Journal of Cardiology Cases最新文献

{"title":"Severe cibenzoline toxicity in hypertrophic obstructive cardiomyopathy successfully managed with extracorporeal membrane oxygenation and percutaneous transluminal septal myocardial ablation — A case report","authors":"Hiroto Yagasaki MD ,&nbsp;Takeki Suzuki MD, MPH, PhD ,&nbsp;Keitaro Watanabe MD ,&nbsp;Shunichiro Warita MD, PhD ,&nbsp;Makoto Iwama MD, PhD ,&nbsp;Toshiyuki Noda MD, PhD, FJCC","doi":"10.1016/j.jccase.2025.02.006","DOIUrl":"10.1016/j.jccase.2025.02.006","url":null,"abstract":"<div><div>Cibenzoline (CBZ), a class I antiarrhythmic drug, is used for patients with hypertrophic obstructive cardiomyopathy (HOCM). However, it requires careful monitoring in patients with renal dysfunction for potential toxicity. We present a case of severe CBZ toxicity in a 72-year-old woman with HOCM, previous ascending aortic dissection repair, and renal dysfunction. She was maintained on CBZ 300 mg daily despite fluctuating renal function. She presented with acute respiratory distress. On presentation, she was found to have bradycardia with QRS prolongation (340 ms) and markedly elevated CBZ levels (1973 ng/mL, therapeutic range: 200–800 ng/mL). She developed sudden cardiac arrest in the emergency room. Following cardiac arrest, venoarterial extracorporeal membrane oxygenation (VA-ECMO) was initiated. Direct hemoperfusion yielded minimal reduction in CBZ levels. With increased urine output, CBZ levels normalized, accompanied by electrocardiographic improvement. After VA-ECMO withdrawal, percutaneous transluminal septal myocardial ablation (PTSMA) was performed as definitive treatment to eliminate CBZ dependency. This case illustrates the complex interaction between HOCM, renal impairment, and CBZ toxicity, emphasizing the importance of careful drug monitoring in patients with renal impairment. Additionally, it demonstrates the potential role of PTSMA as a definitive treatment for selected patients with HOCM at high risk of medication-related complications.</div></div><div><h3>Learning objectives</h3><div><ul><li><span>•</span><span><div>Understand the pharmacokinetics of cibenzoline and its need for monitoring in elderly patients with hypertrophic obstructive cardiomyopathy (HOCM) and impaired renal function.</div></span></li><li><span>•</span><span><div>Recognize the mechanisms of cibenzoline toxicity and its acute management strategies, including mechanical circulatory support and direct hemoperfusion.</div></span></li><li><span>•</span><span><div>Identify the acute management strategies and long-term treatment options for complications arising from medical therapy in patients with HOCM.</div></span></li></ul></div></div>","PeriodicalId":52092,"journal":{"name":"Journal of Cardiology Cases","volume":"31 6","pages":"Pages 158-161"},"PeriodicalIF":0.0,"publicationDate":"2025-06-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"144184695","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}

{"title":"Sudden cardiac death unveiling a mitral annular disjunction in a young man: The role of multimodal cardiac imaging for diagnosis","authors":"Waly Niang Mboup MD,&nbsp;Clément N'guyen Ngok MD,&nbsp;Jean-François Riviere MD,&nbsp;Nicolas Delarche MD","doi":"10.1016/j.jccase.2025.03.004","DOIUrl":"10.1016/j.jccase.2025.03.004","url":null,"abstract":"<div><div>Non-ischemic structural heart diseases, such as mitral annular disjunction (MAD) and/or mitral valve prolapse (MVP), are known to increase the risk of arrhythmias and sudden cardiac death (SCD). We report a rare case of SCD in a young patient, secondary to ventricular fibrillation, revealing a MAD. A 20-year-old man was admitted to our hospital after a sudden cardiac arrest at home. Immediate cardiopulmonary resuscitation was initiated with chest compressions. Semi-automatic defibrillator tracings demonstrated ventricular fibrillation, and three rounds of defibrillation were performed with return of spontaneous circulation within approximately 15 min. Electrocardiogram showed T-wave inversion in inferior-lateral leads. Transthoracic echocardiography and cardiac magnetic resonance imaging revealed a MAD and a bileaflet MVP without obvious valve regurgitation. The patient was diagnosed with ventricular fibrillation, likely resulting from MAD. He was initiated on a daily regimen of bisoprolol and received a subcutaneous implantable cardioverter-defibrillator. In-hospital evolution was favorable. Cognitive impairment was noted, leading to a referral for rehabilitation. In conclusion, this case involves a cardiac arrest likely caused by valvular heart disease. Multimodal cardiac imaging contribution is fundamental in the diagnosis of MAD with MVP, which is an uncommon cause of cardiac arrest.</div></div><div><h3>Learning objectives</h3><div>This case highlights the utility of multimodal cardiac imaging in mitral annular disjunction (MAD) diagnosis and the management of emergency care. Isolated MAD is not an indication for implantable cardioverter-defibrillator (ICD) placement, but the sudden death due to ventricular fibrillation that was recovered in this patient makes the implantation of a prophylactic ICD worth considering.</div></div>","PeriodicalId":52092,"journal":{"name":"Journal of Cardiology Cases","volume":"31 6","pages":"Pages 186-188"},"PeriodicalIF":0.0,"publicationDate":"2025-06-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"144184429","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}

