Journal of Pediatric Neurosciences最新文献

筛选
英文 中文
Leukoencephalopathy with Calcifications and Cysts in a Child with Progressive Hemiparesis-A Case Report. 儿童进行性偏瘫伴钙化和囊肿的脑白质病1例报告。
IF 0.5
Journal of Pediatric Neurosciences Pub Date : 2021-10-01 DOI: 10.4103/jpn.JPN_113_20
Minhaj Shaikh, Sarbesh Tiwari, Taruna Yadav, Pawan K Garg, Pushpinder S Khera
{"title":"Leukoencephalopathy with Calcifications and Cysts in a Child with Progressive Hemiparesis-A Case Report.","authors":"Minhaj Shaikh,&nbsp;Sarbesh Tiwari,&nbsp;Taruna Yadav,&nbsp;Pawan K Garg,&nbsp;Pushpinder S Khera","doi":"10.4103/jpn.JPN_113_20","DOIUrl":"https://doi.org/10.4103/jpn.JPN_113_20","url":null,"abstract":"<p><p>With the advent of modern neuroimaging, the imaging features of various leukoencephalopathies have been unraveled in the past two decades. Leukoencephalopathy with calcifications and cysts (LCC) is one such rare autosomal recessive disorder with marked clinical heterogeneity and a striking but characteristic imaging appearance-diffuse white matter changes, intraparenchymal cysts, and calcifications. The calcifications in LCC are characteristically nodular, dense, bulky, and predominantly located in gray nuclei of the central brain (basal ganglia, thalami) and cerebellum (dentate nuclei). We describe a case of a 9-year-old boy with progressive left hemiparesis and seizures, which on imaging showed characteristic features of LCC. We further review the neuroimaging features of LCC and its differential diagnoses.</p>","PeriodicalId":46746,"journal":{"name":"Journal of Pediatric Neurosciences","volume":"16 4","pages":"277-280"},"PeriodicalIF":0.5,"publicationDate":"2021-10-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://www.ncbi.nlm.nih.gov/pmc/articles/PMC9757512/pdf/","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"10749745","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"OA","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
引用次数: 0
Clinical Phenotype of FASTKD2 Mutation. FASTKD2突变的临床表型。
IF 0.5
Journal of Pediatric Neurosciences Pub Date : 2021-10-01 DOI: 10.4103/jpn.JPN_199_20
Ritesh Shah, Seema Balasubramaniam
{"title":"Clinical Phenotype of FASTKD2 Mutation.","authors":"Ritesh Shah,&nbsp;Seema Balasubramaniam","doi":"10.4103/jpn.JPN_199_20","DOIUrl":"https://doi.org/10.4103/jpn.JPN_199_20","url":null,"abstract":"<p><p>Mitochondrial disorders (MIDs) are frequently multisystemic in nature and cause significant morbidity and mortality. Accurate assessment of mitochondrial disease prevalence has been difficult in the past. Primary MIDs are due to mutations in mitochondrial DNA (mtDNA) or nuclear DNA (nDNA)-located genes. Here we report cases of two siblings who presented to the pediatric emergency department with status epilepticus. Initially, the elder sibling was treated for metabolic encephalopathy and viral encephalitis, during his admission to the hospital. On treatment with multiple antiepileptic drugs, the status epilepticus subsided. A provisional diagnosis of mitochondrial encephalomyopathy with lactic acidosis and stroke-like episodes was made. Magnetic resonance imaging showed diffusion restriction in the left temporal lobe, insular cortex, and left lentiform nucleus, which completely resolved on follow-up after 1 month. His sudden demise in May 2019 due to status epilepticus, and a similar case presentation in his younger sibling, prompted us to do a genetic analysis test. The exome sequence revealed FASTKD2 mutation, a rare variant. This case report helps in increasing the awareness among the clinicians about the clinical presentation of FASTKD2 mutation case.</p>","PeriodicalId":46746,"journal":{"name":"Journal of Pediatric Neurosciences","volume":"16 4","pages":"319-322"},"PeriodicalIF":0.5,"publicationDate":"2021-10-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://www.ncbi.nlm.nih.gov/pmc/articles/PMC9757524/pdf/","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"10400468","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"OA","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
