Annals of Tropical Paediatrics最新文献

{"title":"Annals of Tropical Paediatrics will become Paediatrics and International Child Health from 2012","authors":"J. Coulter","doi":"10.1179/146532811X13006353133957","DOIUrl":"https://doi.org/10.1179/146532811X13006353133957","url":null,"abstract":"The title of Annals of Tropical Paediatrics: International Child Health will change to Paediatrics and International Child Health with effect from February 2012 with publication of the first issue of Volume 32. A new editorial board has been appointed and members were published in the February 2011 issue. The abbreviated title will be Paediatr Int Child Health. The rationale for the change is as follows. Annals of Tropical Paediatrics publishes papers on topics relevant to developing countries from a wide variety of countries throughout the world, mainly developing or low-income countries, but also from industrialised settings, on subjects relevant to developing countries. Thus, the word ‘tropical’ is no longer considered appropriate to publications from many of those countries which do not have a tropical climate and therefore do not have tropical diseases in the strictest sense. The new title embraces the key elements of the papers we publish: paediatrics, which encompasses clinical and laboratory aspects, and international child health which covers the wide spectrum of topics in community child health. Notwithstanding the majority of their populations being poor, some developing counties are now using advanced medical treatments and surgical and medical procedures at a level seen in industrialised countries. Submissions relating to these are considered for publication as long as they have a broad relevance to practice in low-income countries. However, authors of highly technical papers or reports of very expensive investigations or treatments are usually advised to consider submitting to more specialised journals of paediatrics. Since 2005, the journal has published invited reviews on a variety of subjects at a state-of-the-art level. We are very grateful to these authors for the quality of their work and the support they give the journal. Unsolicited and systematic reviews are also welcome, and those which receive favourable peer review are published. In the past, there have been occasional commentaries on papers in current issues. We now try to publish commentaries regularly, depending, of course, on receipt of papers appropriate for comment. Original articles of high quality are welcome, particularly those which report scientific research and prospective clinical and community studies. Case reports are also encouraged but these must be on unusual conditions or complications, not mere repetition of the known topics which are readily available in textbooks of paediatrics. To be of value to readers, the discussion must be of a high quality based on a substantial review of the literature. It is hoped that the new title will attract an even broader spectrum of paediatrics than the journal has published hitherto. Paediatrics and International Child Health will retain the ethos of the journal when it was first published in 1981 under the editorship of the founder, Ralph Hendrickse: to provide an opportunity for paediatricians working in developing","PeriodicalId":50759,"journal":{"name":"Annals of Tropical Paediatrics","volume":"31 1","pages":"189 - 190"},"PeriodicalIF":0.0,"publicationDate":"2011-08-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://sci-hub-pdf.com/10.1179/146532811X13006353133957","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"65721861","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}

{"title":"Delayed diagnosis of Kawasaki disease: risk factors and outcome of treatment.","authors":"R Sittiwangkul,&nbsp;Y Pongprot,&nbsp;S Silvilairat,&nbsp;C Phornphutkul","doi":"10.1179/1465328111Y.0000000005","DOIUrl":"https://doi.org/10.1179/1465328111Y.0000000005","url":null,"abstract":"<p><strong>Background: </strong>Kawasaki disease (KD) is associated with a high incidence of coronary artery aneurysms in untreated children. Treatment with intravenous immunoglobulin (IVIG) within the 1st 10 days of illness reduces by approximately fivefold the prevalence of coronary artery abnormalities (CAA). Data regarding delayed diagnosis of KD in Thailand have not been reported in the literature.</p><p><strong>Aims: </strong>To determine the prevalence, risk factors and outcome of delayed diagnosis of KD in Thai patients.</p><p><strong>Methods: </strong>We retrospectively reviewed the medical records of patients at Chiang Mai University Hospital diagnosed as KD during 2000-2008. Patients were classified into two groups: Group I were diagnosed ≤10 days of fever and Group II were diagnosed >10 days of fever.</p><p><strong>Results: </strong>Of 170 patients, 150 were in Group I [mean (SD) fever 7 (1·45) d] and 20 (11·7%) in Group II [mean (SD) fever 15 (4) d]. There were no statistical differences between the two groups in age, gender, number of KD clinical manifestations or laboratory results, except that Group II were of lower weight (p = 0·01). Group II were younger (p = 0·09) and had more incomplete criteria (p = 0·09) but the differences were not statistically significant. Group II had a higher incidence of CAA (75% vs 19%) (p<0·001), more severe CAA and more resistant cases (31·2% vs 9·5%) (p = 0·04).</p><p><strong>Conclusion: </strong>Patients with delayed diagnosis of KD have a higher risk of developing CAA and of a more severe outcome for coronary artery disease. Education is needed to make healthcare providers and physicians more aware of KD, especially in small children or those with incomplete KD.</p>","PeriodicalId":50759,"journal":{"name":"Annals of Tropical Paediatrics","volume":"31 2","pages":"109-14"},"PeriodicalIF":0.0,"publicationDate":"2011-01-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://sci-hub-pdf.com/10.1179/1465328111Y.0000000005","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"30189735","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}