{"title":"Incidental acute cholecystitis as a gateway to the diagnosis of AL cardiac amyloidosis","authors":"Haruhiko Higashi MD, PhD ,&nbsp;Yukihiro Miyazaki MD, PhD ,&nbsp;Mitsuharu Ueda MD, PhD ,&nbsp;Shunsuke Tamaki MD, PhD ,&nbsp;Kazuhisa Nishimura MD, PhD ,&nbsp;Katsuji Inoue MD, PhD, FJCC ,&nbsp;Shuntaro Ikeda MD, PhD, FJCC ,&nbsp;Osamu Yamaguchi MD, PhD, FJCC","doi":"10.1016/j.jccase.2025.03.006","DOIUrl":"10.1016/j.jccase.2025.03.006","url":null,"abstract":"<div><div>Amyloid light-chain (AL) amyloidosis is characterized by the deposition of amyloid fibrils in various organs, although gallbladder involvement is rare. We present the case of a 62-year-old female patient initially diagnosed with heart failure with preserved ejection fraction (HFpEF), who incidentally developed acute cholecystitis during her evaluation for HFpEF. Following a cholecystectomy, amyloid deposits were identified in the resected gallbladder. Subsequent investigations confirmed cardiac amyloidosis through imaging, bone marrow analysis, and M-protein detection, leading to a diagnosis of AL λ amyloidosis associated with multiple myeloma. The patient favorably responded to chemotherapy. This case emphasizes the importance of considering gallbladder amyloidosis, which may aid in avoiding unnecessary biopsies and facilitate timely diagnosis and treatment of amyloidosis.</div></div><div><h3>Learning objective</h3><div>Gallbladder amyloidosis is rare. However, recognizing that the gallbladder may be a site of amyloid deposition is crucial in cardiac amyloidosis management. This awareness can help clinicians avoid unnecessary biopsies by considering the gallbladder as a potential source of diagnostic tissue.</div></div>","PeriodicalId":52092,"journal":{"name":"Journal of Cardiology Cases","volume":"31 6","pages":"Pages 182-185"},"PeriodicalIF":0.0,"publicationDate":"2025-06-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"144184507","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}

{"title":"Survival post out-of-hospital cardiac arrest from anomalous right coronary artery: A case report and insight into management","authors":"Haowen Jiang MBBS ,&nbsp;Samuel Ji Quan Koh MBBS, MRCP, MMED ,&nbsp;Jeffrey Lau BA, MD, PhD ,&nbsp;Swee Yaw Tan MBChB, MRCP","doi":"10.1016/j.jccase.2025.02.004","DOIUrl":"10.1016/j.jccase.2025.02.004","url":null,"abstract":"<div><div>Anomalous coronary arteries originating from the opposite sinus of Valsalva are rare causes of sudden cardiac death (SCD), and when present are associated with poor outcomes. We present a rare case of a young man with an anomalous right coronary artery (RCA) presenting with out of hospital cardiac arrest and provide insights into management. A 27-year-old healthy Chinese man presented with out of hospital cardiac arrest during exertion, with initial electrocardiogram and echocardiography unrevealing for an underlying cause of collapse. Subsequent computed tomography coronary angiogram revealed the presence of an anomalous origin of the RCA from the left coronary sinus with acute angle take-off and slit-like orifice, with an inter-arterial course. Other investigations, including flecainide challenge test, cardiac magnetic resonance imaging, and treadmill stress tests were unyielding. He subsequently underwent an uneventful surgery to unroof the origin of the anomalous RCA without the need for an implantable cardiac defibrillator. The patient made a full recovery post-surgery and was discharged well. Anomalous coronary arteries with an inter-arterial course are a rare cause of SCD and where responsible, these anomalies are often found postmortem after SCD. In our case, the patient successfully underwent surgical correction.</div></div><div><h3>Learning objectives</h3><div>Anomalous coronary arteries are a rare cause of cardiac arrest and when present, are often associated with poor neurological and functional outcomes. The long-term management of this group of patients is not well known. After appropriate surgical correction and with preserved cardiac function, an implantable cardiac defibrillator may not always be necessary. This decision should follow a detailed discussion between the cardiologist, cardiothoracic surgeons, and patient.</div></div>","PeriodicalId":52092,"journal":{"name":"Journal of Cardiology Cases","volume":"31 6","pages":"Pages 162-165"},"PeriodicalIF":0.0,"publicationDate":"2025-06-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"144184699","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}

