{"title":"更正“在小鼠中产生OMgp等位基因系列”","authors":"","doi":"10.1002/dvg.23513","DOIUrl":null,"url":null,"abstract":"<p>\n <span>Lee, J.K.</span>, <span>Case, L.C.</span>, <span>Chan, A.F.</span>, <span>Zhu, Y.</span>, <span>Tessier-Lavigne, M.</span> and <span>Zheng, B.</span> (<span>2009</span>), <span>Generation of an <i>OMgp</i> allelic series in mice</span>. <i>Genesis</i>, <span>47</span>: <span>751</span>–<span>756</span>. https://doi.org/10.1002/dvg.20557\n </p><p>In the originally published article, there are errors in the α-Tubulin controls on two Western blots presented in Figure 3a and Figure 4b. Two sets of gels, each in duplicate (a total of four gels), were run at the same time, and all were blotted sequentially with antibodies to OMgp and control α-Tubulin. While OMgp signals gave distinct patterns across different samples depending on the mutant conditions, control α-Tubulin signals gave similar patterns, which led to confusion and therefore errors in the figures in the specific lanes used for α-Tubulin controls. There were no errors for OMgp, which was the protein of interest. The authors have tracked the correct lanes for α-Tubulin controls, and the correct Western blot images for Figure 3a and Figure 4a, along with updated quantifications in Figure 3b and Figure 4b, is shown below. The figure legends remain the same for both figures except for the following: Representative data from one of 3–4 (instead of 3) independent sets of biological samples are shown. These errors had no impact on the scientific conclusions of the paper.</p><p>We apologize for this error.</p>","PeriodicalId":12718,"journal":{"name":"genesis","volume":"61 1-2","pages":""},"PeriodicalIF":2.4000,"publicationDate":"2023-03-20","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://onlinelibrary.wiley.com/doi/epdf/10.1002/dvg.23513","citationCount":"0","resultStr":"{\"title\":\"Correction to “Generation of an OMgp allelic series in mice”\",\"authors\":\"\",\"doi\":\"10.1002/dvg.23513\",\"DOIUrl\":null,\"url\":null,\"abstract\":\"<p>\\n <span>Lee, J.K.</span>, <span>Case, L.C.</span>, <span>Chan, A.F.</span>, <span>Zhu, Y.</span>, <span>Tessier-Lavigne, M.</span> and <span>Zheng, B.</span> (<span>2009</span>), <span>Generation of an <i>OMgp</i> allelic series in mice</span>. <i>Genesis</i>, <span>47</span>: <span>751</span>–<span>756</span>. https://doi.org/10.1002/dvg.20557\\n </p><p>In the originally published article, there are errors in the α-Tubulin controls on two Western blots presented in Figure 3a and Figure 4b. Two sets of gels, each in duplicate (a total of four gels), were run at the same time, and all were blotted sequentially with antibodies to OMgp and control α-Tubulin. While OMgp signals gave distinct patterns across different samples depending on the mutant conditions, control α-Tubulin signals gave similar patterns, which led to confusion and therefore errors in the figures in the specific lanes used for α-Tubulin controls. There were no errors for OMgp, which was the protein of interest. The authors have tracked the correct lanes for α-Tubulin controls, and the correct Western blot images for Figure 3a and Figure 4a, along with updated quantifications in Figure 3b and Figure 4b, is shown below. The figure legends remain the same for both figures except for the following: Representative data from one of 3–4 (instead of 3) independent sets of biological samples are shown. These errors had no impact on the scientific conclusions of the paper.</p><p>We apologize for this error.</p>\",\"PeriodicalId\":12718,\"journal\":{\"name\":\"genesis\",\"volume\":\"61 1-2\",\"pages\":\"\"},\"PeriodicalIF\":2.4000,\"publicationDate\":\"2023-03-20\",\"publicationTypes\":\"Journal Article\",\"fieldsOfStudy\":null,\"isOpenAccess\":false,\"openAccessPdf\":\"https://onlinelibrary.wiley.com/doi/epdf/10.1002/dvg.23513\",\"citationCount\":\"0\",\"resultStr\":null,\"platform\":\"Semanticscholar\",\"paperid\":null,\"PeriodicalName\":\"genesis\",\"FirstCategoryId\":\"99\",\"ListUrlMain\":\"https://onlinelibrary.wiley.com/doi/10.1002/dvg.23513\",\"RegionNum\":4,\"RegionCategory\":\"生物学\",\"ArticlePicture\":[],\"TitleCN\":null,\"AbstractTextCN\":null,\"PMCID\":null,\"EPubDate\":\"\",\"PubModel\":\"\",\"JCR\":\"Q2\",\"JCRName\":\"DEVELOPMENTAL BIOLOGY\",\"Score\":null,\"Total\":0}","platform":"Semanticscholar","paperid":null,"PeriodicalName":"genesis","FirstCategoryId":"99","ListUrlMain":"https://onlinelibrary.wiley.com/doi/10.1002/dvg.23513","RegionNum":4,"RegionCategory":"生物学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":null,"EPubDate":"","PubModel":"","JCR":"Q2","JCRName":"DEVELOPMENTAL BIOLOGY","Score":null,"Total":0}
Correction to “Generation of an OMgp allelic series in mice”
Lee, J.K., Case, L.C., Chan, A.F., Zhu, Y., Tessier-Lavigne, M. and Zheng, B. (2009), Generation of an OMgp allelic series in mice. Genesis, 47: 751–756. https://doi.org/10.1002/dvg.20557
In the originally published article, there are errors in the α-Tubulin controls on two Western blots presented in Figure 3a and Figure 4b. Two sets of gels, each in duplicate (a total of four gels), were run at the same time, and all were blotted sequentially with antibodies to OMgp and control α-Tubulin. While OMgp signals gave distinct patterns across different samples depending on the mutant conditions, control α-Tubulin signals gave similar patterns, which led to confusion and therefore errors in the figures in the specific lanes used for α-Tubulin controls. There were no errors for OMgp, which was the protein of interest. The authors have tracked the correct lanes for α-Tubulin controls, and the correct Western blot images for Figure 3a and Figure 4a, along with updated quantifications in Figure 3b and Figure 4b, is shown below. The figure legends remain the same for both figures except for the following: Representative data from one of 3–4 (instead of 3) independent sets of biological samples are shown. These errors had no impact on the scientific conclusions of the paper.
期刊介绍:
As of January 2000, Developmental Genetics was renamed and relaunched as genesis: The Journal of Genetics and Development, with a new scope and Editorial Board. The journal focuses on work that addresses the genetics of development and the fundamental mechanisms of embryological processes in animals and plants. With increased awareness of the interplay between genetics and evolutionary change, particularly during developmental processes, we encourage submission of manuscripts from all ecological niches. The expanded numbers of genomes for which sequencing is being completed will facilitate genetic and genomic examination of developmental issues, even if the model system does not fit the “classical genetic” mold. Therefore, we encourage submission of manuscripts from all species. Other areas of particular interest include: 1) the roles of epigenetics, microRNAs and environment on developmental processes; 2) genome-wide studies; 3) novel imaging techniques for the study of gene expression and cellular function; 4) comparative genetics and genomics and 5) animal models of human genetic and developmental disorders.
genesis presents reviews, full research articles, short research letters, and state-of-the-art technology reports that promote an understanding of the function of genes and the roles they play in complex developmental processes.