{"title":"Deafness: malaria as a forgotten cause.","authors":"S Z Zhao,&nbsp;I J Mackenzie","doi":"10.1179/146532811X12925735813724","DOIUrl":"https://doi.org/10.1179/146532811X12925735813724","url":null,"abstract":"<p><strong>Background: </strong>Ototoxicity from antimalarials is a well publicised cause of deafness and a great deal of time and resources are spent assessing it in relation to new drugs. The effect of the malaria parasite itself on hearing is, however, poorly documented and most evidence is anecdotal. This paper aims to collate existing evidence of this association.</p><p><strong>Methods: </strong>Two systematic literature searches were performed on Ovid Medline, first for 'malaria' and 'hearing loss' or 'hearing impairment' or 'deafness', and secondly for 'cerebral malaria' and 'neurologic' or 'neurological' or 'neurocognitive sequelae'. The articles were then individually studied for relevance.</p><p><strong>Results: </strong>Malaria has been implicated as a rare cause of hearing loss in various studies, but recommendations and hypotheses have not been taken seriously or investigated. Searches also returned numerous studies of neurological sequelae after cerebral malaria, a small proportion of which observed hearing impairments on follow-up. However, no attempt was made to distinguish between treatment and disease as the cause. A few antimalarial drug trials which assessed hearing before treatment found unexplained hearing loss which improved with elimination of the parasite.</p><p><strong>Conclusion: </strong>Evidence from this review suggests that the falciparum parasite is a potential cause of hearing loss. Malaria is a disease of such high prevalence that even if only a small proportion of survivors develop this impairment the effects on children's education could be detrimental. More attention should be focussed on investigating this association as the clinical and pathophysiological implications are potentially considerable.</p>","PeriodicalId":50759,"journal":{"name":"Annals of Tropical Paediatrics","volume":"31 1","pages":"1-10"},"PeriodicalIF":0.0,"publicationDate":"2011-01-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://sci-hub-pdf.com/10.1179/146532811X12925735813724","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"29621467","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}

{"title":"Crimean-Congo haemorrhagic fever: peritoneal and pleural effusion.","authors":"G Şensoy,&nbsp;G Çaltepe Dinler,&nbsp;G Kalkan,&nbsp;A Ateş,&nbsp;N Belet,&nbsp;D Albayrak","doi":"10.1179/1465328111Y.0000000011","DOIUrl":"https://doi.org/10.1179/1465328111Y.0000000011","url":null,"abstract":"<p><p>In Crimean-Congo haemorrhagic fever (CCHF), haemorrhagic manifestations are usually petechiae and ecchymoses on mucous membranes and skin. Rarely, there is bleeding from the nose, gingiva, gastro-intestinal tract, genito-urinary tract, brain and lungs. A 13-year-old boy with CCHF presented with gastro-intestinal bleeding and developed peritoneal and pleural effusion. He made a complete recovery with supportive treatment and ribavirin, without requiring chest or peritoneal fluid drainage. To our knowledge, this is the first report of CCHF associated with peritoneal and pleural fluid.</p>","PeriodicalId":50759,"journal":{"name":"Annals of Tropical Paediatrics","volume":"31 2","pages":"169-72"},"PeriodicalIF":0.0,"publicationDate":"2011-01-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://sci-hub-pdf.com/10.1179/1465328111Y.0000000011","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"29885580","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}