{"title":"Cardiac arrest in an (apparently) healthy heart: A case report of an undiagnosed arrhythmogenic cardiomyopathy","authors":"Paolo Pollice MD, PhD ,&nbsp;Antonio Gianluca Robles MD, PhD ,&nbsp;Domenico Riccardo Rosario Chieppa MD ,&nbsp;Saverio Pollice MD ,&nbsp;Francesco Bartolomucci MD, PhD","doi":"10.1016/j.jccase.2025.03.005","DOIUrl":"10.1016/j.jccase.2025.03.005","url":null,"abstract":"<div><div>Arrhythmogenic cardiomyopathy is a rare and insidious disease that can be misdiagnosed with the common first-line diagnostic techniques. It can also primarily debut with sustained ventricular arrhythmias that can lead to cardiac arrest. We report the case of a 49-year-old triathlon athlete who during strenuous physical exercise experimented cardiac arrest. After successful resuscitation he was admitted to our unit: electrocardiogram, emergency echocardiogram, and coronary angiography did not show anything abnormal. Holter monitoring showed premature ventricular complexes with two different morphologies and cardiac magnetic resonance allowed us to make diagnosis of biventricular arrhythmogenic cardiomyopathy previously unrecognized. A subcutaneous implantable cardiac defibrillator was positioned for secondary prevention. Our case shows that in athletes a deep process of diagnostic screening is mandatory and this must include also cardiac magnetic resonance in case of element of clinical suspicion such as premature ventricular complexes at Holter monitoring and/or low voltages on limb leads in baseline 12‑lead electrocardiogram. A network of basic life emergency support measures is fundamental in every setting in which sport at competitive and non-competitive levels is performed.</div></div><div><h3>Learning objectives</h3><div>A deep diagnostic cardiologic screening for young athletes is important especially in case of premature ventricular complexes at electrocardiographic Holter monitoring and/or in clinical suspicion of an underlying cardiomyopathy. In selected cases the execution of cardiac magnetic resonance imaging is essential to permit the correct diagnostic assessment of a previously undiagnosed cardiomyopathy as the arrhythmogenic cardiomyopathy preventing dangerous (also lethal) clinical presentation.</div></div>","PeriodicalId":52092,"journal":{"name":"Journal of Cardiology Cases","volume":"31 6","pages":"Pages 178-181"},"PeriodicalIF":0.0,"publicationDate":"2025-06-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"144184436","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}

