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Child nephrology and urology最新文献

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Hypertension in the neonatal period. 新生儿期高血压。
Child nephrology and urology Pub Date : 1992-01-01
D L Blowey, B A Warady, U Alon
{"title":"Hypertension in the neonatal period.","authors":"D L Blowey, B A Warady, U Alon","doi":"","DOIUrl":"","url":null,"abstract":"","PeriodicalId":77067,"journal":{"name":"Child nephrology and urology","volume":"12 2-3","pages":"113-8"},"PeriodicalIF":0.0,"publicationDate":"1992-01-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"12794194","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
引用次数: 0
Pediatric post-transplant hypertension: a review of current standards of care. 儿童移植后高血压:当前护理标准的回顾。
Child nephrology and urology Pub Date : 1992-01-01
J A Ingelfinger, E D Brewer
{"title":"Pediatric post-transplant hypertension: a review of current standards of care.","authors":"J A Ingelfinger, E D Brewer","doi":"","DOIUrl":"","url":null,"abstract":"","PeriodicalId":77067,"journal":{"name":"Child nephrology and urology","volume":"12 2-3","pages":"139-46"},"PeriodicalIF":0.0,"publicationDate":"1992-01-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"12794198","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
引用次数: 0
Hypercholesterolemia in rats with chronic renal insufficiency not aggravated by recombinant human growth hormone. 重组人生长激素未加重慢性肾功能不全大鼠的高胆固醇血症。
Child nephrology and urology Pub Date : 1992-01-01
F Santos, J C Chan, R J Krieg, K Niimi, J D Hanna, J W Foreman, A H Perkins
{"title":"Hypercholesterolemia in rats with chronic renal insufficiency not aggravated by recombinant human growth hormone.","authors":"F Santos,&nbsp;J C Chan,&nbsp;R J Krieg,&nbsp;K Niimi,&nbsp;J D Hanna,&nbsp;J W Foreman,&nbsp;A H Perkins","doi":"","DOIUrl":"","url":null,"abstract":"<p><p>The lipid metabolic disorders in chronic renal insufficiency (CRI) are related to increased hepatic lipid synthesis, reduced triglyceride removal coupled with insulin insensitivity and impaired lipoprotein lipase activity. Growth hormone is lipolytic, and the effects of recombinant human growth hormone (rhGH) on the hypercholesterolemia of CRI are unsettled. To test this question, we gave rhGH for 14 days at a dosage of 3 units/day intraperitoneally to two-stage, 5/6 nephrectomized, male Sprague-Dawley rats (n = 18) compared to sex- and age-matched control (n = 27) and CRI (n = 40) rats. At the end of the study, CRI rats and those treated with rhGH had a similar degree of renal impairment, as assessed by serum concentrations (mean +/- SEM) of urea nitrogen (49 +/- 3 vs. 54 +/- 4 mg/dl), creatinine (0.9 +/- 0.0 vs. 1.0 +/- 0.1 mg/dl) and cumulative food intake (311 +/- 8 vs. 290 +/- 12 g). Serum urea nitrogen (16 +/- 4 mg/dl) and creatinine (0.4 +/- 0.1 mg/dl) concentrations as well as food intake (412 +/- 9 g) of control rats were significantly (p < 0.0001) different. Serum cholesterol concentration of CRI rats treated with rhGH (87 +/- 3 mg/dl) was not higher than those of CRI rats (81 +/- 2 mg/dl, p < 0.1338) but was significantly higher than in control rats (55 +/- 3 mg/dl, p < 0.0001). CRI rats treated with rhGH showed a similar serum albumin concentration and lower serum glucose than CRI rats (0.9 +/- 0.1 vs. 0.9 +/- 0.0 g/dl and 144 +/- 4 vs. 163 +/- 3 mg/dl, p < 0.0001).(ABSTRACT TRUNCATED AT 250 WORDS)</p>","PeriodicalId":77067,"journal":{"name":"Child nephrology and urology","volume":"12 4","pages":"182-5"},"PeriodicalIF":0.0,"publicationDate":"1992-01-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"12643863","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
引用次数: 0
Sudden death in cystinosis. 胱氨酸病猝死
Child nephrology and urology Pub Date : 1992-01-01
B Lettgen, M Bald, W Rascher
{"title":"Sudden death in cystinosis.","authors":"B Lettgen,&nbsp;M Bald,&nbsp;W Rascher","doi":"","DOIUrl":"","url":null,"abstract":"<p><p>We report on an 8-year-old boy with infantile cystinosis. The patient died in hospital for unknown reason after cardiopulmonary arrest although resuscitation was started immediately. As opposed to other cases [1-3], neither laboratory investigations nor autopsy in out patient gave an explanation for the sudden death.</p>","PeriodicalId":77067,"journal":{"name":"Child nephrology and urology","volume":"12 4","pages":"228-30"},"PeriodicalIF":0.0,"publicationDate":"1992-01-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"12643871","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