{"title":"Effect of cryptosporidial and giardial diarrhoea on social maturity, intelligence and physical growth in children in a semi-urban slum in south India.","authors":"S S R Ajjampur,&nbsp;B Koshy,&nbsp;M Venkataramani,&nbsp;R Sarkar,&nbsp;A A Joseph,&nbsp;K S Jacob,&nbsp;H Ward,&nbsp;G Kang","doi":"10.1179/1465328111Y.0000000003","DOIUrl":"https://doi.org/10.1179/1465328111Y.0000000003","url":null,"abstract":"<p><strong>Background: </strong>Early childhood diarrhoea is a major cause of infant morbidity and mortality in developing countries. Recurrent and persistent diarrhoea affect growth and cognition in children as young as 6 years.</p><p><strong>Objectives: </strong>To evaluate the effect of early childhood cryptosporidial and giardial diarrhoea on growth and development in children in a semi-urban slum in India. This is the first report of such assessment at 3 years of age.</p><p><strong>Methods: </strong>This study was undertaken on 116 children who were part of an ongoing birth cohort study (n=452) of rotaviral and cryptosporidial diarrhoea between June and December 2005. Social quotients (SQ) assessed by the Vineland Social Maturity Scale, intelligence quotients (IQ) assessed by the Seguin Form Board Test, physical growth parameters and sociodemographic data in 84 children with a history of cryptosporidial or giardial diarrhoea were compared with those of 32 without diarrhoea.</p><p><strong>Results: </strong>Children with a past history of giardial diarrhoea showed a trend towards lower SQ (p=0.09) and had significantly lower IQ (p=0.04) and increased wasting (p=0.04). Cryptosporidial diarrhoea was not associated with poor IQ, SQ or physical growth.</p><p><strong>Conclusion: </strong>This study demonstrates the long-term effect of protozoan diarrhoea, especially that caused by giardia, on both intelligence and physical growth in Indian children as early as 3 years of age and re-inforces the need for early detection and prevention of early childhood protozoan diarrhoea.</p>","PeriodicalId":50759,"journal":{"name":"Annals of Tropical Paediatrics","volume":"31 3","pages":"205-12"},"PeriodicalIF":0.0,"publicationDate":"2011-01-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://sci-hub-pdf.com/10.1179/1465328111Y.0000000003","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"30025006","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}

{"title":"Longitudinal study of vitamin D status in the 1st 6 months of life.","authors":"H Narchi,&nbsp;J Kochiyil,&nbsp;R Zayed,&nbsp;W Abdulrazzak,&nbsp;M Agarwal","doi":"10.1179/1465328111Y.0000000017","DOIUrl":"https://doi.org/10.1179/1465328111Y.0000000017","url":null,"abstract":"<p><strong>Unlabelled: </strong>Although hypovitaminosis D has been reported in the neonatal period and infancy, there is currently little information on the longitudinal changes in vitamin D status throughout early infancy.</p><p><strong>Aim: </strong>To estimate, in Al Ain, UAE, the prevalence of vitamin D deficiency and longitudinal changes and risk factors in infants between birth and 6 months of age.</p><p><strong>Methods: </strong>Serum 25-OH-vitamin-D levels were measured after birth and 6 months later in 27 infants of mothers of Middle Eastern or Asian origin who were pregnant between the months of September and November 2007.</p><p><strong>Results: </strong>At delivery, mean (SD) maternal serum 25-OH-vitamin-D level was 35.5 nmol/L (24.7); five mothers (22%, 95% CI 0.7-43) had adequate serum levels (>50 mmol/L), 11 (48%, 95% CI 27-70) insufficient levels (25-50 nmol/L) and seven (30%, 95% CI 13-53) deficient (<25 nmol/L) levels. Serum 25-OH-vitamin-D levels were adequate in eight infants (30%, CI 14-50%), insufficient in 13 (48%, CI 28-60%) and deficient in six (22%, CI 8.5-42%). Despite recommendations, none had received any vitamin D supplementation since birth. Despite the high prevalence of hypovitaminosis D at birth and the lack of pharmacological supplementation, the number of infants with adequate levels at 6 months of age rose to 20 (87%, CI 66-97%). No infant had deficiency (CI 0-21%) and three (13%, CI 27-33%) had insufficiency. Adequate levels were detected in four infants who were partially breastfed [mean (SD) 108.5 (20.7) nmol/L] and in only 84% of the 19 exclusively breastfed infants [mean (SD) 96.2 (44.5) nmol/L] but the difference was not statistically significant. Although serum levels improved at 6 months, it occurred more slowly in exclusively breastfed infants.</p><p><strong>Conclusion: </strong>In the absence of vitamin D supplementation, guidelines for vitamin D supplementation in infancy still need to be followed because the mechanisms for normalisation are not clearly understood.</p>","PeriodicalId":50759,"journal":{"name":"Annals of Tropical Paediatrics","volume":"31 3","pages":"225-30"},"PeriodicalIF":0.0,"publicationDate":"2011-01-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://sci-hub-pdf.com/10.1179/1465328111Y.0000000017","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"30025011","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}

