重新配方的免疫编辑在癌变灶的建立中作为激动剂和允许因子

lawrence M Agius
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引用次数: 0

摘要

作为初始癌变焦点的旁分泌和自分泌重建的弥漫性现象包括诱导免疫监视丧失的免疫编辑衍生现象。免疫监测试图定义与炎症反应和相互作用密切相关的损伤的宪法识别。从进一步符合慢性炎症损伤的角度来看,由于单个肿瘤细胞的抗原性丧失,重构中的应答元件失效。这种激动剂作用的重现被进一步促进为容许性元素损伤,将这种容许性转化为逃避免疫系统的肿瘤细胞的出现。表征是在复杂的相互作用系统中引起伤害,在试图重建时由多个分子事件调节。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
Re-Formulated Immuno-Editing as Agonist and Permissive Elements in Establishment of Foci of Carcinogenesis
Diffusional phenomena of paracrine and autocrine reconstitution in a focus of initial carcinogenesis include the derivative phenomenon of immuno-editing in the induced loss of immuno-surveillance. Immuno-surveillance is attempted definition of the constitutional identification of an injury that relates intimately with inflammatory response and interactivity. In terms further conforming to an injury in chronic inflammation, the responsive elements at reconstitution fail as a result of loss an antigenicity of individual tumor cells. Recapitulation of such agonist action is further promoted as permissive elemental injury that transforms such permissiveness to the emergence of tumor cells that evade the immune system. Characterization is injury-provoking within the systems of complex interactivity as modulated by multiple molecular events at attempted reconstitution.
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