抗n -甲基- d -天冬氨酸受体脑炎8年后系列单纯疱疹病毒1型和人疱疹病毒7型脑炎。

Yoonhyuk Jang, Jeong-Min Kim, Jangsup Moon, Kyung-Il Park, Soon-Tae Lee, Keun-Hwa Jung, Sang Kun Lee, Kon Chu
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引用次数: 1

摘要

抗n -甲基- d -天冬氨酸受体(NMDAR)脑炎是由疱疹病毒脑炎引起的。人类7型疱疹病毒(HHV-7)是最近发现的一种疱疹病毒,其神经侵犯的报道很少。我们报告一例抗nmdar脑炎在复发性单纯疱疹病毒1型和HHV-7型相关的疱疹脑炎8年后在免疫功能正常的宿主中检测到。我们的病例提示抗nmdar脑炎可能由HHV-7脑膜脑炎在免疫功能正常的成年人中引发,有疱疹病毒脑炎病史的患者应警惕监测。
本文章由计算机程序翻译,如有差异,请以英文原文为准。

Anti-N-methyl-D-aspartate receptor encephalitis 8 years after serial herpes simplex virus type 1 and human herpesvirus type 7 encephalitis.

Anti-N-methyl-D-aspartate receptor encephalitis 8 years after serial herpes simplex virus type 1 and human herpesvirus type 7 encephalitis.

Anti-N-methyl-D-aspartate receptor (NMDAR) encephalitis is triggered by herpesvirus encephalitis. Human herpesvirus type 7 (HHV-7) is a recently described herpesvirus for which neuroinvasion has been reported rarely. We report a case of anti-NMDAR encephalitis detected 8 years after recurrent herpes encephalitis associated with herpes simplex virus type 1 and HHV-7 in an immunocompetent host. Our case suggests that anti-NMDAR encephalitis may be triggered by HHV-7 meningoencephalitis in immunocompetent adults, and patients with a history of herpesvirus encephalitis should be vigilantly monitored.

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