{"title":"Bilateral symmetrical facial swelling owing to tuberculous gummas.","authors":"S Shukla,&nbsp;S Singh,&nbsp;V Puri,&nbsp;D Verma,&nbsp;L Jain","doi":"10.1179/1465328111Y.0000000034","DOIUrl":"https://doi.org/10.1179/1465328111Y.0000000034","url":null,"abstract":"<p><p>Metastatic tuberculous abscesses and gummas are unusual forms of cutaneous tuberculosis. They result from haematogeneous spread of the mycobacterium from a primary focus during a period of impaired immunity. A 5-year-old boy is reported who presented with spinal tuberculosis and bilateral subcutaneous swelling of the cheeks owing to tuberculous gummas.</p>","PeriodicalId":50759,"journal":{"name":"Annals of Tropical Paediatrics","volume":"31 4","pages":"363-5"},"PeriodicalIF":0.0,"publicationDate":"2011-01-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://sci-hub-pdf.com/10.1179/1465328111Y.0000000034","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"40117807","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}

{"title":"Perinatal tuberculosis: two unusual cases.","authors":"S Basu,&nbsp;A Kumar,&nbsp;B K Das","doi":"10.1179/1465328110Y.0000000009","DOIUrl":"https://doi.org/10.1179/1465328110Y.0000000009","url":null,"abstract":"<p><p>Two immunocompromised infants with perinatal tuberculosis are reported. Both presented with bilateral miliary mottling of the lungs. The first was a 4-month-old boy with a history of prolonged administration of prednisolone. He had tuberculous meningitis. Diagnosis was confirmed by PCR in serum and CSF. His mother had tuberculous endometritis. The second was a 1-month-old boy with perinatal HIV infection. Diagnosis was made by PCR in serum. Both parents were HIV-infected and had open pulmonary tuberculosis.</p>","PeriodicalId":50759,"journal":{"name":"Annals of Tropical Paediatrics","volume":"31 1","pages":"81-6"},"PeriodicalIF":0.0,"publicationDate":"2011-01-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://sci-hub-pdf.com/10.1179/1465328110Y.0000000009","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"29621855","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}