{"title":"Rapid regression of marked left ventricular septal hypertrophy following immunosuppressive therapy in cardiac sarcoidosis","authors":"Ayaka Fujita MD ,&nbsp;Masashi Amano MD, PhD ,&nbsp;Yurie Tamai MS ,&nbsp;Makoto Amaki MD, PhD ,&nbsp;Hideaki Kanzaki MD, PhD, FJCC ,&nbsp;Yoshiaki Morita MD, PhD ,&nbsp;Takeshi Kitai MD, PhD ,&nbsp;Chisato Izumi MD, PhD, FJCC","doi":"10.1016/j.jccase.2025.02.008","DOIUrl":"10.1016/j.jccase.2025.02.008","url":null,"abstract":"<div><div>A 46-year-old man presented with asymmetric remarkable left ventricular (LV) septal hypertrophy (maximal wall thickness: 24 mm) and complete atrio-ventricular block. Systemic sarcoidosis with a cardiac lesion was diagnosed by a supraclavicular lymph node biopsy. Following pacemaker implantation, the patient received immunosuppressive therapy with corticosteroids and methotrexate. One week after starting treatment, echocardiography and cardiac magnetic resonance (CMR) imaging showed reduced LV septal hypertrophy. At the 6-month follow-up, further thinning of the basal septal wall and enlargement of the LV with a decreased ejection fraction were observed, despite resolution of abnormal uptake in ¹⁸F-fluorodeoxyglucose positron emission tomography. A reduction in T2 values on CMR indicated that the initial hypertrophy was associated with edematous and inflammatory changes. Our findings suggest that there were heterogeneous lesions in the myocardium, such as edematous lesions responsive to immunosuppressive therapy and fibrotic lesions progressing to LV wall thinning.</div></div><div><h3>Learning objective</h3><div>Cardiac sarcoidosis with asymmetrical and remarkable hypertrophy is rare in the clinical setting, and using multimodality imaging in addition to histological findings is necessary for diagnosing this condition. Multimodality imaging including echocardiography and cardiac magnetic resonance are useful for evaluating myocardial characterization and confirming remarkable changes in left ventricular wall thickness and dysfunction after immunosuppression therapy.</div></div>","PeriodicalId":52092,"journal":{"name":"Journal of Cardiology Cases","volume":"31 6","pages":"Pages 174-177"},"PeriodicalIF":0.0,"publicationDate":"2025-06-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"144184435","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}

{"title":"Fatal coronary vasospasm following oral triptan intake","authors":"Idir-Yanis Djellal MD,&nbsp;Sohaïb Mansour MD,&nbsp;Brahim Berdaoui MD,&nbsp;Sophie Samyn MD,&nbsp;Jose Castro Rodriguez MD,&nbsp;Georgiana Pintea Bentea MD","doi":"10.1016/j.jccase.2025.02.007","DOIUrl":"10.1016/j.jccase.2025.02.007","url":null,"abstract":"<div><div>Triptans, commonly used for migraine treatment, induce vasoconstriction by activating 5HT1B/1D and 5HT1F receptors. Although several cases of coronary vasospasm associated with sumatriptan have been reported, few describe life-threatening arrhythmias and cardiac arrest. We present a case of fatal coronary vasospasm caused by triptan use in a young patient with minimal cardiovascular risk. A 30-year-old man with morbid obesity was admitted for unstable angina. He reported chest pain episodes starting one month earlier, coinciding with the initiation of oral sumatriptan for migraines. On the second day of hospitalization, he took 100 mg of sumatriptan. Ninety minutes later, he developed recurrent chest pain that progressed to cardiac arrest caused by a ventricular arrhythmia storm. Left anterior descending artery vasospasm with ST-segment elevation myocardial infarction was identified, and intracoronary nitrate and adenosine relieved the spasm. However, resuscitation was ineffective due to morbid obesity, leaving the patient in profound cardiogenic shock, followed by distributive shock from prolonged low-flow time. Despite maximal hemodynamic support, he succumbed to multiorgan failure. This case highlights the potentially fatal cardiovascular risk of triptans, even in low-risk patients, and underscores the need to discontinue triptans at the first sign of chest pain.</div></div><div><h3>Learning objectives</h3><div>Coronary vasospasm, a rare but severe complication of triptans, can lead to life-threatening arrhythmias and cardiac arrest, even in patients with low cardiovascular risk. Diagnosing vasospasm is challenging, as angiograms may appear normal during symptom-free intervals. This case highlights the need for heightened clinical suspicion when chest pain follows triptan use, emphasizing the importance of recognizing this risk to ensure prompt management and avoid fatal outcomes.</div></div>","PeriodicalId":52092,"journal":{"name":"Journal of Cardiology Cases","volume":"31 6","pages":"Pages 170-173"},"PeriodicalIF":0.0,"publicationDate":"2025-06-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"144184701","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}

