Religiosity predicts prosociality, especially when measured by self-report: A meta-analysis of almost 60 years of research.

IF 4 2区 医学 Q2 CHEMISTRY, MEDICINAL
ACS Infectious Diseases Pub Date : 2024-03-01 Epub Date: 2024-02-26 DOI:10.1037/bul0000413
John Michael Kelly, Stephanie R Kramer, Azim F Shariff
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Abstract

This meta-analysis explores the long-standing and heavily debated question of whether religiosity is associated with prosocial and antisocial behavior at the individual level. In an analysis of 701 effects across 237 samples, encompassing 811,663 participants, a significant relationship of r = .13 was found between religiosity and prosociality (and antisociality, which was treated as its inverse). Nevertheless, there was substantial heterogeneity of effect sizes, and several potential moderators were explored. The effect was most heavily moderated by the type of measurement used to assess prosocial or antisocial behavior. Religiosity correlated more strongly with self-reported prosociality (r = .15) than with directly measured prosocial behavior (r = .06). Three possible interpretations of this moderation are discussed, namely, that (a) lab-based methods do not accurately or fully capture actual religious prosociality; (b) the self-report effect is explained by religious self-enhancement and overreports actual prosociality; or (c) both religiosity and self-reported prosociality are explained by self-enhancement. The question of whether religiosity more strongly positively predicts prosociality or negatively predicts antisociality is also explored. This moderation is, at most, weak. We test additional potential moderators, including the aspect of religiosity and type of behavior measured, the ingroup or outgroup nature of the recipient, and study characteristics. Finally, we recommend a shift in how researchers investigate questions of religiosity and prosociality in the future. (PsycInfo Database Record (c) 2024 APA, all rights reserved).

宗教信仰可预测亲社会性,尤其是通过自我报告进行测量时:对近 60 年研究的荟萃分析。
本荟萃分析探讨了宗教信仰是否与个人层面的亲社会行为和反社会行为相关这一长期存在且备受争议的问题。在对 237 个样本、811663 名参与者的 701 个效应进行分析后发现,宗教信仰与亲社会性(以及作为其反义词的反社会性)之间存在 r = .13 的显著关系。然而,效应大小存在很大的异质性,研究人员对几个潜在的调节因素进行了探讨。用于评估亲社会或反社会行为的测量类型对该效应的调节作用最大。与直接测量的亲社会行为(r = .06)相比,宗教信仰与自我报告的亲社会性(r = .15)的相关性更强。我们讨论了对这种调节作用的三种可能解释,即:(a)基于实验室的方法不能准确或全面地捕捉到实际的宗教亲社会性;(b)自我报告效应是由宗教自我提升和过度报告实际亲社会性所解释的;或(c)宗教信仰和自我报告的亲社会性都是由自我提升所解释的。此外,我们还探讨了宗教信仰对亲社会性的预测是更积极还是更消极的问题。这种调节作用最多是微弱的。我们还测试了其他潜在的调节因素,包括宗教信仰的方面和所测量的行为类型、受试者的内群体或外群体性质以及研究特点。最后,我们建议研究人员改变今后研究宗教信仰与亲社会性问题的方式。(PsycInfo Database Record (c) 2024 APA, 版权所有)。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
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来源期刊
ACS Infectious Diseases
ACS Infectious Diseases CHEMISTRY, MEDICINALINFECTIOUS DISEASES&nb-INFECTIOUS DISEASES
CiteScore
9.70
自引率
3.80%
发文量
213
期刊介绍: ACS Infectious Diseases will be the first journal to highlight chemistry and its role in this multidisciplinary and collaborative research area. The journal will cover a diverse array of topics including, but not limited to: * Discovery and development of new antimicrobial agents — identified through target- or phenotypic-based approaches as well as compounds that induce synergy with antimicrobials. * Characterization and validation of drug target or pathways — use of single target and genome-wide knockdown and knockouts, biochemical studies, structural biology, new technologies to facilitate characterization and prioritization of potential drug targets. * Mechanism of drug resistance — fundamental research that advances our understanding of resistance; strategies to prevent resistance. * Mechanisms of action — use of genetic, metabolomic, and activity- and affinity-based protein profiling to elucidate the mechanism of action of clinical and experimental antimicrobial agents. * Host-pathogen interactions — tools for studying host-pathogen interactions, cellular biochemistry of hosts and pathogens, and molecular interactions of pathogens with host microbiota. * Small molecule vaccine adjuvants for infectious disease. * Viral and bacterial biochemistry and molecular biology.
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