{"title":"Enteric protozoa and human potential.","authors":"R L Guerrant,&nbsp;R B Oria,&nbsp;S R Moore,&nbsp;R Scharf,&nbsp;A A M Lima","doi":"10.1179/146532811X13006353133911","DOIUrl":"https://doi.org/10.1179/146532811X13006353133911","url":null,"abstract":"The paper by Ajjampur et al. in this issue confirms and extends several previous studies from developing countries which show alarming and robust associations between enteric infections, child growth and neuro-development.1 Their findings that symptomatic giardiasis and diarrhoea predicted wasting and poor performance on a test of cognition (and perhaps lower ‘social quotient’) are profoundly important and add to the increasing evidence that early childhood enteric infections have long-term growth and developmental consequences which must be addressed if children are to achieve their full potential.2–10 \u0000 \u0000Indeed, evidence is mounting that intestinal infections might have harmful effects not only on childhood stunting — itself an important predictor of IQ — but also on cognitive development which might be independent of the effect of diarrhoea on malnutrition. In an analysis of IQ data from 184 countries, Eppig et al. have recently shown that infectious diseases were the most powerful predictor of mean national IQ, even when temperature, GDP per capita and educational indicators were taken into account.11 Furthermore, although ‘nutritional stress’ correlated with national IQ, even this correlation was lost when the effects of infectious diseases were removed. Although this analysis did not assess enteric infections or diarrhoea per se, their specific suggestion that diarrhoeal diseases may lead infectious diseases in causing lifelong detrimental effects on brain development is strongly supported by original reports from our group over the last decade.2–7 As with Ajjampur and colleagues’ data on the correlation between wasting and ‘social quotients’, our data suggest that stunting (height-for-age Z-score ≤2 SD below the median) at age 2 years may be a surrogate marker of heavy early childhood diarrhoea burdens and a key predictor of later cognitive development. Further follow-up studies of national HAZ-2, wasting and IQ are needed. \u0000 \u0000Similar to earlier findings in children with intestinal helminthiases by Stephenson et al. and Nokes et al., 12–14 we and others have reported that early childhood diarrhoea and Cryptosporidium infections have lasting effects on child growth and cognitive development.2–5,8,9 Furthermore, similar to the results of Eppig et al., we find that diarrhoea, independent of its significant effects on growth, impairs cognitive development (Pinkerton et al, submitted). Clearly, more studies of the long-term impact of major enteric infections, with or without overt diarrhoea, are urgently needed to assess adequately the magnitude and mechanisms by which these devastating developmental impairments occur. \u0000 \u0000We and others have identified stunting as one of the best surrogate markers of the deleterious effects of early childhood diarrhoeal illness; the decline in cognitive function is apparent 4–8 years later.2,3,5,15 In Ajjampur et al.’s study, the lack of significant correlation between cognitive function and stunting when cumula","PeriodicalId":50759,"journal":{"name":"Annals of Tropical Paediatrics","volume":"31 3","pages":"201-3"},"PeriodicalIF":0.0,"publicationDate":"2011-01-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://sci-hub-pdf.com/10.1179/146532811X13006353133911","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"30025004","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"OA","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}

{"title":"Evaluation of glomerular and tubular renal function in neonates with birth asphyxia.","authors":"S Kaur,&nbsp;S Jain,&nbsp;A Saha,&nbsp;D Chawla,&nbsp;V R Parmar,&nbsp;S Basu,&nbsp;J Kaur","doi":"10.1179/146532811X12925735813922","DOIUrl":"https://doi.org/10.1179/146532811X12925735813922","url":null,"abstract":"<p><strong>Background: </strong>Acute kidney injury (AKI) is one of the commonest manifestations of end-organ damage associated with birth asphyxia.</p><p><strong>Objective: </strong>To evaluate glomerular and tubular dysfunction in neonates with moderate to severe birth asphyxia.</p><p><strong>Design: </strong>Prospective cohort study.</p><p><strong>Setting: </strong>Neonatal unit of a teaching hospital.</p><p><strong>Methods: </strong>Subjects were inborn neonates of ≥34 completed weeks of gestation with an APGAR score <7 at 1 min after birth. Renal function tests including serum electrolytes were measured daily until 96 hrs of life along with urinary output. Fractional excretion of sodium (FeNa), renal failure index (RFI), urinary myoglobin and creatinine clearance (CrCl) were calculated using timed urine collection. Staging of AKI was undertaken using Acute Kidney Injury Network criteria (AKIN). PRIMARY OUTCOME MEASUREMENT: Recovery of glomerular function.</p><p><strong>Results: </strong>A total of 2196 neonates were born during the study period (September 2006 to April 2007), 44 of whom met the inclusion criteria. Data from 36 neonates were available for final analysis. AKI developed in 9·1% (1/11) infants with moderate asphyxia and 56·0% (12/25) infants with severe asphyxia, making a total incidence of 41·7%. AKI persisted in 16·6% neonates at 96 hours of life. Ten neonates (27·7%) had serum creatinine levels >1·5 mg/dl. In neonates with AKI, tubular function (Fe Na, RFI, urinary myoglobin) was significantly deranged until 72-96 hrs of life. One infant died and one who was critically ill was discharged against medical advice; both had AKI.</p><p><strong>Conclusion: </strong>It is feasible to use AKIN staging for evaluating AKI in neonates with birth asphyxia.</p>","PeriodicalId":50759,"journal":{"name":"Annals of Tropical Paediatrics","volume":"31 2","pages":"129-34"},"PeriodicalIF":0.0,"publicationDate":"2011-01-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://sci-hub-pdf.com/10.1179/146532811X12925735813922","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"30190283","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}