引用次数: 0
Pediatric Skull Base Meningioma: Case Report and Review of Literature. 小儿颅底脑膜瘤:病例报告及文献回顾。
IF 0.5
Journal of Pediatric Neurosciences Pub Date : 2021-10-01 DOI: 10.4103/jpn.JPN_264_20
N K Venkataramana, Shailesh A V Rao, B S Sridutt, N Krishna Chaitanya
{"title":"Pediatric Skull Base Meningioma: Case Report and Review of Literature.","authors":"N K Venkataramana,&nbsp;Shailesh A V Rao,&nbsp;B S Sridutt,&nbsp;N Krishna Chaitanya","doi":"10.4103/jpn.JPN_264_20","DOIUrl":"https://doi.org/10.4103/jpn.JPN_264_20","url":null,"abstract":"<p><p>Meningiomas are frequent in adults but rare in children, though they are not uncommon. They are known to occur in the pediatric population in all age groups. In children, meningiomas are usually known to be large, cystic, and even aggressive. Among them, skull base meningiomas constitute a distinct entity. Meningiomas arising from the skull base in those younger than the age of two years are rarely reported in literature. We report one such skull base meningioma, involving the middle and posterior cranial fossa, in a child of one year and eight months. The challenges associated in its diagnosis and management are presented.</p>","PeriodicalId":46746,"journal":{"name":"Journal of Pediatric Neurosciences","volume":"16 4","pages":"354-357"},"PeriodicalIF":0.5,"publicationDate":"2021-10-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://www.ncbi.nlm.nih.gov/pmc/articles/PMC9757519/pdf/","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"10766416","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"OA","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
引用次数: 0
Mitochondrial Ultrastructural Defects in NDUFS3-Related Disorder. ndufs3相关疾病的线粒体超微结构缺陷。
IF 0.5
Journal of Pediatric Neurosciences Pub Date : 2021-10-01 DOI: 10.4103/jpn.JPN_182_20
Debopam Samanta, Aravindhan Veerapandiyan, Thomas A Burrow, Murat Gokden
{"title":"Mitochondrial Ultrastructural Defects in NDUFS3-Related Disorder.","authors":"Debopam Samanta,&nbsp;Aravindhan Veerapandiyan,&nbsp;Thomas A Burrow,&nbsp;Murat Gokden","doi":"10.4103/jpn.JPN_182_20","DOIUrl":"https://doi.org/10.4103/jpn.JPN_182_20","url":null,"abstract":"Complex I, the largest multisubunit enzyme complex of the respiratory chain, has a vital role in the energy production of the cell, and the clinical spectrum of complex I deficiency varies from severe lactic acidosis in infants to muscle weakness in adults. Pathogenic variants of NDUFS3 (constitutes the catalytic core of the complex I) have been reported in a small number of patients with variable phenotypes. We describe a girl with a history of infantile-onset nonepileptic myoclonus, who developed myopathy at the age of 2 years. Next-generation sequencing revealed compound heterozygous for two variants in the NDUSF3 gene. The electron-microscopic study of the skeletal muscle showed an increase in the number of mitochondria inside the myofibers; mitochondria were variably enlarged with some irregularity and were aligned perpendicular to the myofibrils in a stacked-up manner. This is the first description of mitochondrial ultrastructural abnormality in an individual with NDUFS3-related disorder.","PeriodicalId":46746,"journal":{"name":"Journal of Pediatric Neurosciences","volume":"16 4","pages":"299-302"},"PeriodicalIF":0.5,"publicationDate":"2021-10-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://www.ncbi.nlm.nih.gov/pmc/articles/PMC9757518/pdf/","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"10766418","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"OA","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
引用次数: 3
Treatable Neurodegenerative Disorder: Cerebral Folate Transport Deficiency--Two Children from Southern India. 可治疗的神经退行性疾病:脑叶酸运输缺乏症——来自印度南部的两个孩子。
IF 0.5
