甲型流感(NS1A)非结构蛋白1显示菌株特异性dsRNA结合能力。

IF 4 2区 医学 Q2 CHEMISTRY, MEDICINAL
Veronica A Smith, Aubrey R Schall, John W Tomsho
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引用次数: 0

摘要

甲型流感非结构蛋白1 (NS1A)是感染甲型流感病毒(IAV)的宿主细胞内产生的关键毒力因子,由一个n端dsRNA结合域(RBD)和一个c端效应域(ED)组成,由一个柔性连接体连接。虽然NS1A是一种高度混杂的蛋白,具有许多细胞内功能,但其主要功能是非特异性dsRNA结合,使流感逃避我们的先天免疫系统。因此,NS1A一直被认为是一个潜在的药物靶点。该领域先前的研究已经证明了通过RBD形成二聚体以使dsRNA结合的必要性,通过ED相互作用的寡聚化进一步增强了dsRNA的结合。然而,对dsRNA结合的潜在菌株特异性影响的探索很少。大多数现有的研究仅限于A/Udorn/307/1972菌株,通常具有c端尾部缺失。在这里,我们利用荧光极化(FP)和基于荧光的电泳迁移率转移测定(fEMSA)来表征导致1918年“西班牙流感”的H1N1毒株中具有完整c端尾的NS1A的dsRNA结合特性。我们发现A/Brevig Mission/1/1918 NS1A在RBD中含有增强dsRNA结合的特异性残基。我们进一步证明,Brevig Mission和Udorn NS1A都通过ED的高度碱性c端尾部直接与dsRNA结合。这些新的结合相互作用可能导致了1918年流感大流行的致病性增加,并可能对NS1A靶向抗病毒药物产生影响。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
Nonstructural Protein 1 of Influenza A (NS1A) Demonstrates Strain-Specific dsRNA Binding Capabilities.

Nonstructural protein 1 of influenza A (NS1A) is a key virulence factor produced inside host cells infected with Influenza A Virus (IAV) and consists of an N-terminal dsRNA binding domain (RBD) and a C-terminal effector domain (ED), joined by a flexible linker. While NS1A is a highly promiscuous protein with a number of intracellular functions, its primary function is nonspecific dsRNA binding that enables influenza to evade our innate immune system. For this reason, NS1A has long been proposed as a potential drug target. Previous research in the field has demonstrated the necessity of dimer formation through the RBD to enable dsRNA binding, which is further enhanced by oligomerization through ED interactions. However, there has been minimal exploration of potential strain-specific effects on dsRNA binding. Most existing studies are limited to the A/Udorn/307/1972 strain, often with a C-terminal tail deletion. Here we utilize fluorescence polarization (FP) paired with fluorescence-based electrophoretic mobility shift assays (fEMSA) to characterize the dsRNA binding properties of NS1A from the H1N1 strain responsible for the 1918 "Spanish Flu" with an intact C-terminal tail. We show that A/Brevig Mission/1/1918 NS1A contains specific residues in the RBD that enhance dsRNA binding. We further demonstrate that both Brevig Mission and Udorn NS1A bind directly to dsRNA through the highly basic C-terminal tail of the ED. These novel binding interactions may have contributed to the increased pathogenicity of the 1918 flu pandemic and may have implications for NS1A-targeted antivirals.

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来源期刊
ACS Infectious Diseases
ACS Infectious Diseases CHEMISTRY, MEDICINALINFECTIOUS DISEASES&nb-INFECTIOUS DISEASES
CiteScore
9.70
自引率
3.80%
发文量
213
期刊介绍: ACS Infectious Diseases will be the first journal to highlight chemistry and its role in this multidisciplinary and collaborative research area. The journal will cover a diverse array of topics including, but not limited to: * Discovery and development of new antimicrobial agents — identified through target- or phenotypic-based approaches as well as compounds that induce synergy with antimicrobials. * Characterization and validation of drug target or pathways — use of single target and genome-wide knockdown and knockouts, biochemical studies, structural biology, new technologies to facilitate characterization and prioritization of potential drug targets. * Mechanism of drug resistance — fundamental research that advances our understanding of resistance; strategies to prevent resistance. * Mechanisms of action — use of genetic, metabolomic, and activity- and affinity-based protein profiling to elucidate the mechanism of action of clinical and experimental antimicrobial agents. * Host-pathogen interactions — tools for studying host-pathogen interactions, cellular biochemistry of hosts and pathogens, and molecular interactions of pathogens with host microbiota. * Small molecule vaccine adjuvants for infectious disease. * Viral and bacterial biochemistry and molecular biology.
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