathy due to a novel combination of TTN and BAG3 genetic variants: From acute heart failure to subclinical phenotypes","authors":"Irene Bottillo ,&nbsp;Carla Giordano ,&nbsp;Maria Pia Ciccone ,&nbsp;Maria Gemma Pignataro ,&nbsp;Fiammetta Albi ,&nbsp;Gabriella Parisi ,&nbsp;Daniela Formicola ,&nbsp;Simona Grotta ,&nbsp;Federico Ranocchi ,&nbsp;Maria Valeria Giuli ,&nbsp;Saula Checquolo ,&nbsp;Laura Masuelli ,&nbsp;Federica Re ,&nbsp;Silvia Majore ,&nbsp;Giulia d'Amati ,&nbsp;Paola Grammatico","doi":"10.1016/j.carpath.2024.107675","DOIUrl":"10.1016/j.carpath.2024.107675","url":null,"abstract":"<div><p>Dilated cardiomyopathy (DCM) is defined as left ventricular enlargement accompanied by systolic dysfunction not explained by abnormal loading conditions or coronary heart disease. The DCM clinical <em>spectrum</em> is broad, ranging from subclinical to severe presentation with progression to end stage heart failure. To date, different genetic <em>loci</em> have been found to have moderate/definitive evidence for causality in DCM and pathogenic variants in the <em>TTN</em> gene represent the main genetic determinant.</p><p>Here, we describe a family in which the co-occurrence of two genetic hits, one in the <em>TTN</em> and one in the <em>BAG3</em> gene, was associated with heterogeneous clinical presentation ranging from subclinical phenotypes to acute cardiogenic shock mimicking fulminant myocarditis. We hypothesize that at least some specific <em>BAG3</em> genotypes could be related to DCM presenting with acute heart failure and suggest that patients and relatives carrying <em>BAG3</em> pathogenic variants should be addressed to a tertiary-level heart care center.</p></div>","PeriodicalId":9451,"journal":{"name":"Cardiovascular Pathology","volume":"73 ","pages":"Article 107675"},"PeriodicalIF":2.3,"publicationDate":"2024-11-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://www.sciencedirect.com/science/article/pii/S1054880724000711/pdfft?md5=36798fb256d6e87a6374f45ee082b089&pid=1-s2.0-S1054880724000711-main.pdf","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"141765549","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":4,"RegionCategory":"医学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"OA","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
引用次数: 0
Exploring the role of pericardial miRNAs and exosomes in modulating cardiac fibrosis 探索心包 miRNA 和外泌体在调节心脏纤维化中的作用。
IF 2.3 4区 医学
Cardiovascular Pathology Pub Date : 2024-11-01 Epub Date: 2024-06-19 DOI: 10.1016/j.carpath.2024.107671
{"title":"Exploring the role of pericardial miRNAs and exosomes in modulating cardiac fibrosis","authors":"","doi":"10.1016/j.carpath.2024.107671","DOIUrl":"10.1016/j.carpath.2024.107671","url":null,"abstract":"<div><p>The potential of the pericardial space as a therapeutic delivery tool for cardiac fibrosis and heart failure (HF) treatment has yet to be elucidated. Recently, miRNAs and exosomes have been discovered to be present in human pericardial fluid (PF). Novel studies have shown characteristic human PF miRNA compositions associated with cardiac diseases and higher miRNA expressions in PF compared to peripheral blood. Five key studies found differentially expressed miRNAs in HF, angina pectoris, aortic stenosis, ventricular tachycardia, and congenital heart diseases with either atrial fibrillation or sinus rhythm. As miRNA-based therapeutics for cardiac fibrosis and HF showed promising results in several <em>in vivo</em> studies for multiple miRNAs, we hypothesize a potential role of miRNA-based therapeutics delivered through the pericardial cavity. This is underlined by the favorable results of the first phase 1b clinical trial in this emerging field. Presenting the first human miRNA antisense drug trial, inhibition of miR-132 by intravenous administration of a novel antisense oligonucleotide, CDR132L, established efficacy in reducing miR-132 in plasma samples in a dose-dependent manner. We screened the literature, provided an overview of the miRNAs and exosomes present in PF, and drew a connection to those miRNAs previously elucidated in cardiac fibrosis and HF. Further, we speculate about clinical implications and potential delivery methods.</p></div>","PeriodicalId":9451,"journal":{"name":"Cardiovascular Pathology","volume":"73 ","pages":"Article 107671"},"PeriodicalIF":2.3,"publicationDate":"2024-11-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://www.sciencedirect.com/science/article/pii/S105488072400067X/pdfft?md5=fd26a5be0c55954fe94eead47f63d87d&pid=1-s2.0-S105488072400067X-main.pdf","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"141436398","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":4,"RegionCategory":"医学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"OA","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
引用次数: 0
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