Journal of Pediatric Neurosciences Pub Date : 2021-10-01 DOI: 10.4103/jpn.JPN_76_20
Vykuntaraju K Gowda, Balamurugan Natarajan, Varunvenkat M Srinivasan, Sanjay K Shivappa
{"title":"Treatable Neurodegenerative Disorder: Cerebral Folate Transport Deficiency--Two Children from Southern India.","authors":"Vykuntaraju K Gowda,&nbsp;Balamurugan Natarajan,&nbsp;Varunvenkat M Srinivasan,&nbsp;Sanjay K Shivappa","doi":"10.4103/jpn.JPN_76_20","DOIUrl":"https://doi.org/10.4103/jpn.JPN_76_20","url":null,"abstract":"<p><p>Cerebral folate transport deficiency results from impaired folate transport across the blood:choroid plexus:cerebrospinal fluid (CSF) barrier. This leads to low CSF 5-methyltetrahydrofolate (5MTHF), the active folate metabolite. We are reporting two children with this treatable cerebral folate transport deficiency. Case 1: Seventeen-year-old boy presented with delayed milestones followed by regression, seizures, and intention tremors. On examination child had pyramidal and cerebellar signs. Magnetic resonance imaging (MRI) of brain revealed diffuse cerebral and cerebellar atrophy. Targeted next generation sequencing revealed homozygous missense pathogenic variant in <i>FOLR1</i> gene in exon 4 c.382C>T p.R128W, confirming the diagnosis of cerebral folate deficiency. Case 2: Six-year-old male child presented with delayed milestones, myoclonic jerks and cognitive regression from 3 years of age. Child had microcephaly with ataxia. Computed tomography (CT) of brain revealed multifocal calcifications. MRI brain revealed cerebellar atrophy with hyperintense T2 signal changes in the subcortical white matter of frontal and temporal lobes. Genetic testing revealed homozygous variant (c.493+2_493+6delTGAGG) in intron 4 of the <i>FOLR1</i> gene which is a novel pathogenic variant. Both children started on folinic acid and there was a significant improvement in development, behavior, ataxia, and decrease in seizure frequency. In conclusion, cerebral folate transport deficiency should be suspected in every child with global developmental delay, epilepsy, ataxia and neuroimaging showing cerebellar atrophy and calcification. Response to folinic acid supplementation is partial if diagnosed late and treatment initiation is delayed.</p>","PeriodicalId":46746,"journal":{"name":"Journal of Pediatric Neurosciences","volume":"16 4","pages":"273-276"},"PeriodicalIF":0.5,"publicationDate":"2021-10-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://www.ncbi.nlm.nih.gov/pmc/articles/PMC9757528/pdf/","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"10766420","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"OA","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
引用次数: 0
The Dysfunctional Gangway: SZT2-associated Epilepsy with Thick Corpus Callosum. 功能失调的舷梯:与厚胼胝体szt2相关的癫痫。
IF 0.5
Journal of Pediatric Neurosciences Pub Date : 2021-10-01 DOI: 10.4103/jpn.JPN_128_20
Ajith Cherian, Kalikavil Puthanveedu Divya, Harini Pavuluri, Bejoy Thomas
{"title":"The Dysfunctional Gangway: <i>SZT2</i>-associated Epilepsy with Thick Corpus Callosum.","authors":"Ajith Cherian,&nbsp;Kalikavil Puthanveedu Divya,&nbsp;Harini Pavuluri,&nbsp;Bejoy Thomas","doi":"10.4103/jpn.JPN_128_20","DOIUrl":"https://doi.org/10.4103/jpn.JPN_128_20","url":null,"abstract":"<p><p>Mutations in seizure threshold 2 (<i>SZT2</i>) gene on chromosome 1p34.2 are an of late identified cause of epilepsy and epileptic encephalopathy. We report a 3-year-old girl who presented with developmental delay, dysmorphic facies, refractory seizures, and subsequent developmental regression. Despite significant multifocal epileptiform abnormalities on her electroencephalogram, she had a paucity of generalized discharges indicating a functional deficiency of corpus callosum inspite of its increased thickness seen on magnetic resonance imaging. Her clinical exome sequencing revealed a homozygous single base pair duplication in the <i>SZT2</i> gene that resulted in a frameshift mutation and premature truncation of the protein. Our case emphasizes the role of <i>SZT2</i> gene in the diagnostic algorithm of early childhood refractory epilepsy especially in the context of a thick yet dysfunctional corpus callosum.