表达关注:缺氧时乙酰左旋肉碱介导的神经保护归因于erk1 /2- nrf2调节的线粒体生物合成

IF 2.7 3区 医学 Q3 NEUROSCIENCES
Hippocampus Pub Date : 2025-07-01 DOI:10.1002/hipo.70022
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引用次数: 0

摘要

关注表达:K. B. Hota, S. K. Hota, O. P. Chaurasia和S. B. Singh,“缺氧时乙酰-左旋肉碱介导的神经保护归因于erk1 /2- nrf2调节的线粒体生物合成,”海马22,no。4 (2012): 723-736, https://doi.org/10.1002/hipo.20934。上述文章于2011年5月3日在线发表在Wiley在线图书馆(wileyonlinelibrary.com)上,由期刊主编和Wiley期刊有限责任公司达成协议发布。由于在图1D(i)中所示的图像中观察到重叠,因此本关注表达已达成一致。作者提供了一个解释,虽然这个数字的原始图像已经无法访问,但作者提供了一些替代数据。尽管如此,怀疑仍然存在。尽管结论不被认为会受到影响,但该期刊发表此关注表达是为了提醒读者注意对图1D(i)所做的不适当修改。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
EXPRESSION OF CONCERN: Acetyl-L-Carnitine-Mediated Neuroprotection During Hypoxia Is Attributed to ERK1/2-Nrf2-Regulated Mitochondrial Biosynthesis

EXPRESSION OF CONCERN: K. B. Hota, S. K. Hota, O. P. Chaurasia and S. B. Singh, “ Acetyl-L-Carnitine-Mediated Neuroprotection During Hypoxia Is Attributed to ERK1/2-Nrf2-Regulated Mitochondrial Biosynthesis,” Hippocampus 22, no. 4 (2012): 723736, https://doi.org/10.1002/hipo.20934.

This Expression of Concern is for the above article, published online on 03 May 2011 in Wiley Online Library (wileyonlinelibrary.com), and has been issued by agreement between the journal Editor-in-Chief and Wiley Periodicals LLC. The Expression of Concern has been agreed upon due to overlaps observed within the images presented in Figure 1D(i). The authors provided an explanation, and although the original images for this figure were no longer accessible, the authors provided some alternative data. Despite this, doubts still remained. Although the conclusions are not believed to be affected, the journal is issuing this Expression of Concern to alert readers to the inappropriate modifications made to Figure 1D(i).

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来源期刊
Hippocampus
Hippocampus 医学-神经科学
CiteScore
5.80
自引率
5.70%
发文量
79
审稿时长
3-8 weeks
期刊介绍: Hippocampus provides a forum for the exchange of current information between investigators interested in the neurobiology of the hippocampal formation and related structures. While the relationships of submitted papers to the hippocampal formation will be evaluated liberally, the substance of appropriate papers should deal with the hippocampal formation per se or with the interaction between the hippocampal formation and other brain regions. The scope of Hippocampus is wide: single and multidisciplinary experimental studies from all fields of basic science, theoretical papers, papers dealing with hippocampal preparations as models for understanding the central nervous system, and clinical studies will be considered for publication. The Editor especially encourages the submission of papers that contribute to a functional understanding of the hippocampal formation.
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