引用次数: 0
Partial deficiency of cytochrome c oxidase with isolated proximal renal tubular acidosis and hypercalciuria. 细胞色素c氧化酶部分缺乏伴孤立性肾近端小管酸中毒和高钙尿症。
Child nephrology and urology Pub Date : 1992-01-01
H Matsutani, Y Mizusawa, M Shimoda, F Niimura, A Takeda, M Shimohira, Y Iwakawa
{"title":"Partial deficiency of cytochrome c oxidase with isolated proximal renal tubular acidosis and hypercalciuria.","authors":"H Matsutani,&nbsp;Y Mizusawa,&nbsp;M Shimoda,&nbsp;F Niimura,&nbsp;A Takeda,&nbsp;M Shimohira,&nbsp;Y Iwakawa","doi":"","DOIUrl":"","url":null,"abstract":"<p><p>We report the case of a 5-year-old boy with mitochondrial cytopathy due to a partial deficiency of cytochrome c oxidase who had isolated proximal renal tubular acidosis and hypercalciuria. The patient developed hypotonia and blepharoptosis and exhibited growth retardation. Biochemical examination of muscle tissue revealed a partial deficiency of cytochrome c oxidase. He was treated with an alkali, hydrochlorothiazide, and indomethacin. After treatment, metabolic acidosis and hypercalciuria improved, and the patient had a catch-up growth phase. This case emphasizes the importance of performing renal tubular functional investigations and treatment in patients with mitochondrial cytopathy, even in the absence of multiple proximal tubular dysfunction.</p>","PeriodicalId":77067,"journal":{"name":"Child nephrology and urology","volume":"12 4","pages":"221-4"},"PeriodicalIF":0.0,"publicationDate":"1992-01-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"12508996","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
引用次数: 0
Medical management of hypertension in childhood. 儿童期高血压的医学处理。
Child nephrology and urology Pub Date : 1992-01-01
P N Houtman, M J Dillon
{"title":"Medical management of hypertension in childhood.","authors":"P N Houtman,&nbsp;M J Dillon","doi":"","DOIUrl":"","url":null,"abstract":"","PeriodicalId":77067,"journal":{"name":"Child nephrology and urology","volume":"12 2-3","pages":"154-61"},"PeriodicalIF":0.0,"publicationDate":"1992-01-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"12526084","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
引用次数: 0
Evoked potentials before and after anemia correction with recombinant human erythropoietin in end-stage renal disease. 重组人促红细胞生成素治疗终末期肾病患者贫血前后的诱发电位。
Child nephrology and urology Pub Date : 1992-01-01
A Suppiej, G Montini, G Casara, A Polo, G Zacchello, F Zacchello
{"title":"Evoked potentials before and after anemia correction with recombinant human erythropoietin in end-stage renal disease.","authors":"A Suppiej,&nbsp;G Montini,&nbsp;G Casara,&nbsp;A Polo,&nbsp;G Zacchello,&nbsp;F Zacchello","doi":"","DOIUrl":"","url":null,"abstract":"<p><p>Subclinical involvement of the nervous system in uremic adults has been detected by modern neurophysiological techniques. Chronic anemia is one of the possible factors responsible for neural dysfunction in uremia. We evaluated neurophysiological (brainstem auditory and somatosensory evoked potential) abnormalities and their possible modification following anemia correction with recombinant human erythropoietin in 14 children with end-stage renal disease maintained by hemodialysis. Only peripheral and 8th cranial nerve electrophysiological data are significantly abnormal in our patients, and they are not acutely modified by anemia correction. These data confirm the importance of electrophysiological testing of uremic children for detecting nervous system involvement at an early stage and for monitoring the efficacy of its management.</p>","PeriodicalId":77067,"journal":{"name":"Child nephrology and urology","volume":"12 4","pages":"197-201"},"PeriodicalIF":0.0,"publicationDate":"1992-01-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"12643866","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
引用次数: 0
Efficacy of CAPD as the primary treatment for end-stage renal failure in children. CAPD作为儿童终末期肾功能衰竭主要治疗方法的疗效观察。
Child nephrology and urology Pub Date : 1992-01-01
M Barakat, J M Savage, A Burns, M Stewart