</p>","PeriodicalId":46746,"journal":{"name":"Journal of Pediatric Neurosciences","volume":"16 4","pages":"289-292"},"PeriodicalIF":0.5,"publicationDate":"2021-10-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://www.ncbi.nlm.nih.gov/pmc/articles/PMC9757505/pdf/","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"10400469","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"OA","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
引用次数: 1
Posterior Fossa Hematoma Following Minor Trauma in an Infant with Rare Combined Factor V and Factor X Deficiency. 婴儿后窝血肿合并罕见的因子V和因子X缺乏症。
IF 0.5
Journal of Pediatric Neurosciences Pub Date : 2021-10-01 DOI: 10.4103/jpn.JPN_172_20
Indrajit Rana, Laxmi Narayan Tripathy
{"title":"Posterior Fossa Hematoma Following Minor Trauma in an Infant with Rare Combined Factor V and Factor X Deficiency.","authors":"Indrajit Rana,&nbsp;Laxmi Narayan Tripathy","doi":"10.4103/jpn.JPN_172_20","DOIUrl":"https://doi.org/10.4103/jpn.JPN_172_20","url":null,"abstract":"<p><p>Congenital combined deficiency of factor V and factor X deficiency is extremely rare. We report this for the first time in literature in an infant who developed acute subdural hematoma in posterior fossa leading to hydrocephalus.</p>","PeriodicalId":46746,"journal":{"name":"Journal of Pediatric Neurosciences","volume":"16 4","pages":"296-298"},"PeriodicalIF":0.5,"publicationDate":"2021-10-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://www.ncbi.nlm.nih.gov/pmc/articles/PMC9757509/pdf/","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"10400470","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"OA","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
引用次数: 0
Case Report of Congenital Kyphoscoliosis with Myotonic Dystrophy Type 1: Perioperative and Anesthetic Considerations. 先天性脊柱后凸伴1型肌强直性营养不良病例报告:围手术期及麻醉考虑。
IF 0.5
Journal of Pediatric Neurosciences Pub Date : 2021-10-01 DOI: 10.4103/jpn.JPN_119_20
Arpit Agrawal, Tanvi Dhawale, Varinder Kaur, Gouri Rao Passi
{"title":"Case Report of Congenital Kyphoscoliosis with Myotonic Dystrophy Type 1: Perioperative and Anesthetic Considerations.","authors":"Arpit Agrawal,&nbsp;Tanvi Dhawale,&nbsp;Varinder Kaur,&nbsp;Gouri Rao Passi","doi":"10.4103/jpn.JPN_119_20","DOIUrl":"https://doi.org/10.4103/jpn.JPN_119_20","url":null,"abstract":"<p><p>Congenital kyphoscoliosis associated with myotonic dystrophy type 1 (DM 1) is a rare combination and carries challenges of surgical as well as anesthetic intervention. The presence of muscular dystrophy may accelerate progression of scoliosis thus requiring surgical treatment. The objective of this case report was to discuss the perioperative anesthetic and surgical management of such cases.</p>","PeriodicalId":46746,"journal":{"name":"Journal of Pediatric Neurosciences","volume":"16 4","pages":"281-284"},"PeriodicalIF":0.5,"publicationDate":"2021-10-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://www.ncbi.nlm.nih.gov/pmc/articles/PMC9757522/pdf/","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"10392092","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"OA","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
引用次数: 0
Tracheal Tube Kinking Amidst Prone Position During Neurosurgical Procedures: An Attempt to Elucidate the Mechanism. 神经外科手术中俯卧位时气管管扭结:试图阐明其机制。
IF 0.5
Journal of Pediatric Neurosciences Pub Date : 2021-10-01 DOI: 10.4103/jpn.JPN_226_20
Nidhi Singh, Rajeev Chauhan, Rashi Sarna, Anjali Mohan
