神经炎症期间来自神经胶质细胞的大脑来电:细胞外囊泡的作用

IF 5.1 2区 医学 Q1 NEUROSCIENCES
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引用次数: 0

摘要

中枢神经系统(CNS)的功能依赖于神经细胞之间的连接、整合和信息交流。神经胶质细胞和神经元之间的相互作用对神经系统的发育、电传导、突触传递、神经回路的建立和脑平衡等一系列神经功能至关重要。神经胶质细胞是生理和疾病状态下维持大脑功能的关键角色。神经炎症是神经退行性疾病和感染等各种脑部疾病的常见病理过程。神经胶质细胞,包括星形胶质细胞、小胶质细胞和少突胶质细胞,是神经炎症的主要介质,因为它们可以通过释放促炎或抗炎因子来感知和应对脑部损伤。最近的证据表明,细胞外囊泡(EVs)在细胞间通信中起着关键作用,而细胞间通信是生理和病理过程的基础。特别是,神经胶质细胞衍生的细胞外囊泡在调节神经炎症方面发挥着重要作用,它们可以促进或抑制神经胶质细胞和神经元的活化,也可以促进致病蛋白的清除或传播。EVs参与神经退行性疾病,如阿尔茨海默病(AD)、帕金森病(PD)、亨廷顿病(HD)和多发性硬化症(MS),这些疾病的共同特征是神经炎症和氧化应激导致DNA损伤、本综述将剖析神经营养素水平的变化、线粒体损伤和蛋白质动态变化,展示 EVs 如何充当毒性刺激的关键细胞-细胞介质,从而传播变性和细胞死亡信号。因此,本综述侧重于小胶质细胞、星形胶质细胞、少突胶质细胞和神经炎症情况下分泌的 EVs,强调 EVs 对神经元和中枢神经系统功能的影响,同时考虑其有益和有害影响。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
Brain incoming call from glia during neuroinflammation: Roles of extracellular vesicles

The functionality of the central nervous system (CNS) relies on the connection, integration, and the exchange of information among neural cells. The crosstalk among glial cells and neurons is pivotal for a series of neural functions, such as development of the nervous system, electric conduction, synaptic transmission, neural circuit establishment, and brain homeostasis. Glial cells are crucial players in the maintenance of brain functionality in physiological and disease conditions. Neuroinflammation is a common pathological process in various brain disorders, such as neurodegenerative diseases, and infections. Glial cells, including astrocytes, microglia, and oligodendrocytes, are the main mediators of neuroinflammation, as they can sense and respond to brain insults by releasing pro-inflammatory or anti-inflammatory factors. Recent evidence indicates that extracellular vesicles (EVs) are pivotal players in the intercellular communication that underlies physiological and pathological processes. In particular, glia-derived EVs play relevant roles in modulating neuroinflammation, either by promoting or inhibiting the activation of glial cells and neurons, or by facilitating the clearance or propagation of pathogenic proteins. The involvement of EVs in neurodegenerative diseases such as Alzheimer's Disease (AD), Parkinson's Disease (PD), Huntington's Disease (HD), and Multiple Sclerosis (MS)- which share hallmarks such as neuroinflammation and oxidative stress to DNA damage, alterations in neurotrophin levels, mitochondrial impairment, and altered protein dynamics- will be dissected, showing how EVs act as pivotal cell-cell mediators of toxic stimuli, thereby propagating degeneration and cell death signaling. Thus, this review focuses on the EVs secreted by microglia, astrocytes, oligodendrocytes and in neuroinflammatory conditions, emphasizing on their effects on neurons and on central nervous system functions, considering both their beneficial and detrimental effects.

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来源期刊
Neurobiology of Disease
Neurobiology of Disease 医学-神经科学
CiteScore
11.20
自引率
3.30%
发文量
270
审稿时长
76 days
期刊介绍: Neurobiology of Disease is a major international journal at the interface between basic and clinical neuroscience. The journal provides a forum for the publication of top quality research papers on: molecular and cellular definitions of disease mechanisms, the neural systems and underpinning behavioral disorders, the genetics of inherited neurological and psychiatric diseases, nervous system aging, and findings relevant to the development of new therapies.
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