{"title":"Efficacy of CAPD as the primary treatment for end-stage renal failure in children.","authors":"M Barakat,&nbsp;J M Savage,&nbsp;A Burns,&nbsp;M Stewart","doi":"","DOIUrl":"","url":null,"abstract":"<p><p>In the 6 years before this paper, all children presenting with end-stage renal failure (ESRF) in Northern Ireland were electively commenced on continuous ambulatory peritoneal dialysis (CAPD). In that period, 22 patients were treated, 16 received renal transplants and 6 remained on CAPD. Active treatment was withdrawn in 1 child due to obliterative peritonitis. There were no deaths. One incident of peritonitis was documented per 11.6 patient treatment months. Recurring peritonitis lead to catheter replacement in 3 patients. Other problems encountered were catheter exit site infections, hernia formation and in one instance, hydrothorax. Peritonitis was more common in children under 2 years of age as was undernutrition, growth retardation and developmental delay. Home CAPD can be recommended as a viable first option treatment for childhood ESRF where suitable haemodialysis facilities are not easily accessible.</p>","PeriodicalId":77067,"journal":{"name":"Child nephrology and urology","volume":"12 4","pages":"216-20"},"PeriodicalIF":0.0,"publicationDate":"1992-01-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"12643869","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
引用次数: 0
Primary hyperoxaluria type 1: clinical manifestations in infancy and prenatal diagnosis. 原发性高草酸尿1型:婴儿期临床表现及产前诊断。
Child nephrology and urology Pub Date : 1992-01-01
N Illum, L Lavard, C J Danpure, T Horn, H AErenlund Jensen, F Skovby
{"title":"Primary hyperoxaluria type 1: clinical manifestations in infancy and prenatal diagnosis.","authors":"N Illum,&nbsp;L Lavard,&nbsp;C J Danpure,&nbsp;T Horn,&nbsp;H AErenlund Jensen,&nbsp;F Skovby","doi":"","DOIUrl":"","url":null,"abstract":"<p><p>A 9-month-old Pakistani boy of consanguineous parents presented with uraemia preceded by pyuria from 5 weeks of age. He had no history of renal calculi or macroscopic haematuria. Renal biopsy revealed severe calcium oxalate deposition in the tubuli and fibrosis of the interstitial tissue. Liver biopsy demonstrated complete absence of alanine: glyoxylate aminotransferase catalytic activity and immunoreactive protein compatible with a diagnosis of primary hyperoxaluria type 1. He died at the age of 11 months, just before liver transplantation was made possible. Fetal liver biopsy in the mother's subsequent pregnancy showed normal enzymatic activity. Early detection and early replacement of the missing enzyme by liver transplantation are considered to be crucial for the survival of severely affected infants with the acute neonatal form of primary hyperoxaluria type 1. Persistent pyuria could be an early sign of renal damage secondary to accumulation of oxalate crystals.</p>","PeriodicalId":77067,"journal":{"name":"Child nephrology and urology","volume":"12 4","pages":"225-7"},"PeriodicalIF":0.0,"publicationDate":"1992-01-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"12643870","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
引用次数: 0
Urinary excretion of N-acetyl-beta-D-glucosaminidase in proteinuric states. 蛋白尿状态下n -乙酰- β - d -氨基葡萄糖酶的尿排泄。
Child nephrology and urology Pub Date : 1992-01-01
E Ring, G Zobel, W Erwa, M Haim-Kuttnig
{"title":"Urinary excretion of N-acetyl-beta-D-glucosaminidase in proteinuric states.","authors":"E Ring,&nbsp;G Zobel,&nbsp;W Erwa,&nbsp;M Haim-Kuttnig","doi":"","DOIUrl":"","url":null,"abstract":"<p><p>Urinary excretion of N-acetyl-beta-D-glucosaminidase (NAG) was studied in 22 children with proteinuria and a normal glomerular filtration rate. Twelve patients had minimal change nephrotic syndrome (MCNS), 10 children suffered from different types of glomerulonephritis (GN) without tubulointerstitial renal disease. Sixty-six random urine samples were analyzed for protein, creatinine and NAG. There was a strong positive correlation between proteinuria and urinary NAG activity (r = 0.79, p = 0.0001). The raised NAG activities in proteinuric states turned to normal during remission of the disease. Patients with MCNS and GN did not differ significantly and had nearly identical correlation curves. Our results seem to indicate that functional changes in the renal tubular cells probably caused by protein resorption are responsible for this association. Raised NAG activities in proteinuria reflect the activity of the disease but cannot distinguish patients with MCNS and GN.</p>","PeriodicalId":77067,"journal":{"name":"Child nephrology and urology","volume":"12 1","pages":"15-8"},"PeriodicalIF":0.0,"publicationDate":"1992-01-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"12773602","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
引用次数: 0
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