{"title":"Tracheal Tube Kinking Amidst Prone Position During Neurosurgical Procedures: An Attempt to Elucidate the Mechanism.","authors":"Nidhi Singh,&nbsp;Rajeev Chauhan,&nbsp;Rashi Sarna,&nbsp;Anjali Mohan","doi":"10.4103/jpn.JPN_226_20","DOIUrl":"https://doi.org/10.4103/jpn.JPN_226_20","url":null,"abstract":"<p><p>The tracheal tube (TT) kink during the intraoperative period is commonly observed and is worrisome once the positioning is done during neurosurgical procedures. The complications related to tube kink are more in the prone position and the mechanism of this with the neck in flexion has not been explained anywhere. We have made an attempt to elucidate the probable mechanism of this TT kink by using SOLIDWORKS 2020 {3D Computer assisted design (CAD) design software} and the precautions that can be taken to prevent perioperative catastrophe by describing a case of a pediatric patient undergoing a neurosurgical procedure while in the prone position.</p>","PeriodicalId":46746,"journal":{"name":"Journal of Pediatric Neurosciences","volume":"16 4","pages":"332-334"},"PeriodicalIF":0.5,"publicationDate":"2021-10-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://www.ncbi.nlm.nih.gov/pmc/articles/PMC9757508/pdf/","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"10400465","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"OA","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
引用次数: 0
Extramedullary Hematopoiesis Presenting with Thoracic Spinal Cord Compression in a Young Adult with Thalassemia Major: A Case Report. 髓外造血表现为胸椎脊髓压迫的年轻成人地中海贫血:1例报告。
IF 0.5
Journal of Pediatric Neurosciences Pub Date : 2021-10-01 DOI: 10.4103/jpn.JPN_183_20
Prasheelkumar Premnarayan Gupta, Salman T Shaikh, Richa Premnarayan Goyal, Deepak Premnarayan Gupta
{"title":"Extramedullary Hematopoiesis Presenting with Thoracic Spinal Cord Compression in a Young Adult with Thalassemia Major: A Case Report.","authors":"Prasheelkumar Premnarayan Gupta,&nbsp;Salman T Shaikh,&nbsp;Richa Premnarayan Goyal,&nbsp;Deepak Premnarayan Gupta","doi":"10.4103/jpn.JPN_183_20","DOIUrl":"https://doi.org/10.4103/jpn.JPN_183_20","url":null,"abstract":"<p><strong>Background: </strong>Extramedullary hematopoiesis (EMH) refers to the production of blood cellular components at sites other than the bone marrow, namely liver, spleen, and lymph nodes. The common sites associated with this condition are the liver, spleen, and lymph nodes whereas the common conditions associated with it are myelofibrosis, myelodysplasia, thalassemia, sickle cell anemia, and polycythemia vera.</p><p><strong>Case description: </strong>This report describes a young male with thalassemia major, who presented with symptomatic cord compression due to a thoracic intraspinal lesion. It was surgically excised and diagnosed as a case of EMH. The boy recovered fully and has been asymptomatic for six months now.</p><p><strong>Conclusion: </strong>The occurrence of EMH in the thoracic spine is uncommon, whereas symptomatic cord compression as a result of it is even more unusual. Magnetic resonance imaging (MRI) is the diagnostic imaging of choice and treatment options that can be offered are surgical decompression, radiotherapy, hydroxyurea, and transfusion of packed red blood cells (RBCs).</p>","PeriodicalId":46746,"journal":{"name":"Journal of Pediatric Neurosciences","volume":"16 4","pages":"303-306"},"PeriodicalIF":0.5,"publicationDate":"2021-10-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://www.ncbi.nlm.nih.gov/pmc/articles/PMC9757527/pdf/","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"10766421","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"OA","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
引用次数: 1
0
×
引用
GB/T 7714-2015
复制
MLA
复制
APA
复制
导出至
BibTeX EndNote RefMan NoteFirst NoteExpress
×
提示
您的信息不完整,为了账户安全,请先补充。
现在去补充
×
提示
您因"违规操作"
具体请查看互助需知
我知道了
×
提示
确定
请完成安全验证×
相关产品
×
本文献相关产品
联系我们:info@booksci.cn Book学术提供免费学术资源搜索服务,方便国内外学者检索中英文文献。致力于提供最便捷和优质的服务体验。 Copyright © 2023 布克学术 All rights reserved.
京ICP备2023020795号-1
ghs 京公网安备 11010802042870号
Book学术文献互助
Book学术文献互助群
群 号:604180095
Book学术官方微信