No to shinkei = Brain and nerve最新文献_第3页

[Parallel processing of motion vision]. [运动视觉的并行处理]。

No to shinkei = Brain and nerve Pub Date : 2007-01-01

Akira Midorikawa, Mitsuru Kawamura

{"title":"[Parallel processing of motion vision].","authors":"Akira Midorikawa, Mitsuru Kawamura","doi":"","DOIUrl":"","url":null,"abstract":"Several neuropsychological studies have reported dissociation between motion vision and object vision. One patient with motion blindness had a bilateral MT/V5 lesion and could see objects, but could not see the motion of the objects (Zihl et al, 1983). By contrast, some blindsight patients with primary visual cortex lesions cannot see objects but can see their movement (e.g. Riddoch, 1917). These results imply that movement vision and form vision rely on independent mechanisms. However one patient with motion blindness had controversial symptoms concerning motion vision. She could not perceive the movement of objects, although she could walk without colliding with obstacles and could catch incoming objects. It has also been reported that patients with a bilateral parietal lesion had well-preserved primary motion vision, but had problems walking and catching a ball (Vaina, 1998). Therefore, motion for vision and motion for action might have independent mechanisms. Such dissociation has also been noted in patients with Bálint syndrome. Some patients behave like a blind person but can walk and catch a ball, while other patients bump into obstacles while walking. These results indicate that the neural bases of motion vision are distinct from those of form vision and that there are subdivisions of motion vision.","PeriodicalId":19163,"journal":{"name":"No to shinkei = Brain and nerve","volume":"59 1","pages":"37-44"},"PeriodicalIF":0.0,"publicationDate":"2007-01-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"26493979","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}

引用次数: 0

[Neuropsychological approach to visual attention]. [视觉注意的神经心理学方法]。

No to shinkei = Brain and nerve Pub Date : 2007-01-01

Kyoko Suzuki

{"title":"[Neuropsychological approach to visual attention].","authors":"Kyoko Suzuki","doi":"","DOIUrl":"","url":null,"abstract":"Visual experience depends critically on visual attention, which selects a particular aspect of a visual display. Recent clinical, neuroimaging, and animal studies revealed that visual attention was divided into active and passive or top-down and bottom-up attention. Although these dichotomies are clear-cut in definition, visual attention could be modulated by many factors. Detailed observation of brain-injured patients provides with evidence for dynamic and fine control of visual attention. We observed patients with dorsal simultanagnosia and that with callosal disconnection syndrome. Patients with dorsal simultanagnosia demonstrated that extent of visual attention was dynamically changed depending on the level of visual processing. Despite the ability to read a kanji character and to describe its components correctly, a patient could not notice a component that he had just written and could not assemble individual components to make up a correct kanji character. He could point to an overlapping area of two figures. But once he started to color the overlapping area, he missed the margin of the area and colored much larger area. Another patient with dorsal simultanagnosia missed borderlines between columns of a newspaper and read letters continuously across columns. In contrast, he could point to lines between figures or meaningless patterns easily. These findings indicated that visual attention was directed automatically to meaningful characters. A patients with callosal disconnection syndrome demonstrated left unilateral spatial neglect only when he used his right hand to draw figures. Right hand movement, controlled by the left hemisphere, elicited visual attention to the right hemispace, resulting in the left unilateral spatial neglect. Thus visual attention is not simply top-down or bottom up, but is implicitly affected by the visual recognition as well as motor component of the task.","PeriodicalId":19163,"journal":{"name":"No to shinkei = Brain and nerve","volume":"59 1","pages":"23-30"},"PeriodicalIF":0.0,"publicationDate":"2007-01-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"26493976","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}

引用次数: 0

[Adult-onset citrullinemia]. (成人citrullinemia)。

No to shinkei = Brain and nerve Pub Date : 2007-01-01

Shu-ichi Ikeda

{"title":"[Adult-onset citrullinemia].","authors":"Shu-ichi Ikeda","doi":"","DOIUrl":"","url":null,"abstract":"Adult-onset citrullinemia (CTLN2) is a rare hereditary metabolic disorder characterized by highly increased concentration of citrulline and ammonia in the plasma, which is ascribed to a deficiency of argininosuccinate synthetase (ASS), one of the urea cycle enzymes mainly located in the liver. Neurological manifestation in CTLN2 patients closely resemble those of hepatic encephalopathy and in the past, most patients usually followed rapidly deteriorating clinical courses and died of severe brain edema within a few years after onset. However, in 1995 the first CTLN2 patient who was successfully treated by living-related liver transplantation was reported and since then more than 30 patients had underwent this operation in our country, showing good outcomes. No primary defect had not been found within ASS gene locus, but the causative gene of this disorder is now identified as the \"citrin gene\", which might act as a aspartate/glutamate transporter in mitochondria. Different phenotypes are seen in the individuals with a citrin deficiency: neonatal intrahepatic cholestasis, juvenile-onset chronic pancreatitis and hepatocellular carcinoma without cirrhosis can precede the appearance of CTLN2. The precise pathogenesis of this disease that includes the relationship between the mutations of citrin gene and a deficiency of hepatic ASS activity remains unclear.","PeriodicalId":19163,"journal":{"name":"No to shinkei = Brain and nerve","volume":"59 1","pages":"59-66"},"PeriodicalIF":0.0,"publicationDate":"2007-01-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"26550706","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}

引用次数: 0

[Autopsied case of 89-year-old female who was complicated with dementia 14 years from the onset of spastic gait in her total 27 years of clinical course (clinical conference)]. 【89岁女性27年临床病程中伴痴呆14年的尸检病例(临床会议)】。

No to shinkei = Brain and nerve Pub Date : 2007-01-01

Madoka Yoshimura

引用次数: 0

[Problems and plans in a new diagnosis procedure combination in neurological deseases. 2. Neurosurgical diseases]. 神经系统疾病新诊断程序组合存在的问题与对策。2. 神经外科疾病)。

No to shinkei = Brain and nerve Pub Date : 2006-12-01

Naoyuki Hattori, Yoichi Katayama

引用次数: 0

[5-ALA fluorescence guided tumor resection]. [5-ALA荧光引导肿瘤切除术]。

No to shinkei = Brain and nerve Pub Date : 2006-12-01

Fumio Yamaguchi, Akira Teramoto, Hiroshi Takahashi