{"title":"Usefulness of exercise stress echocardiography in a patient with unilateral pulmonary branch stenosis","authors":"Ryohei Yokoyama MD ,&nbsp;Yoshihiko Kodama MD, PhD ,&nbsp;Kazunari Takamura MD ,&nbsp;Masako Takahashi MD ,&nbsp;Miyo Tanaka MT ,&nbsp;Nozomi Watanabe MD, PhD, FJCC ,&nbsp;Hiroshi Moritake MD, PhD","doi":"10.1016/j.jccase.2025.02.003","DOIUrl":"10.1016/j.jccase.2025.02.003","url":null,"abstract":"<div><div>Exercise stress echocardiography (ESE) is a feasible and valuable tool for evaluating subclinical pulmonary hypertension (PH). However, its utility in patients with unilateral pulmonary branch stenosis remains unclear. We present a case involving a 17-year-old patient with left pulmonary branch stenosis who exhibited exercise-induced PH in the contralateral pulmonary artery as detected by ESE. Standard echocardiography was unable to visualize the left pulmonary artery clearly; therefore, computed tomography was performed, revealing a left pulmonary branch stenosis with a minimum diameter of 4.2 mm. Resting echocardiography showed a pressure gradient of 17 mmHg, calculated using the tricuspid regurgitant velocity. During ESE with a prone ergometer, the slope of the mean pulmonary arterial pressure to systemic cardiac output was 3.1 mmHg/L/min, meeting the diagnostic criteria for exercise-induced PH. The patient underwent stent implantation to treat the left pulmonary branch stenosis. Follow-up ESE demonstrated improvement, with the slope of the mean pulmonary arterial pressure to systemic cardiac output decreasing to 1.5 mmHg/L/min. These findings underscore that ESE is both feasible and effective for assessing subclinical unilateral pulmonary branch stenosis.</div></div><div><h3>Learning objective</h3><div>Patients with congenital unilateral peripheral branch pulmonary artery stenosis usually do not have pulmonary hypertension at rest, and identifying patients who require treatment is challenging. Exercise stress echocardiography can detect latent pulmonary hypertension of the contralateral pulmonary artery in some patients, providing valuable insights for determining treatment indications and evaluating the efficacy of catheter interventions for the stenotic lesion.</div></div>","PeriodicalId":52092,"journal":{"name":"Journal of Cardiology Cases","volume":"31 6","pages":"Pages 155-157"},"PeriodicalIF":0.0,"publicationDate":"2025-06-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"144184689","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}

{"title":"A case with combination of pyoderma gangrenosum and cardiac sarcoidosis successfully implanted with a dual-chamber pacemaker","authors":"Toshihiko Akasaka MD, PhD ,&nbsp;Aiko Takami MD ,&nbsp;Ryo Higuchi MD ,&nbsp;Kazuyoshi Ogura MD, PhD ,&nbsp;Hiroshi Nasu MD ,&nbsp;Kazuhiro Yamamoto MD, PhD, FJCC","doi":"10.1016/j.jccase.2025.02.005","DOIUrl":"10.1016/j.jccase.2025.02.005","url":null,"abstract":"<div><div>A patient with ulcerated skin lesions of unknown etiology on his left leg underwent pacemaker implantation for atrioventricular block caused by cardiac sarcoidosis (CS). After initiation of corticosteroid for CS, his skin lesions improved. The lesions were diagnosed as pyoderma gangrenosum (PG) from the findings of a skin biopsy and the reaction to corticosteroid. PG causes skin necrosis associated with immune system disorders after slight stimulation. Although we worried about wound healing after pacemaker implantation, the wound healed well under the corticosteroid treatment. This is the first report of successful pacemaker implantation in a patient with both PG and CS.</div></div><div><h3>Learning objective</h3><div>Conventional transvenous pacemakers can be implanted for pyoderma gangrenosum patients by early initiation of corticosteroid.</div></div>","PeriodicalId":52092,"journal":{"name":"Journal of Cardiology Cases","volume":"31 6","pages":"Pages 166-169"},"PeriodicalIF":0.0,"publicationDate":"2025-06-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"144184700","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}

{"title":"","authors":"Takuya Nishimura MD,&nbsp;Yuri Ochi MD,&nbsp;Naoki Arima MD,&nbsp;Kenta Sugiura MD,&nbsp;Takayoshi Hirota MD,&nbsp;Toru Kubo MD, FJCC,&nbsp;Naohito Yamasaki MD,&nbsp;Hiroaki Kitaoka MD, FJCC","doi":"10.1016/j.jccase.2025.02.009","DOIUrl":"10.1016/j.jccase.2025.02.009","url":null,"abstract":"","PeriodicalId":52092,"journal":{"name":"Journal of Cardiology Cases","volume":"31 5","pages":"Page 153"},"PeriodicalIF":0.0,"publicationDate":"2025-05-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"143892213","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}